Skeletal Flashcards

(55 cards)

1
Q

What is osteochondrosis?

A

Developmental disorder of medium and large breed rapidly growing dogs that is characterized by abnormal endochondral ossification of epiphyseal cartilage in the shoulder, elbow, stifle, and hock joints.

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2
Q

What are some predilection sites for OC (Osteochondrosis?)

A
  1. Caudal humeral head
  2. Medial humeral condyle
  3. Lateral and medial femoral condyle
  4. Lateral and medial trochlea of talus
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3
Q

What are the radiographic signs of OC?

A
  1. Flattening or concavity of subchondral bone, and joint surfaces
  2. Subchondral bone lucency and adjacent sclerosis
  3. Osteophytosis
  4. Joint effusion
  5. Joint mice (+/- mineralized flap)
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4
Q

What is the cause of OC?

A

The exact cause is unknown but excessive nutrition, rapid growth, trauma, and a hereditary component are suspected to be contributing factors.

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5
Q

How would you make a patient with OC comfortable?

A
NSAIDs
ex:) Carprofen 2.2 mg/kg PO BID
ex:) Meloxicam 0.1 mg/kg/day PO
JOINT FLUID MODIFIERS
ex:) Polysulfated glycosaminoglycan (Adequan)  4.4 mg/kg IM twice weekly for 4 weeks
WEIGHT LOSS
DIET/EXERCISE RESTRICTION
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6
Q

What is the prognosis for recovery with OC in the shoulders?

A

Excellent

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7
Q

What is the prognosis for recovery with OC in the stifle?

A

Good

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8
Q

What is the prognosis for recovery with OC in the elbow and tarsal joints?

A

Fair

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9
Q

How do you treat OC?

A

Surgery to excise cartilage flaps or free floating fragments and curettage of subchondral bone to stimulate fibrocartilage formation.

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10
Q

What is osteochondritis dissecans (OCD)?

A

An inflammatory condition that occurs when the diseased cartilage from OC separates from the underlying bone.

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11
Q

What types of dogs are typically affected by OCD?

A

Medium to large breed rapidly growing young dogs (6-9 months)

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12
Q

What is the free floating piece of necrotic cartilage that has broken off referred to as?

A

Joint mice (Joint fragment)

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13
Q

What is Degenerative Joint Disease (DJD)?

A

Refers to arthritis or osteoarthritis which is the result of the gradual deterioration of the articular cartilage within one or more of the joints. The smooth resilient cartilage degenerates becoming brittle over time. With severe DJD, the degenerated cartilage may actually split away from the bone and become loose within the joint. NOT AN INFLAMMATORY DISEASE. But you can get synovial inflammation (synovitis).

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14
Q

What types of patients typically develop DJD?

A

Obese

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15
Q

Radiographically what will you see in dogs with DJD?

A

Irregular articular cartilage surface and new bone production. You may also get bone on bone from loss of cartilage. You may also seen osteophytes (forms at synovial or articular margins), effusion, increased joint space, or on the contrary a decrease in joint space, soft tissue swelling, and subchondral sclerosis.

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16
Q

What are the 2 different types of osteoarthritis?

A
  1. Primary (idiopathic)

2. Secondary (DJD)

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17
Q

What species is primary (idiopathic) osteoarthritis more common in?

A

Cats

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18
Q

What are the 2 subcategories of secondary osteoarthritis (DJD)?

A
  1. Developmental (OCD, hip dysplasia)

2. Acquired (trauma or neoplasia)

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19
Q

How do you treat osteoarthritis?

A

Tramadol, Gabapentin, & NSAID (Rimadyl (Carprofen), Onsior (Robenacoxib), Polysulfated glycosaminoglycans (Adequan)), Neutraceuticals (Glucosamine/Chondroitin (Cosequin) Avocado/Soybean Unsaponafiables (Dasequin), Omega 3 Fatty Acids+ surgery (salvage procedures) like joint replacement, arthroplasty, arthrodesis or amputation of the limb. Should also encourage obese patients to lose weight, have a modified exercise regimen, physical rehabilitation therapy (cold/heat therapy, passive range of motion, massage, swimming, treadmill exercise, acupuncture.)

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20
Q

What is hip dysplasia?

A

Abnormal formation of the coxofemoral joint that in it’s more severe form can eventually cause crippling lameness and painful arthritis of the joints. It is a genetic trait that is affected by environmental factors. Characterized by instability in 1 or both hip joints which leads to DJD, osteoarthritis and subluxation of the hip.

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21
Q

What is the cause of hip dysplasia?

A

It is a polygenic, complex, multifactorial disease.

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22
Q

What breeds are most commonly effected?

A

Large breeds: German Shepherds, Golden Retrievers and Lab Retrievers.

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23
Q

What are the clinical signs of hip dysplasia?

A

Bunny hopping gait, positive ortolani sign if there is a hip subluxation or hip laxity (should be done in a sedated patient b/c it can cause pain), pain on extension of hips, palpation of crepitus over the hips and atrophy of leg muscles.

24
Q

Radiographically what will we see with hip dysplasia?

A

Morgan line (osteophyte formation at insertion of joint capsule), <50% coverage of femoral head, remodeling, thickening and sclerosis of femoral neck, perichondral osteophyte formation, remodeling and sclerosis of the acetabulum.

25
What is the onset of clinical signs with hip dysplasia?
3-8 months of age but there is bimodal age distribution so signs may occur later in life as well.
26
What are treatment options for hip dysplasia?
NSAIDs (Rimadyl, [Carprofen], Onsior [Robenacoxib], Deramaxx [Deracoxib], Previcox [Firocoxib]), Chondroprotective agents [Cosequin or Dasequin], Surgery (Juvenile Pelvic Symphysiodesis, Triple Pelvic Osteotomy, Total Hip Replacement, FHO (Femoral Head and Neck Ostectomy).
27
Does a veterinarian have to be certified to take x-rays for OFA?
No
28
What does OFA stand for?
Orthopedic Foundation of Animals
29
What is the earliest age you can evaluate an animal for OFA hip?
2 years old
30
Is the scoring system quantitative or qualitative for OFA hip?
Qualitative (7 point system from excellent to severe, based on evaluation by 3 radiologists.)
31
Is OFA hip recognized by the AKC?
Yes
32
Does a veterinarian have to be certified to shoot PennHIP x-rays?
Yes
33
What is the earliest age you can evaluate an animal for PennHIP?
16 weeks
34
Is the scoring system quantitative or qualitative for PennHIP?
Quantitative (There is a calculated Distraction Index 0-1 with 0 being the best and it is evaluated by a trained veterinarian.)
35
What is elbow dysplasia?
Disease of the elbows of dogs caused by growth disturbances in the elbow joint.
36
What causes elbow dysplasia?
It is a multifactorial developmental anomaly of the canine elbows that may have been caused by a defect in cartilage growth, trauma, genetics, exercise, diet and so on...
37
What types of dogs does elbow dysplasia typically effect?
Large breed dogs: Lab Retrievers, Golden Retrievers, German Shepherds and Rottweilers.
38
What are the 4 classic underlying causes of elbow dysplasia?
1. OC/OCD of the medial aspect of the humeral condyle 2. Ununited anconeal process of the ulna 3. Fragmented medial coronoid process of the ulna 4. Incongruity of the elbow joint
39
What is one consequence of elbow dysplasia?
Early onset of DJD/OA.
40
When should the anconeal process fuse in dogs?
5.5 months of age
41
What is the most common cause of elbow dysplasia out of the 4 causes listed?
Fragmented medial coronoid process of the ulna. (Very difficult to see radiographically).
42
What is panosteitis?
Short lived, self limiting and painful condition characterized by limping and (shifting leg) lameness and effects the long bones of young dogs. The bone inflammation can affect one or more legs in dogs or cats.
43
What is the typical onset of age for panosteitis?
5-12 months
44
What are the radiographic signs of panosteitis?
Increased medullary opacity, beginning as nodular and then increasing to diffuse. The lesion often begins around the nutrient foramen, smooth continuous periosteal new bone occurs in a minority of patients, cortical thickening can persist and cause remodeling.
45
What types of dogs are predisposed to panosteitis?
German Shepherds and Basset Hounds
46
How would you treat panosteitis?
There is no treatment for this disease as it is usually self limiting but you can prescribe NSAIDs to relieve any discomfort.
47
What is Hypertrophic Osteodystrophy (HOM)?
A bone disease that occurs in large breed fast growing dogs sometimes referred to as metaphyseal osteopathy. Occurs from a decrease in blood flow to the metaphysis leading to failure of ossication and necrosis and inflammation of the cancellous bone. The disease is usually bilateral and affects the distal radius and ulna, and tibia in particular.
48
What do you see radiographically with HOM?
Double physis sign typically in the metaphysis of distal radius, ulna and tibia.. Radiolucent line parallel to the physis. Radiographs may go from looking normal to abnormal in only 48 hours. In advanced stages you may get irregular periosteal new bone formation along the metaphysis. You may also see physeal closure and retarded growth.
49
What are the clinical signs of HOM?
Ranges from a mild self limiting disease to severe multisystemic, life threatening illness. Pyrexia, malaise, pain and swelling over the metaphyseal region and lameness may be seen.
50
What is the typical onset of age for HOM?
Young dogs (7 weeks-8 months)
51
What dog breeds are predisposed to HOM?
Great Danes, Boxers, German Shepherds, Irish Setters, Weimeraners
52
How would you treat HOM?
Supportive care and possibly corticosteroids or NSAIDs.
53
What is Aseptic Necrosis of the Femoral Head?
Legg-Calve-Perthes. It is deterioration of the femoral head associated with ischemia and avascular necrosis of the bone. Infarction of the bone leads to collapse of the femoral head and neck, followed by revascularization, resorption and remodeling. The lesion is often bilateral.
54
What types of dogs is Legg-Calve-Perthes often seen in?
Toy, Miniature and Small breeds: and there may be a genetic component in Manchester Terriers.
55
What are the radiographic signs of Legg-Calve-Perthes?
Lysis of the femoral head and neck, widening of the joint space due to hyperplastic cartilage, microfracture and deformation of the femoral head, secondary DJD and muscle atrophy.