Sabiston GERD and HH Flashcards
(40 cards)
LES is made up of four anatomic structures
1-The intrinsic musculature of the distal esophagus
2- Sling fibers of the gastric cardia
3-The crura of the diaphragm
4- increased intra abdominal pressure is transmitted to the GEJ
Best time to measure LES
at mid-expiration or end-expiration.
Hypotensive LES is frequently associated with
hiatal hernia because of displacement of the GEJ into the posterior mediastinum
A type I hernia
when the GEJ migrates cephalad into the posterior mediastinum.
This occurs because of laxity of the phrenoesophageal membrane
water brash phenomenon
Regurgitation of gastric contents to the oropharynx and mouth > produce a sour taste that patients will describe as either acid or bile
Two mechanisms may lead to extraesophageal symptoms of GERD
-proximal esophageal reflux and microaspiration of gastroduodenal contents cause direct caustic injury to the larynx and lower respiratory tract
-distal esophageal acid exposure triggers a vagal nerve reflex that results in bronchospasm and cough
GERD with extraesophageal symptoms should be referred to
a laryngologist or a pulmonologist to determine if a nongastrointestinal condition is causing these symptoms
In patients with asthma and GERD
antireflux surgery appears to be more effective than medical therapy at managing pulmonary symptoms
Proximal esophageal reflux with microaspiration of acid and nonacid gastric contents can lead to
Idiopathic pulmonary fibrosis > severe, chronic, and progressive lung disease that generally results in death
Tx : Laparoscopic antireflux surgery
Signs of proximal esophageal reflux and regurgitation of gastric contents
-erosion of their dentition (revealing yellow teeth caused by the loss of dentin)
-injected oropharyngeal mucosa
-signs of chronic sinusitis
-constantly drinks water ( To clear Acid )
-sit leaning forward > To flattens diaphragm, narrows the anteroposterior diameter of the hiatus, and increases the LES pressure
pathognomonic for GERD ( By Endoscopy )
peptic strictures and LA class C and D esophagitis
LA class A and B esophagitis should undergo
pH testing
HH measurement in Endoscopy
hernia should be measured in both cranial-caudal and lateral dimensions
most common cause of dysphagia in GERD
is a reflux-associated inflammatory process of the esophageal wall
What can cause a false-negative pH study
presence of a tight stricture may prevent reflux of acid
The majority of peptic strictures are effectively treated with
-dilation and PPI therapy
Refractory peptic strictures are defined as strictures that recur despite dilation and PPI therapy > Tx with LARS / steroid injections
Another cause of dysphagia in patients with GERD
Schatzki ring >
submucosal fibrotic bands (as opposed to mucosal strictures).
Ineffective esophageal motility
at least 50% weak or failed peristaltic swallows on high-resolution manometry.
patients with GERD and ineffective esophageal motility > (Toupet or Dor) fundoplication should be performed
Nissen fundoplication > greater postoperative dysphagia
For barret Based on endoscopic measurements
long segment (≥3 cm) and short segment (<3 cm).
present with typical symptoms of GERD Tx
8-week course of PPI therapy is recommended
How long to stop PPI for ambulatory pH monitoring
patients must stop PPI therapy 1 week
PPI use has been associated with the following
-loss of bone density and risk of fracture
-dementia
-myocardial infarction
-micronutrient (magnesium, iron, B-12) deficiencies
-Clostridioides difficile infection
-kidney disease
-interactions with antiplatelet medication
Intraoperative Management of Short Esophagus
-unilateral vagotomy > additional 1 to 2 cm of esophageal length
-division of both vagus nerves > 3 to 4 cm of additional esophagus
Operative Complications of LARS
-Pneumothorax
When identified intraoperatively, the pleura should be closed with a suture
postoperative radiograph should be obtained.
If a pneumothorax is identified on this radiograph, the patient may be maintained on oxygen therapy to facilitate its resolution