Surgical bowel disorders Flashcards

(38 cards)

1
Q

Ischemic Bowel Disease

A

● Ischemic Bowel Disease (also known as Mesenteric Ischemia) occurs when blood supply to the intestine is decreased or cut off.
● It could be an acute problem or a
chronic problem (acute versus chronic
mesenteric ischemia).
● Because of the close association with
atherosclerotic disease, chronic is considered a condition of the elderly

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2
Q

Pathophysiology of Ischemic Bowel Disease

A

■ Embolism- Arterial blood clot becomes lodged.
■ Thrombosisatherosclerotic disease or
dissection or SMV clot
■ Low Flow- Insufficient blood pressure to
maintain adequate blood flow can occur with CHF, shock, or vasoconstrictive Rx (digoxin)
● Aka nonocclusive mesenteric ischemia

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3
Q

Ischemic Bowel Disease S&S (Chronic vs. Acute)

A

Chronic: presents similarly to “angina,”
where significant pain starts 10-30 minutes after eating food
Acute: comes on suddenly
Both:
○ Abdominal pain out of proportion to the physical exam findings in an elderly patient is classic.
○ Later in the process, involuntary guarding, rebound tenderness, and bloody stool may be positive.
○ A gangrenous bowel can rupture and result in hemodynamic shock

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4
Q

Ischemic Bowel Disease diagnosis:

A

○ Plain film X-rays may reveal dilated loops
of bowel and free air
○ CTA of the abdomen can rule out other
causes of severe abdominal pain, but may
also reveal the bowel distention and
intestinal edema of ischemic injury.
○ Mesenteric Angiography (arterial access) can provide very precise anatomic diagnosis
(and treatment) of vascular compromise.
(Gold Standard)

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5
Q

Gold standard testing for Ischemic Bowel Disease

A

Mesenteric Angiography (arterial access)

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6
Q

Ischemic Bowel Disease Management - ACUTE

A

○ Acute ischemia requires emergent Tx.
■ IV hydration is an important therapy.
■ Most patients require extensive bowel
resection of the diseased bowel.
■ Open laparotomy is preferred

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7
Q

Ischemic Bowel Disease Management - Chronic

A

○ Chronic ischemia with viable intestine may
be candidates for Angioplasty with
Revascularization. (consult vascular)
■ Anticoagulation is sometimes needed

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8
Q

Postoperative adhesions are the most common etiology of

A

Small Bowel Obstruction

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9
Q

Etiology of Small bowel obstruction

A

● Small bowel obstruction is relatively common and can occur
secondary to multiple processes.
● Postoperative adhesions are the most common etiology.
● The second most common etiology is incarceration and strangulation
of a hernia.
● Tumors, intussusception, and volvulus are less-common causes.

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10
Q

Common hernia locations:

A

● Umbilical
● Inguinal
● Ventral
● Incisional

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11
Q

Small Bowel Obstruction pathophysiology

A

■ Obstruction of intestinal motility, which leads to intestinal dilation and potential for perforation
■ Strangulation of intestinal tissue, which can lead to ischemic injury and the potential for necrosis and perforation

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12
Q

Small Bowel Obstruction S&S

A

○ Nausea and vomiting (60-80%)
○ Obstipation
○ Initial abdominal cramping followed by constant severe pain when necrosis is occurring.
○ Cramping abdominal pain often has a
“crescendo-decrescendo” pattern, classically.
○ Distention, diarrhea, inability to flatulate
○ High-pitched bowel sounds are common early
○ GU exam: check for hernias and rectal bleeding

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13
Q

Diagnosis of a Small Bowel Obstruction

A

○ Upright Chest/ Abdomen X-ray is the most
common 1st diagnostic study. (75% sens)
● Abdomen/Pelvis CT w/ IV and PO
contrast (if tolerated)

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14
Q

When is emergency surgery needed in a small bowel obstruction

A

Pneumoperitoneum is seen on CT

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15
Q

Small bowel obstruction Management-Nonsurgical (3Ns)

A

■ Change to NPO diet-bowel rest
■ IV hydration Normal Saline
■ NG tube for gastric decompression

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16
Q

Management-Surgical of small bowel obstructions - When do we do surgery?

A

■ Signs of ischemic bowel/sepsis
■ Possibility of perforation
■ No improvement after 3-7 days
○ Monitor labs daily
■ CBC
■ CMP
■ Glucose q6
○ Consider TPN (total parenteral nutrition)

17
Q

Causes of large bowel obstructions:

A

○ Tumors
○ Volvulus
○ Adhesions
○ Strictures
○ Foreign objects

18
Q

Volvulus

A

● Volvulus is the twisting or malrotation of the intestine, small intestine most common in children.
● This is a similar concept to testicular torsion, where the twisting may produce ischemia.
● Bowel obstruction also may occur.
● In adults, most commonly occurs in the sigmoid area of the bowel, followed by cecal region.
● Ischemic injury and bowel perforation are the two largest and significant concerns.

19
Q

S&S of a volvulus

A

-Majority present with acute presentation
○ Tachycardia, fever, and constant severe pain accompany ischemia (with concern for necrosis)

20
Q

Diagnosis of a volvulus

A

○ Abdominal X-ray is the initial diagnostic
study of choice.
■ Reveals intestinal distention
■ Often described as “double bubble”

21
Q

What is this?

A

Pneumoperitoneum - surgical emergency

22
Q

Volvulus diagnosis

A

○ Barium Enema-used if no evidence of
peritonitis and inconclusive x-ray
○ CT usually not needed unless suspect
cecal volvulus or pneumoperitoneum
not apparent on x-ray
○ Labs not conclusive, but can point to
complications:
■ CBC: check WBC
■ CMP: check electrolytes

23
Q

Management of a Volvulus

A

○ Regardless, definitive treatment is
surgery either elective or emergent
○ Emergent surgical intervention as an open laparotomy is usually
indicated, with reduction of the rotation and resection.

24
Q

Indications for emergent volvulus surgery

A

○ Cecal volvulus as endoscopic
decompression unlikely
○ Unsuccessful endoscopic sigmoid
decompression
○ Evidence of ischemia/peritonitis

25
Intussusception
The invagination of one segment of the bowel into a segment adjacent to it- Appropriately called “telescoping.” ● 95% of cases occur in young children, and the cause is not discernible ○ When it occurs in adults, the cause is almost always colon cancer
26
______ intussusception is most commonly seen in children ages 3 months to 3 years- condition of the infant
Idiopathic
27
Main concern with intussusception
Necrosis of the involved portion of bowel
28
Signs and Symptoms of Intussusception
○ Nearly all infants present with an acute onset of colicky abdominal pain with crying and possibly vomiting ○ A “sausage-like” abdominal mass may be palpated on examination . ○ The classic “Currant Jelly stools” are a characteristic finding in late disease
29
Diagnosis of intussusception
○ Barium/air enema can be diagnostic and therapeutic for infants. ○ CT is the Dx tool of choice in adults
30
Management of Intussusception
○ All patients with suspected intussusception should be hospitalized. ○ Air or barium enema is often times completely curative for infants ○ Surgery is usually required for children older than 3 and all adults. Can be open or laparoscopic
31
Toxic Megacolon
Toxic Megacolon is the pathologic distention of the colon with accompanying clinical signs of toxicity ○ Sometimes called Toxic Colitis +/- megacolon
32
The most common causes of toxic megacolon
○ Ulcerative Colitis ○ Crohn’s Disease ○ Acute Colitis (C diff)
33
Pathophysiology of toxic megacolon
○ The pathophysiologic mechanism of development is not fully understood, but seems to be related to intestinal immobility ○ Results in severe non-obstructive dilation of the colon- Could be a segment or the entire length of the colon.
34
S&S of Toxic Megacolon
○ The patient often appears “toxic,” almost as if septic. ○ Clinical presentation may include: ■ Fever ■ Prostration (lying down/weak) ■ Severe abdominal pain/distension ■ AMS ■ Tachycardia ○ Abdomen may be rigid with rebound tenderness.
35
Toxic Megacolon Diagnosis
○ Abdominal plain-film X-rays- Reveals significant colonic distention, often greater than 6 cm in the transverse colon. ○ Three of the following as well: ■ Fever > 38C (100.4F) ■ Tachycardia (usually >120 bpm) ■ Leukocytosis (usually > 10.5) ■ Anemia (due to bloody diarrhea) ○ PLUS one of these: Electrolyte abnl, AMS, hypotension, or dehydration
36
Treatment of toxic megacolon includes 3 main goals
○ Reduce colonic distention to prevent perforation ○ Correct fluid/electrolyte disturbances ○ Treat toxemia and underlying etiology
37
Management of toxic megacolon
○ NPO diet and NG tube for gastric decompression. (Bowel Rest) ○ Broad-spectrum IV antibiotics should be initiated. ○ Urgent decompression of the colon, usually by surgical resection, is usually necessary. ■ Open Laparotomy ■ May be entire colectomy
38
Ostomy care includes
○ Send with supplies and HH/wound care referral ○ Order PT ○ Possible reversal after 3-6 months ○ THIS IS A BIG DEAL FOR PATIENTS