Respiratory CIS Flashcards

(59 cards)

1
Q

most common cause of wheezing in kids

A

-viral infections

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2
Q

what time of day is more suggestive of asthma

A

coughing that’s worse in middle of the night

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3
Q

risks for developing asthma

A
  • RSV infection prior to 6 months of age

- FH of any atopy- allergic rhinitis, eczema

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4
Q

common triggers for asthma

A
  • virus
  • allergies
  • exercise
  • cold air
  • cigarette smoke
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5
Q

CXR in a child with asthma or reactive airway dz (RAD)

A
  • atelectasis (auscultated as dec breath sounds)
  • hyperinflation of both lungs
  • perihilar thickening
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6
Q

capillary refill- in healthy child and child with asthma or RAD

A
  • > 2 sec = very concerning

- healthy- nearly instantaneously!!

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7
Q

best way to obtain blood gases in pediatric pt

A

capillary blood gases

  • quicker and less distressing than arterial gas (which is more accurate for O2)
  • cant use PaO2 from them
  • useful only for pH and CO2
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8
Q

pediatric pt with asthma exacerbation- admitted to?

A

-PICU- due to significant risk for decompensation

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9
Q

options for maintenance IVF in children

A
  • 1/2 NS in >1 yo or 1/4 NS in <1 yo
  • NS is reserved for bolusing
  • in peds usually K is added to IVF
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10
Q

when is it appropriate to intubate an asthmatic (pediatric) pt- treatments to add to intubation?

A
  • time to intubate- when b/w irritable and obtunded
  • ADD terbutaline drip, Mg, Theophylline, subcutaneous epinephrine, heliox (breathing as that is a mixture of helium and oxygen, less resistance and easier to breath), or BiPAP– in order to NOT INTUBATE an asthmatic
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11
Q

Why don’t you want to intubate asthmatics?

A

-they can’t exhale- so you force breaths in with the vent, they get fuller and fuller until they either get b/l pneumothorax or acute right heart collapse and die

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12
Q

which population of asthmatics has the highest mortality

A

-adolescents- b/c they dont carry their rescue inhaler with them

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13
Q

signs of resp distress in a pediatric pt

A
  • inspiratory and expiratory wheezing
  • nasal flaring and tachypnea
  • subcostal, intercostal, and suprasternal retractions
  • stridor
  • sniffing or tripod positioning
  • dec air movement (after albuterol hear wheezing, means improving)
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14
Q

treatment considerations

A
  • albuterol nebulizer or inhaler (rescue inhaler- bronchodilator- short acting Beta-2 agonist)
  • inhaled corticosteroids
  • oral corticosteroids
  • oxygen (put on first if hypoxic)
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15
Q

what would indicate concern for CF?

A
  • poor height and weight
  • clubbing
  • foul-smelling stools
  • recurrent pneumonia
  • edema
  • failure to thrive
  • test with Sweat chloride test!!`
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16
Q

sudden stridor in a child makes you think of what?

A

-foreign body aspiration

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17
Q

O2 options to consider

A
  • NC up to 5 Liter
  • simple face mask at 5-6L
  • NRB at 10-15 L/min
  • bag valve mask
  • bipap
  • intubation
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18
Q

asthma under control- green- YES

A
  • daytime sx- 3 times or less/week
  • nighttime sx- none
  • reliever- 3 times or less/week
  • physical activity- normal
  • able to go to school- yes
  • peak expiratory flow- 85-100%
  • Stay controlled and avoid triggers
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19
Q

asthma under control- yellow- NO

A
  • daytime sx- >3 times/week
  • nighttime sx- some nights
  • reliever- >3 times/week
  • physical activity- limited
  • able to go to school- maybe
  • peak expiratory flow- 60-85%
  • Adjust meds
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20
Q

asthma under control- red- NOT AT ALL

A
  • daytime sx- continuous and worsening
  • nighttime sx- continuous and worsening
  • reliever- relief less than 3-4 hrs
  • physical activity- very limited
  • able to go to school- no
  • peak expiratory flow- <60%
  • call for help
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21
Q

obstetrical history taking- GTPAL

A
  • Gravidity- number of total pregnancies
  • Term births- term deliveries (38 wks)
  • Preterm births- viability up to 37 wks
  • Abortions/miscarriages
  • Living children
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22
Q

total hysterectomy

A

-take uterus and ovaries, usually but not always take the cervix too

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23
Q

CAD risk factors

A

-emotional stress and no exercise

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24
Q

S2 splitting during cardiac exam- cause

A
  • physiologic splitting of S2 on inspiration can be normal
  • persistent splitting of S2 during inspiration and expiration- HD in adults (RBBB), RV pressure overload situations (acute massive PE)
25
- physiologic splitting of S2 on inspiration can be normal - persistent splitting of S2 during inspiration and expiration- HD in adults (RBBB), RV pressure overload situations (acute massive P
rales
26
what is an invasive and highly accurate way of measuring BP constantly?
arterial line
27
checking for inherited thrombophilia- labs
checking for inherited thrombophilia- labs
28
most common findings on an EKG in a pt with a PE?
- nonspecific ST-T wave abnormalities | - sinus tachycardia
29
gold standard imaging choice for PE?
CTA (CT angiogram) of chest (PE protocol) | -need to consider the stability of pt before taking them to radiology!
30
EKG- indicative of PE
-S1 Q3 T3
31
what is tPA?
- recombinant tissue type plasminogen activator (alteplase)- "clot buster" - enzyme produced by a number of tissues, including endo cells- binds to fibrin, which inc its affinity for plasminogen and enhances plasminogen activation - systemic thrombolysis is preferred- can give rapidly (as compared to taking to cath lab for angiogram and direct injection into the clot itself)
32
Indications for thrombolytic therapy in venous threomboembolism
- hypotension related to PE - severe hypoxemia - substantial perfusion defect - right ventricular dysfxn assoc with PE - extensive DVT
33
hormones are what?
pro-thrombotic | -hormone replacement therapy- premarin (Estrogen)
34
what is Factor V Leiden?
mutant form of coagulation factor V - FVL mutation renders factor V insensitive to the actions of activated protein C (aPC), a natural anticoagulant - FVL mutations- inc risk of venous thromboembolism - extremely common!!- many pts will never have a VTE
35
Virchow's Triad
VTE occurs as a result of: - alterations in blood flow (stasis) - vascular endothelial injury - hypercoagulable state (inherited or acquired)
36
Wells Criteria
``` (assessment for PE) -clinical sx of DVT- 3 -other dx less likely than PE- 3 -HR > 100- 1.5 -immobilization or surgery in prev 4 wks- 1.5 -previous DVT/PE- 1.5 -Hemoptysis- 1 -malignancy- 1 SCORE -high >6 -moderate 2-6 ```
37
contraindications to fibrinolytic therapy?
- prior intracranial hemorrhage - structural cerebral vascular lesion - malignant intracranial neoplasm - ischemic stroke within 3 months - suspected aortic dissection - active bleeding or bleeding diathesis - close-head trauma in last 3 months
38
types of shock
- hypovolemic - cardiogenic - distributive - obstructive
39
what is MAP
mean arterial P - diastolic BP + [(systolic BP - diastolic BP) / 3] - >65 = good perfusion to all organs - <65 = hypotension/hypoperfusion
40
dd for Tb
- NTM (nontuberculosis mycobacterial infection- M kansaii) - fungal infection - lung cancer - lymphoma
41
NTM (nontuberculosis myobacterial infection- M kansaii)- sx, dx
- fatigue, dyspnea, occasional hemoptysis | - sputum Cx and molecular diagnostics
42
fungal infection- sx, dx
- PNA, nodules, cavitation | - dx- Cx results, regional exposure
43
lung cancer- sx, dx
- fevers, cough, chest pain, hemoptysis, dyspnea | - dx- histopathology
44
lymphoma- sx, dx
- fevers, night sweats, weight loss | - dx- histopathology
45
pts at risk for tb
- close contact with someone who has active tb - immigrants from endemic areas - jail, nursing homes, homeless shelters, healthcare facilities - medically underserved, poor populations - IV drug abuse - HIV/AIDS
46
PPD >5 mm induration
- HIV - close contact with actively infected person - CXR with fibrotic changes consistent with TB - immunosuppression
47
PPD > 10 mm induration
- clinical conditions that INC risk of react- silicosis, DM, chronic renal failure, malignancies - children < 4 - from country with high prevalance - jail, healthcare facilities, homeless shelters
48
PPD > 15 mm induration
-healthy individual > 4 with low likelihood of true TB infection
49
signs/sx of active tb
-fever, night sweats, cough (>2 wks), weight loss, lymphadenopathy
50
workup for tb
- Sputum culture - sputum staining - PPD skin test - IGRA - CXR - NAAT
51
TB- sputum culture
- tb is a microbiological diagnosis!! - 3 separate morning sputum samples - takes 6-8 wks * culture is the gold-standard for dx!!!!!
52
TB- sputum staining
Acid-fast bacillus!! - Rhodamine-auramine stain- initial screening - Ziehl-Neelsen and/or Kinyun stain- confirmatory
53
tb- IGRA- indication
- diagnostics of LATENT tb | - pts who have received a BCG vaccination and those with a positive PPD in whom latent tb is suspected
54
TB- CXR
-cavitary lesions- apex of lung
55
TB- NAAT
-detects INH and rifampin resistance
56
TB- drug therapy and SE's
- Isoniazid- peripheral neuropathy (give pt Vit B6)!! - Rifampin- red/orange body fluids - Pyrazinamide- gout, joint aches - Ethambutol- optic neuritis, color-blindness * monitor CMPs- kidney and liver fxn!!!
57
clinical dx of active TB
- clinical sx - risk factors - order radiography - sputum culture - dont delay treatment when awaiting sputum culture!!
58
PPD + with no BCG vaccine
- check CXR- make sure there's no active TB | - treat as latent tb- 9 months of INH
59
PPD + with BCG vaccine
- check CXR and IGRA - if IGRA positive- treat as latent tb - if IGRA negative- NO active or latent tb