Rhinology Flashcards

1
Q

List the different components of inspection for nose examination

A

external- lesions, scars, alignment
internal- lift tip of nose- septal deviation, mucosal lesions, oral examination of hard/soft palate, dentitions, oropharynx

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2
Q

Which further investigations would you suggest in nose exam OSCE

A

blood tests- allergens
autoimmune screen
imagine- CT, MRI
smell test
skin prick testing for allergens

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3
Q

List four aspects of rhinology specific questions for history

A

Nasal obstruction – unilateral / bilateral, intermittent / persistent, onset, duration
Sense of smell – normal, decreased, absent
Nasal discharge – anterior / posterior, unilateral / bilateral, watery / mucoid
Sneezing / Itch – triggers, persistent or seasonal
Facial pain – site, onset, duration, triggers, alleviating factors, type

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4
Q

State three red flags for nose symptoms

A

Unilateral symptoms – obstruction, sero-sanguinous discharge, altered sensation
Alteration of vision – diplopia, opthalmoplegia (suggests invasion of orbit)
Pain – face, retro-orbital
Swelling – face, maxilla (dentures not fitting)
Epistaxis – persistent cases suspicious

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5
Q

List two causes of nasal obstruction

A

Infective - Viral infection – “the common cold”
Autoimmune - Rhinitis – Allergic / non-allergic, persistent / seasonal
Inflammatory - Nasal polyps – Bilateral with hyposmia
Traumatic - Deviated nasal septum
Neoplasms – Squamous cell carcinoma, inverted papilloma

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6
Q

Discuss two causes of rhinorrhoea= runny nose

A

Allergic rhinitis – Watery, bilateral, associated with sneezing + nasal itch +/- eye symptoms
Chronic rhinosinusitis - > 12 weeks of obstruction / discharge / pain / decreased smell
Head trauma – CSF leak, unilateral, increased with position / straining – Ix β2-transferrin
Viral infection – the common cold
Nasal foreign body – unilateral, offensive is indicative

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6
Q

List two causes of hyposmia= reduced ability to smell

A

Rhinosinusitis – cardinal symptom of chronic rhinosinusitis
Nasal polyps – “Conductive loss” – blockage of airflow to olfactory mucosa
Viral infection / idiopathic – post influenza virus infection – “senori-neural loss”
Neoplasms – Conductive or sensorineural if from olfactory mucosa
Head trauma – shearing of olfactory nerves as pass through cribiform plate

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7
Q

List three differentials for facial pain

A

Acute sinusitis – unilateral, fever, purulent discharge <14 days
Neoplasms – associated paraesthesia, other unilateral symptoms
Trigeminal neuralgia – lancinating pain, trigger points, treat with carbemezapine
Dental infection – poor dentition, dull ache, needs dental exclusion
Migraine – unilateral, visual effects, triggers, nausea & vomiting

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8
Q

List two differentials for epistaxis

A

Idiopathic – spontaneous, can be associated with infection
Trauma – nose picking / post surgical
Neoplasms – recurrent persistent cases this should be considered
Anti-coagulants – increase risk especially with supra-theraputic levels
Bleeding disorders – primary or secondary, need treatment in recurrent cases

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9
Q

What is the management of nasal polyps?

A

intranasal steroids + saline douching
if not
systemic steroids
if not
surgery

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10
Q

State one complication of nasal fracture

A

septal haematoma
epistaxis

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11
Q

What is the time frame for nasal fracture for manipulaion post injury

A

<14 days

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12
Q

Which infections is associated with nasopharyngeal cancer?

A

EBV

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13
Q

Which population are at increased risk of nasopharyngeal cancers?

A

chinese population

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14
Q

How do nasopharyngeal cancers present

A

unilateral otitis media with effusion
neck lump
epistaxis
nasal obstruction

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15
Q

List two benign sinonasal tumours

A

Inverted papilloma – aka scheridan’s papilloma, local erosive, recurrence a problem
Fibroma – Juvenile nasal angiofibroma – adolescent male, nasal obstruction + epistaxis
Haemangioma – rare paediatric
Meningioma
Adenoma

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16
Q

List two malignant sionasal tumours

A

Squamous cell carcinoma – smoking, soft woods
Adenocarcinoma – hard wood working
Olfactory neuroblastoma – decreased smell arise from olfactory mucosa
Malignant melanoma – mucosal type, poor prognosis
Fibrosarcoma

17
Q

State two congenital anomalies of the nose

A

Nasal dermoid cyst – midline, hair tuft in pit, can extend intracranially, surgical excision
Nasal encephalocele – skull base defect, brain (non-functioning) / meninges in nasal cavity
Choanal atresia – posterior nose remains occluded, bony / membranous
Haemangioma – vascular lesion, self limiting, cosmetically disfiguring
Arrhinia – non-formation of nose (very rare)

18
Q

Name two systemic diseases associated with nasal symptoms

A
  1. Wegener granulomatosis – destructive vasculitis, c-ANCA + ve, kidneys + lungs
  2. Sarcoidosis – Nasal obstruction, ACE +ve serology, Lungs (hilar lymphadenopathy)
  3. Churg-Strauss syndrome – Vasulitis + Asthma, GI tract p-ANCA +ve
  4. Chronic Infective disease – TB, Leporsy, Syphillis
  5. Cystic fibrosis – chronic sinusitis + polyposis (in children indicative of CF)
19
Q

What is the most common ENT emergency presentation?

A

epistaxis

20
Q

Which group is most commonly affected by epistaxis admissions?

A

Elderly >70

21
Q

Classification of epistaxis?

A

primary
secondary- anticoagulants
acute
chronic
adult
childhood

22
Q

What is the most important part of history for epistaxis?

A

OTC medications
warfarin
antiplatelets

trauma

alcohol

hypertension

23
Q

What is a genetic condition that causes epistaxis?

A

Hereditary haemorrhagic telangiectasia (HHT)

24
Q

What is the most common site of bleeding in the nose?

A

Kesselbach’s plexus/little’s area

25
Q

Which is the most common artery that is ligated in epistaxis?

A

sphenopalatine artery

26
Q

What is the management of epistaxis?

A

decongestant

adrenaline patch
silver nitrate cautery

27
Q

What is the algorithm for epistaxis

A

resus
examination
ID vessel
Seal

28
Q

If vessel not located, what should you do to stop the bleeding?

A

anterior packing (tampon in the nose)

29
Q

If packing doesn’t work, what could you do?

A

surgery
septal surgery (if difficult access)
arterial ligation

30
Q

Sphenopalatine artery branches off from which major artery?

A

external carotid artery (then from internal maxillary artery)

31
Q

Can you stop warfarin in patient with epistaxis?

A

yes if they have something like AF, but not if they have metallic heart valve

32
Q

What is floseal?

A

for secondary epistaxis
-gletain and human thrombim, helps form clot

33
Q

Can you stop antiplatelets in someone with epistaxis?

A

yes if secondary CDV prevention but no if post-MI or recent insertion of stent…

34
Q

Contraindication for TXA?

A

prev thrombosis

35
Q

orbito-ethmoid fracture, which artery is at risk of bleeding?

A

anterior ethmoid artery

36
Q

Causes of adult recurrent epistaxis?

A

liver disease
medication
nasal tumour

37
Q

What is the mode of inheritance of hereditary haemorrhagic telagectasia?

A

AD

38
Q

Management of HHT?

A

arterial ligation
antifibronlytic agent
TXA
selective embolisation
anticoagulate
young’s procedure??

39
Q

What is Samter’s triad?

A

The association of asthma, aspirin sensitivity and nasal polyposis is known as Samter’s triad.