skin infections & wounds Flashcards

1
Q

given in tetanus prone wounds when the patient’s immune status is unknown or partial

A

tetanus immune globulin

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2
Q

given in traumatic wounds if tetanus immune status is unknown

A

tetanus toxoid

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3
Q

what are tetanus prone wounds?

A

gross devitalized tissue, obvious contamination, frostbite, missile injuries, and injuries >6hrs old

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4
Q

best antibiotic choice for an MRSA infection resistant to vancomycin

A

linezolid

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5
Q

treatment for severe hidradenitis suppurativa

A

wide excision with healing by secondary intention

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6
Q

recurrent chronic inflammatory skin condition due to follicular occlusion:

A

hidradenitis suppurativa

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7
Q

acute purulent infection of the fingertip pad (pulp)

A

felon

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8
Q

most common organism of infection in felon

A

staph aureus

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9
Q

what does full thickness skin graft include

A

epidermis and complete dermis; no subcutaneous tissue

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10
Q

what is primary graft contraction?

A

degree to which a graft shrinks in surface area after harvesting but before grafting

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11
Q

what is secondary graft contraction?

A

degree to which a graft shrinks during healing

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12
Q

which has less primary contraction - full thickness or STSG?

A

STSG

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13
Q

which has less secondary contraction - full thickness or STSG?

A

full thickness

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14
Q

which has greater rates of engraftment (graft survival)?

A

STSG

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15
Q

infection spreading along lymphatic channels that presents with red linear streaking proximal to the site of infection and travels toward the regional lymph nodes which may also be enlarged and tender

A

acute lymphangitis

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16
Q

most common organism and treatment of acute lymphangitis

A

group A strep; tx with penicillin

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17
Q

mupirocin is indicated for treatment of ___ wound infections

A

MRSA

18
Q

how is a full thickness skin graft harvested and repaired?

A

excised sharply with knife, defatted to encourage uptake; wound is closed primarily

19
Q

most common pathogen of paronychia

A

staph aureus

20
Q

order and stages of healing for skin grafts:

A

imbibition, inosculation, revascularization

21
Q

treatment of paronychia (nailbed infection):

A

incision and drainage of nailbed to evacuate purulence + abx; incision made longitudinally and laterally, parallel to nail bed

22
Q

characteristics of Hurley I hidradenitis suppurativa:

A

localized abscesses without sinus tracts or scarring; first line treatment is clindamycin gel

23
Q

characteristics of Hurley II hidradenitis:

A

multiple abscesses and sinus tracts separated by normal appearing skin

24
Q

characteristics of Hurley III hidradenitis:

A

diffuse disease with multiple interconnected sinus tracts and abscesses involving the entire anatomic area with scarring

25
Q

form of cellulitis caused by group A strep that causes raised lesions with sharply demarcated borders between infected and normal skin:

A

erysipelas

26
Q

skin and soft tissue infection confined to the epidermis that usually involves the face or extremities; consists of discrete vesicular lesions and crusted plaques; usually caused by beta hemolytic strep or staph aureus

A

impetigo

27
Q

deep ulcerated form of impetigo

A

ecthyma

28
Q

pressure ulcer stage: intact skin that is reddened

A

stage 1

29
Q

pressure ulcer stage: blisters or breaks in dermis or partial thickness loss of dermis

A

stage 2

30
Q

pressure ulcer stage: full thickness tissue loss with visible subcutaneous fat but no exposed bone, tendon or muscle

A

stage 3

31
Q

pressure ulcer stage: exposed bone, joint, muscle, or tendon

A

stage 4

32
Q

pressure ulcer stage: covered by slough or eschar, true depth can’t be determined without debridement

A

unstageable

33
Q

subcutaneous abscess of the fingertip pulp

A

felon; treat with I&D and abx

34
Q

nodular cellulitis that is often mistaken for fungal infection; due to innoculation of bacteria (usually S. aureus) in the wound of immunocompromised patient

A

botryomycosis

35
Q

TNF alpha inhibitor shown to have benefits for patients with moderate to severe hidradenitis

A

adalimumab

36
Q

clindamycin mechanism of benefit in NSTI

A

provides coverage for panton-valentine leucocidin toxin (PVL)

37
Q

histologic features of hidradenitis

A

follicular hyperkeratosis, plugging, and dilation; lymphocytic perifolliculitis; in chronic phase can demonstrate psoriasform hyperplasia of the interfollicular epithelium or inflammatory infiltrate of dermis and subcutis

38
Q

medical treatments for hidradenitis:

A
smoking cessaion
zinc gluconate
resorcinol sulfur topical
methylprednisolone 1 week taper
IV ceftriaxone followed by rifampin, moxifloxacin, and metronidazole
dapsone or cyclosporine
TNF blocker adalimumab
39
Q

Initial treatment of paronychia:

A

warm compresses and antibiotics

40
Q

Surgical treatment of hidradenitis:

A

total excision of all hair bearing skin with STSG

41
Q

First line therapy for Hurley stage 1 hidradenitis:

A

clindamycin gel