Tachycardia Flashcards

(52 cards)

1
Q

people that present with tachycardia and heart palpitations should get what test

A

12 lead EKG

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2
Q

what is narrow complex tachycardia

A
  1. sinus tachycardia
  2. AV nodal re-entrant tachycardia
  3. AV reciprocating tachyc ardia orthodromic
  4. multifocal atrial tachy
  5. atrial fibrillation
  6. atrial flutter
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3
Q

what is wide complex tachycardia

A
  1. AVRT - antidromic - WPW
  2. ventricular tachycardia
  3. ventricular fibrillation
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4
Q

etiologies of sinus tachy

A
  • exercise
  • ansxiety
  • pain
  • exposure to stimulants (caffeine)
  • volume depletion (dehydration/sepsis)
  • anemia
  • hypoxia
  • hyperthyroidism
  • pulm embolism
  • pericarditis
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5
Q

symptoms of sinus tachy

A

asymptomatic
heart palpitations
SOB (esp with exertion)

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6
Q

symptoms of sinus tachy in pts with heart dz

A
heart palpitations 
SOB
chest discomfort
lightheadedness
fatigue
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7
Q

treatment of sinus tachycardia

A

treat underlying cause:

  1. dehydration - IV fluid
  2. pain - pain meds
  3. pulm embolism - anticoagulants
  4. sepsis - treat source
  5. anxiety - consider anxiolytics

**No specific treatment for the tachycardia itself

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8
Q

what is supraventricular tachycardia

A
  • regular, rapid rhythm
  • narrow complex (originates above ventricles)
  • no discernible p waves
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9
Q

examples of supraventricular tachycardia

A

atrioventricular nodal reentry tachycardia
orthodromic AV reciprocating tachycardia
junctional tachycardia

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10
Q

symptoms of SVT

A
  • sudden onset racing heart palpitations
  • lightheadedness, pre syncope, syncope
  • SOB
  • anxiety
  • if underlying heart dz: chest pain
  • often self limiting, ends abruptly as well

begins suddenly and ends suddenly

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11
Q

management of persistent SVT stable pts:

A
  • vagal maneuvers
  • carotid massage (listen for bruit, risk of stroke esp >50yo)
  • adenosine (initial dose 6mg IVP, then 12mg IVP, then 12mg IVP)
  • CCB or BB
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12
Q

tx for pts with frequent attacks of SVT

A

consult EP –> confirm aberrant pathway –> radiofrequency catheter ablation

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13
Q

management of persistent SVT unstable pts:

A

vagal maneuvers then if unsuccessful immediate DC cardioconversion

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14
Q

what is DC & how does it work

A

direct cardioconversion

  • medical procedure which converts cardiac arrhythmias to normal sinus rhythm using electricity
  • two electrode pads are placed on the pt (chest and back)
  • electrode pads are connected to a machine via cables
  • the cardioverter delivers a shock which causes momentary depolarization of most cardiac cells allowing the sinus node to resume normal pacemaker activity
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15
Q

what is (AVNRT)

  • which gender more predominant
  • triggered by?
  • HR
A

atrioventricular nodal reentrant tachycardia

  • most common cause of SVT
  • 75% female
  • reentrant circuit around the AV node
  • healthy hearts and sick hearts
  • triggered by exertion, caffeine, alcohol
  • HR 140-280 regular
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16
Q

what is AVRT

A

atriventricular reciprocating tacycardia

  • accessory pathway
  • healthy and sick hearts
  • wide or narrow QRS complexes
  • seen in WPW
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17
Q

tx of stable pts with narrow complex (orthodromic) AVRT

A
  • vagal maneuvers
  • adenosine
  • CCB or BB
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18
Q

if orthodromic AVRT is associated with afib/flutter how would you treat?

A

avoid nodal blocking meds, use class Ia, Ic II antiarrhythmics - procainamide

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19
Q

how would you treat orthodromic AVRT in unstable pts

A

immediate cardioversion

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20
Q

orthodromic AVRT

antidromic AVRT

A
  • orthodromic = narrow complex

- antidromic = wide complex

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21
Q

antidromic AVRT is difficult to distinguish from what?

A

ventricular tachycardia

22
Q

immediate tx of antidromic AVRT: unstable vs stable if you know it’s a preexcitation syndrome

A

immediate DC in unstable pts

procainamide in stalbe pts

23
Q

subsequent tx of antidromic AVRT

A

catheter radiofrequncy ablation of accessory pathway

24
Q

what is WPW

A

-pre-excitation syndrome involving an accessory pathway

25
how many pts are symptomatic in WPW
25-50%
26
range of symptoms in WPW
palpitations to syncope
27
most common cause of WPW on EKG
paroxysmal supraventricular tachycardia which is usually AVRT
28
distribution of people affected by WPW
bi modal - early childhood - early adulthood
29
1/3 of pts with WPW also have
afib - potentially serious arrhythmia in WPW pts - afib may result in rapid ventricular response - subsequent degeneration to ventricular fib = sudden death
30
management of WPW with normal rate and stable
- catheter ablation of accessory pathway (standard of care) | - medical therapy if unable (BB, adenosine, antiarrhythmics - procainamide, amiodarone)
31
management of WPW with fast rate
same as other forms of AVRT - unstable: DC - stable: procainamide - subsequent: catheter radiofrequency ablation
32
what is multifocal atrial tachycardia | -therapy?
``` >=3 p wave morphologies 100-150bpm -therapy involves tx of underlying medical problems (typically pulm dz) -verapamil -flecainide or propafenone ```
33
what is ventricular tachycardia (sustained) - associated with what? - rate? - complication of what conditions?
-fast wide complex rhythm -often associated with structural heart dz -frequently associated with syncope >= consecutive ventricular premature beats -usual rate 160-240bpm -moderately regular -frequent complication of MI and dilated cardiomyopathy
34
symptoms of ventricular tachycardia
- asymptomatic - heart palpitation - lightheadedness - chest pain - SOB - diaphoresis - near syncope - syncope - sustained LOC - pulseless electrical activity (death)
35
tx of acute ventricular tachycardia - pulse present
pulse present: - if vtach causes hypotension, heart failure, myocardial ischemia = DC - if pt is stable = amiodarone 150mg IV bolus followed by continuous infusion - implantable cardioverter-defibrillator
36
tx of acute ventricular tachycardia - pulseless
- CPR - DC - epinephrine
37
what is NSVT
3+ consecutive ventricular beats - a rate of >120 bpm - duration of less than 30s
38
NSVT - symptoms - how is it found? - prognosis in healthy vs structural heart dz - potential marker for what?
- common - asymptomatic - dx on routine EKG or exercise stress test - usually benign - potential marker for development of sustained ventricular arrhythmias and sudden death - in presence of structural heart dz, more serious pronogisis
39
goals of tx for NSVT
- prevent sustained arrhythmia and sudden death | - eliminate symptoms
40
NSVT and associated diseases
- hypertrophic cardiomyopathy - 25% - idiopathic dilated cardiomyopathy - 80% - valvular heart dz - 20% of pts with MVP, MR, AS - chorinc coronary heart dz - left ventricular dysfunction
41
management of NSVT | -frequent vs infrequent
1. if infrequent, no specific intervention except: - optimize electrolytes, BB, manage underlying conditions/risk modification 2. frequent: consider amiodarone
42
what is torsades de pointes
a type of vtach
43
what is torsades de pointes triggered by
hypokalemia, hypomagnesemia, drugs that prolong QTc
44
meds that prolong QTc - antiarrhythmic agents
antiarrhythmic agents: amiodarone flecainide sotalol
45
meds that prolong QTc - antipsychotics
``` chlorpromazine haloperidol olanzapine quetipine risperidone ```
46
meds that prolong QTc - antibiotics
macrolides (azithro) | quinolones (levo/cipro)
47
meds that prolong QTc - antidepressants
citalopram | tricyclic antidepressants
48
tx of torsades de pointe
unstable - prompt defibrillation | stable - IV magnesium (1st line); temp transvenous overdrive pacing if no response to magnesium
49
ventricular fibrillation
often associated with severe CAD and caused by acute MI (ACS) - sudden death may be initial manifestation of coronary dz in 20% of pts - pts are pulseless and unresponsive
50
causes of ventricular fibrillation
- myocardial ischemia and infarction - heart failure - hypoxemia or hypercapnia - hypotension/shock - electrolyte imbalances - stimulants (drugs, caffeine) - preceded by Vtach
51
associated conditions with V fib
- LVH - HOCM (hypertrophic obstructive cadriomyopathy) - CHF - AS - brugada syndrome
52
tx of v fib
- cpr - defibrillation - if pulse regained = coronary arteriogrphy , (cardiac cath) to view and tx AAD - implantalbe cardioverter-defibrillator