respiration Flashcards

(33 cards)

1
Q

respiration D

A

oxidation of food molecules to release energy

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2
Q

aerobic resp D

A

oxidation of food molecules in the presence of oxygen to release of a relatively large amt of energy

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3
Q

anerobic resp D

A

oxidation of food molecules wihtout oxygen to release of a relatively small amt of energy

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4
Q

where does aerobic resp occur?

A

mitochondria of cells
(enzymatic rxn)

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5
Q

anaerobic resp during exercise

A

-muscles contract vigorously –> high energy demand
-(rate of breathing and heart rate increases so) rate of respiration increases
-rate of aerobic resp cannot increase any further and energy demand continues to increase
-anerobic resp occurs to provide additional energy required

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6
Q

after exerise:

A

accumulation of lactic acid that is removed in liver
-heart rate remains high
-rate and depth of breathing remains high (lactic acid transported faster to liver)

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7
Q

nostril F

A

hair and mucous layer: trap dust and foreign particles

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8
Q

pharynx F

A

throat area (openings to trachea and oesophagus)

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9
Q

larynx F
(vocal cords)

A

cartilaginous structure

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10
Q

trachea F
(windpipe)

A

C-shaped rings of cartilage
-prevents collapse
-keeps airways open

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11
Q

epithelium of airways (trachea and bronchus)

A

goblet cells and cililated cells

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12
Q

goblet cells

A

produce mucus that trap dust and bacteria

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13
Q

cilia

A

sweep mucus up bronchi and trachea into larynx

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14
Q

properties of alveoli (5)

A
  1. numerous alveoli
  2. many blood capillaries
  3. one-cell thick
  4. thin film of water
  5. maintenance of steep concentration gradient
    –> increase rate of diffusion of gases between alveoli and blood capillaries
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15
Q

one-cell thick

A

short distance for quick diffusion of gases

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16
Q

thin film of water

A

gases dissolve and diffuse easily into blood

17
Q

maintenance of steep conc gradient btwn alveoli and blood capillaries

A

-blood constantly flowing: keep conc of O2 low and CO2 high in blood capillaries
-constant breathing: conc of O2 high and CO2 low in alveoli space

18
Q

pulmonary arteriole

A

deoxygenated blood from heart

19
Q

pulmonary venule

A

oxygenated blood going to the heart

20
Q

process of O2 diffusion (at alveoli)

A

O2 molecules diffuse from alveolar space into RBCs in blood capillaries
(oxygenated carried towards left atrium by pulmonary vein)

21
Q

o2 diffusion at tissues

A

simple diffusion of o2 molecules from RBCs (unbind with haemoglobin) into body cells

22
Q

CO2 diffusion

A

CO2 molecules diffuse from blood in capillaries into alveoli

23
Q

exhalation

A
  1. diaphragm relaxes, arches up
  2. external intercostal muscles relax, internal intercostal muscles contract
    + rib cage: downwards and inwards
  3. thoracic volume decreases
  4. pressure in thoracic cavity increases above atmospheric pressure, lungs are compressed, air is forced out of lungs
24
Q

inhalation

A
  1. diaphragm contracts, flattens down
  2. external intercostal muscles contract, internal intercostal muscles relax
    + rib cage: upwards and outwards
  3. thoracic volume increases
  4. pressure in thoracic cavity decreases below atmospheric pressure, air is forced into lungs
25
carbonic anhydrase
enzyme found in RBCs that catalyses conversion of carbon dioxide and water into carbonic acid (or vice versa: reversible rxn)
26
at tissues (body cells)
-simple diffusion of co2 molecules from body cells into blood -co2 + h2o converted into carbonic acid (in RBCs) catalysed by carbonic anhydrase -CA dissociates to form bicarbonate and hydrogen ions (in blood plasma) --> bc of high conc of co2 in body cells
27
at lungs
-bicarbonate and hydrogen ions readily react to form carbonic acid -CA converted to co2 & h2O, catalysed by carbonic anhydrase (in RBCs) -co2 molecules diffuse from RBC to blood plasma into alveoli to be removed via exhalation --> bc of low conc of co2 in lungs
28
tobacco smoke components (3)
-nicotine -tar -CO
29
nicotine effects
-increases risk of blood clotting, higher risk of coronary heart disease
30
tar effects
-carcinogens -paralyses cilia lining airways: dust particles trapped in mucus lining cannot be removed --> higher risk of bronchitis & emphysema
31
CO effects
binds tightly with haemoglobin in RBCs to form carboxyhaemoglobin -less haemoglobin available to bind with co2 --> reduced o2 transport around body
32
emphysema
-walls btwn alveoli break down due to persistent violent coughing -total SA for GE decreases --> slower rate of diffusion of o2 into blood --> difficulty breathing
33
chronic bronchitis
-tar coats cilia, becomes paralysed -excessive mucus secreted by goblet cells -trapped dust and bacteria stay in respiratory tract --> bacteria grow and multiply -persistent coughing --> airways become inflamed