SIM Lab Quiz 4 Flashcards

(76 cards)

1
Q

early PPH

A

first 24 hrs after childbirth
loss of > 500mL

diagnosed when provider determines blood loss is greater than normal

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2
Q

late PPH

A

after 24 hours post-birth

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3
Q

% of women who will experience PPH

A

10%

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4
Q

early PPH causes

A

uterine atony
lacerations
hematomas

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5
Q

late PPH causes

A

hematomas
subinvolution
retained placental tissue

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6
Q

PPH risk factors

A
macrosomia
polyhydramnios
operative vaginal delivery
augmented labor
ineffective contractions
prolonged 1st/2nd stage
precipitous labor/birth
general anesthesia
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7
Q

indications of possible early PPH

A
  • 10% decrease in hematocrit post birth
  • saturation of peripad w/in 15 min
  • boggy fundus after massage
  • tachycardia
  • decrease in BP
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8
Q

uterine atony

A

decreased tone of uterine muscle - primary cause of early PPH

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9
Q

assessment findings of uterine atony

A
boggy fundus
peripad saturation w/in 15 min
slow/steady or sudden/massive bleeding
blood clots
pale, clammy skin
anxiety, confusion
tachycardia
hypotension
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10
Q

medical management of uterine atony

A

oxy, methergine, hemabate - uterine contractions

IV - hypovolemia

blood replacement - hemorrhagic shock

surgical interventions

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11
Q

nursing actions for uterine atony

A
  • review risk factors for PPH
  • assess for displaced uterus (distended bladder)
  • assess fundus firmness (massage)
  • assess lochia (amount, clots)
  • review lab tests (HgB, Hct)
  • notify provider
  • oxy, methergine, hemabate
  • blood transfusion monitoring
  • emotional support
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12
Q

methergine

A

Indication: PPH s/t uterine atony or subinvolution

Action: stimulates contraction of uterine smooth muscle

Side effects: N/V/cramps

Route/dose: IM/IV, 200mcg every 2-4 hr

Caution: check BP - HTN contraindicated

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13
Q

hemabate

A

Indication: PPH that is unresponsive to oxy or methergine

Action: uterine contraction

Side Effects: N/V/D/fever

Route/doe: IM, 250 mcg every 15-90 min (total

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14
Q

lacerations

A

2nd most common cause of early PPH

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15
Q

common sites for lacerations

A

cervix
vagina
labia
perineum

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16
Q

laceration risk factors

A

fetal macrosomia
operative vaginal delivery
precipitous labor/birth

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17
Q

laceration assessment findings

A

firm, midline uterus w/ heavy bleeding

steady bleeding w/o clots

tachycardia

hypotension

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18
Q

medical management of lacerations

A

visual inspection
suturing
IV meds for pain

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19
Q

nursing actions for lacerations

A
review risk factors
vitals
blood loss
notify provider
pain meds
pelvic exam preparation
emotional support
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20
Q

hematoma

A

occurs when blood collects w.in connective tissue of vagina or peineal areas r/t vessel that ruptures and continues to bleed

hard to diagnose degree of blood loss

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21
Q

hematoma risk factors

A

episiotomy
forceps
prolonged 2nd stage

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22
Q

hematoma assessment findings

A
  • severe pain in vagina/perineal area
  • not managed w/ normal postpartum pain management
  • tachy and hypotension
  • heaviness/fullness of vagina or rectal pressure if in vagina
  • in perineum - swelling, discoloration, tenderness
  • hematomas of 200-500 ml can displace uterus and cause uteirne atony
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23
Q

medical management of hematoma

A

evaluated and monitored if small

surgically excised if large

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24
Q

nursing actions w/ hematoma

A
review risk factors
ice for 24 hrs
assess pain
ask pt to verbalize pain, heaviness
monitor for BP decrease
monitor for HR increase
pain meds
review lab reports
notify provider
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25
subinvolution of uterus
uterus does not decrease in size nor descend into pelvis usually later in postpartum period
26
uterine subinvolution risk factors
fibroids endometritis retained placental tissue
27
assessment findings of uterine subinvolution
soft, large uterus lochia returns to rubra, heavy back pain
28
medical management of uterine subinvolution
D&C for retained placental tissue methergine for fibroids antibiotic for endometritis
29
nursing actions for uterine subinvolution
``` risk factors monitor pt pt education S/Sx: increased bleeding, clots, lochia change reduce risk for infection discharge meds ```
30
retained placental tissue
most common cause of LPH - when small portions of placenta (cotyledons) remain attached to uterus during 3rd stage can interfere with involution of uterus and lead to endometritis
31
cotyledons
small portions of placenta still in uterus
32
risk factors for retained placental tissue
manual removal of placenta
33
assessment findings for retained placental tissue
``` sudden profuse bleeding after 1st wk subinvolution of uterus high temp uterine tenderness pale tachy hypotension ```
34
medical management of retained placental tissue
D&C to remove tissue | IV antibiotic therapy (endometritis)
35
nursing actions for retained placental tissue
- risk factors - monitor - review labs - patient education (bleeding) - teach fundus assessment/massage - assess fundus/lochia every hr for 4 hrs after hemorrhage - oral/IV fluid intake - prevent overdistended bladder - assist w/ ambulation - rest - uninterrupted rest - emotional support - food high in iron
36
chronic hypertension
HTN before conception or before 20th wk of gestation high risk of preeclampsia
37
preeclampsia-eclampsia
pre: HTN w/ proteinuria after 20th wk eclampsia: convulsive stage of disease
38
preeclampsia superimposed on chronic HTN
new-onset proteinuria proteinuria before 20th wk sudden uncontrolled HTN
39
gestational hypertension
high BP for first time mid-pregnancy w/o proteinuria diagnosis is made postpartum and is relatively benign w/o underlying physiological changes
40
arteries in normotensive pregnancies
spiral arteries of uterus are remodeled by invasion of endovascular trophoblast cells of placenta, which allows them to widen to accommodate 10-fold increase in blood flow
41
arteries in hypertensive pregnancies
remodeling of spiral arteries of uterus is incomplete arteries remain thick-walled, resulting in suboptimal placental perfusion
42
uteroplacental perfusion
can be diminished 50% before onset of preeclamptic symptoms
43
ischemia of uteroplacental tissue
results in endothelial cell dysfxn, resulting in multiorgan endothelial cell damage and dysfxn triggers vasospasm w/ poor tissue perfusion to all organs increased peripheral resistance and elevated BP
44
liver in preeclampsia
fat deposits - epigastric pain HELLP
45
HELLP
Hemolysis Elevated Liver enzymes Low Platelets
46
hepatic involvement in preeclampsia
periportal hemorrhagic necrosis of liver subcapsular hematoma
47
GFR in preeclampsia
glomerular endothelial damage fibrin deposition resulting ischemia reduce GFR - protein excreted
48
coagulation system in preeclampsia
activated - thrombocytopenia
49
endothelial damage in preeclampsia
damage to brain | fibrin deposition, edema, hemorrhage
50
retinal arterial spasms in preeclampsia
blurring, double vision | photophobia, scotoma
51
leakage of serum protein in preeclampsia
decreased serum albumin | tissue edema
52
pulmonary edema in preeclampsia
volume overload r/t to left ventricular failure s/t high vascular resistance
53
preeclampsia risk fx
``` nulliparity younger than 19; older than 35 obesity multiple gestation family hx preexisting htn or renal disease previous preeclampsia or eclampsia DM ```
54
preeclampsia risks for mother
``` cerebral edema/hemorrhage/stroke disseminated intravascular coagulation pulmonary edema congestive HF hepatic failure renal failure abruptio placenta ```
55
preeclampsia risk for newborn
``` prematurity IUGR LBW stillbirth fetal intolerance r/t decreased placental perfusion ```
56
preeclampsia assessment findings
``` HTN proteinuria elevated liver fxn diminished kidney fxn altered coagulopathies ```
57
first-line drugs for preeclampsia
hydralazine (vasodilator) methyldopa (mechanism unknown) labetalol (beta blocker)
58
second-line drug for preeclampsia
nifedapine (Ca channel blocker)
59
mag loading dose
4-6 g in 100ml IV fluid over 15-20 min
60
mag continuous infusion
2g/hr in 100 ml IV fluid for maintenance
61
mag lab eval
measure serum magnesium level at 4-6 hrs maintain 4-7 mEq/L
62
mag duration
IV should continue 24 hrs post delivery
63
mag antidote
calcium gluconate or calcium chloride 5-10 mEq IV over 5-10 min
64
eclampsia
occurrence of seizure activity in presence of preeclampsia
65
eclampsia triggers
``` cerebral: vasospasm hemorrhage ischemia edema ```
66
eclampsia warning signs
``` persistent headaches epigastric pain N/V hyperreflexia w/ clonus restlessness ```
67
care during eclampsia seizure
``` stay with pt call for help As and Bs anticipate suction prevent injury record time, length, type notify provider ```
68
maternal side effects on mag
``` nausea flushing diaphoresis blurred vision lethargy hypocalcemia depressed reflexes respiratory distress/arrest cardiac dysrhythmias decreased platelet aggregation circulatory collapse ```
69
fetal side effects on mag
``` decreased variability in FHR respiratory depression hypotonia decreased suck reflex magnesium toxicity ```
70
nursing actions for patient on mag
vital signs before infusion and every 5-15 min during loading dose, then 30-60 min until stable assess DTRs every 2 hrs monitor I/Os strictly monitor mag levels monitor for mag toxicity seizure/resuscitation precautions cardiac monitoring FHR monitor - continuous neonatal team
71
post-seizure assessment
``` maternal and fetal status assess airway supplemental oxygen IV access mag sulfate quiet environment ```
72
HELLP risks for woman
abruptio placenta renal failure liver hematoma/rupture death
73
HELLP risk for fetus
preterm | death
74
HELLP assessment findings
general malaise, nausea, RUQ pain unexplained bruising, mucosal bleeding, petechiae, bleeding from injection/IV sites altered lab tests
75
HELLP medical management
immediate delivery of fetus and placenta resolution generally 48 hrs postpartum platelet replacement
76
HELLP nursing actions
``` assessment r/t preeclampsia lab tests notify physician platelet replacement education emotional support ```