Skin Flashcards
(48 cards)
functions skin
regulation of heat - blood vessel constrict/dilate
sensation
production vitamin d - sun
regulation toxins and fluid levels - sweat (wastes ammonia and urea)
protection
composition of self image
layers skin
epidermis
- no blood supply, renew q 4-6 weeks, outmost layer dead
dermis
- thicker than epidermis, prov strength and elasticity from elastin and collagen (helps with healing), contains vessels
subcutaneous (hypodermis)
- fat tissue, varies weight sex age, cushion internal organs, dermis to muscle and bone
structures within skin
melanin - protect UV rays, give skin color
melanocyte - makes melanin
sebaceous gland - lubricates hair and skin
arrector pili muscle - hair erect, goose bumps
free nerve ending - stim sense pain
basal cells - join dermis and epidermis, single layer active cells, produces new cells constantly push way to surface
keratinocytes
perfusion
Positive Effects
Adequate perfusion allows oxygen and nutrients to nourish the skin, promoting a healthy protective skin barrier.
Negative Effects
Poor or inadequate perfusion decreases the supply of oxygen and nutrients to the skin, causing it to deteriorate, producing a weak barrier. A prolonged decrease in oxygen to the skin produces the following results:
Decreased collagen formation
Reduced production of cells that make collagen (fibroblasts)
Decreased number of white blood cells (leukocytes)
Reduced migration of cells that help the skin regenerate
Vascular diseases, like peripheral vascular disease and heart failure, can also produce these effects.
moisture/hydration
Positive Effects
Adequate moisture allows the skin to stay hydrated and intact.
Negative Effects
Too much moisture for extended periods of time causes the skin to erode from the process of maceration (softening due to excess moisture). The moisture can be stool, urine, drainage from wounds, saliva, mucus, or sweat. Excess moisture can allow bacteria to multiply and break down skin, leading to an increased risk for infection.
A lack of moisture leads to cracking of the skin, leading to skin breakdown and increased risk for infection.
nutrition
Positive Effects
Adequate intake of protein, carbohydrates, fats, vitamins, and minerals allows the skin to repair itself quickly. An appropriate number of calories is also necessary to maintain healthy skin.
Negative Effects
Several deficiencies weaken the skin, specifically lack of sufficient calories, protein, vitamin C, vitamin A, zinc, and copper. A lack of calories is especially harmful because damage to the skin requires additional calories to heal; vitamin A, zinc, and copper are needed for healing, too. Protein is especially significant because fibroblasts need protein to make collagen. Vitamin C is necessary for collagen formation, too. Malnutrition that causes an unintentional weight loss of 5% or more and a low body mass index (BMI) can adversely affect skin and healing.
medications
Positive Effects
Antibiotics allow the body to fight invading pathogens, thus preventing or eliminating infection.
Negative Effects
Steroids interrupt the inflammatory process (are antiinflammatory), making patients prone to infections and slow healing.
Some antibiotics, antidepressants, and antipsychotics cause photosensitivity, which makes the skin more sensitive to sunlight, resulting in redness/sunburn, rash/hives, and/or itchiness.
negative skin effects
shearing/friction prolonged pressure diabetes smoking UV rays
friction/shearing
Friction: Rubs the skin against something, like a bed or medical device, and damages the top layer of the skin
Shear: Combines effects of gravity with friction, like when the sitting patient “slides” down in bed, and causes opposing forces (skin “sticks” to bed but gravity and the patient’s weight pull the patient down), leading to blood vessels being stretched and compressed, reducing blood supply to the area
prolonged pressure
Leads to capillaries collapsing → ischemia → skin damage → subcutaneous tissue and muscle damage → tissue death (if pressure is relieved at the beginning of ischemia, damage usually does not occur)
Produces damage if the pressure level is low for an extended period and if the pressure is high for a short period
Produces damage to bony prominences and certain pressure areas based on position
Produces damage if the skin is re-exposed to the same, or even less, pressure
diabetes
Affects large blood vessels (macrovascular)
Affects small blood vessels (microvascular)
Alters the acidic pH of the skin, making it less effective at fighting microorganisms
Thickens vessel walls and obstructs blood flow, leading to reduced oxygen and nutrients to the cells
Decreases collagen synthesis and strength
Impairs the immune system: produces weak leukocytes and macrophages
smoking
Decreases hemoglobin’s capability to transport oxygen
Causes vessels to constrict and increases clotting ability of the blood due to nicotine, leading to decreased oxygen to cells
Causes wrinkles around mouth
UV lights
Includes two major types: ultraviolet light A (UVA) and ultraviolet light B (UVB)
UVB penetrates only the epidermis
UVA penetrates more deeply, into the dermis
Induces degenerative changes in skin cells
Alters skin DNA, increasing risk for skin cancer
effects aging
atatched
Which statement about ultraviolet light is accurate?
Ultraviolet light A (UVA) penetrates the dermis.
UVA penetrates the dermis.
wound classifications
integrity of skin
- open or closed
cause
- Intentional: surgery, radiation therapy
- Unintentional: accident
description
- Arterial ulcer, Pressure injury, Gunshot wound
depth
- Superficial, Partial thickness, Deep or full-thickness
healing time
- acute or chronic
contamination level
- clean or dirty
classification skin integrity
open
- Skin is broken, such as in a cut, puncture, or surgical incision.
Other examples include pressure injuries or ulcers on the skin from vascular diseases.
These wounds can be prone to infection.
closed
- The skin is not broken (intact), for example, a bruise or a hematoma.
Closed wounds can be just as serious as open wounds.
Some wounds that allow the skin to remain intact can cause serious injury internally. A nurse cannot judge the severity of the injury or tissue damage by whether the wound is open or closed.
wound depth
superficial and partial thickness
- Superficial wounds affect only the top layer of skin (epidermis).
Partial-thickness wounds extend through the epidermis to the dermis but do not penetrate the subcutaneous layer.
Superficial and partial-thickness wounds generally heal quickly without scarring unless factors, such as infection, interrupt the healing process.
deep
Deep wounds injure all layers of the skin: the epidermis, dermis and subcutaneous layer.
Full-thickness wounds can penetrate the bone, muscle, or other structures.
These wounds usually heal slowly and form scar tissue, and they can even become a chronic type of wound.
contamination level
see
classifications burns
Chemicals, heat, radiation, electricity, and frostbite can cause burns. If a patient is burned over a large portion of the body, infection and fluid and electrolyte imbalances can be severe. Burns, like wounds, have classifications.
superficial
- Injures the epidermis, causing pain, redness (which will blanch), and no edema
partial thickness
-Injures the epidermis and part (called superficial-partial thickness) or all (called full or deep-partial thickness) of the dermis, leading to blisters, redness, and extreme pain
full thickness
-Injures all three layers, causing the tissue to be dry, white, brown, charred (black); causes no pain (nerves were burnt in injury); requires surgery
stages pressure injury (1-4)
Pressure injuries, caused by pressure from bony prominences and/or shearing and friction, can damage the skin to varying degrees. Pressure injuries are classified by type of visible tissue. This is also called staging.
stage 1
- Intact skin with no blisters
Nonblanchable erythema or persistent redness in the area of pressure (abnormal reactive hyperemia)
Painful area that differs in firmness and temperature from surrounding tissues
stage 2
-Partial-thickness wound involving the top two layers of skin: epidermis and dermis
Wound bed that is shallow, superficial, and pink
Includes intact or ruptured blisters from pressure that have not yet cratered
stage 3 -Full-thickness wound extending into the subcutaneous tissue (third layer of skin), but not into fascia, muscle, or bone Possible undermining (tissue loss around edges and under intact skin, which forms a lip around the wound) May include tunneling, also called a sinus tract (narrow channel extending from wound)
stage 4
- Full-thickness wound that is deeper than a stage 3 wound
Involves exposure of muscle, bone, tendons, and cartilage
Infection of the bone (osteomyelitis), if exposed, likely to occur
unstagable
- Full-thickness wound with necrotic tissue (eschar)
Inability to assess wound depth or involvement of underlying tissues
Removal of necrotic tissue necessary before staging occurs
suspected deep tissue injury
-Area of intact skin with purple or maroon area or a blood-filled blister
Similar to stage 1 injuries but difficult to detect in darker-skinned patients
Depth of tissue damage not readily visible on observation
Progresses rapidly, exposing deeper layers of tissue damage, even if treated rapidly and correctly
types healing
primary intention
- Healing that is swift and uncomplicated in an acute wound with minimal scar tissue
secondary intention
- Healing that starts from the bottom and sides of the wound until new tissue fills the wound bed; larger amounts of tissue loss that take longer to heal
tertiary intention
- Healing in which the wound is initially left open, causing a delay between injury and closure
phases wound healing
Wounds progress through several phases. How quickly healing occurs can be affected by complicating factors, such as comorbidities, depth of wound, and wound contamination. The three phases of wound healing are inflammatory, which includes hemostasis (forming clot); proliferative; and maturation/remodeling.
inflammatory
proliferative
maturation/remodeling
inflammatory phase
- This phase starts with the body’s initial recognition of and response to a wound—bleeding.
Bleeding activates the coagulation cascade, which involves platelets releasing growth factors to form a clot and stop bleeding (hemostasis).
Growth factors, in conjunction with cytokines, are also released during this phase, which initiates wound healing.
Blood vessels dilate, leaking fluid into the area of injury (wound), resulting in the classic signs of inflammation: pain, redness (erythema), warmth, swelling, and limited function in the area of injury.
Macrophages and neutrophils move into the area of injury and begin the process of removing bacteria and debris, which cleans the wound bed for the repair process.
The inflammatory phase lasts approximately 3 days.