T2 Exam Flashcards

(35 cards)

1
Q

Corneal Ulcer Classification

A

Superficial/uncomplicated

Complicated
- Non-healing >7days
- Penetrating
- Perforating

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2
Q

Corneal ulcer diagnosis

A

History, CS, ocular exam, positive fluorescein stain

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3
Q

How to treat a superficial, small, non-complicated ulcer sustained <2days ago?

A
  1. ABs e.g. chloramphenicol, tricin
  2. Systemic analgesia e.g. phenylbutazone
  3. Atropine (once off)

revisit in 2-3days

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4
Q

Corneal ulceration surgical options?

A
  1. debridement of melting and stromal abscesses
  2. Grafting
  3. Temporary tarsorrhaphies
  4. Enucleation
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5
Q

Steps when presented with a pruritic horse. (6)

A
  1. Skin scraping (superficial & deep)
    - ectoparasites
    Positive: treat
    Negative: ascaricidal trial
  2. Fungal culture
    - detrmatophyte
    Positive: treat
    Negative: rule out
  3. Bacterial infection
    - cytology of papule fluid
    - AB trial
  4. Consider allergies
    - insect bite trial
  5. Adverse food reactions (food allergy)
    - elimination diet
  6. Atopic dermatitis
    - serological/intradermal allergy testing
    - response to therapy
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6
Q

Drugs for pain relief in a colic case

A

xylazine: 0.4-0.6mg/kg IV
(double dose for IM)

Flunixin 1.1mg/kg
- Only if we have a diagnosis otherwise masks pain so can’t cage prognosis

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7
Q

Fluid Therapy Calculation

A

Deficit= %dehydration x BW
Maintenance= 60ml/kg/day (adult)
= 100ml/kg/day (foal)
Ongoing losses= estimated NG reflux, Diarrhea frequency x volume

First half within first 3-6hrs (~5-10l/hr)
second half within 24hrs (1-2l/hr)

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8
Q

How much fluid would a 500kg horse require if its 10% dehydrated with high volume diarrhoea (1L every 2hrs) in the first 24hrs?

A

Deficit
= 0.1 x 500
= 50L

maintenance
= 0.06x500
= 30L

Deficit
= 1 x 12
= 12L
total = 92L

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9
Q

What are the radiographic signs of physeal dysplasia? (7)

A
  • Physeal widening
  • Irregular physis outline
  • Sclerosis
  • Bone lipping
  • Asymmetry of metaphysis
  • Wedging of epiphysis
  • Asymmetry of cortical bone
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10
Q

At what age must you correct angular limb deformities of the Fetlock and Carpus/Tarsus?

A

Fetlock = 30-60days (closes ~90days)
Carpus/Tarsus = 6weeks-4m (closes around 6m)

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11
Q

Pyrrolizidine Alkaloid Toxicosis pathogenesis.

A

PAs alkylate nucleic acids and protein prevents cell division and protein synthesis results in formation of MEGALOCYTES. Fibrous tissue replaces parenchyma when megalocytes die = liver fibrosis CHRONIC NODULAR LIVER HYPOPLASIA

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12
Q

PA toxicosis Histopathological findings (3)

A

megalocytosis, bridging portal fibrosis and biliary hyperplasia.

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13
Q

Common plants containing PAs

A
  • Paterson’s curse (Echium plantagineum)
  • Rattlepod (Crotalaria crispata)
  • Ragwort (Senecio spp) - not in AU
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14
Q

CS of hepatobiliary disease (6 common/6 uncommon)

A

Common = Icterus, weightloss, depression/lethargy, anorexia, colic, fever.

Less common = photosensitization, diarrhea, ventral oedema, ascites, encephalopathy, bleeding diathesis.

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15
Q

Liver enzymes for hepatocellular injury

A

AST, SDH, GLDH

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16
Q

Liver enzymes associated with Biliary injury/cholestasis

17
Q

Suspected causes of Neonatal Maladjustment Syndrome

A

Maternal disease e.g.
- anaemia, lung/cardiac disease, cardiovascular shock (toxemia)

Placental disease
- premature placental separation, placental insufficiency, placentitis

Stage II labour
- placenta previa (red bag delivery), uterine inertia, dystocia, oxytocin

18
Q

5 main differential for sick looking foals

A
  1. Sepsis
  2. Uroperitoneum
  3. Neonatal maladjustment syndrome
  4. Neonatal isoerythrolysis
  5. Prematurity
19
Q

Most important bacterial cause of sepsis in foals

20
Q

In what circumstance would a horse with cardiac disease be unsafe to ride?

A
  1. Diastolic murmurs
    • Aortic regurg until proven otherwise
    • Risk of ventricular ectopy
  2. Systolic murmurs
    • mitral valve regurg may lead to AF
  3. Bilateral murmurs
    • VSD unsafe in unrestrictive
  4. Uncommon/sudden-onset murmurs
21
Q

What are the types of perineal lacerations and their management?

A

First degree
- superficial mucosa and muscles of labia and vestibule
- local wound care e.g. AB’s, AI’s
- most will heal spontaneously

Second degree
- constrictor vulvae muscle & perineal body only
- debridement, superficial suturing, Caslick’s
- AB’s and AI’s

Third degree
- perineal body, anal sphincter and rectum
- delay corrective surgery for 6 weeks
- manual daily evacuation of vagina
- surgery = 2 stages
1. recreate shelf between rectum and
vagina
2. tension relieving sutures to restore
perineal body

22
Q

What are the 4 grades of rectal tears?

A

Grade 1: mucosa & submucosa
Grade 2: mucosa, submucosa & muscular
layer prolapse
Grade 3: all layer except serosa (subtype
a) or mesentery (subtype b)
Grade 4: full thickness tear

23
Q

Management of type 1 & 2 rectal tears?

A

CAREFULLY evacuate rectum and pack defect
Conservative management
- BS AB’s e.g. pent & gent
- tetanus toxoid
- flunixin
- supportive care e.g. IVFT
- Fast + stool softeners

24
Q

Management of type 3 & 4 rectal tears?

A

REFER to surgical facility
Surgical management
- in-dwelling rectal liner
- suture per rectum
- colostomy
- laparoscopic repair
- euthanasia if extensive contamination

25
IgG levels in foals and what levels indicate successful passive transfer vs partial vs failure?
>8g/L = PROTECTED 4-8g/L = PARTIAL PROTECTION (protected if well and in a clean environment <4g/L = FAILURE of passive transfer
26
When should you check passive transfer levels in foals?
<24hrs of age maximum absorption <6hrs
27
What are the complications associated with castration?
1. Hemorrhage 2. Oedema/swelling 3. Infection (can lead to scirrhous cord) 4. Herniation/Evisceration 5. Hydrocoele 6. Persistent masculine behaviour
28
What is the drug of choice to treat AF in horses?
Quinidine Sulphate
29
What are the consequences of Quinidine Sulphate toxicity?
Non-cardiac - colic/diarrhoea/inappetence - stertor/nasal oedema - weakness/ataxia Cardiac - SVT - Hypotension - VT
30
How do colic signs appear in a horse vs a foal?
Horse: - recumbency - stretching - bruxism - flehmen response - arching of neck - flank watching/biting - pawing -kicking at belly - rolling - posturing to lay down - sweating - posturing to urinate Foal: - Lie on their back - bruxism
31
How to diagnose foal meconium impaction?
Black to dark brown, pasty - firm faeces Passage should be completed by 48hrs Diagnostic test: - digital rectal palpation - US/RADS for a proximal compaction
32
How to diagnose foal meconium impaction?
Black to dark brown, pasty - firm faeces Passage should be completed by 48hrs Diagnostic test: - digital rectal palpation - US/RADS for a proximal compaction
33
Management of a meconium impaction
Supportive care - IVFT or oral fluids if CS consistent with dehydration. - Analgesia e.g. flunixin or meloxicam - Enema e.g. soapy water then acetylcysteine
34
How to administer an enema in a foal
- warm soapy water - foal in lateral recumbency - lubricated foley catheter - 500ml-1L for 50kg foal - administered by gravity flow - max twice!
35
What to do if fluid enema fails in a foal with a meconium impaction?
Acetylcysteine enema - decreases viscosity of meconium - kept in place for 30-45mins with foal sedated in lateral recumbency. - excellent prognosis