rhythm notes Flashcards

1
Q

A fib

Rate
Rhythm
P wave –
PR interval –
WRS interval –
ST segment –

A

Rate – varies (atrial rate > 400 bpm, ventricular rate 100-175 bpm)
Rhythm – irregular
P wave – absent, not before QRS
PR int – none
QRS – normal
ST segment

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2
Q

v fib
Rate
Rhythm
P wave –
PR interval –
WRS interval –
ST segment –

A

Rate – not measurable
Rhythm – irregular
P wave – none
PR int – none
QRS – none
ST segment –

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3
Q
A

A fib

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4
Q
A

NSR

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5
Q

Rate – varies (atrial rate > 400 bpm, ventricular rate 100-175 bpm)
Rhythm – irregular
P wave – absent, not before QRS
PR int – none
QRS – normal
ST segment

A

A fib

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6
Q

Rate – varies (atrial rate may be >250 bpm, ventricular rate slower)
Rhythm – irregular or regular
P wave – absent
PR int – none
QRS – normal
ST segment -

A

A flutter

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7
Q
A

VTACH

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8
Q

Rate – not measurable
Rhythm – irregular
P wave – none
PR int – none
QRS – none
ST segment –

A

V fib

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9
Q

NSR
Rate
Rhythm
P wave –
PR interval –
WRS interval –
ST segment –

A

Rate 60-100
Rhythm regular
P wave – present, one before every QRS
PR interval – normal
WRS interval – normal
ST segment – normal

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10
Q

Rate 150-250
Rhythm - regular or irregular
P wave – none
PR int – none
QRS - WIDE
ST segment –

A

VTACH

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11
Q

A fib and A flutter
3. s/s?
- depends on (3)
- typically, s/s of _____

which is most common dysrhythmia?

which one do we see prevalence increases with age?

A
  • depends on
  • ventricular rate
  • how long rhythm has been present
  • CV status
  • typically, s/s of tachydysrhythmia ***
  • most common dysrhythmia = A fib
  • prevalence increases with age = A fib
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12
Q

Rate – not reported in rate, count number of ______
Rhythm – regular or irregular (name the underlying rhythm)
P wave – absent on the _____
PR interval – none
QRS – WIDE
ST segment –

A

PVC

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13
Q

PAC

Rate
Rhythm
P wave –
PR interval –
WRS interval –
ST segment –

A

Rate – not reported in rate, count number of PACs
Rhythm – regular or irregular (name the underlying rhythm)
P wave – present, before QRS, but looks different on the PAC
PR interval – N/A (don’t measure)
QRS – normal
ST segment – normal

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14
Q

what could cause sinus brady?
1. excessive vagal nerve stimulation or inhibition?
2. Carotid sinus massage
3. Vomiting/gagging
4. Valsalva maneuvers
5. anxiety
6. Eyeball pressure
7. Administration of parasympathomimetic drugs
8. hypoxia
9. digoxin toxicity
10. Hyp___kalemia
11. low BP
12. MI
13. may be a normal rhythm in athletes and during sleep

A
  1. excessive vagal nerve stimulation (causes rest and digest)
  2. Carotid sinus massage
  3. Vomiting/gagging
  4. Valsalva maneuvers
    X5. anxiety
  5. Eyeball pressure
  6. Administration of parasympathomimetic drugs
    X8. hypoxia
  7. digoxin toxicity
  8. Hyperkalemia – slows depolarization
    X11. low BP
  9. MI
  10. may be a normal rhythm in athletes and during sleep
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15
Q
A
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16
Q
A

PAC

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17
Q

Rate >130
Rhythm – regular
P wave – not discernable
PR int – not measurable
QRS – normal
ST segment

A

PSVT

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18
Q
A

a flutter

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19
Q

PSVT
1. what would be the next best nursing action or related nursing interventions or treatment?
1st
2nd
third

A

1st vagal maneuvers – trigger vagal response/PNS, brings pt out of PSVT
- Valsalva maneuver – most effective, hold breath 10-15 secs, should see JVD, then resume breathing
- Coughing
- Carotid sinus massage – HCP only
Or diving reflex/cold water submersion

2nd (If that doesn’t work) adenosine
- IV push followed with rapid NS flush (may use stop cock)
- Warn pt may see pause on rhythm strip – flat line
- Onset is 10-40 secs
- Duration – 1-2 mins
- very short half life

3rd (if that doesn’t work and pt becomes hemodynamically unstable)
- cardioversion, synchronized switch on

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20
Q
A

PSVT

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21
Q

Rate 60-100
Rhythm regular
P wave – present, one before every QRS
PR interval – normal
WRS interval – normal
ST segment –

A

NSR

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22
Q
A

V fib

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23
Q
A

sinus arrhythmia

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24
Q

PVC
1. what would be the next best nursing action or related nursing interventions, How is this treated?

Nursing action:
(2)

Treatment:
(2)

A

Nursing action:
- monitor for new PVC or increasing frequency
- contact HCP if new PVC or increasing frequency (could be turning into VTACH)

Treatment:
- treat the cause
- drugs
- beta blockers
- lidocaine
- amiodarone

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25
VTACH 2. what could cause this rhythm? - MI - CAD - electrolyte imbalance - HF - fever - drug toxicities 3. s/s? 4. where does the impulse originate in this rhythm?
2. - MI - CAD - electrolyte imbalance - HF X- fever - drug toxicities 3. - will be symptomatic very quickly unless converts back to other rhythm 4. - ectopic focus within the ventricles takes controls and fires repeatedly - no atrial contractions occurring (very decreased cardiac output)
26
sinus arrhythmia 1. what would be the next best nursing action or related nursing interventions, How is this treated? 2. what could cause this rhythm? 3. s/s? 4. where does the impulse originate in this rhythm?
1. what would be the next best nursing action or related nursing interventions, How is this treated? - Normal cardiac rhythm - document - No treatment necessary 2. what could cause this rhythm? - Seen in young healthy people - A result from changes in intrathoracic pressure w/ breathing. 3. s/s? none 4. where does the impulse originate in this rhythm? Sinus/sinoatrial/SA node
27
4. where does the impulse originate in PAC?
ectopic pacemaker (group of cells in the heart, other than the SA node, that spontaneously generates electrical impulses) in atrium fires before SA node fires
28
sinus brady 1. what would be the next best nursing action, related nursing interventions, how is this treated? 1st 2nd, 2nd 3rd 4th
1st check if pt is symptomatic - asymptomatic = 2nd monitor (may be normal in some people like athletes and during sleep) - symptomatic*** 2nd atropine - Vagolytic = blocks vagus nerve, will increase HR 3rd transcutaneous pacing of heart (temp) 4th pacemaker (permanent)
29
If QRS is absent = (2)
V fib Asystole
30
A fib 2. what could cause this rhythm? - can occur with any underlying heart disease - electrolyte imbalance - hypoxia - cardiac surgery - fever
- can occur with any underlying heart disease - electrolyte imbalance - hypoxia - cardiac surgery X- fever
31
sinus brady
32
causes of sinus tachy? 1. vagal inhibition or stimulation? 2. physical activity 3. digoxin toxicity 4. anxiety 5. pain 6. Hyperkalemia 7. stress 8. fever 9. anemia 10. hypoxia 11. Administration of parasympathomimetic drugs 12. low BP 13. dehydrate 14. Valsalva maneuvers 15. hypovolemia 16. low SV 17. MI 18. Carotid sinus massage 19. HF
1. vagal inhibition 2. physical activity X3. digoxin toxicity 4. anxiety 5. pain X6. Hyperkalemia 7. stress 8. fever 9. anemia = r/t lack of RBC to oxygenate 10. hypoxia X 11. Administration of parasympathomimetic drugs 12. low BP = sinus tachy is a compensatory mechanism for low BP 13. dehydrate = low BP X14. Valsalva maneuvers 15. hypovolemia= low BP 16. low SV = low BP 17. MI = low BP X18. Carotid sinus massage 19. HF = low BP
33
NSR 1. what would be the next best nursing action or related nursing interventions, how is this treated? 2. what could cause this rhythm? 3. s/s? 4. where does the impulse originate in this rhythm?
Normal cardiac rhythm, document, No treatment necessary Normal cardiac rhythm following normal conduction pattern none Sinus/sinoatrial/SA node
34
2. what could cause PAC? - benign (common) - electrolyte imbalance - CVD - stress - cardiac stimulants – caffeine - atrial pathology (any disease or abnormality that affects the atria of the heart, includes: A fib, A flutter) - fever
- benign (common) - electrolyte imbalance X- CVD - stress - cardiac stimulants – caffeine - atrial pathology (any disease or abnormality that affects the atria of the heart, includes: A fib, A flutter) X- fever
35
rate >100 rhythm - regular p wave – present and before every QRS PR interval – normal QRS – normal ST segment – normal
sinus tachy
36
v fib 1. what would be the next best nursing action or related nursing interventions, How is this treated? (3) 4. where does the impulse originate in this rhythm?
1. - CPR - ACLS - defibrillation (synch switch turned off, no QRS) 4. - ventricles are quivering - no effective contractions = NO cardiac output
37
PVC
38
A flutter Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate – varies (atrial rate may be >250 bpm, ventricular rate slower) Rhythm – irregular or regular P wave – absent PR int – none QRS – normal ST segment -
39
VTACH 1. what would be the next best nursing action or related nursing interventions, How is this treated? (4)
1- check if patient has pulse or is pulseless - treatment depends on pulse (perfusion) or pulseless (no perfusion) 2- ACLS 3- anti-dysrhytmic drug – beta blocker, calcium channel blockers, amiodarone 4- electrolyte replacement
40
sinus tachy 3. s/s 4. where does the impulse originate in this rhythm?
*** Sinus/sinoatrial/SA node
41
Rate – not reported in rate, count number of ____ Rhythm – regular or irregular (name the underlying rhythm) P wave – present, before QRS, but looks different on the ____ PR interval – N/A (don’t measure) QRS – normal ST segment – normal
PAC
42
sinus brady Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate – <60 Rhythm – regular P wave – normal PR interval – normal QRS complex – normal ST segment – normal
43
P wave and PR interval will be normal in only (4)
Sinus brady Sinus tachy Sinus arrythmia Normal sinus rhythm
44
sinus arrhythmia Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate – varies Rhythm – irregular P wave – normal PR interval – normal QRS complex – normal ST segment – normal
45
PSVT Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate >130 Rhythm – regular P wave – not discernable PR int – not measurable QRS – normal ST segment
46
PAC 1. what would be the next best nursing action or related nursing interventions or treatment? nursing action: (2) Treatment: (2)
nursing action: - Monitor for new or increasing PACs - contact the HCP if new or increasing PACs (could indicate pt is about to convert to A fib) Treatment: - benign = no treatment - if atrial pathology is the cause = same treatment as A fib
47
sinus tachy 1. what would be the next best nursing action, related nursing interventions, how is this treated? (2)
- treat the cause Ex: -If FVD = fluid volume replacement - If in pain = give analgesic - If febrile = give anti-pyretic - If panic attack/anxiety = give benzo/anxiolytic - give beta adrenergic blockers – reduce HR and myocardial oxygen consumption
48
Rate – varies Rhythm – irregular P wave – normal PR interval – normal QRS complex – normal ST segment – normal
sinus arrhythmia
49
PVC 2. what could cause this rhythm? - if it’s an isolated PVC – may be benign - stimulants - vomiting - electrolyte imbalance - Hypoxia - fever - exercise - emotional stress - CVD 4. where does the impulse originate in this rhythm?
- if it’s an isolated PVC – may be benign - stimulants X- vomiting - electrolyte imbalance - Hypoxia - fever - exercise - emotional stress - CVD 4. ectopic focus in the ventricles (not the atria) - it comes earlier (premature) than the QRS should come - doesn’t follow a normal rhythm or p wave
50
VTACH Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate 150-250 Rhythm - regular or irregular P wave – none PR int – none QRS - WIDE ST segment –
51
PSVT 2. what could cause this rhythm? - overexertion - fever - emotional stress - stimulants - digitalis toxicity - various forms of heart disease
- overexertion X- fever - emotional stress - stimulants - digitalis toxicity - various forms of heart disease
52
If QRS is wide = (2)
PVC V Tach
53
A fib 4. where does the impulse originate in this rhythm?
- NOT the SA node (it’s being taken over and is no longer the pacemaker of the heart) - multiple ectopic foci firing all at the same time = loss of effective atrial contraction/kick (p wave) - total disorganization of atrial electrical activity
54
PSVT 3. s/s? - depends on (2) - s/s of 4. where does the impulse originate in this rhythm?
3. s/s? - depends on - How long it lasts - How fast the ventricular rate is (tachycardia), if it’s too high CO is reduced - s/s of *** 4. ectopic focus anywhere above bifurcation of bundle of His, anywhere in atria (“supraventricular”)
55
Rate – <60 Rhythm – regular P wave – normal PR interval – normal QRS complex – normal ST segment – normal
sinus brady
56
A flutter 2. what could cause this rhythm? - can occur with any underlying heart condition - electrolyte imbalance - hypoxia 4. where does the impulse originate in this rhythm?
- can occur with any underlying heart condition - electrolyte imbalance X- hypoxia a single ectopic focus firing = loss of effective atrial contraction/kick (p wave)
57
sinus brady 3. s/s 4. where does the impulse originate in this rhythm?
3. *** 4. Sinus/sinoatrial/SA node
58
PVC Rate Rhythm P wave – PR interval – WRS interval – ST segment –
Rate – not reported in rate, count number of PVCs Rhythm – regular or irregular (name the underlying rhythm) P wave – absent on the PVC PR interval – none QRS – WIDE ST segment –
59
A fib and A flutter (and PAC if not a benign cause) 1. what would be the next best nursing action or related nursing interventions or treatment? 1st 2nd 3rd (if that didnt work) 4th (if that didnt work)
1st - check if they are hemodynamically stable and if they are symptomatic 2nd - if hemodynamically unstable (VS are not ok): synchronized cardioversion/cardiovert/life pack and synchronized switch is turned on 2nd - if hemodynamically stable (VS are ok), but symptomatic***: Slow ventricular rate with either IV - calcium channel blockers - beta blockers - digitalis - amiodarone (“Bolus and start a drip” may be ordered) 48 hours later 3rd - planned cardioversion - with anticoagulation therapy (warfarin/coumadin) before cardioversion for 3-4 weeks AND after cardioversion for 3-4 weeks - TEE may be performed before cardioversion 4th - long term anti coagulation required - drug of choice - warfarin/coumadin - alternatives – dabigatran, apixaban, rivaroxaban, eboxaban
60
warfarin/coumadin vs alternatives – dabigatran, apixaban, rivaroxaban, eboxaban 1. anticoagulants 2. Have to monitor INR regularly 3. Don’t require routine lab testing 4. Antidote – vitamin K 5. More expensive 6. May have to takes >once per day 7. Contraindicated with renal dysfunction 8. No antidote
W/C & A 1. anticoagulants W/C 2. Have to monitor INR regularly A 3. Don’t require routine lab testing W/C 4. Antidote – vitamin K A 5. More expensive A 6. May have to takes >once per day A 7. Contraindicated with renal dysfunction A 8. No antidote
61
A fib vs A flutter single ectopic focus firing multiple ectopic focus firing
A flutter - single ectopic focus firing A fib - multiple ectopic focus firing
62
which do you check for pulse with
VTACH
63
treat the cause for (2)
PVC, sinus tachy
64
atropine ___ HR treats ___ beta blockers lidocaine amiodarone ___ HR treats ___ adenosine ___ HR treats ___ beta blockers CCB amiodarone ___ HR treats ___ beta blockers CCB amiodarone digitalis ___ HR treats ___
increases sinus brady decreases PVC decreases PSVT decreases VTACH decreases A fib