Stomach

Question 1

Cardia and antrum luined with?

G cells secrete? parietal cells? chief?

Answer 1

Question 2

Gastropathy and acute gastritis

gastritis is what process? reffered to as acute when? gastropathy when? what is it casues? 4

sympotms- common? 3 severe? 6

Path-gastric pH? protects gastric mucosa? pH? result of? physical barrier? replacement of these cells every?

what is an alkaline tide?

Answer 2

Question 3

Gastropathy/acute gastritis/chronic

path- disruption of what mechanisms? 4

N- effect what?

U/P- works how?

R- what? 2

DO- account for increased what?

chemicals?

Answer 3

Question 4

Question 5

Gastropathy/actue gastritis 3

Morph- lamina propria show? foveolar cell? charactersitic what? what lymphocye?

presence of what above basement membrane is abnormal?

in the lumen? acute erosive hemorrhagic gastritis?

clin- actue gastritis vs gastropathy? nsaid induced repsonds to? pain where? bile reflux in comparison?

Answer 5

Question 6

Stress-related mucosal disease

occurs in what patients?

stress ulcer- in what individuals?

curling? location?

Cushing? location? high incidence of?

Path-realted to? why? upreg of what? 2 increased what is protective?

Answer 6

Question 7

Stress-related mucosal disease

morph- acute ulcers shape? size? base color? why? number?

micro- transition? vs chronic peptic ulcers?

Answer 7

Question 8

Stress-related mucosal disease

clin- most of what pop? phrophylatic? clin outcome determined by?

non stress related- 2? casued by?

Answer 8

Question 9

Chronic gastritis

most common cause? other major cause?

symptoms compared to acute? whats uncommon?

Answer 9

Question 10

H. pylori gastritis

shape? present in what type of ulcers? what part of stomach often affected?

epidem- race? living? primary carrier? transmision route?

Path- often presents as what type of gastritis? with what abnormality? increased risk of what ulcer if in antrum?

in body and fundus- associated with what?

Answer 10

Question 11

H.pylori chronic gastritis

path- virulence due to? 4

what gene is often in gastric cancer risk?

cytokines? iron?

Answer 11

Question 12

H.pylori morph

location? tropism for what type of tissue?

most often found whre? lamina propria large numbers of what cell?

loing standing extend to where? atrophy associated with?

Answer 12

Question 13

H. Pylori

Clin- testing via? 3

biopsy analyzed by? 3

treatments?

Answer 13

Question 14

Autoimmune Gastritis (chronic gastritis)

location in stomach?

characterized by? 5 anti to? reduced? hyperplasia of? dificiency?

Path- loss of? responsible for? abseence stims? vit defic? leads to? reduced seum leads to chief cell death? damage compared to H.pylori?

type of cells against pareital? specific against?

Answer 14

Question 15

Answer 15

Question 16

Autoimmune Gastritis

morph- type of damage? where? thinned? RBC?

loss of what cells? hyperlasia demonstarated with proteins such as? can go to what cancer?

Answer 16

Question 17

Autoimmune gastritis

clin- gastric atrophy? anemia?

age? gender? associated with what diseases?

B12 can cause other than megaloblasts?

cured by replacement?

Answer 17

Question 18

Complication of chronic gastritis

PUD- what is it? mostly associated to?3

most common? lesser acid secretion in? why cant affect other areas?

aicd secreted by what can also cause? 2 esophagus? via?

epidem- raising or falling? but?

Answer 18

Question 19

PUD

path- results from imbalances between?

morph- most common whre? gastric where? grouped? size and depth? describe ulcer?

if heaped up? present in serosa? perforation seen via?

base of ulcer is? infiltrate? bleeding risk? malignant transformation?

Answer 19

Question 20

PUD- clin

recurrent? morbidity? symptoms? shitty causes? pain worse when? timing?

recurrence?

complications table

Answer 20

Question 21

Hypertrophic gastropathies characterized by?

Menetrier- associated with excessive? low?

symptoms?

pediatric vs adult? cancer in?

morph- enlargement of? where? spared? most characteristic?

tratment?

Answer 21

Question 22

Question 23

Zollinger-ELlison

caused by? secretes? found where? present with?

most remarkable feature anatomically? due to?

treatment? malignant?

sporadic? gene?

Answer 23

Question 24

INflammatory and hyperplastic polyps

common? age? size to resect?

morph- shape? surface? what is irregular dilated and elongated?

Answer 24

Question 25

Fundic Gland Polyps occur in what pops? 2 increasing due to? how? morph- occur where? shape? group? composed of? lined by?

Answer 25

Question 26

Gastric adenoma increaes with? age? dysplasia? occur on background of? adenocarcinoma risk related to? morph- grouped? location? type of cells? exhibit? high grade dysplasia?

Answer 26

Question 27

Gastric Adenocarcinoma common in stomach? 2 types? differ how? early symptoms? this results in? late symptoms? epidem- high incidence where? help in these areas? metastasis common? more common in wich groups? US rates?

Answer 27

Question 28

Gastric adenocarninoma cause in reduction due to? cancer where on the rise? due to?

Answer 28

Question 29

Gastric Adenocarcinoma path hereditary? familial gene? encodes for? also present in? so? diffuse higher chance with which mutation? seen in majority sporadic? intestinal type/sporadic- mutations in what? so loss of? other suppressor genes? 3 cytokines?

Answer 29

Question 30

Gastric adenocarcinoma morph- mainly where? intestinal- type of tumor? type of structures? penetrate where? diffuse- cells? cohesive? glands? large what? both have lots of what ?

Answer 30

Question 31

Gastric adenocarc clin- type in high risk areas? age? gender? diffuse- precursor lesions? gender? prognositc indicators? 5 year?

Answer 31

Question 32

Lymphoma most common location? type of cell? called? often what virus?

Answer 32

Question 33

Lymphoma path- arise at what sites? or? in stomach most often due to? so treatment? translocations 3 11-18 brings what? the other two? net effect for all 3? H.pylori does this through? differ in treatment? become more aggressive via?

Answer 33

Question 34

Lymphoma morph location? infiltrate of? lesions? follicles? B-cell markers? do not express? immunoglobulins? clin- symptoms

Answer 34

Question 35

Carcinoid tumor from components of? mainly found where? associated with? 4 can be caused by what therapy? morph- mucosa? color? hardness? in bowells? cytoplasm? markers?

Answer 35

Question 36

Carcinoid tumor clin- age? symptoms determiend by? if confined to intestine? so carcinoid sydrome associated to? prognostic factor? forgut? midgut? hindgut?

Answer 36

Question 37

Gastrointestinal Stromal Tumor type of tissue? named via? most common of stomach? arise from what cells? epidem- odd presentation? age? chldren realted to? triad? more GISt in what individuals?

Answer 37

Question 38

Gastrointestinal stromal tumor path- gain of fucntion? lesser gen emutation? mutation of what 2 detectable in what size? chromosomes?

Answer 38

Question 39

Gastrointestinal stromal tumor clin- blood? treatment? prognosis?

Answer 39