Spinal Cord Injury Flashcards

(47 cards)

1
Q

clinical effects of complete spinal cord injury

A

all voluntary movement and sensory gone

reflex function in all segments are suspeded

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2
Q

definition of level of injury

A

most caudal segment with normal sensation and muscle strength of 3/5 or better with the level immediately above 5/5

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3
Q

complete vs incomplete lesion

A

complete: no preservation of any motor or sensory function
incomplete: any residual motor or sensory function more than 3 segments below the level of injury

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4
Q

signs of incomplete cord injury

A

any sensation or voluntary movement in LE
sacral sparing (anus sensation, perineum, voluntary anal contraction)
preservation of sacral reflexes not counted

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5
Q

what is spinal shock

A

profound loss of all spinal reflexes below level of injury + complete paralysis and anesthesia

NOT THE SAME AS COMPLETE CORD INJURY

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6
Q

presentation of spinal shock

A

flaccid paralysis
areflexia
hypotonic paralysis of bowel and bladder
hypotension, anhidrosis, flushed warm peripheral skin
hypotension without compensatory tachy (if high cervical lesion)

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7
Q

triad of neurogenic shock

A

hypotension
bradycardia
hypothermia

more common in injuries above t6 due to secondary disruption of sympathetic outflow from t1-l2

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8
Q

t/f less complete lesions and slow developing lesions result in little to no spinal shock

A

true

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9
Q

mechanisms of spinal cord injury

A

vertical compression
hyperflexion
rotational
hyperextension

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10
Q

characteristics of vertical compression

A

creates axial node on vertebra –> vertebral body is compressed –> burst fracture

usually in lumbar

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11
Q

characteristics of hyperreflexion injury

A

causes wedge fractures and stretched interspinous ligaments

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12
Q

characteristics of rotational injuries

A

shearing injury that causes fracture

results in tearing of posterior ligamentous structures and displacement of vertebrae

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13
Q

characteristics of hyperextension injury

A

causes a disruption in the anterior longitudinal ligament or buckling of ligamentum flavum into spinal canal

can rupture intervertebral discs

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14
Q

asia impairment scale

A

table 4

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15
Q

pathology in acute paraplegia syndrome

A

squeezing or shearing in spinal cord –> destruction of gray and white matter + hemorrhage –> traumatic necrosis

healing –> gliotic focus or cavitation + hemosiderin and iron
in months/years: progressive cavitation happens (traumatic syringomyelia or fluid build up) –> delayed central or incomplete transverse cord syndrome

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16
Q

t/f in most traumatic lesions, the lateral parts suffer greater injury

A

false, the central parts

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17
Q

features of cord injury in c1-c3

A

vasomotor and respiratory collapse = ventilator support

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18
Q

features of cord injury in c4-c5

A

quadriplegia/tetraplegia with preserved respiratory function

complete paralysis below the neck

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19
Q

features of cord injury in c5-c6

A

sparing of shoulder muscles with partial paralysis of arm and hands
loss of biceps and brachioradialis reflexes
paralysis of lower body

20
Q

features of cord injury in c7 and c8

A

c7: biceps sparing, loss of triceps reflex
c8: triceps sparing, paralyzed fingers and wrist flexion

21
Q

high vs low cervical cord injury

22
Q

features of t6 injury

A

paraplegia (xiphoid process)

23
Q

features of t9 injury

A

paralysis of lower abdominal muscles
loss of superficial abdominal reflexes
(+) beevor sign

24
Q

high vs low thoracic cord injury

A

high: spares UE, impaired breathing, ileus, paraplegia
low: spares abdominal muscles, normal breathing,

25
lumbar cord injury (l1)
paraplegia
26
incomplete injuries of spinal cord
central cord syndrome anterior spinal cord syndrome (anterior 2/3) posterior spinal cord syndrome (posterior column) brown sequard syndrome (hemisection)
27
most common type of incomplete spinal cord injury
central cord syndrome - symmetrical incomplete quadriparesis, disproportionately affecting ue
28
tracts affected in central cord syndrome
cervical corticospinal | cervical lateral spinothalamic
29
pathophysio of ccs
fall forward and hit chin = hyperextension usually with preexisting narrowing of spinal canal spinal cord rubs against disc osteophyte = anterior compression inward buckling of ligamentum flavum = posterior compression
30
clinical presentation of ccs
loss of pain and temperature in the distribution level of the injury expansion of lesion = weakness at level of sensory loss
31
syringomyelia in ccs
fluid filled cavitation in the cord - loss of pain and temp sensation (cape-like sensory loss) - suspended sensory loss - weakness of muscles in arms with atrophy and hyporeflexia - later: cst involvement with brisk reflexes in legs, spasticity and weakness
32
etiology of syringomyelia in ccs
trauma | chiari type 1 malformation
33
clinical presentation of syringomyelia
central cord syndrome (arms weaker than legs) dissociated sensory loss areflexic weakness in ue
34
management of syringomyelia
syringo-subarachnoid shut
35
another type of lesion that can have similar presentation with ccs
intramedullary tumor
36
clinical presentation of anterior spinal cord syndrome
bilateral muscle weakness (cst) bilateral loss of pain and temp sensation (stt) urinary incontinence (descending autonomic tracts) posterior column sparing
37
etiology of ascs
anterior spinal artery infarction 2/3 (thromboembolism, trauma, vertebral burst fracture) intervertebral disc herniation radiation myelopathy spinal metastasis (fracture or sc compression) cervical spine injury w/ wedge and burst fractures (bone compressing)
38
progression of symptoms in ascs
``` back pain numbness of limbs difficulty walking urinary urgency paralysis ```
39
least favorable prognosis in all of syndromes
ascs preservation of some sensory or motor function below the level of injury are good prognostic factors
40
clinical presentation of posterior spinal cord syndrome
loss of proprioception and vibration sense no weakness bladder dysfunction
41
clinical presentation of brown sequard syndrome
ipsilateral: weakness, lmn symptoms at level, umn symptoms below, loss of proprioception, vibrationi, light touch, and tactile sense contralateral findings: loss of pain and temperature sensation
42
etiology of brown sequard syndrome
knife or bullet ms tumor
43
t/f cauda equina syndrome is part of incomplete cord injuries
false
44
clinical presentation of cauda equina syndrome
radicular pain in sciatic distribution that worsens with coughing or sneezing severe radicular sensory deficits in legs and saddle area lmn deficit: flaccid paresis of le with areflexia, urinary and fecal incontinence, impaired sex
45
clincial presentation of conus syndrome
``` detrusor areflexia with urinary retention and overflow incontinence fecal incontinence impotence saddle anesthesia loss of anal reflex ```
46
management for conus syndrome
open surgery to remove disk that comes out
47
clinical presentation of poliomyelitis
lower motor neuron weakness (invasion of anterior horn cells) flaccid paralysis atrophy areflexia