Smoking and Periodontal Disease Flashcards

(42 cards)

1
Q

what organs of the body is smoking harmful to?

A

almost every organ

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2
Q

what are the diseases associated with smoking?

A
  • lung cancer
  • heart disease
  • stroke
  • emphysema
  • bronchitis
  • cancers of the oral cavity, bladder, kidney, stomach, liver, cervix
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3
Q

approximately half of long term smokers will…. ?

A
  • die early as a result of smoking
  • loose an average of 20 years of life
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4
Q

what diseases cause the most deaths in smokers?

A
  • lung cancer
  • chronic obstructive pulmonary disease
  • coronary artery disease
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5
Q

what are the phases and contents of tobacco smoke?

A
  • gaseous phase and solid (particulate) phase
  • thousands of noxious chemicals
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6
Q

what is the particulate phase?

A

nicotine “tar” - benzene and benzopyrene
- tar is inhaled with smoke and in its condensed form it is the sticky brown substance that stains fingers and teeth

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7
Q

what is nicotine and what does it cause?*

A

a highly addictive compound that causes
- a raise in blood pressure
- increased heart rate
- increased respiratory rate
- peripheral vasoconstriction

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8
Q

where is nicotine found and how is it absorbed?

A
  • found in the tobacco leaf
  • when a cigarette is lit it evaporates and is inhaled and absorbed in the lungs where it travels to the brain (10-19 secs)
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9
Q

how does cigarette smoking contribute to periodontal disease?

A

cigarette smoking is associated with lower oxygen delivery in the periodontal pockets which is favorable for growth of anaerobic bacteria
- favors early acquisition and colonization of perio pathogens in oral biofilms

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10
Q

what are the types of smokeless tobacco products?

A
  • ground/grated + salts and water as “moist snuff” in a tea bag like sachet that is placed under the lip
  • powder “dry snuff” that is snorted
  • coarsely cut and placed into the cheek
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11
Q

what is Snus and what cancer is it linked to?

A
  • smokeless tobacco popular in Sweden that claims to have lower levels of carcinogens
  • link to pancreatic cancer but not mouth or lung
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12
Q

what is the protocol for documenting cigarette smoking in the dental field?

A
  • all patients must be asked their smoking status or use (be specific)
  • current smokers: ask about current and past smoking
  • former smokers: ask about past smoking
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13
Q

why is it important to ask a current smoker about their current AND past usage?

A

cumulative exposure
- someone who says they currently only smoke 1 cig a day may have been smoking a pack a day in the past

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14
Q

How is smoking a major risk factor for periodontitis?

A

it effects the prevalence, extent and severity of the disease
- almost half of periodontitis cases attribute to smoking due to the impact of smoking on initiation, progression and management

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15
Q

smoking has an adverse impact on the clinical outcome of…?

A

non surgical and surgical therapy as well as the long-term success of implant placement

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16
Q

what human systems does smoking effect and what does this cause?

A
  • immune system
  • cellular and humoral inflammatory systems
  • affects soluble molecules in bodily fluids to fight infections and inflammation
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17
Q

what effects does smoking have on gingivitis?

A
  • the development of inflammation in response to plaque accumulation is reduced in smokers
    (less clinical gingival inflammation in smokers)
18
Q

what effects does smoking have on periodontitis?

A
  • major risk factor for increasing the prevalence and severity of periodontal destruction
  • increase in pocket depth, attachment loss and alveolar bone loss
  • smokers are 4x more likely to have periodontitis
19
Q

how does the odds of having periodontitis decline in relation to smoking?

A

the more years that a person has quit smoking declines periodontitis odds

20
Q

current smokers are ____ times more likely to have SEVERE periodontitis

21
Q

how is bone loss effected by smoking in periodontitis patients?

A
  • bone loss is 2x as rapid in smokers than non smokers even in the presence of plaque control
  • tooth loss is also increased
  • clinical attachment loss increases
22
Q

why does smoking cause pleasure?

A

when nicotine is inhaled the brain releases dopamine and adrenaline requiring increasing amounts to achieve same level of pleasure due to tolerance

23
Q

how does smoking effect bacterial-host interactions?

A
  • alters the interactions leading to more extensive periodontal breakdown
  • an imbalance between bacterial challenges and host response
24
Q

how is the subgingival biofilm different in smokers and non smokers?

A

higher levels of tannerella forsythia and 2.3x more likely to harbor harbor T. forsynthia
- also increase in A. actinomyeces and P. gingivitis

25
what are the immune-inflammatory responses to smoking?
- increased periodontal destruction - alterations in neutrophil numbers and functions - increase in TNF-a, PGE2, neutrophil elastase and MMP-8
26
what is the mechanism of the immune-inflammatory response to smoking?
nicotine increases the secretion of PGE2 by monocytes in response to LPS - smoking alters the response of neutrophils to bacterial changes by increasing the release of tissue destructive enzymes leading to increased periodontal tissue destruction
27
why are the clinical signs of inflammation less prominent in smokers?
reduced vascular response in the gingiva leads to lower gingival blood flow, reduced gingival crevicular fluid (GCF), and decreased bleeding on probing (BOP) which result is less visible inflammation, even when tissue destruction is occurring
28
what is E-liquid used in electronic cigarettes?
- liquid composed of diluents (propylene glycol or vegetable glycerin), nicotine and flavorings with 13-18 mg/ml of nicotine
29
what was the conclusion on long term e-cigarette smoking and its hazard to health?
unlikely to exceed 5% of the harm from smoking normal tobacco
30
what are the oral health effects of e-cigarettes?
cytotoxicity and DNA damage has been seen but risks are currently unknown and predicted to be less than that of tobacco smoking
31
why do smokers not respond well to non-surgical periodontal therapy?
- they have less probing depth reduction after non-surgical periodontal therapy - less pronounced clinical attachment level gain
32
what is the effect of smoking and surgical therapy and implant outcomes?
- less favorable response - less pocket reduction and less gain in clinical attachment - negative impact on the outcomes of guided tissue regeneration
33
smokers who undergo maintenance periodontal therapy tend to respond?
poorer - smokers tend to have deeper pockets and less gain of attachment - 90% of poor responders to periodontal therapy are smokers
34
what are the 5 A's for smoking cessation that dentists can ask?
- ask - about tobacco use every visit - advise - all tobacco users at every visit - assess - willingness to quit - assist - patient in quitting - arrange - follow up contact
35
what are the smoking cessation stages of change?
- pre-contemplation: not ready - contemplation: planning to quit, no date set - preparation: planning to quit, date set - action: quit - maintenance: no use for 6 months
36
is quitting cold turkey more effective?
no - both medication and counseling can be 4x more effective
37
what are the effects of will power, self help, brief intervention and NRT on smoking cessation?
- WP: least effective - SH: can increase quitting compared to no innervation, only by a small amount - BI: delivered by dentist or physician can increase rate of quitting by 40 to 90% - NRT: increase the rate of quitting 50-70%
38
what are examples of NRT products?
- patches - lozenges - nasal spray - inhaler
39
what are the two drugs used for smoking cessation?
- Varenicline: nicotine receptor partial agonist; reduce withdraw - Bupropion: antidepressant at high doses and smoking cessation at lower doses
40
what are the nicotine withdraw symptoms?
* Depressed mood * Insomnia * Irritability * Anxiety * Difficulty concentrating * Restlessness * Decreased heart rate * Increased appetite/weight gain
41
what is the time course of nicotine withdraw?
* Begins few hours after last use * Usually peaks in a few days, or up to 1w * Lasts about 4 weeks * “Cravings” last longer
42
what are the reasons to use medications to quit smoking?
- assist in becoming tobacco free - comletement other treatment - reduce cravings and withdraw severity ---> concentrate on psychosocial treatment