S-Z Flashcards

(40 cards)

1
Q

What is School refusal?

A

Child motivated refusal to attend school and behavioral difficulties when in school.

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2
Q

What is the aetiology of School refusal?

A

Separation anxiety disorder persisting over 3-4y, environmental factors such as depravation, abuse, neglect, bullying, and academic performance related.

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3
Q

What is the epidemiology of School refusal?

A

Peaks at age of school changes, 5-11, sometimes 14-15. Higher in lower SES. May coexist with separation anxiety disorder, depression etc.

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4
Q

What would you find in the history/ exam of School refusal?

A

Structured interview

School avoidance pattern: refusing to go, leaving early, being ill, crying, clinging, tantrum, distressed during school.

· Internalization: worrying, fatigue, somatization, stress.

· Externalization: crying, clinging, screaming, behavioral

Screen for depression (low mood, anhedonia, unworthiness feeling), other organic causes, and for behavioral problems (sleep, eating, conduct disorder, substance abuse).

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5
Q

How would you investigate School refusal?

A

If organic cause suspected, FBR, TFT, urine dip, glucose.

Urine toxicology if indicated and ?substance abuse

MRI/CT if neurological cause suspect. Assess hearing and vision if concerned.

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6
Q

What is the management of School refusal?

A

Early stepwise return to school with positive reinforcement. CBT.

Medical TX if medical problem, SSRI (fluoxetine) may be appropriate in children with signs of depression.

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7
Q

What are the complications and prognosis of School refusal?

A

Deteriorating school performance, social isolation, family issues.

Likely to solve unless long standing.

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8
Q

What is Sleep related disorders?

A

Night terrors: disturbance in the stricture of sleep

Nightmares: frightening dreams

Difficulty sleeping without parent.

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9
Q

What is the aetiology of Sleep related disorders?

A

Night terrors: may be due to fever, stress, medication, lack of sleep.

Nightmares: stressful event, drugs, fever, fhx.

Difficulty: separation anxiety.

Associated with LD, depression, PTSA, ASD, ADHD.

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10
Q

What is the epidemiology of Sleep related disorders?

A

NT usually 4-12y onset, NM usually 3-6yo, DIFF common in toddlers.

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11
Q

What would you find in the history/ exam of Sleep related disorders?

A

NT: recurrent episodes of intense crying and fear about 1h30 during the night after falling asleep. Lasts 2 min. Following terror, hard to rouse, disoriented up to 10 min. During terror may have tachypneic, sweating, but does not remember in the morning. During the transition from non-REM to REM sleep with sudden autonomic activation.

MN: usually occur in the middle of the night. Threat to the child, loss of control or dear of injury. Child is alert on waking. Stress in the day. Occur during REM.

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12
Q

How would you investigate Sleep related disorders?

A

EEG if associated nocturnal seizures.

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13
Q

What is the management of Sleep related disorders?

A

Parental reassurance.

NT: ensure sleeping in safe environment, regular bedtimes, wake child after 1h30 to avoid terror, keep a record of times when occurring.

NM: relax child, psych consultant if possible PTSD etc.

DIFF: routines for sleeping, in extreme cases sedate for a few nights followed by increasing length of item between leaving room and returning, until child falls asleep before return.

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14
Q

What are the complications and prognosis of Sleep related disorders?

A

Distress.

Usually outgrown.

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15
Q

What is Tics?

A

TICS: stereotyped movement of muscle groups with no clear function

Tourette’s: chronic idiopathic syndrome with both motor and vocal tics beginning before adulthood.

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16
Q

What is the aetiology of Tics?

A

Genetic: higher concordance in twins, higher in first degree relative.

Acquired: ? Abs to GBS?

Associated with ADHD in 30%, OCD in 20%.

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17
Q

What is the epidemiology of Tics?

A

3-15% of children. Declining by adolescence. Usual onset 7-9y. Tourette 1%.

18
Q

What would you find in the history/ exam of Tics?

A

Simple: brief movements of few muscle groups, may be transient <1y or chronic>1y.

Complex tics: coordinated movements involving several muscle groups, or coprolalia (outburst of obscenities). Worsened by stress and reduced by absorbing activities.

Tourette: Multiple motor and vocal tics. Occur more than once a day for over a year, vary in nature. Rage attacks may occur, with unpredictable outbursts, out of proportion to stimulus, threatening destruction and self injury, with near immediate remorse.

?due to reduction in basal ganglia inhibition of undesired motor programs.

19
Q

How would you investigate Tics?

A

Usually none. In specific cases, exclude things. ASOT may be required if sudden onset. TFT to exclude hyperthyroidism. Serum ceruloplasmin to screen for Wilsons. EEG to check absence seizures.

Assess mental health, ADHD and OCD.

20
Q

What is the management of Tics?

A

MDT approach: supportive parental education, behavioral and psychotherapy to reduce habit

Medical treatment may include neuroleptic drugs in low dose or dopamine agonists.

Treat comorbid psych disease (OCD: SSRI, ADHD: atomoxetine if tics present)

21
Q

What are the complications and prognosis of Tics?

A

Stigma, embarrassment, social withdrawal

Worsen throughout childhood but usually remit by 18.

22
Q

What is Visual impairment?

A

Decrease visual acuity (<6/18 on Schnellen). Blind if <3/18).

23
Q

What is the aetiology of Visual impairment?

A

Developed countries mainly genetic, developing countries mainly acquired.

24
Q

What is the epidemiology of Visual impairment?

A

10-20/10000 UK.

25
What would you find in the history/ exam of Visual impairment?
Lack of eye contact, no responsive smiling and sharing by 6wk, no social bonding, random eye movements, squint, abnormal perceptual development, delays. May be normal if impairment is of cortical origin. However, may lack fixation and visual tracking or have persistent nystagmus. Squint and cataracts would be seen inmmediately using ophthalmoscope at baby check. (no red reflex)
26
How would you investigate Visual impairment?
Use Snellens chart if possible. (>5y) slit lamp exam. CT/MRI if indicated.
27
What is the management of Visual impairment?
Maximise development of signs and compensatory responses as soon as possible. Correct refraction errors. Advise patient families on providing non visual stimulation. Ensure safe environment for the child. Special schooling may be required for severe impairment.
28
What is Strabismus?
Abnormal alignment of both eyes. As a result, lock in position without focus on a single point. Most commonly horizontal (convergent or divergent) but may be vertical (hypertropia upward, or hypotropia downward).
29
What is the aetiology of Strabismus?
Failure to develop binocular vision. Non paralytic: more common, due to refractive error in one or both eyes. Paralytic: Rare and due to paralysis of motor nerves. When onset is rapid, may be due to SOL such as tumor.
30
What is the epidemiology of Strabismus?
4/100.
31
What would you find in the history/ exam of Strabismus?
Neonates often look squinty due to overconvergence. Strabismus is only defined in infants >2-3months. May be intermittent. Corneal light reflection test: torch over eyes, if you can see in in symmetrical place on middle of pupils on both sides there is no squint. If not on middle of pupil, squint. Cover test: squinting eye moves to take up fixation when the normal eye is covered. Fundoscopy and neurological examination important (remember – SOL!)
32
How would you investigate Strabismus?
CT/MRI brain if suspect SOL.
33
What is the management of Strabismus?
Refer all children to ophthalmologist. 1. Correct any underlying issue i.e. cataracts 2. Correct any refractive error with glasses. 3. Treat amblyopia with patch occlusion therapy if it occurs. Non paralytic strabismus may be controlled with glasses that correct for overconvergence (long sightedness). Congenital paralytic squints require surgery ASAP to enable good visual function.
34
What is Cataracts?
Opacificaiton of lens at birth.
35
What is the aetiology of Cataracts?
Familial: usually AD Congenital infection: TORCH Drugs: corticosteroids. Metabolic: hypocalcaemia, galactosaemia, DM Chromosomal: Downs, Turners, trisomy13, trosimy18. Idiopathic in 1/3
36
What is the epidemiology of Cataracts?
1/250
37
What would you find in the history/ exam of Cataracts?
Congenital cataracts present at birth but may not be identified until later. Some cataracts static, some progressive. Loss of red reflex, white reflex in pupil instead (DDx: retinoblastoma, ROP, cataracts)
38
How would you investigate Cataracts?
Slit lamp exam, investigate to exclude retinoblastoma and ROP.
39
What is the management of Cataracts?
Surgical removal of cataract, ideally before 2 months.
40
What are the complications and prognosis of Cataracts?
Amblyopia if surgery is delayed, strabismus, glaucoma post surgery. Vision is irreversibly impaired if not treated, great prognosis with treatment.