Simulation cases Flashcards

(39 cards)

1
Q

autonomic features of hypoglycaemia

A
  1. sweating
  2. palpitations
  3. tremor
  4. hunger
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2
Q

neurological features of hypoglycaemia

A
  1. confusion
  2. drowsiness
  3. behavioural changes
  4. speech abnormalities
  5. incoordination
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3
Q

other symptoms of hypoglycaemia can include

A

nausea

headache

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4
Q

normal reference range for fasting plasma glucose

A

4 - 5.8mmol/l

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5
Q

what defines hypoglycaemia

A

plasma glucose of less than 3mmol/l

in hospitalised patients <4.0 mmol should be treated if patient is symptomatic

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6
Q

SBARR handover

A

Situation
- who, where, when, what aspect of patients management you need advice on, current working diagnosis

Background
- relevant pmh, surgical hx, medication, allergies, investigation results

Assessment
- vitals, clinical examination, overal clinical impression

Recommendations
- suspected diagnosis, what needs to happen, time frame

Response and review

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7
Q

AIRWAY (hypoglycaemic patient)

A

can the patient talk !?

no:
- airway compromise: cyanosis, see-saw breathing, accessory muscle use, diminished breath sounds

  • open mouth and inspect: secretions or foreign body
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8
Q

how would you open airway?

A

head-tilt-chin-lift manoeuvre

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9
Q

what would you do in airway if patient has suffered significant trauma with potential spinal involvement ?

A

jaw thrust

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10
Q

other interventions for airway?

A
  • nasopharyngeal airway

- oropharyngeal airway (Guedel)

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11
Q

breathing assessment for hypoglycaemic patient

A
  • resp rate

- patients with severe hypoglycaemia may develop slow, irregular pattern of breathing

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12
Q

investigations for hypoglycaemic patient

A
  • arterial blood gas to quantify degree of hypoxia

- CXR if abnormalities are heard on x-ray

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13
Q

pulse and blood pressure: hypoglycaemia patient

A
  • tachycardia in the context of hypoglycaemia

bradycardia is a late sign often precedes cardiac arrest

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14
Q

investigations and procedure hypoglycaemia patient:

A

intravenous cannula

blood tests:

  • fbc (anaemia and infection)
  • U&Es to assess renal function and electrolyte levels
  • CRP to screen for evidence of infection
  • serum glucose

ECG

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15
Q

interventions for hypoglycaemia patient

A

if patient conscious:
- glucose gel by mouth

  • repeat capillary blood glucose 10-15mins after
  • if still hypoglycaemic administer more

if patient unconscious

  • intravenous glucose
  • when consciousness regained then oral glucose
  • IV access not possible then do IM glucagone
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16
Q

disability assessment of hypoglycaemic patient

A

AVPU

  • alert
  • verbal
  • pain
  • unresponsive
17
Q

The Cushing’s triad

A
  • bradycardia
  • irregular respirations
  • hypertension

physiological response tor raised ICP , attempts to improve perfusion

18
Q

what Is coning?

A

herniation of cerebellar tonsils through foramen magnum

compression of brainstem

19
Q

when does the classical ‘blown pupil’ appearance occur in traumatic brain injury patients

A
  • herniation of uncus of temporal lobe
  • through tentorial notch
  • leads to compression of oculomotor nerve
  • blown pupil appearance
20
Q

primary brain injury

A
  • initial injury caused to brain tissue

- skulls fractures, haematoma formation

21
Q

secondary brain injury

A
  • indirect damage to brain injury post primary insult

- inadequate perfusion of brain

22
Q

interventions for hypoxia and hypercapnia

A
  • try and maintain oxygen sats 94-98%

- intubation in patients unable to protect their airway

23
Q

intervention for patients who have hypotension and are hypovolaemic

A
  • resus with fluids or blood products

- vasopressors

24
Q

intervention for patients with cerebral oedema and raised ICP

A
  • avoid tight c-spine collars
  • position 30 degrees to aid venous drainage
  • mannitol or hypertonic saline to reduce ICP
  • intubation and hyperventilation strategies
25
intervention for expanding haematoma
- reverse clotting abnormalities - tranexamic acid if 3hrs since injury - neurosurgical intervention
26
intervention for hypoglycaemic patient or hyperglycaemic patient
- maintain bg within normal range | - with insulin or dextrose
27
imaging for head injuries
- CT head scan - pathologies: intracranial bleeds, EDH, SDH, SAH, ICH - brain contusion - skull fractures - cerebral oedema
28
if opiod toxicity is suspected as cause for patients reduced level of consciousness then what intervention should be given
naloxone
29
Exposure : traumatic head injury
Active bleeding - estimate total blood loss - rate of blood loss - hypovolaemic shock: hypotension, t.cardia, presyncope and syncope - temperature
30
intervention for haemorrhage
- if actively bleeding seek senior input - need for blood products - 2 large bore iv access - bloods
31
typical symptoms of opiod overdose include:
- nausea - vomiting - confusion - drowsiness
32
clinical signs of opiod overdose
- decreased level of consciousness - respiratory depression - pin point pupils
33
what is the initial dose of naloxone given iv in opiod toxicity patients?
- 400 micrograms
34
as naloxone rapidly reverses effects of opioids, it can precipitate symptoms of:
- opiod withdrawal | - including pain, confusion and agitation
35
key components of unconscious patients:
- GCS - pattern of breathing - size and reactivity of pupils - eye movement, oculovestibular response s - motor responses (tone, reflexes and posturing) - meningism
36
what is antepartum haemorrhage?
- bleeding from or into genital tract | - occurring from 24 weeks of pregnancy
37
most important causes of antepartum haemorrhage are
- placenta praaevia | - placental abruption
38
initial assessment in patient with antepartum haemorrhage
- weigh blood loss - maternal examination: vitals, gentle abdominal palpation, assess pain, rigidity and foetal presentation, size and movement
39
foetal surveillance in antepartum haemorrhage
- CTG