Stroke Flashcards

(98 cards)

1
Q

speech/language deficits =

A

left brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

spatial/ perceptual deficits =

A

right brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

quick/impulsive =

A

right brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

slow/cautious =

A

left brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

performance memory deficits =

A

right brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

language memory deficits =

A

left brain damage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

describe the double whammy of hemorrhagic stroke

A
  • tissue starved of oxygen/nutrients

- bleeding damages surrounding tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

prevalence of ischemic stroke

A

70-80%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

prevalence of hemorrhage stroke

A

20-30%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

thrombus/embolus are associated with

A

ischemic stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

3rd leading cause of death

A

stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

hemorrhagic stroke mortality

A

38%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

ischemic stroke mortality

A

12%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

1 cause of disability

A

Stroke

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

what percentage of stroke survivors require LTC

A

26%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

atherosclerosis

A

plaque formation with accumulated lipids, carbs, calcium, etc on arterial walls

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

arterial narrowing occurs commonly at

A
  • origin of the common carotid artery
  • transition from ICA> MCA
  • main bifurcation of MCA
  • junction of vertebral a w/ basilar a.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

hemorrhagic stroke

A

abnormal bleeding into extravascular areas of the brain from a ruptured cerebral vessel

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

hemorrhagic stroke leads to

A
  • increased ICP

- restricted distal blood flow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

which form of imaging better detects a stroke

A

MRI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

which form of imaging is more commonly used on suspected stroke pt. and why

A

CT b/c it is inexpensive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

ischemic stroke

A

partial or total blockage of vessels

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

how long is the recovery of surrounded area damaged by the ischemic stroke

A

3-4 weeks

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

thrombosis

A

thrombus (blood clot) from platelet adhesion and aggregation of plaques

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
cerebral embolism
traveling blood clot formed elsewhere that lodged in a cerebral artery
26
strokes can also be caused by
low systemic profusion pressure (hypotension)
27
most common location of ischemic strokes
MCA
28
MCA stroke occurs when
embolic blood clots from the heart or ICA > MCA
29
Right cortical damage =
spatial recognition problems
30
Left cortical damage =
language problems (expressive and receptive)
31
Left CVA (dominant)
- R. Hemiparesis - R. sensory loss - aphasia - dysarthria - difficulty read, write, calculate
32
Right CVA (non-dominant)
- L. Hemiparesis - L. sensory loss - spatial disorientation - L visual field deficit
33
R. CVA behavioral deficit
- spatial perceptual - quick/impulsive - overestimate ability, underestimate disability - emotion labile - disturbed body image, depth perception - difficulty in loud, cluttered environment
34
R. CVA perceptual deficit
- unilateral neglect - anosognosia - apraxia
35
anosognosia
lack of self awareness, unaware of disability
36
L. CVA behavior deficit
- processing info (language/reading comprehension) - easily discouraged - slow, cautious, anxious - depression is common
37
Left brain
- logic - language - numbers - analysis - reasoning
38
Right brain
- color - image - symbol - imagination - special visualization
39
ACA impairments
- contralateral sensory loss - hemiparesis of contralateral let/foot - cognitive impairment - imitation and bimanual task problem - slow, delay, lack of spontaneity - urinary incontinence - abulia - distractibility
40
abulia
inability to make decisions
41
damage to posteromedial aspect of superior frontal gyrus
urinary incontinence
42
damage to corpus callosum
problems with imitation and bimanual tasks
43
what are some ACA cognitive impairments
- preservation | - confusion
44
damage to primary motor cortex/ internal capsule
hemiparesis of contralateral leg/foot
45
damage to medial cortex, primary sensory
contralateral sensory loss
46
cerebellar ataxia
motor disorder when planning amplitude is to large
47
sensory ataxia
cant feel the limb - if they can see the limb they can better mange the movement
48
MCA impairments
- contra sensory loss - contra hemiparesis of face, arm, leg - homonymous hemianopsia - deviation of head/eyes to side of lesion - contra limb sensory ataxia - motor speech (expressive) - receptive aphasia - perceptual dysfunctions - loss of conjugate gaze to opposite side
49
damage to the optic radiation in internal capsule =
homonymous hemianopsia
50
homonymous hemianopsia
visual field loss on the same side of both eyes from damage of contralateral brain
51
damage to the parietal lobe =
sensory ataxia of contra limb
52
damage to broca's area
difficulty with motor (expressive) speech
53
damage to Wernicke's area
receptive aphasia
54
receptive aphasia
difficulty understanding written and spoken language
55
damage to the parietal sensory association cortex
perceptual dysfunctions
56
where do the ICA branch
at the base of the brain in the Circle of Willis
57
ICA stroke involves
lesions of both MCA & ACA
58
ICA impairments
- widespread deficits - massive edema - possible brain herniation, coma and death - incomplete lesions can produce mixed ACA and MCA signs
59
PCA impairments
- contra homonymous hemianopsia - contra sensory loss - thalamic syndrome - dominant hemi lesion (left) affect language/memory - non dominant hemi lesion (Right) = prosopagnosia - involuntary movements - visual symptoms (blur, focusing, graying) - hypothalamus (smell and emotion)
60
thalamic syndrome
pain (any sensory info can be perceived as pain)
61
prosopagnosia
inability to recognize familiar faces
62
thalamic sensory syndrome occurs from a lesion in the
PCA: VPL thalamus
63
thalamic sensory syndrome impairments
- sensation (touch, pain, temp) - transient/persistent sx (Numb early, hyperesthesia and pain later) - involves face arm, leg on one side - over dramatic pain from a touch stimulus - delayed onset - postural changes/depression
64
vertebrobasilar a. impairments
- vertigo - visual change - ataxia, diplopia - dysphagia, dysarthria - medial medullary syndrome - lateral medullary syndrome - basilar artery syndrome (locked in syndrome) - medial inferior pontine syndrome - lateral inferior pontine syndrome
65
cerebellar symptoms
- gait ataxia - dysarthria - nystagmus - head ache - amnesia - bilateral field deficits - motor/sensory loss in all 4 limbs
66
basal ganglia signs
- hypotonia - flaccid paralysis - impaired ambulation/gait - movement disorders
67
movement disorders associated with basal ganglia
- dyskinesia - hemiballism (subthalamic lesion) - flinging motion - bradykinesia - dystonia - chorea - tremors
68
brain stem symptoms
- vertigo - CN sx - ipsi motor and sensory CN signs w/ contra hemiplegia/hemianesthesia - bilateral hemiparesis - gait ataxia - dysarthria-clumsy hand syndrome
69
dysarthria- clumsy hand syndrome
slurring of speech plus clumsiness of hands
70
pontine lesion
- double vision, sensation of ear canal blockage - severe dysarthria/hx of vertigo or gait instability - horizontal nystagmus (Sustained)
71
midbrain lesion
cerebral peduncle = ipsi oculomotor paralysis w/ contra hemiplegia
72
Weber Syndrome
ipsi oculomotor paralysis w/ contra hemiplegia
73
lacunar stroke
- occlusion of small perforating a. - sx can be sudden and progressive - cortical symptoms
74
most common brainstem lesion
dysarthria clumsy hand syndrome PONS
75
80% of this stroke are "clinically silent"
lacunar stroke
76
lacunar stroke signs
- pure motor/ hemiparesis - ataxic hemiparesis - dysarthria/clumsy hand - pure sensory - mixed sensorimotor
77
what percentage of lacunar stroke have hyper- reflexia and Babinski
33-50%
78
ataxia hemiparesis
combo of cerebellar/motor sx - weakness - clumsy ipsi side of body (leg) - nystagmus
79
dysarthria/clumsy hand
pons lesion - unilateral lower face weakness - ipsi hemiparesis - arm ataxia - ipsi hyper-reflexia & positive babinski
80
CVA clinical impairments
- motor - somatosensory - visual - multi sensory integration - perceptual - cognitive/affect - language/ comprehension - speech/swallow - behavior change - balance/posture/ positioning
81
UE flexion synergy
- scap retract/elevate - shoulder flex/ABD/ER - elbow flex - forearm supinate - wrist/finger flex
82
LE flexion synergy
- hip flex/ABD/ER - knee flex - ankle DF/invert - toe DF
83
LE extension synergy
- hip ext/ADD/IR - knee ext - ankle PF/invert - toe PF
84
UE extension synergy
- scap protract/depress - shoulder ext/ADD/IR - elbow ext - forearm pronate - wrist/finger extension
85
Brunnstorm Stages 1
flaccidity
86
Brunnstorm Stage 2
synergies/ hypertonicity begin, some voluntary movement
87
Brunnstorm Stage 3
more voluntary control, more hypertonicity
88
Brunnstorm Stage 4
movement out of synergy as hypertonicity declines
89
Brunnstorm Stage 5
synergies lose dominance, more complicated isolated patterns are learned
90
Brunnstorm Stage 6
hypertonicity disappears & movement/ coordination is near normal
91
what is used as indicator of arm function following stroke
return of grip strength w/n 24 days
92
typical appearance of arm early post stroke
- arm hangs by side, IR, elbow extended, forearm pronated | - inf shoulder sublux
93
typical appearance of rib early post stroke
convex lateral curve on affected side
94
typical standing appearance early post stroke
- pelvis tilted down - hip and knee flex - weight shifted to strong side - PF
95
typical appearance early post stroke
WEAK, HYPOTONIC
96
arm appearance over time
- body in flexion | - one shoulder lower
97
standing appearance over time
leans on AD for balance
98
hypertonicity and mm imbalance over time can cause
- hip hiking, rotation toward affected side, hip flex/IR - knee extension, post pelvic tilt with hip ABD and knee flex - no heel strike, loss of hip extension (difficulty advancing limb) - fisting fingers and toes