Surgical Trauma And Emergency Flashcards

(47 cards)

1
Q

Site for needle insertion and chest drain in tension pneumothorax

A

Adult 5th intercoastal space in mid axillary line
Children 2nd intercoastal space mid clavicular line

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2
Q

Size of chest tube for tension pneumothorax or hemorrhoids

A

28-32Fr gauze

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3
Q

Burn fluid

A

For thermal
2ml/kg×surface area
For electrical
4 no/kg×surface area

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4
Q

Type of chest drain in traumatic pneumothorax

A

Under water seal

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5
Q

Beck’s triad is for what and components

A

Pulsus paradoxus, also known as paradoxical pulse, is a condition where a person’s systolic blood pressure, stroke volume, and pulse wave amplitude drop abnormally during inhalation. It’s a sign of heart or lung disease and is the opposite of what would normally happen, as blood pressure usually increases when someone inhales.

For cardiac tamponade
Hypotension
Raised JVP
Muffled heart sound
And
There will be a pulsus paradoxus

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6
Q

Boerhaave’s syndrome causes and sign symptoms

A

Complete rupture at lower thoracic esophagus
Hamman’s sign -
Crunching sound upon auscultation of heart due to pneumomediastinum
Chest pain
Shock
Subcutaneous emphysema

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7
Q

Mallory Weiss syndrome causes and sign symptoms

A

Incomplete tear only affecting muchos and submucosa
Tear on the gastric side of gastroesophageal junction which may extend to distal esophagus
Repeated vomiting then
Hematemesis

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8
Q

What to do if a patient of perforated peptic ulcers came with sepsis

A

Approach as ATLS protocol

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9
Q

DD for epigastric pain

A

Peptic ulcers
Perforated peptic ulcer
Pancreatitis
Cholecystitis
Myocardial infarction
diabetes may cause of DKA

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10
Q

Primary treatment of MI

A

Antiplatet
Thrombolysis
Angioplasty

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11
Q

Sign symptoms of pulmonary embolism and ECG changes and confirmatory investigation

A

Hemoptysis
Hypoxia
Small pleural effusion
ECG
S wave in lead 1
Q wave in lead 3
T inversion in lead 3
Investigation
CTPA

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12
Q

Collapsing signs of pulmonary embolism
And management

A

Chestpain
Hypotension
Tachycardia
Breathlessness
Desaturation
Mx
Patient is in peri arrest state so make thrombolysis with alteplase
Heparin administration upto achieving INR of 2-3. Then stop heparin and start warfarin 4-6 weeks with temporary risk factors
Oral anticoagulant only after confirmation of venous thrombosis

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13
Q

When to suspect fat embolism and
Management

A

After long bone fractures
Liposuction
Mx
Fat embolism syndrome (FES) is generally treated with supportive care in the hospital, often in the intensive care unit. Treatment focuses on maintaining intravascular volume and ensuring good arterial oxygenation. Some treatments include:
Oxygen
One of the fastest and easiest treatments for respiratory problems. You may be given oxygen or need help breathing with mechanical ventilation.
Intravenous fluids
Helps remove damaging free fatty acids from the body. Human albumin is recommended to restore blood volume and bind to fatty acids to reduce lung injuries.
Drugs
Your doctor may prescribe steroids or the blood thinner heparin, but these drugs have not been proven to be highly effective.
Surgical management
This includes early stabilization of long-bone fractures, rigid fixation within 24 hours, and appropriate surgical technique.

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14
Q

Massive pulmonary embolism Mx
Vs
Small embolism

A

British thoracic society guideline
Massive
3mg bolus of alteplase
Heparin
Thrombus fragmentation
IVC filter insertion
Small
Heparin if high probability of embolism before imagine like unfractionated heparin in massive pulmonary embolism where rapid reversal is needed. If not then LMWH cause it has the same efficacy and is easier to administer upto reaching INR 2-3
Oral anticoagulant should be used only after confirmation of embolism upto 4-6 weeks with temporary risk factors,3 months for idiopathic,6 months for any other reason

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15
Q

GCS components

A

Eye 4
Spontaneous
Response to verbal command
Response to pain
No eye opening
Verbal 5
Oriented(knows who and where he is)
Confused(answers,disoriented)
Inappropriate words(no conversation)
Incompetent sounds
No verbal response
Motor 6
Obeys command
Localizes response to pain
Withdrawal response to pain
Flexion to pain(decorticate-flex, int.Rotat)
Extremely to pain(decerebrate-ex,ext.rot)
No motor response
#below 8 intubate
*Inapplicable have
Macroglossia
Laryngeal edema
Puffy eyes
Hematoma eye

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16
Q

Organized hematoma in splenic injury

A

Don’t disrupt it

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17
Q

Infectious organism after splenectomy
And measures to be taken

A

Encapsulated
Streptococcus pneumoniae
Hemophylus influenzae
Neisseria meningitidis
Vaccine and penicillin
Hib
Pneumoniae
Meningitis

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18
Q

Blood film post splenectomy

A

Reticulocyte
Howell jolly bodies
Heinz bodies

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19
Q

Must common cause subarachnoid hemorrhage

A

Ruptured berry aneurysm
Looks like a berry in a stalk
Along the circle of Willis
Associated with autosomal dominant polycystic kidney disease

20
Q

Lethal triad

A

*Injury>exposure>hypothermia >coagulopathy>acidosis
*Injury>haemorrhage>hypoperfusion
>Acidosis>coagulopathy>hypothermia
*Injury>hemorrhage>coagulopathy
>Acidosis>hypothermia

21
Q

Stages of hypothermia

A

Mild- conscious, shivering(32-35°C)
Moderate-drowsy, not shivering(28-32)
Severe-uncooked, not shivering (20-28)
Profound-no vital signs (<20)

22
Q

Response stages of hypothermia
Response stages of hyperthermia

A

Hypo
Vasoconstriction(aw37:an35.5)>non shivering metabolic thermogenesis(aw36:an35)>shivering thermogenesis(aw35.5:an34)
*N B.aw=awake,an=anesthetized
Hyper
Sweating(aw37:an37.5)>vasodilation (aw37.5:an38)

23
Q

Rx of hypothermia
ECG changes

A

Rewarming
Mild- external
Serve -active core : peritoneal lavage, hemostasis, cardiac bypass
ECG
J wave
Arrhythmia

24
Q

Antidote of prilicaine

A

IV methylene blue

25
Antidote of lignocaine
Intralipid
26
Repeating technique of vessels and type of suture
Figure of 8 with 5-0 prolene
27
Rate of air change in laminar floor OT
300 times per hour
28
What to use facial reconstruction
Local flap Or Full thickness graft
29
Adverse effect of central line on lungs
May cause pneumothorax
30
Which coronary artery may be associated with abdominal aortic aneurysm and ECG finding
Right coronary artery So Inferior MI in ECG
31
Chest x-ray changes in ruptured thoracic aorta confirmatory investigation and Mx
Widening mediastinum Trachea and esophagus shifted to right Obliteration of space between aorta and pulmonary artery May be rib fractures and hemothorax CT, angiography, aortogram Tx is emergency endovascular repair
32
Dx and Tx of achalasia cardiac
Investigation Ph Manometry Barium swallow x-ray Endoscopy Tx Balloon dilatation Botulinum toxin Cardiomyotomy
33
If suspecting intracranial or intracerebral gardenia what to do
First surgery Then Antiplatet or anticoagulant
34
Beck's triad and cause
Elevated venous pressure Reduced arterial pressure Reduced heart sound And maybe associated with pulsus paradoxus Cause Cardiac tamponade even with 100 ml blood
35
Le Fort's classification of mid face fractures
1-no speak 2-no see 3-be hear
36
Criteria of immediate CT
Loss of consciousness for more than 5 minutes Amnesia for more than 5 minutes Abnormal drowsiness 3 or more episodes of discrete vomiting Clinical suspicious of non accidental inquiry Part traumas seizure nutty no history of epilepsy GCS<14 in adults GCS<15 in children below 1 year old or with bruise/swelling/laceration >5cm Sign of base of skull fractures -hemotympanum -CSF otorrhea or rhinorrhea -battle sign -halo sign Focal neurological deficit Fall from height>3 meter High speed injury like RTA
37
Tx of boerhaave's syndrome
Surgery of within 12 hours Beyond that controlled fistula
38
Antidote of opioid toxicity
Naloxone But risk of rebound pain *Rebound release of catecholamines may cause hypertension, tachycardia, and ventricular arrhythmias. Pulmonary edema has also been observed after naloxone administration. Small doses of naloxone can precipitate a withdrawal syndrome in subjects that are opioid-dependent.
39
Compartment syndrome
Compartment syndrome can occur in any muscle, but it's most common in the forearms and lower legs. Symptoms can be difficult to detect, and can become severe within a few hours of an acute injury. Some early signs of compartment syndrome include: Pain Pain that's more severe than expected from the injury, and that doesn't go away after taking pain medication or raising the affected area. The pain may feel like a deep ache or burning sensation, and it can get worse when you move the affected body part. Swelling The muscle may swell or bulge, or you may have difficulty moving the affected body part. Other sensations You may experience numbness, tingling, or a burning feeling under your skin (paresthesia). The skin may also appear pale. Other signs of compartment syndrome include: tightness, weakness, and pins and needles. Numbness and paralysis are later signs of compartment syndrome, and usually indicate permanent tissue injury. Compartment syndrome can be caused by fractures, severe contusions, crush injuries, or reperfusion injury after vascular injury and repair.
40
Virtual and disarthia suggest what type of vascular injury and if sudden deterioration of consciousness what may be main cause
Posterior circulation Basilar artery occlusion
41
Signs of occlusion of anterior circulation
Hemiparesis Hemisensory loss Homonymous hemianopia Higher cognitive dysfunction Dysphagia
42
Confirm CSF
Transferrin test Halo sign on gauze *Fluid containing CSF is classically described to make a “halo” or “double-ring” pattern on gauze or linen.
43
Allergic/anaphylactic shock Mx
Remove allergen ABC survey Adrenaline 1:1000 IM repeat every 5 minutes if no response Chlorpheniramine Hydrocortisone
44
Causes of Addisonian crisis
Sepsis or surgery causing acute exacerbation of chronic deficiency like Addison's or hypopituitarism Adrenal haemorrhage in Waterhouse Fredricksen syndrome Fulminant meningococcemia Steroid sudden withdrawal
45
Mx of Addisonian crisis
Hydrocortisone 100mg IM or IV Normal saline 1L over 30 to 60 minutes or dextrose if hypoglycemia Oral replacement after 24 hours with maintainance dose over 3-4 days
46
ECG in pulmonary embolism
s1 q3 t3 ,tall r in v1,t inversion in v123, peak p pulmonale in inferior leads,RBB, right axis deviation, right ventricular strain, atrial arrhythmia
47
Cushing triad
Hypertension—due to sympathetic activation after raised ICP Bradycardia Bradypnea