Sedation + Behavior Guidance + Abuse/Neglect + Pain Management Flashcards
(241 cards)
Goals of procedural sedation
- Promote patient welfare + safety
- Facilitate provision of quality care
- Minimize extremes of disruptive behavior
- Promote positive psychological response to tx
Child has ~__ alveoli at birth which grows to ~__ by adulthood
Child has ~20 million alveoli at birth which grows to ~300 million by adulthood
Respiratory system is the last to develop
Young children have less surface area for gas exchange
Hypercarbia + children
Children tolerate greater incidences of hypercarbia, unlike adults, where hypercarbia drives immediate increased respiratory response.
Hypercarbia may be associated w/ onset of cardiac arrhythmias
Pediatric diaphragms
Pediatric diaphragm is less capable of displacing large volumes in vertical dimensions.
Children have relatively small functional reserve capacity.
Likewise, children have relatively larger post-expiration volume.
Pediatric vs. adult airways
- ⇡airway resistance
- Glottis (vocal cords) are positioned more anteriorly + cephalad
- Narrowest portion of the upper airway is at level of cricoid cartilage (below vocal cords)
- A relatively larger tongue + epiglottis
- A larger head to body size ratio
- Mandible is less developed
- Potential for significant lymphoid tissue obstructing:
- Nasopharynx
- Oropharynx
- Larygopharynx
What affects medication regimen selection in sedation?
- Extent of tx
- Child temperament
- Age/parental expectations
Patient selection for sedation
- Traditional techniques have been unsuccessful in managing behavior
- Patient ASA I or ha a medical condition that is well-controlled ASA II
- Patient is pre- or uncooperative
- Extent of tx: ultimately determined by amount of LA allowed by weight
- Needle phobic; excessively fearful older child
- Older child w/ poor experiences or coping abilities
- Distance traveled even for patients w/o behavioral problems
Prematurity + sedation
- Reduced surfactant; alveoli less patent
- Less surface area for qualitative gas exchange
- Poor qualitative + quantitative gas exchange
- ⇡incidence of early life intubation
- Altered neural reflex pathways; ⇡gag reflex
- Potentially at greater risk for laryngospasm
- Hospitalization; ventilator use
Atopic triad
- Asthma
- Eczema
- Food allergy
May suggest IgE hyper-responder caution
C-spine precautions w/ sedation
Down syndrome
Arnold-Chiari malformation
ASA pre-procedure fasting guidelines for sedation
- No solid foods, non-human milk, and infant formula up to 8hr
- Clear liquids = 2hr minimum fasting period
- For children +6mo
- Clear liquids include anything w/o pulp/particulate
- Breast milk = 4hr minimum fasting period
- Infant formula = 6hr minimum fasting period
- Non-human milk = 6hr minimum fasting period
- Light meal = 6hr minimum fasting period
ADD/ADHD medications + sedation
- Should be taken following NPO guidelines
- Bladder empty prior to giving meds
- Baseline vital signs
LA toxicity
Initial signs of mild LA toxicity may mimic effects of N2O/O2 or may be misinterpreted as painful response/maladaptive behavior
- Lidocaine: CNS + CV effects
- 4.4mg/kg w/ or w/o epi
- Septocaine: CNS + CV + immune effects
- 4-5mg/kg (7mg/kg listed by manufacturer but also noted that should not be used on children <4yo; 5mg/kg noted for children)
Causes + effects of LA toxicity
- Causes of OD
- Intravascular injection
- Excess dosage delivered to patient
- Effects
- CNS excitement followed by depression, seizures, disorientation, loss of consciousness
- CV system depressed
- ⇣myocardial contractility
- ⇣cardiac output
- CV collapse
Minimal level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel
- Cognitive Function: May be impaired
- Physiological Function: Not affected
- Monitor: Observation only; intermittent
- Personnel: Not specified
Moderate level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel
- Cognitive Function:
- Depression
- Responds to light tactile stimulation
- Physiological Function:
- Patent, self-correcting airway
- Ventilation + CV function is adequate
- Monitor:
- O2 sat
- HR
- Intermittent BP + RR (no designated period of recording)
- Capnography recommended if patient is not capable of appropriate bi-directional verbal interactions
- EKG + defib available (should)
- Personnel:
- Person responsible for monitoring other than operator
- May do other tasks
Deep level of sedation: Cognitive Function, Physiological Function, Monitor, Personnel
- Cognitive Function:
- Depression
- Not easily aroused
- Physiological Function:
- Potential loss of airway reflexes
- CV may be affected
- Monitor:
- O2 sat
- HR
- EKG
- Capnography recommended
- Intermittent BP + RR (recorded q5min)
- Personnel:
- Person responsible solely for monitoring
Sedation - fatal trio
- Hypovolemia = Patients are NPO, verify “not over NPO” but as close to guidelines as possible
- Hypoxia = Recognize respiratory obstruction/distress immediately
- Hypercapnia = If hypoxia persists, hypercapnia may result + make patient more prone to cardiac arrhythmias
How often do you calibrate inhalation equipment (sedation)
Annually
Where do you do submucosal injection
B/w 1st and 2nd primary molar in maxillary vestibule
N2O-O2 induction + recovery
- Induction = 5min
- Recovery = 10min
Relative contraindications for N2O-O2 (may be used following med consult)
- Wheezing (moderate-severe asthma)
- Nasopharyngeal obstruction
- Tuberculosis, cystic fibrosis
- Sickle cell disease (due to lowered oxygen tension in blood)
- Acute otitis media
- Methionine synthetase deficiency
- 1st trimester pregnancy
Other contraindications: COPD, narrow angle glaucoma, pneumothorax, small obstruction, middle ear surgery, retinal surgeries
Chronic exposure/abuse of N2O-O2
May result in peripheral neuropathies
Chloral hydrate
- Sedative/hypnotic
- CNS depression – minimal CV or respiratory effect
- Mucosal irritant
- Gastric irritation a side effect; unpleasant taste
- Onset: 30-60min
- Peak: 60min
- Duration: 5hr
- Working time: up to 60min
- 10-50mg/kg PO to 1g max (as sole agent; reduced doses if used in combination w/ other sedatives)
- Metabolized to trichlorethanol in liver; excreted by kidney
- Arrhythmias in higher doses (usually >75mg/kg)
- No reversal agent
- When combined w/ other sedates, this is why CH dose should be loweer