Spinal Cord Injury Flashcards

1
Q
  • Major health problem
  • Over 200,000 people living w/a SCI in the US
  • 12,000-14,000 new injuries each year
A
  • 1/2 of all SCI occur to the cervical spine & another large portion to the T11-L2 area
  • Trauma is the leading cause of SCI
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2
Q
  • Occurs more in males
    > Traumatic injury r/t inc risk-taking behavior
  • Ages 16-30 account for more than 1/2 of new SCI each yr
  • ETOH/substance use
  • Warmer mos
A

Non-traumatic injury etiology incl

  • osteoporosis fx’s
  • tumors
  • infarction & hemorrhage
  • myelitis from infection or non-infection
  • spondylosis
  • syringomyelia
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3
Q
  • Spinal nerves exit between vertebrae
  • There’s a sensory & a motor axon
A
  • Sensory stimulation from the receptors in the skin/organs send a signal to the brain & back down the motor neuron to illicit a purposeful movement (e.g., writing, catching a ball, putting on clothes, etc.)
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4
Q

Autonomic reflexes

  • Peripheral sensory nerve impulses arc around spinal cord & return to the muscles in a particular organ, via motor neuron bypassing the brain
  • This is a much faster process but not controlled; e.g., touching a hot pan
A
  • The sensory root sends the signal around the spinal cord & back to the motor neuron to contract the muscle, moving the hand away from the hot pan
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5
Q

Spinal Cord

! W/trauma, fractured pieces may be sharp & sever or crush nerve tissue

A

Level of injury we need to consider location

  • Cervical 1-7
  • Thoracic 1-12
  • Lumbar 1-5
  • Sacral 1-5
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6
Q

Mechanisms of Injury: Primary

  • Hyperflexion
  • Hyperextension
  • Axial loading (vertical compression)
  • Excessive rotation
  • Penetrating injury
A
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7
Q

2 Categories of Injury: Primary

  • Is c/b the acceleration/deceleration applied to the spine
A

Secondary

  • Is c/b hemorrhage, ischemia, hypovolemia, & general impaired tissue perfusion from neurogenic shock
  • Local edema peaks in 2-3 days & subsides in 7; causes pressure on the cord & dec perfusion
  • Microvascular destruction causes more neuronal damage
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8
Q
  • The manifestations of SCI will depend on the type & lvl of injury

> Incomplete
- Some function preserved below the lesion

> Complete
- Paraplegia & tetraplegia

A

SCI - Level of Injury

  • Area of actual injury, NOT area of function
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9
Q
  • Inj classified according to the area of damage
    > Central, lateral, anterior, or peripheral
  • Damage may have occurred @ C4 but able to move muscles below lvl of inj if inj was incomplete
A
  • Neurologic lvl
    > The lowest lvl @ which functions are normal
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10
Q

?

Is utilized to determine the highest neurological lvl of normal function & is NOT the lvl of inj; spec neurological function is determined by following the body’s dermatomes

A

ASIA scale

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11
Q

Dermatomes that map sensation for incomplete injuries

A

These syndromes are exceptions to sensation & function

  • Anterior cord syndrome
  • Central cord syndrome
  • Posterior cord syndrome
  • Brown-Sequard syndrome
  • Cauda Equina syndrome
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12
Q

SCI - Anterior Cord

  • Usually from the anterior spinal artery syndrome; there’s an artery that feeds the anterior portion of the spinal cord & a problem w/perfusion & circulation to this artery will lead to this
A

SCI - Central Cord

  • Loss of sensation greater in upper extremities
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13
Q

SCI - Posterior Cord

  • Damage @ the back of the spinal cord
  • Pt may have good muscle power
  • Pain & temp sensation
  • May have difficulty w/coordination of limbs
A

SCI - Brown-Sequard

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14
Q

?

Loss of motor/sensory function in various patterns w/the potential for recovery w/regeneration of peripheral nerves; neurogenic bladder/bowel

Etiology
- pressure on nerves r/t trauma, fx, abscess, hematoma, ruptured disc, tumor, foreign object

Sx’s
- Incl lbp, alteration in LE sensation, dec motor strength LE, neurogenic bladder, loss of anal wink (aka anal reflex), saddle area numbness

Treatment = surgical decompression

A

Cauda equina syndrome

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15
Q

SCI - Diagnostics

  • x-ray, CT, MRI
  • Myelogram (examines subarachnoid space)
  • Continuous ECG monitoring
  • Sensory/motor function assessment
  • Reflex assessment
A

Emergency Management

  • ABC’s incl signs of hemorrhage
  • Think about jaw thrust vs. head tilt technique (CPR)
  • Maintain c-spine
  • Log rolling
  • 1 person must always assume control of the head
  • LOC (GCS)
  • Determine lvl of inj
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16
Q

Documentation of Consciousness

  • Pain is elicited by sternal rub (can cause bruising), supraorbital pressure, trapezius muscle squeeze or mandibular pressure
  • GCS: 15 is best score, 3 is worst score
A
17
Q

SCI: Acute Phase - Management

! Prevent further inj
- May or may not see methylprednisolone use (some recent research doesn’t recommend; inc hyperglycemia & stress ulcers)

  • Oxygen/mech support
  • Skeletal fx reduction & traction
  • NGT & Foley
A
  • Ventilation - may not be able to endotracheal intubate d/t cervical inj (do not want to hyperextend); may need tracheostomy
  • NGT for dec peristalsis (build-up of gastric juices)
  • Foley for bladder flaccidity
  • These are from spinal shock
18
Q
  • Hemodynamic monitoring
    > may need dextran or other volume expanders or vasopressors (dopamine HCl (inotropin); isoproterenol)
A
19
Q

___ and possible ___ for bradycardia

A

atropine sulfate; pacemaker

20
Q

SCI - Traction

  • HOB raised; bed on casters so that it can be wheeled to x-ray dept; awaiting poss MRI or surgery
A
  • Raise the bed to apply counter traction; skin care as ordered
21
Q

Halo Fixation w/Jacket

  • Often used s/p fx or arthrodesis
  • Bone fragments or laminae (discs) laminectomy may be surgically removed to prevent further inj
A
  • Pins are inserted into the skull - monitor for loosening pins or manifestations of infection may lead to osteomyelitis & subdural abscess
  • Pin care by hospital policy or physician order
  • Do not loosen screws that hold device in place
  • Check skin for breakdown = 1 finger breadth
22
Q

SCI - Complications

! Spinal shock
! Neurogenic shock
! DVT & thrombophlebitis
! Orthostatic hypotension
! Autonomic dysreflexia
! Pressure ulcers

A
23
Q

?

Complete but temporary loss of autonomic, reflex, motor & sensory activity below the lvl of injury; 2° to damage of the cord

Often lasts <48 hrs but may last for wks

! Flaccid paralysis
! Loss of DTRs & perianal reflexes
! Loss of motor & sensory function

A

Spinal shock

24
Q

?

Loss of ANS function below the lvl of inj

Can lead to cardiovascular changes

! Orthostatic hypotension
! Bradycardia
! Inability to sweat below the lvl of inj

A

Neurogenic shock

25
Q

?

A true medical emergency

Exaggerated autonomic response to stimuli

! Pulsating HA
! Piloerection
! Diaphoresis (forehead)
! Bradycardia

A

Autonomic dysreflexia

26
Q

Autonomic dysreflexia - Treatment

✔️ Remove triggers
✔️ Sit pt up
✔️ Vasodilators (Nifedipine, nitrates)

A

Prevent AD
- Loose clothing
- Meticulous skin care and bowel & bladder care

27
Q

Pressure sores

  • Decubiti r/t dec sensation & failure of motor function
  • Special air/gel/sand beds
  • Repositioning & chair padding
  • Electronic chairs may be shifted to lean back to change pressure points
A

Other problems & treatments

! Spasticity
* baclofen (Lioresal); tizanidine (Zanaflex)

  • intrathecal baclofen pump - directly released into CSF & pump placed in abd

s/e: if d/c’d too abruptly, client may have seizures or hallucinate
! if too much circulating systemically, may cause sedation, changes in mental status & fatigue

! Sexual dysfunction
* sildenafil (Viagra)

28
Q

Complications

! Osteopenia/osteoporosis
- Vertical weight-bearing; PT to slow bone & muscle loss & contractures
- Vit D & Ca supplements; Boniva

! Heterotrophic ossifications
- celecoxib (Celebrex) prevents boney overgrowth

A

! Renal stones

! Psychosocial
- Depressed over many losses = body functions, freedom to move around, career, family dynamics, sexual intimacy, etc.

! DVT

29
Q

?

Is an anticholinergic for spastic bladder; helps to dec bladder spasms & inc bladder capacity

s/e: dizziness, drowsiness, agitation, headache, constipation, dry mouth, tachycardia, blurred vision, urine retention, hyperthermia

A

Detrol

30
Q

?

Cholinergic used for dec bladder tone

Urinary retention c/b neurogenic bladder

s/e: HA, lacrimation, abd discomfort, diarrhea, salivation, urgency, flushing, sweating, hypothermia

A

urecholine (Bethanechol, Duvoid)

31
Q
  • Bladder schedule - offer scheduled times to void; straight cath @ scheduled times
  • Possibly bladder scan post voids if distention or discomfort
A
  • Goal = to dec risk of AD & poss bladder perforation
  • Teach pts manifestations of UTI’s & pyelonephritis

! Watch for skin breakdown

32
Q

Complications - Neurogenic Bowel

Colonic stimulants
- Inc peristalsis
- bisacodyl (Dulcolax), senna (Senokot)

Hyperosmolar
- fleet enema

A

Bulking agents
- Metamucil, Citrucel = inc fiber & fecal content; promotes bacterial growth

Stool softeners
- Colace, Surfak

33
Q
  • Osmotic agents
    > Isotonic less risk for dehydration & electrolyte imbalance than other laxatives (hyperosmolar, colonic stimulants)
A
  • Bowel schedule
    > Sit @ reg times
34
Q

Cervical injuries

! C2 through C3 usually fatal

  • C4 - phrenic nerve disrupted = diaphragm & resp muscles involved
A
  • C5 & below = movement @ shoulder lvl
35
Q

Thoracic injuries

  • Depending on lvl of inj, there may be loss of movement w/chest, trunk, bladder, & legs
  • Paraplegia
A
  • Autonomic dysreflexia w/T6 & above
36
Q

Lumbar & Sacral injuries

  • Loss of movement & sensation to LE
  • Neurogenic bladder w/S2 & S3 inj
A
  • Sexual dysfunction in males above S2