Spine Trauma Flashcards

(122 cards)

1
Q

When is Removal of cervical collar WITHOUT radiographic studies is allowed

A
  • patient is awake, alert, and not intoxicated AND
  • has no neck pain, tenderness, or neurologic deficits AND
  • has no distracting injuries
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2
Q

What to look for in the trauma setting on an X-Ray to R/O cervical Fx

A
  • soft-tissue swelling
  • Hypo-lordosis
  • disk-space narrowing or widening
  • widening of the interspinous distances
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3
Q

Incidence of Iatrogenic SCI?

A

it is estimated that 3-25% of all spinal cord injuries occur after initial traumatic episode due to improper immobilization and transport.

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4
Q

What is the pathophysiology of SCI?

A

◦ primary injury

  • damage to neural tissue due to direct trauma
  • irreversible

secondary injuryinjury to adjacent tissue due to

  • decreased perfusion
  • lipid peroxidation
  • free radical / cytokines
  • cell apoptosis

methylprednisone used to prevent secondary injury by improving perfusion, inhibiting lipid peroxidation, and decreasing the release of free radicals

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5
Q

What are the risk factors for vertebral artery injury

A
  • Atlas fractures
  • Facet dislocations
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6
Q

What is the prognosis of SCI?

A

only 1% have complete recovery at time of hospital diagnosis

conus medullaris syndrome has a better prognosis for recovery than more proximal lesions

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7
Q

What is the definition of tetraplegic, paraplegic, Complete injury and incomplete injury

A

Tetraplegia: arms, trunk, legs, and pelvic organs

Paraplegia: Arm function is preserved

Complete injury: an injury with no spared motor or sensory function below the affected level.

patients must have recovered from spinal shock (bulbo-cavernosus reflex is intact) before an injury can be determined as complete

classified as an ASIA A

incomplete injury

an injury with some preserved motor or sensory function below the injury level

incomplete spinal cord injuries include

  • anterior cord syndrome
  • Brown-Sequard syndrome
  • central cord syndrome
  • posterior cord syndrome
  • conus medullaris syndromes
  • cauda equina syndrome
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8
Q

What are the steps for ASIA Classification?

A
  1. Determine if patient is in spinal shock
    * check bulbocavernosus reflex
  2. Determine neurologic level of injury

lowest segment with intact sensation and antigravity (3 or more) muscle function strength

in regions where there is no myotome to test, the motor level is presumed to be the same as the sensory level.

  1. Determine whether the injury is COMPLETE or INCOMPLETE

COMPLETE defined as (ASIA A)

no voluntary anal contraction (sacral sparing) AND

0/5 distal motor AND

0/2 distal sensory scores (no perianal sensation) AND

bulbocavernosus reflex present (patient not in spinal shock)

INCOMPLETE defined as

voluntary anal contraction (sacral sparing)

sacral sparing critical to determine complete vs. incomplete

OR palpable or visible muscle contraction below injury level OR

perianal sensation present

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9
Q

ASIA Grades

A
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10
Q

What are the Stages of spinal shock?

A

Phase 1 -

hypo-reflexic

0 to 48 hours

Areflexia/hypo-reflexic

Phase 2 -

initial reflex return

1-2 days

polysynaptic reflexes return (bulbo-cavernous reflex)

monosynaptic (patellar) remain absent

Phase 3 -

initial hyper-reflexia

1-4 weeks

Phase 4 - spasticity

1 to 12 months

characterized by altered skeletal performance

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11
Q

What SCI require intubation?

A

above C5

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12
Q

What should seat belt sign (abdominal ecchymoses) raise suspicion for?

A

flexion distraction injuries of thoracolumbar spine

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13
Q

Recommended initial medical treatment?

A
  • DVT prophylaxis
  • Hypotension should be avoided
  • Decubitus ulcer prevention
  • acute closed reduction with axial traction
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14
Q

What are the surgical indications from GSW

A

Most incomplete SCI (except GSW)

decompress when patient hits neurologic plateau or if worsening neurologically

decompression may facilitate nerve root function return at level of injury (may recover 1-2 levels)

Most complete SCI (except GSW)

stabilize spine to facilitate rehab and minimize need for halo or orthosis

decompression may facilitate nerve root function return at level of injury (may recover 1-2 levels)

consider for tendon transfers

e.g. Deltoid to triceps transfer for C5 or C6 SCI

GSW with

progressive neurological deterioration with retained bullet within the spinal canal

cauda equina syndrome (considered a peripheral nerve)

retained bullet fragment within the thecal sac

CSF leads to the breakdown of lead products that may lead to lead poisoning

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15
Q

Function C1-C3 SCI

A
  • Ventilator dependent with limited talking.
  • Electric wheelchair with head or chin control
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16
Q

Function C3-C4

A
  • Initially ventilator dependent, but can become independent
  • Electric wheelchair with head or chin control
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17
Q

Function C5 SCI

A
  • Ventilator independent
  • Has biceps, deltoid, and can flex elbow, but lacks wrist extension and supination needed to feed oneself
  • Independent ADL’s; electric wheelchair with hand control, minimal manual wheelchair function
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18
Q

SCI FUNCTION

A
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19
Q

What is the prognosis for complete injuries- Incomplete injuries- Conus medullaris syndrome?

A

• Complete Injuries

Improvement of one nerve root level can be expected in 80% of patients

improvement of two nerve root levels can be expected in 20% of patients

only 1% have complete recovery at time of hospital diagnosis

Incomplete Injuries

trends of improvement include

the greater the sparring, the greater the recovery

patients that show more rapid recovery have a better prognosis

when recovery plateaus, it rarely resumes improvement

Conus Medullaris syndrome:

has a better prognosis for recovery than more proximal lesions

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20
Q

What are the complications of SCI?

A
  1. Skin problems
  2. Venous Thromboembolism
  3. Urosepsis: common cause of death; strict aseptic technique when placing catheter; don’t let bladder become overly distended
  4. Sinus bradycardia: most common cardiac arrhythmia in acute stage following SCI
  5. Orthostatic hypotension: occurs as a result of lack of sympathetic tone
  6. Autonomic Dysreflexia; potentially fatal; presents with headache, agitation, hypertension; caused by unchecked visceral stimulation; check foley; disimpact patient; radiographs of lower extremity if there is concern for undiagnosed fracture
  7. Major depressive disorder: ~11% of patients with spinal cord injuries suffer from MDD; MDD in spinal cord injury patients is highly associated with suicidal ideation in both the acute and chronic phase.
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21
Q

3 things to check with Autonomic dysreflexia

A

unchecked visceral stimulation; check foley; disimpact patient; radiographs of lower extremity if there is concern for undiagnosed fracture

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22
Q

What are the 4 incomplete SCI

A
  1. Anterior cord syndrome
  2. Brown-Sequard syndrome
  3. central cord syndrome
  4. posterior cord syndrome
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23
Q

What is the most common ISCI? Population?

A

Central Cord Syndrome

  • Elderly with minor extension injury mechanisms
    • due to anterior osteophytes and posterior infolded ligamentum flavum
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24
Q

What is the pathophysiology of Central cord syndrome?

A

spinal cord compression and central cord edema

selective destruction of lateral cortico-spinal tract white matter

hands and upper extremities are located “centrally” in cortico-spinal tract

Weakness with hand dexterity most affected

Hyper-pathia

Burning in distal upper extremity

motor deficit worse in UE than LE (some preserved motor function)

hands have more pronounced motor deficit than arms

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25
What is the prognosis of central cord syndrome?
good prognosis although full functional recovery rare usually ambulatory at final follow up usually regain bladder control upper extremity and hand recovery is unpredictable and patients often have permanent clumsy hands
26
How does recovery occurs with central cord syndrome?
◦ recovery occurs in typical pattern * lower extremity recovers first * bowel and bladder function next * proximal upper extremity next * hand function last to recover
27
Particularities of anterior cord syndrome
* Worst prognosis; 10-20% chance of motor recovery * motor dysfunction + dissociated sensory deficit below level of SCI * flexion/ compression injury * lower extremity affected more than upper extremity
28
Brown Sequard Mechanism Symptoms Prognosis
* Penetrating trauma * Ipsilateral deficit LCS tract * Motor function * dorsal columns * proprioception * vibratory sense * contralateral * deficitLST: pain, temperature * spinothalamic tracts cross at spinal cord level (classically 2-levels below)
29
True or False Improved Neuro outcome with early \<24hours decompression
True Fehlings et al. performed a multicenter prospective cohort study on the timing of intervention for spinal cord injuries. They found improved neurological outcome for patients with complete or incomplete spinal cord injuries that were decompressed within 24 hours compared to those that were decompressed at a later time. Additionally, they found no increased risk of mortality or complications for patients who underwent early surgical intervention. **STASCIS**
30
Definition of ASIA B
no motor function preserved more than 3 levels below the affected neurological level is consistent with an ASIA B category injury.
31
Evidence on administering a dose of Methylprednisolone 30 mg/kg bolus followed by a 5.4 mg/kg/hr infusion x 24 hours
It is not supported by current literature - recent studies have shown an increased risk of complications with no clear evidence of benefits
32
What is Autonomic Dysreflexia? Treatment
increase in systolic blood pressure of at least 20% associated with a change in heart rate and accompanied by at least one of the following signs (sweating, piloerection, facial flushing), or symptoms (headache, blurred vision, nasal congestion) due to a stimulus such as overdistended bladder or bowel impaction. due to sympathetic decentralization leads to altered regulation of the autonomic function, despite the presence of intact parasympathetic (vagal) afferent and efferent pathways in patients with SCI. Guidelines for treatment of autonomic dysreflexia include 1) patient immediately placed in a sitting position if the person is supine. 2) clothing or constrictive devices need to be loosened 3) troubleshoot etiologies for bladder distention or bowel impaction 4) a SBP \>150 mmHg may need to be treated with nifedipine or nitrates 5) close monitoring of symptoms, blood pressure, and heart rate for at least 2 hours.
33
What is the most important predictor of her neurologic outcome
Severity of initial neurologic injury
34
When is Posterior deltoid-to-triceps transfer is considered
COMPLETE spinal cord injuries at C5 or C6 with 5/5 delt/biceps, but 0/5 triceps. Activities such as dressing, controlling a power wheelchair and supporting oneself while sitting are dependent on the balanced forces provided by the triceps muscle. The transfer involves detaching the posterior deltoid muscle and anchoring the tendon sutured into the triceps muscle. It is the best choice as it will allow for opposing elbow extension to his maintained bicep function - which will help patients to perform reaching movements and improve level of functional independence.
35
2 types of Occipito-cervical instability
Traumatic occipito-cervical dislocation: most patients die of brainstem destruction Acquired occipito-cervical instability: Down's syndrome; occipital condyle hypoplasia results in limited AOJ motion and basilar invagination
36
Associated Conditions with Occipito-cervical Instability & Dislocation
* Atlanto-axial instability: also seen in Down syndrome patients * neurologic deficits * vertebral or carotid artery injuries * Down Syndrome
37
Atlas C1 anatomy
* Ring containing two articular lateral masses * No vertebral body or a spinous process * anatomic variation: incomplete formation of the posterior arch is a relatively common; does not represent a traumatic injury * Ligamentous structures: transverse ligament; paired alar ligaments; apical ligament ; tectorial membrane
38
Types of Atlanto-occipital dislocation
**Type I** Anterior occiput dislocation **Type II** Longitudinal dislocation **Type III** Posterior occiput dislocation
39
Measurements done in Atlanto-Occipital dislocation
- **_Powers ratio_** = C-D/A-B C-D: distance from basion to posterior arch A-B: distance from anterior arch to opisthion significance ratio ~ 1 is normal if \> 1.0 concern for * anterior dislocation ratio \< 1.0 raises concern for * posterior atlanto-occipital dislocation * odontoid fractures * ring of atlas fractures - **_Harris rule of 12_** basion-dens interval or basion-posterior axial interval \>12mm suggest occipito-cervical dissociation
40
Posterior Occipito-cervical fusion
**_Approach:_** midline posterior approach to base of skull Instrumentation - rigid occipito-cervical screw-rod or plate construct - aim for 3 uni-cortical occipital screws on each side of the midline (total 6 screws in occiput) - some institutions prefer bi-cortical screws but they come at increase risk extend to C2 or lower with polyaxial pedical screws to achieve fixation - **_Safe zone for Occipital screws_** : within 20mm lateral to the external occipital protuberance along the superior nuchal line **Structures at risk:** ***major dural venous sinuses*** located below the external occipital protuberance
41
structure at the greatest risk of injury with perforation of the anterior cortex at C1
Internal carotid artery
42
2 Processes that might lead to Atlanto-axial instability
Degenerative and traumatic processes
43
Adult and Pediatric causes of C1-C2 instability
**_Adult causes_** **_Degenerative_** Down's syndrome Rheumatoid Arthritis Os Odontoideum **_Traumatic_** Type I odontoid fracture (very rare) Atlas fractures Transverse ligament injuries **_Pediatric causes_** **_Degenerative_** JRA Morquio's Syndrome lysosomal storage disorder **_Trauma/infection_** rotatory Atlanto-axial subluxation
44
C1-C2 instability Radiographics parameters
* flexion-extension x-rays atlanto-dens interval (ADI) adult parameters \> 3.5mm considered unstable \> 10mm indicates surgery in RA * space-available-cord (SAC) = posterior atlanto-dens-interval (PADI) in adults with RA \< 14 mm associated with increased risk of neurologic injury and is an indication for surgery * sum of lateral mass displacement if \> 8.1 mm, then a transverse ligament rupture is assured and the injury pattern is considered unstable
45
Management of os odontoideum
Whether os odontoideum is congenital or the residual of a traumatic process is controversial. Most authors support a post-traumatic etiology; however, some evidence exists to support a congenital origin. - Asymptomatic patients may be managed with cessation of contact sports alone. - Neurologic findings and widened ADI are both indications for a posterior C1-C2 fusion.
46
What is a hangman's fracture?
bilateral fracture of pars inter-articularis of C2 Traumatic Spondylo-listhesis of Axis
47
What is the mechanism of hangman's fracture?
◦ Hyperextension + Distraction injuries: leads to fracture of pars secondary flexion: tears PLL and disc allowing subluxation
48
What is the classification for Hangman's fracture and treatment according to fracture type
**_Levine and Edwards Classification_** 1. Type 1: \< 3mm displacement: Rigid collar 2. Type 2: \>3.5mm displacement: Halo/Surgery 3. Type 2A: angulated \>11deg: Reduction+Halo 4. Type 3: Type 1 with associated bilateral facet dislocation: Surgical reduction of facet + stabilization
49
3 techniques for C2-C3 stabilization
* anterior C2-3 interbody fusion * posterior C1-3 fusion * bilateral C2 pars screw osteosynthesis
50
What is the mechanism of an atlas fracture?
Hyperextension lateral compression axial compression
51
What are the associated conditions with C1 fx
◦ spine fracture 50% have an associated spine injury 40% associated with axis fx
52
What is the Prognosis of C1?
◦ stability dependent on degree of injury and healing potential of transverse ligament
53
Classification of C1 Fractures
**Type I** Isolated anterior or posterior arch fracture. A "plough fracture is an isolated anterior arch fracture caused by a force driving the odontoid through the anterior arch. Stable. Treat with hard collar. **Type II** Jefferson burst fracture with bilateral fractures of anterior and posterior arch resulting from axial load. Stability determined by integrity of transverse ligament. If intact, hard collar. If disrupted, halo vest (for bony avulsion) or C1-2 fusion (for intrasubstance tear)(see Dickman classification below). **Type III** Unilateral lateral mass fx. Stability determined by integrity of transverse ligament. If stable, treat with hard collar. If unstable, halo vest.
54
Radiographics measurements for C1 Fractures
**_atlantodens interval (ADI)_** \< 3 mm = normal in adult (\< 5mm normal in child) 3-5 mm = injury to transverse ligament with intact alar and apical ligaments \> 5 mm = injury to transverse, alar ligament, and tectorial membrane **_lateral mass displacement_** if sum of lateral mass displacement is \> 7 mm (8.1mm with radiographic magnification) then a transverse ligament rupture is assured and the injury pattern is considered unstable
55
How to determine stability of C1 Fx
Transverse ligament integrity
56
Non operative management of C1 Fx Operative Management of C1 Fx
Hard collar vs. halo immobilization for 6-12 weeks posterior C1-C2 fusion vs. Occipitocervical fusion
57
Anderson and D'Alonzo Classification Grauer Classification of Type II Odontoid fractures
Type I Oblique avulsion fx of tip of odontoid. Due to avulsion of alar ligament. Although rare, atlantooccipital instability should be ruled out with flexion and extension films. Type II Fx through waist (high nonunion rate due to interruption of blood supply). Type III Fx extends into cancellous body of C2 and involves a variable portion of the C1-C2 joint.à Type IIA Non-displaced/minimally displaced with no comminution. Treatment is external immobilization Type IIB Displaced fracture with fracture line from anterosuperior to posteroinferior. Treatment is with anterior odontoid screw (if adequate bone density). Type IIC Fracture from anteroinferior to posterosuperior, or with significant comminution. Treatment is with posterior stabilization.
58
Treatment algorithm of C2
Os Odontoideum Observation **Type I** Cervical Orthosis for 6-12 weeks **Type II Young** Halo if no risk factors for nonunion Surgery if risk factors for nonunion **Type II Elderly** Cervical Orthosis if not surgical candidates Surgery if surgical candidates **Type III** Cervical Orthosis; no evidence to support Halo over hard collar
59
Indications for anterior odontoid osteosynthesis
* Type II fractures with risk factors for nonunion AND * acceptable alignment and minimal displacement * oblique fracture pattern perpendicular to screw trajectory * patient body habitus must allow proper screw trajectory
60
3 posterior stabilization for C2 Fx
1. **Sublaminar wiring techniques (**Gallie or Brooks) require postoperative halo immobilization and rarely used 2. **posterior C1-C2 transarticular screws construct** contraindicated in patients with an aberrant vertebral artery 3. **posterior C1 lateral mass screw and C2 pedicle screw construct** modern screw constructs do not require postoperative halo immobilization
61
What are the risk factors for C2 non-union?
* ≥ 6 mm displacement (\>50% nonunion rate): strongest reason to opt for surgery * age \> 50 years * fx comminution * fracture gap \> 1 mm * angulations \> 10° * delay in treatment ( \> 4 days) * posterior re-displacement ( \> 2 mm) * smoker
62
When does the secondary ossification center of C2 fuse?
C2 develops from five ossification centers: - body - two neural arches - odontoid - secondary ossification center The subdental (basilar) **synchondrosis** is an initial cartilagenous junction between the dens and vertebral body that does not fuse until ~**6 years of age**. The **secondary ossification center** appears around age 3 and fuses with the odontoid at around 12 years of age.
63
Important consideration when performing C1-C2 trans-articular screws
aberrant vertebral artery The vertebral artery is an important consideration when performing posterior cervical spine surgery. Injury to this artery can lead to stroke and death. Normally the vertebral artery travels superiorly in the transverse foramen of C6 to C2. At C2 the artery deviates laterally to the pass through the transverse foramen of C1 and then wraps medially on the superior surface of the posterior arch of C1 before ascending into the foramen magnum. Anomalous variants of the vertebral arery may be present in up to 30% of individuals, and may be intraosseous or extraosseous. Intraosseous variants may be injured during posterior cervical stabilization techniques. In patients with an aberrant vertebral artery, C1-C2 transarticular screws are contraindicated due to the risk of injury to the aberrant vertebral artery.
64
What are the criterias of instability for lateral cervical mass fracture separation?
\>3.5mm displacement \>10deg kyphosis \>10deg rotation difference compared with adjacent vertebra
65
Surgical treatment of lateral cervical mass fracture separation
1. posterior decompression and 2 level instrumented fusion 2. 2 level ACDF 3. Anterior and posterior decompression and fusion
66
Mechanism of injury cervical lateral mass fracture?
hyperextension, lateral compression and rotation
67
4 types of subaxial cervical fractures Mechanism Prognosis Treatment
**_1- compression fracture_**: without retropulsion into canal; often associated with posterior ligamentous injury **_2- burst fracture :_** fracture extension through posterior cortex with retropulsion into the spinal canal; often associated with posterior ligamentous injury; often associated with complete and incomplete spinal cord injury; unstable and usually requires surgery **_3- Flexion teardrop fracture:_** Anterior column failure in flexion/compression Posterior portion of vertebra retropulsed posteriorly Posterior column failure in tension Larger anterior lip fragments may be called 'quadrangular fractures' Prognosis: associated with SCI Treatment: unstable and usually requires surgery **_4- Extension teardrop avulsion:_** fracture characterized by small fleck of bone is avulsed of anterior endplate usually occur at C2 must differentiate from a true teardrop fracture Mechanism: extension Prognosis: stable injury pattern and not associated with SCI Treatment: cervical collar
68
What are the indications for non-operative treatments for subaxial cervical fractures?
* Stable mild compression fractures (intact posterior ligaments & no significant kyphosis) * anterior teardrop avulsion fracture
69
Surgical treatment indication for subaxial cervical fractures?
1. compression fracture with 11 degrees of angulation or **25% loss of vertebral body height** 2. unstable burst fracture with cord compression 3. unstable tear-drop fracture with cord compression **_Treatment_** : **_1- Decompression_**: Early decompression (\< 24 hours) has been shown to improve neurologic outcomes compared with delayed (\>/ 24 hours) decompression 2- Anterior decompression, corpectomy, strut graft and fusion with instrumentation OR Posterior decompression, & fusion with instrumentation if significant injury to posterior elements and anterior decompression not required
70
Location of the majority of cervical facet dislocations and fractures
17% C7-T1 junction
71
Which facet is most commonly fractures in cervical facets fx/dislocation?
more frequently involves superior facet
72
Uni vs bilateral facet dislocation on Xray
unilateral facet dislocation leads to ~25% subluxation on xray associated with monoradiculopathy that improves with traction inferior facet of the cephalad vertebrae encrouches the neuroforamina bilateral facet dislocation leads to ~50% subluxation on xray often associated with significant spinal cord injury (~80% of cases)
73
Mechanism of injury of cervical facet dislocation
▪ flexion and distraction forces +/- an element of rotation rotational moment associated with unilateral facet dislocation
74
Clinical presentation unilateral versus bilateral facet dislocations?
**_monoradiculopathy_** seen in patients with unilateral dislocations C5/6 unilateral dislocation presents with a C6 radiculopathy weakness to wrist extension numbness and tingling in the thumb C6/7 unilateral dislocation presents with a C7 radiculopathy weakness to triceps and wrist flexion numbness in index and middle finger **_spinal cord injury_**—\> bilateral dislocations
75
Timing Of MRI in cervical facet dislocations
Controversial ## Footnote ▪ an MRI should always be performed prior to open reduction or surgical stabilization if a disc herniation is present with compression on the spinal cord, then you must go anterior to perform a anterior cervical diskectomy
76
fixation in lateral cervical facet fracture versus in cervical facet dislocation?
important to identify as cervical lateral mass fracture separations require fusing two levels while a facet dislocation only requires fusing a single level
77
indicaitons for emergent closed reduction, emergent MRI, then urgent surgical stabilization
▪ bilateral facet dislocation with deficits in awake and cooperative patient ▪ unilateral facet dislocation with deficits in awake and cooperative patient
78
Problem of Halo immobilization in lower cervical spine
▪ requires close radiographic follow-up; risk of re-dislocation or subluxation morbidly obese patients may not fit or be adequately stabilized in a halo brace
79
Factors to take into consideration for surgical treatment
1. Disc herniation 2. Difficulty to reduce from the front
80
Closed reduction technique for cervical facet dislocation
▪ Application of Gardner-Wells tongs 1 cm above the pinna and in line with the external auditory meatus below the equator of the skull avoids pin migration and slippage gradually increase axial traction with the addition of weights usually in 5 to 10 lbs increments can add up to 140 lbs of weight or 70% body weight average weigh required for reduction ~9.4 to 9.8 lbs per segment above the injury level a component of cervical flexion can facilitate reduction flexion moment can be created with pulley system or posterior placement of the Gardner-Wells tongs pins once reduced, decrease traction weight be 10-15 lbs and apply an extension moment to the cervical spine adjusting pulley system placing pad underneath thorax perform serial neurologic exams and plain radiographs after addition of each weight additionabort if there is over distraction of the spinal segment \>1.5 times that if the adjacent uninjured disc space can switch to carbonfiber Gardner-Wells tongs if need to obtain MRI in traction traction limit ~80 lbs abort if neurologic exam worsens and obtain immediate MRI
81
Indications for anterior cervical discectomy and fusion +/- open reduction
▪ Facet dislocations reduced through closed methods with a MRI showing cervical disc herniation with significant compression on the spinal cord Unilateral facet dislocations that fail closed reduction with a disc herniation with significant compression on the spinal cord
82
Techniques for anterior reduction of facets
▪ unilateral dislocations can be reduced by distracting vertebral bodies with **caspar pins** and then rotating the proximal pin towards the side of the dislocation bilateral dislocations are reduced by placing **converging Caspar pins (10-20° angle) and then compressing the ends together to unlock the facets** posterior directed force applied to rostral vertebral body with currette alternatively, lamina spreaders applied to the endplates not effective for reducing bilateral facet dislocations
83
Technique for posterior reduction of cervical facet dislocation
**▪ reductionPenfield 4 inserted between facets and used to lever back into position** can remove the superior aspect of the superior facet of the caudad vertebrae to facilitate difficult reductions distraction of the affected level between the affected spinous processes or lamina with use of lamina spreaders usually have to fuse two levels due to inadequate lateral mass purchase at level of dislocation
84
More severe neurological outcomes unilateral or bilateral cervical dislocations?
Bilateral
85
An awake and cooperative patient presents to the emergency room with bilateral C5-6 facet dislocation. Prior to the CT scan he had an ASIA Impairment Scale of E. Upon returning from the CT scanner he has an ASIA Impairment Scale of D. What is the most appropriate first step in management?
immediate closed reduction with cervical traction
86
For what type of fractures is an Halo orthosis ideal for?
Ideal for upper C-spine injury Ideal for controlling **_Rotation at the atlantoaxial joint_** Allows intercalated paradoxical motion in the subaxial cervical spinetherefore not ideal for lower cervical spine injuries (**_lateral bending least controlled_**) **"snaking phenomenon"** recumbent lateral radiograph shows focal kyphosis in mid-cervical spine yet, upright lateral radiograph shows maintained lordosis in mid-cervical spine
87
Absolute and Relative contraindications for Halo Vest?
**_Absolute_** * cranial fractures * infection * severe soft-tissue injury especially near proposed pin sites **_Relative_** * Polytrauma * severe chest trauma * barrel-shaped chest * obesity * advanced age recent evidence demonstrates an unacceptably high mortality rate in patients aged 79 years and older (21%)
88
Imaging before Halo application
CT scan prior to halo applicationindications clinical suspicion for cranial fracture children younger than 10 to determine thickness of bone
89
Technique for Adult and Pediatric Halo Vest
**_Adults_** Torque: tighten to 8 inch-pounds of torque 4 pins 2 anterior pins safe zone is a **1 cm region just above the lateral one third of the orbit (eyebrow) at or below the equator of the skull** this is anterior and medial to temporalis fossa/temporalis muscle this is lateral to supraorbital nerve 2 posterior pins: placed on opposite side of ring from anterior pins followup care can have patient return on day 2 to tighten again proper pin and halo care can be done to minimize chance of infection **_Pediatric Technique_** Pediatrics torque more pins with less torque total of 6-8 pins lower torque (**2-4 in-lbs or "finger-tight**")
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8 Complications of Halo Vest
Higher complications in children (70%) than adults (35%) 1- Loosening (36%): treated with retightening; if continues to loosen, should be treated with pin exchange 2- Infection (20%)posterior pin in temporalis fossa because pins hidden in hairline bone is thin temporalis muscle moves with chewing can be treated with **oral antibiotics if pin not loose** if pin infection and loose then pin should be removed 3- Discomfort (18%): treated by loosening skin around pin 4- Dural puncture (1%) 5- Abducens nerve (Cranial Nerve VI) palsy most commonly injured cranial nerve with halo traction injury; diplopia; loss of lateral gaze on affected side; observation most resolve spontaneously 6- Supraorbital nerve palsy: injured by medially placed anterior pins 7- Supra-trochlear nerve palsy: injured by medially placed anterior pins 8- Medical complications pneumonia ARDS arrhythmia
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Placing a pin for a halo vest orthosis in the red zone places what structures at risk?
Supraorbital and supratrochlear
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Consequences of using a higher halo torque Halo vest in pediatric population
Due to reduced thickness of the pediatric skull, **higher rate of pin site infections** and **skull puncture**, and unreliability of many torque wrenches, current recommendations are to use a higher number of pins (8 to 12) with lower insertional torque (1 to 5 in-lb) in case pins need to be removed or exchanged.
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What is the most common neuro complication of Halo vest application?
Cranial nerve VI palsy
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Most common location for thoraco-lumbar dislocation? Mechanims of injury
Thoraco-lumbar junction Acceleration/deceleration injuries Hyperflexion, rotation and shearing
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TLICS classification
**_1- Morphology_** * compression (+1 point) * burst (+2 points) * rotation/translation (+3 points) * distraction (+4 points) **_2- Neurologic status_** * Intact (0 point) * Nerve root (+2 points) * Incomplete Spinal cord or conus medullaris injury (+3 points) * Complete Spinal cord or conus medullaris injury (+2 points) * Cauda equina syndrome (+3 points) **_3- posterior ligamentous complex integrity_** * intact (0 point) * no interspinous ligament widening seen with flexion views. MRI shows no edema in interspinous ligament region * suspected/indeterminate (+2 points) * MRI shows some signal in region of interspinous ligaments * disrupted (+3 points) * widening of interspinous distance seen TLICS treatment implications score \< 4 points nonsurgical management score = 4 points nonsurgical or surgical managment score \> 4 points surgical management indicated
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Mechanism of injury of a chance Fracture
flexion-distraction injury associated with a center of rotation ***_anterior to the spine (frequently being associated with bowel or other abdominal organ injury)._***
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Associated Fractures with Thoraco-lumbar Burst Fractures
**_concomitant spine fractures:_** occurs in 20% traumatic durotomy lamina fracture is associated with dural tear and entrapped nerve roots be aware when spit spinous process **_chest and intra-abdominal injuries:_** common flexion-distration and fracture-dislocations bowel rupture, major vessel injury, upper urinary tract injury, hepatic, splenic, and pancreatic lacerations long bone fractures
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Consideration of Conservative Treatment in thoraco-Lumbar Burst fracture
▪ patients that are **neurologically intact and mechanically stable** **posterior ligament complex preserved** **no focal kyphosis on flexion and extension lateral radiographs** **kyphosis \< 30° (controversial)** **vertebral body has lost \< 50% of body height (controversial)** TLICS score = 3 or lower
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Consideration for surgical treatment of bust fractures according to level of injury
**_posterior approach favored when_** below conus --\> possible to medialize thecal sac to perform decompression of canal / posterior corpectomy and expandable cage injury to PLC fracture dislocations **_anterior/direct lateral approach favored when_** above the conus medullaris (above L2) allow for thorough decompression of the thecal sac substantial vertebral body comminution in order to reconstitute the anterior column kyphotic deformity \>30° chronic injuries greater than 4-5 days from the injury
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Anterior Approach to the spine according to the level of injury
**_lumbar spine:_** **_Anterior Retroperitoneal or transperitoneal approach_** Left paramedian incision suitable for levels **below L1** **_thoracolumbar junction : lateral lumbotomy_** suitable for injuries at **T11-L1** left-sided approach to avoid liver obstructing access **_thoracic spine: lateral thoracotomy_** right-sided approach to avoid major vessels appropriate for injuries **above T11**
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Complications of posterior fusion spine for Brust Fx
**_1- Entrapped nerve roots and dural tear_** from associated lamina fractures can be iatrogenic from decompression decreased risk of dural tears with anterior approach due to improved visualization of the thecal sac during decompression requires closure primarily or reinforced with dural patch prolonged recumbency postoperatively **_2- Pain_** most common; over distraction with instrumentation **_3- Progressive kyphosis_** common with unrecognized injury to PLL increased comminution of the vertebral body loss of anterior column support **_4- Flat back_** leads to pain, a forward flexed posture, and easy fatigue post-traumatic syringomyelia **_5- Surgical site infection_**: can occur in up to 10% of cases trauma predisposes to infection catabolic state increased soft tissue damage requires irrigation and debridement with culture specific antibiotics **_6- Pseudo-arthrosis_**: can result from overdistraction instrumentation **_7- Iatrogenic neurologic injury_** can occur in 1% of cases causes include over medialized pedicle screws inadvertant manipulation of the spinal cord
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TLSO for burst fracture? Does it make a difference
Bailey et al. conducted a multicenter, randomized controlled trial of T11-L3 burst fractures without neurologic deficit treated with or without TLSO and early mobilization. They found equivalent disability scores between the groups at 3 months, indicating that the discomfort and deconditioning associated with brace use might be avoidable.
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Most common site for burst fractures?
T12 and L2
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What is the Most common fragility fracture ?
Osteoporotic Vertebral Compression Fracture
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What are the Changes seen in osteoporotic bone?
bone is normal quality but decreased in quantity cortices are thinned cancellous bone has decreased trabecular continuity
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bone mineral density in the lumbar spine (BMD): Peak-
peaks at: between 33 to 40 yrs in women; between 19 to 33 years in men correlate well with bone strength and is a good predictor of fragility fracture
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Definition of osteoporosis
T score below -2.5
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What is complication related to vertebral compression fractures?
compromised pulmonary function increased kyphosis can affect pulmonary function **each VCF leads up to 9% reduction in FV** increased risk of mortality from pulmonary dysfunction
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What is the prognosis after a vertebral compression fracture?
mortality 1-year mortality ~ 15% (less than hip fx) 2-year mortality ~20% (equivalent to hip fx)
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What is the use of MRI in vertebral compression fractures?
* acute vs chronic nature of compression fracture * injury to anterior and posterior ligament complex * spinal cord compression by disk or osseous material * cord edema or hemorrhage
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What variables should raise suspicion for metastatic cancer to the spine?
* fractures above T5 * atypical radiographic findings * failure to thrive and constitutional symptoms * younger patient with no history of fall
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3 treatments for Vertebral Compression Fractures?
1- Observation, bracing and medical management 2- Kyphoplasty 3- Surgical decompression and stabilization
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What is the conservative treatment of vertebral compression fractures?
Observation, bracing, and medical management majority of patients can be treated with observation and gradual return to activity PLL intact (even if \> 30 degrees kyphosis or \> 50% loss of vertebral body height) Calcitonin- Biphosphonates- Extension orthosis
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When can calcitonin be used for vertebral compression fractures?
if the fracture is less than five days old ; calcitonin can be used for four weeks to decrease pain
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Surgical decompression and stabilization indications in vertebral compression fractures?
▪ very rare in standard VCF progressive neurologic deficit PLL injury and unstable spines techniqueto prevent possible failure due to osteoporotic bone consider long constructs with multiple fixation points consider combined anterior fixation
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What is the difference between Kyphoplasty vs. Vertebroplasty
under fluoroscopic guidance; percutaneous trans-pedicular approach used for cannula **_Vertebroplasty_** PMMA injected directly into cancellous bone without cavity creation performed when cement is more liquid requires greater pressure because no cavity is created increased risk of extravasation into spinal canal is greater Indications: controversial; AAOS recommends strongly against the use of Vertebroplasty in 2011 but then changed their stance in 2014 based on recent studies Outcomes: randomized, double-blind, placebo-controlled trials have shown no beneficial effect of Vertebroplasty ; Vertebroplasty has higher rates of cement extravasation and associated complications than Kyphoplasty **_Kyphoplasty_** cavity created with expansion device (e.g., balloon) prior to PMMA injection performed when cement is more viscous may be possible to obtain partial reduction of fracture with balloon expansion Indications: **_patient continues to have severe pain symptoms after 6 weeks of nonoperative treatment:_** AAOS recommend may be used, but recommendation strength is limited
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When to consider kyphoplasty?
patient continues to have severe pain symptoms after 6 weeks of nonoperative treatment:
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What is Kummell’s disease?
Vertebral body osteonecrosis (aka Kummell's disease) Delayed post-traumatic osteonecrosis
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What is that sign?
The **MR fluid sign** is highly suggestive of an osteoporotic vertebral compression fracture (VCF). 1. **MR signs suggestive of a benign, osteoporotic VCF** include: * band-like low T1 signal * fluid sign * retropulsion of a vertebral bone fragment. * Acute fractures show low signal on T1-weighted and high signal on T2-weighted and STIR images. **_2- MR signs suggestive of malignant VCF_** * complete replacement of normal marrow signal * convex posterior vertebral border * pedicle involvement * focal paraspinal mass * epidural mass.
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AAOS recommendations clinical practice guidelines for osteoporotic spinal compression fractures
1. "strong recommendation": was AGAINST the use of vertebroplasty. 2. "moderate recommendation: The use of calcitonin for acute injuries (presenting within 5 days of symptom onset) received a Calcitonin directly inhibits osteoclast activity by binding to surface cell-surface receptors. 3. limited evidence or were simply inconclusive: ibandronate or strontium use, bed rest and opiate analgesics, L2 nerve block for a L3 or L4 fracture, exercise program, electrical stimulation, kyphoplasty, use of a brace, improvement of kyphosis angle, any treatment in a patient with concomitant neurological deficits.
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Results of studies on vertebroplasty
Buchbinder et al. performed of randomized, double-blind, placebo-controlled trial. Patients were given a sham procedure to greaten the double blind effect. Outcomes were assessed at 1 week and at 1, 3, and 6 months. The primary outcome was overall pain (on a scale of 0 to 10, with 10 being the maximum imaginable pain) at 3 months. They found vertebroplasty did not result in a significant advantage in any measured outcome at any time point. There were significant reductions in overall pain in both study groups at each follow-up assessment.
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mortality rate of fragility fractures in DECREASING order?
Hip fracture \> vertebral compression fracture \> distal radius fracture