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[PHY2] 2ND SHIFT > Renal Physiology 4 > Flashcards

Renal Physiology 4 Flashcards

1
Q

Main ICF Ion

A

Potassium

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2
Q
  • Total Intake of K+
  • Total Output of K+
A
  • Total Intake: 100 mEq/day
  • Total Output: 100 mEq/day
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3
Q

QC

K+ Output via
1. Urine
2. Feces

A

1 > 2

Urine: 92 Feces: 8

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4
Q

QC

Blood K+ Distribution
1. ECF
2. ICF

A

1 < 2

ECF: 59 mEq ICF: 3920mEq

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5
Q

QC

K+ shifts from ECF to ICF if the Na/K ATPase Pump is
1. Activated
2. Deactivated

A

A: 1 > 2

Pump needs to be activated to transport 2 K+ In.

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6
Q

VR

  1. Ingestion of K+ rich food
  2. Na/K ATPase Pump
A

A: Increase in 1 will Increase 2

Increase in 1 will increase ECF K conc = need to be moved to the ICF

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7
Q

QC

ECF K+ Conc after a meal of a:
1. Normal Person
2. Diabetic Person

A

B: 1 < 2

kaya need nya mag insulin to increase rin yung pag secrete ng K

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8
Q
  1. Insulin
  2. ICF K+
A

A: Increase in 1 will Increase 2

1 increases Na/K ATPase Pump activity kasi need i excrete

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9
Q
  1. Aldosterone
  2. K Reabsorption
A

B: Increase in 1 will Decrease 2

Aldosterone secretes K+; 1 increases Na/K ATPase Pump activity

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10
Q

Conn Syndrome
1. Hyperkalemia
2. Hypokalemia

A

B: 1 < 2

Conn = Excess Aldosterone = more K secretion

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11
Q

VR

  1. Addison’s Disease
  2. ECF K+ Levels
A

A: Increase in 1 will Increase 2

Addison’s = deficiency in Aldosterone = Less K secretion

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12
Q
  1. Epinephrine Secretion
  2. K+ Excretion
A

A: Increase in 1 will Increase 2

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13
Q

QC

Na/K ATPase Activity
1. B1 stimulation
2. B2 stimulation

A

B: 1 < 2

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14
Q
  1. Propanolol
  2. Na/K ATPase Activity
A

B: Increase in 1 will Decrease 2

Propanolol is a Beta Blocker

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15
Q

Propanolol
1. Hyperkalemia
2. Hypokalemia

A

A: 1 > 2

Propanolol (Bblocker) = less ATPase activity = less K excretion = high K

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16
Q
  1. H+ concentration
  2. Na/K ATPase activity
A

A: Increase in 1 will Increase 2

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17
Q

Alkalosis
1. Hyperkalemia
2. Hypokalemia

A

B: 1 < 2

Alkalosis = high K secretion

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18
Q
  1. Blood Sugar Glucose
  2. ECF K+
A

B: Increase in 1 will Decrease 2

high sugar = inc in insulin = increase atpase activity = ECF to ICF

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19
Q
  1. Blood Pressure
  2. ECF K+
A

Increase in 1 will Increase 2

High BP = Low aldosterone = low atpase act = no ecf to icf movement

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20
Q

ECF K+ Concentration
1. Hypertension
2. Hypotension

A

A: 1 > 2

Hypertension = no aldosterone = no movement of K from ECF to ICF

21
Q

Plasma K+ Levels in
1. Acidosis
2. Alkalosis

A

A: 1 > 2

Acidosis = inhibit atpase = k remains in ecf

22
Q

K+ Reabsorption
1. Acidosis
2. Alkalosis

A

A: 1 > 2

Acidosis = high k ecf = high reabsorption

23
Q

K+ Excretion
1. Acidosis
2. Alkalosis

A

B: 1< 2

alkalosis = low ecf k = low reab = high excretion

24
Q
  1. RBC Lysis
  2. Plasma K+ Level
A

A: Increase in 1 will Increase 2

25
Q
  1. Strenous Exercise
  2. Plasma K+ Levels
A

A: Increase in 1 will Increase 2

Strenous exercise = rbc lysis = increase K+

26
Q
  1. ECF Osmolarity
  2. Plasma K+ Concentration
A

A: Increase in 1 will Increase 2

high ecf osmo = water will follow = K+ will follow

27
Q
  1. ECF Osmolarity
  2. K+ Reabsorption
A

A: Increase in 1 will Increase 2

28
Q

Renal K+ excretion or retention is determined by:

A

Rate of K+
1. Filtration
2. Reabsorption
3. Secretion

29
Q

VR

  1. Dietary K+
  2. Renal K+ Secretion
A

A: Increase in 1 will Increase 2

30
Q
  1. Tubular Flow Rate
  2. Renal K+ Secretion
A

A: Increase in 1 will Increase 2

31
Q
  1. Aldosterone
  2. K+ Secretion
A

A: Increase in 1 will Increase 2

32
Q
  1. Blood Pressure
  2. K+ Secretion
A

B: Increase in 1 will Decrease 2

Inc BP = No Aldosterone = Less K Secretion

33
Q

Tubular K+ Reabsorption
1. PCT
2. TALH

A

1 > 2
65% > 27%

34
Q

Mechanism of Tubular Reabsorption of K+ Processes in:
* PCT:
* TALH
* Late DT & CD

A
  • PCT: Solvent Drag
  • TALH: NKCC2 Transporter
  • Late DT & CD: H/K ATPase
35
Q

Mechanisms of Tubular K Secretion in the ff
* Principal Cells:
* Type B Cells
* Type A Cells

A
  • Principal Cells: ROMK and BK
  • Type B Cells: H/K ATPase
  • Type A Cells: H/K ATPase
36
Q

Flow-stimulated K+ Secretion
1. ROMK
2. BK

A

B: 1 < 2

37
Q

Conductance of
1. ROMK
2. BK

A

B: 1 < 2

38
Q

Calcium Activated
1. ROMK
2. BK

A

B: 1 < 2

39
Q

Major K Secretion Channel
1. ROMK
2. BK

A

A: 1 > 2

40
Q
  1. ECF K
  2. K Secretion
A

A: Increase in 1 will Increase 2

41
Q
  1. K+ ICF
  2. K+ Secretion
A

A: Increase in 1 will Increase 2

42
Q
  1. Intake of K
  2. K+ Channels Synthesis
A

A: Increase in 1 will Increase 2

43
Q
  1. Volume Expansion
  2. K Secretion
A

A: Increase in 1 will Increase 2

44
Q
  1. Excessive Na Intake
  2. K Secretion
A

C: Increase in 1 will not affect 2

inc in Na will be counteracted by dec in aldosteron = homeostasis

45
Q
  1. Na+ Intake
  2. Aldosterone secretion
A

B: Increase in 1 will Decrease 2

to counteract its increased k secretion effect

46
Q
  1. Diuretics
  2. ECF K+
A

B: Increase in 1 will decrease 2

Diuretics increase k secretion

47
Q
  1. Acute Acidosis
  2. K+ Secretion
A

B: Increase in 1 will Decrease 2

1 decreases Na/K Pump = decrease 2

48
Q
  1. Chronic Acidosis
  2. K+ Secretion
A

A: Increase in 1 will Increase 2

1 increases Distal Tubule Flow/Delivery = Increase 2

[PHY2] 2ND SHIFT (6 decks)

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