Shock Flashcards

(33 cards)

1
Q

catecholamines cause _____, _____, and ____ resulting in _______

A

increased HR, increased contractility, and vasoconstriction; resulting in increased CO

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2
Q

Baroreceptors

A

(pressure receptors) located in the carotid sinus and aortic arch.

A decreased MAP causes decreased stretching of the baroreceptors which results in sympathetic response of the ANS

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3
Q

Chemoreceptors

A

located in the aortic arch and carotid arteries

  • receptive to oxygen changes in blood
  • regulate BP and HR
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4
Q

What is the compensation for metabolic acidosis?

A

increased RR, which causes resp alkalosis

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5
Q

vasoconstriction increases _____, ____& ______

A

preload, SVR, and CO

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6
Q

what is systemic vascular resistance (SVR)?

A

how hard the heart has to pump to get blood out into the system

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7
Q

when the kidneys release rennin what happens?

A

it leads to the conversion of angiotensin I to angiotensin II (vasoconstrictor)

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8
Q

what does aldosterone do?

A

causes retention of Na and H2O= ^BP, preload, and CO

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9
Q

an increase in Na triggers…

A

release of ADH

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10
Q

Shock causes “pre-renal” acute renal failure. Ischemia of tubule epithelium leads to…

A

vasoconstriction, decreased GFR, *oliguria, and azotemia

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11
Q

What is stage 1 of shock also called?

A

Compensatory stage, Non-Progressive

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12
Q

what do glucocorticoids do?

A
  • save water
  • increase blood glucose
  • decrease inflammation
  • decrease immune response
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13
Q

What is stage 2 of shock also called?

A

Progressive shock

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14
Q

what are some initial s/sx of the progressive shock stage?

A

decreased LOC, confusion, thirsty, dizzy, restless, nauseated

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15
Q

What are some things that are going on during the 2nd stage of shock?

A
  • Anaerobic metabolism leading to metabolic acisosis
  • Cellular damage (Na enters cell, K leaves cell, cell ruptures)
  • Coagulation defects- blood clots
  • Cardiovascular changes- incrased HR, increase in O2 demands, circulating volume is decreased by 25-35%, vasoconstriction
  • Signs of failure of more than one organ may be apparent
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16
Q

**2 early signs of shock

A

decreased LOC and tachycardia

17
Q

Causes of hypovolemic shock

A
trauma
anticoagulants
diuretics
dehydration
third spacing
draining wounds and burns (weeping)
water loss from vomiting, diarrhea, sweating
diabetes insipidus
18
Q

*S/sx of hypovolemic shock

A
  • altered mentation: lethargy to unconsciousness
  • *rapid and deep or shallow respirations
  • cool, clammy skin
  • tachycardia (pulse weak and thready)
  • decreased BP
  • decreased CO
  • decreased urinary output
19
Q

Diagnostic findings with hypovolemic shock

A
  • low hematocrit
  • decreased hemoglobin
  • decreased RBCs and platelets
  • elevated serum electrolytes, elevated creatinine and BUN
  • increased urine SG (>1.02)
  • decreased pH
20
Q

what kind of fluids do you give the hypovolemic pt?

A

Isotonic (Normal Saline, Lactated Ringers)

also may need to give blood products

21
Q

NSG care for hypovolemic pt

A
  • Elevate lower extremities to prevent venous pooling and enhance return to heart
  • Assess extent of fluid loss
  • O2
  • monitor VS, neuro stat, cardiac rhythm
  • monitor hemodynamic parameters and I&Os
  • admin blood products and monitor H&H
  • monitor for pulm congestion
  • observe for signs of impending coagulopathy, petechie, bruising, and blood or oozing from gums or puncture sites
22
Q

Complications that can occur from hypovolemic shock

A

renal damage
cerebral anoxia
death

23
Q

Causes of Cardiogenic Shock

A
  • impaired tissue perfusion as a result of cardiac dysfuction (pump failure)
  • MI or severe CHF
  • cardiogenic drugs, penetrating wounds, papillary muscle rupture, cardiac tamponade, septal rupture, cardiomyopathy, myocarditis, valvular disease, or dysrhythmias, tension pneumothorax
  • Heart or Vessels are obstructed/compressed
24
Q

what happens when SVR decreases?

A

the ventricle does not fully eject volume. Pressure backs up in the system to the lungs causing pulmonary congestion

25
S/sx of cardiogenic shock
``` thready, rapid pulse distended neck veins arrhythmias chest pain cool, pale, moist skin oliguria decreased mentation ```
26
Pulmonary s/sx of cardiogenic shock
``` dyspnea increased RR crackles, possible wheezing ABGs show decrease in PaO2 respiratory alkalosis ```
27
How does morphine help decrease the work load of the heart?
decreases preload and afterload, vasodilates
28
List the vasoconstrictors
dopamine, epinephrine, norepinephrine, phenylephrine, vasopressin
29
List the vasodilators
nitroprusside (for HTN emergencies), nitro, ACE inhibitors
30
List the Inotropic agents
amrinone, milrinone, dobutamine
31
What are the 3 types of distributive/normovolemic shock?
neurogenic, anaphylactic, and septic
32
S/sx of septic shock
``` decreased LOC tachycardia fever skin warm and dry rash desquamation (peeling & redness of palms) decreased urinary output decreased BP decreased CO ```
33
Hematologic alterations happening with septic shock
1) endotoxins cause platelet aggregation 2) these cause obstruction to blood flow 3) clotting factors get depleted 4) result: DIC