Rehab Test #3 - Spinal Cord, Brain Injury, EKG Flashcards
(44 cards)
C5-
Elbow Flexors
C6-
Wrist Extensors
C7-
Elbow Extensors
C8-
Finger Flexors
T1-
Finger Abductors
L2-
Hip Flexors
L3-
Knee Extensors
L4-
Ankle Dorsiflexors
L5-
Long Toe Extensors
S1-
Ankle Plantar Flexors
American Spinal Injury Association (ASIA) Impairment Scale
A= comlpete: no motor or sensory function is presesrved in the sacrel segment S4-S5
B=incomplete: sensory but not motor function is preserved below the neurologic level and includes the sacral segments s4-s5
C=incomplete: motor function is presereved below the neurologic level, and more than half of key muscles below the neurologic level have a muscle grade less than 3
D= incomplete: motor function is preserved below the neurologic level, and at leawst half of key muscles below the neurologic level have a muscle grade of 3 or more
E= normal: motor and sensory functions are normal/
Initial injury
primary vs secondary
Primary
Actual physical disruption of axons
Secondary
Ischemia, hypoxia, microhemorrhage, and edema
Spinal shock
Temporary neurologic syndrome characterized by ↓ reflexes and flaccid paralysis below level of injury
Neurogenic shock
Loss of vasomotor tone characterized by hypotension, vasodilation, and bradycardia (≥T6)
Define spinal cord injury
Spinal cord injury (SCI) occurs when the axons of the spinal cord are damaged
Tetraplegia -
quadriplegia, is paralysis caused by illness or injury that results in the partial or total loss of use of all four limbs and torso
Paraplegia –
lower parts of your body, does not affect the arms
Upper Motor Neuron (UMN)
Cervical injuries
Cause loss of cerebral control over all motor activity below level of injury
Causes SPASTIC paralysis
Reflex bladder/spastic; rectal digital stimulation (by suppository)
Lower Motor Neuron (LMN)
Thoracic, Lumbar and Sacral injuries (L1 & below)
Causes destruction of the reflex arc
Causes FLACCID paralysis
Areflexive bladder/Autonomous/flaccid; manual bowel evacuation
Need rectal digital stimulation
autonomic dysreflexia
Autonomic dysreflexia
Also known as autonomic “hyperreflexia”
Massive uncompensated cardiovascular reaction mediated by the sympathetic nervous system in response to visceral stimulation (≥ T6 injury)
S/S Hypertension (greater than 20 mmHg over patient’s baseline systolic), throbbing headache, blurred vision, and flushing, marked diaphoresis, and piloerection above level of injury; cool, pallor below level of injury.
MEDICAL EMERGENCY
Elevate HOB, find & remove irritant (empty bladder, loosen constricted clothing), alpha-blockers and/or nitro as ordered.
Prevention is key – excellent bowel, bladder, and skin care.
Traumatic brain injury
A serious form of head injury
Disruption of brain tissue resulting from an impact to the head in which the head hits, is hit by, or is penetrated by an object
Closed injury vs. open injury
Describe the incidence of traumatic brain injury
5.3 million Americans currently live with disability from brain injury
1.7 million new cases each year
1,365, 000 seen in ER
275,000 are hospitalized
52,000 die
Male > female
Males 14-24 at highest risk
Mild brain injury
Glasgow 13 to 15
Loss of consciousness 20 minutes or less
Posttraumatic amnesia less than 1 hour
Hallmark symptom is headache, neuroimaging is usually negative, may take weeks to months to resolve
Moderate Brain Injury (10%)
Glasgow 9-12
Loss of consciousness greater than 20 minutes
Posttraumatic amnesia 1-24 hours
Symptoms depend on area of brain affected. Cerebral edema and cerebral hemorrhage often seen on imaging.
Usually requires some rehab, return to preinjury lifestyle possible but likely to have some long-term neurologic deficits