Shoulder Flashcards

(31 cards)

1
Q

shoulders are complex

A

} Lots can go wrong and numerous structures can cause shoulder pain
} To assess and treat shoulders effectively you need to
} Understand the anatomy } Joints
} Muscles
} Ligaments etc
} Understand the complexity of movement and control } Joint interactions
̈ Any painful or weak link can decrease effectiveness of shoulder function } Complex co-operative muscle interactions
̈ Any single weakness can disrupt kinetic sequencing } Be able to effectively differentially diagnose
} Assessment is the key!!!

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2
Q

4 joints of the shoulder

A
} Sternoclavicular
} Acromioclavicular
} Glenohumeral
} Scapulothoracic
} All 4 joints work co- operatively
} ROM largely produced by ST & GH...
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3
Q

sternoclavicular joint

A

} Clavicle → mechanical strut for scapula through its attachment to sternum
} Links axial skeleton to appendicular skeleton
} Primary purpose → position scapula optimally to receive head of humerus
} Highly stable – ligamentous structure
} Injury due to large forces } Commonly = fracture
BUT
} Potential SC joint dislocation ̈ Anterior vs posterior….

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4
Q

acromioclavicular joint

A

} Stabilised by
} Superior & inferior acromioclavicular
joint capsular ligaments
} Coraclavicular ligament
} Articular disc (of varying form, mostly present)
} Deltoid & upper trapezius muscles
} Proposed rotational adjustment
motions
} Optimally aligns scapula against thorax
} Adds to scapula motion
} Clinically important – don’t neglect!

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5
Q

glenohumeral joint

A
} Multiaxial synovial joint
} Design favours mobility over
stability
} Producing extensive ROM with ST
} Bony fit offers little to no stability
} Glenoid fossa covers approx 1/3 HOH
} * Mechanical integrity maintained by muscles and capsular ligaments
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6
Q

glenohumeral joint

A

} Provided by:
} Joint capsule & capsular ligaments
} Inherently loose capsular ligaments
̈ superior GH, middle GH, inferior GH ligs (esp ant & posterior bands in functional abd position)
} Reinforce capsule to assist maintenance of negative intra- articular pressure
} Taut in varying positions → please review!
} Coracohumeral ligament } Glenoid labrum
} Expands glenoid cavity } Rotator cuff muscles
} LH biceps brachii

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7
Q

scapulothoracic joint

A

} Specific ST ROMs
} BUT important element of overall shoulder biomechanics } Essential involvement in GH ROM & overall shoulder function

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8
Q

scapulohumeral rhythm

A

• *If there is one movement you can assess well…
• Consider ALL joints & relevant muscles
• Overall ratio of 2:1 = GH:ST
• Initial movement = purely GH
• Clavicle retracts & posteriorly rotates
• Scapula tilts & ER
• GH ER
• Why??
• Supraspinatus contracts - taut superior
capsule & depression of HOH
• Auxillary pouch stretches → inferior
sling for HOH

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9
Q

GH joint muscles

A
} Glenohumeral joint } Deltoid
} Anterior, middle & posterior } Coracobrachialis
} Biceps brachii
} Latissimus dorsi
} Pec major
\+
} Rotator cuff
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10
Q

rotator cuff musculature

A
} Supraspinatus (!!!)
} Superior roll of HOH
} Compression into fossa
} Restricts superior translation
} Infraspinatus, teres minor, subscap } Depression force on HOH
} Infraspinatus, teres minor
} External rotation of humerus
abduction
    } *Counteract pull of deltoid → elevation of HOH } Deltoid vs suraspinatus injury
} Consider ability to abduct...
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11
Q

Scapulothoracic joint muscles

A

} Elevators
} Upper trapezius
} Levator scapulae } Rhomboids

} Depressors
} Lower trapezius
} Latissimus dorsi } Pectoralis minor } Subclavius

} Protractors
} Serratus anterior

} Retractors
} Middle trapezius
} Rhomboids
} Lower trapezius

} Upward rotators
} Serratus anterior
} Upper & lower trapezius

} Downward rotators } Rhomboids
} Pectoralis minor

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12
Q

Shoulders are tricky

A

} Difficult to differentially diagnose
} Complex joint interplay
} Complex co-operative muscular control
} Special tests often don’t have high specificity & sensitivity
} Regularly painful > 90o
} Mechanical vs non-mechanical pain
} Pain can be referred from other regions
} May not be MSK issue BUT due to more serious problem…
} Some tips for success
} Be thorough in your history taking } Assess effectively
} Get your patient to do their work!

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13
Q

Hx

A

} Try to pinpoint the site of pain
} Easier said than done!
} AC & bicipital pain often localised
} Define pain
} Type, severity…
} Sensation } Neural
} Referred
} “Dead arm” – baseball pitchers → labral injury
} Onset of pain
} Acute or insidious
} If MOI
} Shoulder position provides clues to structures injured
} Night pain common in some presentations
} Aggravating & easing
} Impact on ADLs

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14
Q

PE

A

} Takes the usual form and covers all bases } LOADS of special tests to choose from…
} Diagnose effectively
} What is your hypothesis?
} What are your differential diagnoses?
} What is contributing to the pain and presentation? } All joints and muscles!!!

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15
Q

6 categories of shoulder pain

A
} Rotator cuff (including impingement)
 } Labral injury
} Instability
} Stiffness
} AC joint pathology
} Referred pain } Cx
} Tx
} Visceral
} Nerve lesions: e.g suprascapular nerve, axillary nerve, long thoracic nerve
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16
Q

referred pain from viscera

A

} Cardiac dysfunction
} Diaphragm dysfunction, lung disease
} Pneumoperitoneum/Perforated ulcer } Disease of the oesophagus
} Aneurysm of the subclavian artery
} Gallbladder and spleen (left shoulder) } Axillary vein thrombosis

17
Q

differential Dx

A

} Pancoasts tumor – pulmonary apex, non small cell Ca

} Bone tumor (young) } More commonly 2o

18
Q

referred pain

A
} Commonly } Cx spine
} Tx spine
} Associated musculature...
BUT
} Shoulder dysfunction can alternatively result in
} Traps pain and fatigue
} Pain radiation into } Neck
} Posterior scapula } Upper arm
} Forearm
} Wrist and hand
19
Q

rotator cuff pathology

A
} One of the most common shoulder problems you will treat
} Supraspinatus most commonly affected
} Tendinopathy
} As per tendon continuum...
} Calcification may occur } Most commonly within
supraspinatus
} Investigation of choice } MRI
} Alternatively US
20
Q

Rotator cuff pathology clinical presentation

A
} Pain often considerable } Rest
} Seemingly minor movements } Night
} Potential for associated instability & “dead arm”
} Pain with overhead activities
} < 90o painfree
} Painful arc – 70-120o
} Catch/difficulty on return
} TOP supraspinatus tendon at insertion point
} If supra tendinopathy.... } ↓ IR – consider HBB
ROM...
} Pain on
} Empty can > full can
} Impingement tests
} EOR passive flexion
21
Q

impingement syndrome

A

} Impingement syndrome - ?overuse, tendinopathy of supraspinatusàpainàRC muscle dysfunctionàpotenital bursitis
} 3 types of impingement
} 1 ̊ External
} Encroachment of subacromial space from superior structures } Spurs may be seen on X-Ray
} 2 ̊ External
} Inadequate muscle stabilisation of scapula
} Internal (glenoid)
} Overhead sports à undersurface of RC against posterosuperior surface
of glenoid during late stage cocking,
̈ Overuse may cause pathological process and superior labrum injury

22
Q

Rx and rehab for impingement

A
} Pain relief
} Mobilisation, STT, medication...
} Correction of abnormalities
} GH instability
} Muscular weakness and imbalances
} ST tightness
} Posterior capsule tightness
} Impaired scapulohumeral rhythm
} Correct training errors or functional errors
23
Q

Labral lesions

A

} Excess traction on labrum through LHB } FOOSA
} Classification
} SLAP – stable vs unstable, 4 grades
} Non-SLAP – degenerative, flap, vertical labral tears, Bankart
} Bankart – unstable lesion anterior inf labrum, repetitive dislocations } Hill-Sachs – damage to posterior humeral head

24
Q

Labrum assessment and management

A

} Clinical presentation SLAP } TOP anterior shoulder
} Pain on resisted biceps contraction – Dynamic labral shear, O’Brien’s
} Management
} Stable SLAP/nonSLAP = arthroscopic debridement
} Unstable SLAP/non-SLAP = arthroscopic fixation
} Conservative: >50 yo; young, minor SLAP; ?non-overhead sports
} NSAIDs, scapula stabilisers/dynamic stabilisers, capsule stretching } AVOID heavy weights early

25
GH instability
} Post-traumatic, atraumatic or combination } Most commonly anterior (90%) > posterior > inferior or multidirectional } Due to } Trauma – FOOSA } Anterior = abd + ER } Posterior = flex + add + IR } Inferior = traction } Multidirectional } Atraumatic – overhead athletes, ligamentous laxity } Combination – traumatic episode in lax shoulder
26
anterior instability clinical presentation
``` S & S + Assessment }S&S } Recurrent dislocation or subluxation } may incr in frequency &/or occur with minor activities } Shoulder pain } Impingement of RC à weakening & inadequate stabilisation cycle } “Dead arm” } Potential catching – labral detachment } Assessment } Apprehension&relocation } Load & shift (Drawer) } Pain & apprehension noted on Anterior Drawer } Minor instability – perform ant drawer + abduction & ER Treatment } Atraumatic } Muscular stabilisation } Correct technique in athletic population } Surgery if required } Capsular shift } Traumatic } } Correct underlying mechanism Surgery – esp for recurrent anterior subluxation } Bankart repair } Bone graft if Hill-Sachs’ lesion present } Nil tendon transfer in athletes ̈ Loss of ER ROM + ↓ power ```
27
posterior and multidirectional instability
Multidirectional } Combination of 2-3 instabilities } Most commonly atraumatic BUT can be traumatic } ExtremeROMsordirectblow(rare) }S&S } Generalisedligamentouslaxity–hypermobility syndrome } +ve on all instability tests } Apprerehension;Drawer;Sulcus } PainintranslationinmidROMs } Alteredmuscleactivationplaysconsiderablerole } Scap retraction = decr pain } Rx } Relief of symptoms } Therapeuticexerciseprogram–avoidstretching! } Possiblysurgery–results???? Posterior } Most commonly atraumatic as part of multidirectional } Subluxation & dislocation may damage post labrum } Clinical presentation } May be able to voluntarily sublux } Posterior Drawer = marked displacement
28
adhesive capsulitis
} No consensus on aetiology – 3 theories } Inflammatory } Fibrosing } Algoneurodystrophiccondition } Reflex sympathetic dystrophy } Idiopathic or post-traumatic } Decalcification of HOH } Presentation } 40-60 yo, female > male } Diabeticàhigher incidence } Evaluate ER PROM in neutral } May last 18 months+ } 3 phases } Phase 1: onset of severe pain, but movement not stiff. } Phase 2: pain at EOR and night, movement more restricted } Phase3:paindecreasing,slowlyincreasingmovement;“thawing”
29
adhesive capsulitis management
} Exclude primary causes } Assessment by orthopaedic surgeon } Intra-articular cortisone injection may be helpful in early stage } Late stage: } MUA – may lead to intra-articular damage } Capsulotomy } Physiotherapy } No evidence that PT impacts on outcome BUT } Assists in managing pain and symptoms } Education essential
30
AC J dislocations
} Clinically → inherently susceptible due to: • Sloped joint • High probability of large shear force } Mechanism: direct fall on the shoulder with arm in adduction } Common in rugby codes... } Presentation } Pain directly over AC joint } All extremes of movement aggravate pain } Joint sprain } Gr I = damage to capsule & acromioclav lig } Gr II = rupture acromioclav & damage to coracoclav lig } Gr III = rupture acromioclav & coracoclavic ligs
31
management of ACJ dislocation
• Mx = RICE, isometric strength, taping particularly on RTS