Tendinopathies Flashcards

1
Q

What is a tendinopathy

A

it is a failed healing response of the tendon, associated with overuse in and around tendons

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2
Q

Definitions: tendinopathy, tendonitis, tendinosis

A
  1. General term that describes tendon degeneration
  2. refers to a painful tendon with histological signs of inflammation within the tenon
  3. is a localised intrinsic degeneration of unknown aetiology, usually characterised by localised swollen tendon nodes
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3
Q

Load types of tendinopathy: Compressive

A

A tendon can be squashed against a bone (e.g. proximal hamstring tendon against ischial tuberosity)

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4
Q

Load types of tendinopathy: shear and friction

A

movement of the tendon sheath on the tendon can cause sheath irritation, e.g. AT sheath moves with repeated DF/PF

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5
Q

Load types of tendinopathy: tensile

A

store and release energy like a a spring for power

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6
Q

Load types of tendinopathy: combination

A

A combination of compressive, tensile and shear/friction.
e.g. hockey platers running in hip flex have a combination of loads on their proximal hams tendon

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7
Q

first stage of tendinopathy continuum: Reactive tendinopathy

A

Non-inflammatory proliferative response in cell matrix. Due to compressive or tensile overload.
Cell changes shape-> more cytoplasmic organelles=increase protein function,
collagen integrity maintained, short-term adaptations- thickens the tendon to reduce stress and increase stiffness, the potential to revert back to normal tendon

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8
Q

second stage of tendinopathy continuum- Tendon disrepair

A

Chronic overload of RT.
Increased protein production- separation of collagen and disorganisation within cell matrix
great breakdown physiologically
Visible on MRI & US
May be increase vascularity and neural ingrowth in tendon
Reversibility possible with correct load and management

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9
Q

third stage of tendinopathy continuum- Degenerative tendinopathy

A

Areas of cell death due to apoptosis, trauma, tenocyte exhaustion
Large areas of matrix-disordered and vessels, matrix breakdown and little collagen
little capacity for reversibility
focal swelling and pain
high risk of rupture
clinically this is present in older individuals with ongoing problems with tendinopathy or younger individuals who have continued to overload

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10
Q

fourth stage of tendinopathy continuum- reactive on degnerative

A

hybrid of reactive and degenerative pathology
Structually (normal) part of the tend may drift in and out reactive phase
degenerative portion: unable to transmit tensile load
manage as a reactive tendon

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11
Q

what does the tendinopathy continuum mean clinically

A

Interventions should be tailored to the stage of pathology
exercise and load management fundamental to management
if correct stage not identified= symptoms may get worse

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12
Q

clinical presentations of tendinopathies: reactive

A

rapid onset, load substantially exceed previous exposure, easily aggravated by exercise and slow to settle
Painful and less common

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13
Q

clinical presentations of tendinopathies: reactive on degenerative

A

old adult (50-60)
past history of load related excaerbation
onset after overload
variable swelling
less irritable
painful and common

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14
Q

clinical presentations of tendinopathies: degenerative

A

older (30-60)
long history of minimal symptoms
variable swelling and lumps/bumps
unloading strategies
Not painful

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15
Q

concepts to consider with tendinoapathies

A

tendon don’t like compression or too much stretch-shortening cycles
tendons are highly responsive to mechanical loading
chronic, habitual tendon loading needed for adaptations
Effective training program should apply high loading intensity over long duration (>12/52)

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16
Q

Treatment: education

A

pain doesnt= harm
restore the balance between load and tissue capacity
important for compliance
kinesiophobia
manage expectations
slow to settle but quick to aggravate

17
Q

Patient advise- RC tendinoapthy

A

avoid excessive, unaccustomed activity above shoulder height, take regular breaks from repetitive shoulder movement, pillow under arm and behind patient to prevent them rolling on that side at night

18
Q

Patient advise- Tennis elbow

A

when gripping/lifting try and keep palm facing upwards
modify movement/activities that bring on pain

19
Q

Patient advise- hamstring

A

avoid excessive hip flexion
avoid repeated stretching of hamstring

20
Q

Patient advise- gluteal

A

avoid sitting with legs crossed, avoid standing with weight on one leg, avoid laying on it at night
sleep position advise- duvet folded over under patient, pillow between leg

21
Q

Patient advise- patella

A

avoid excessive running or jumping movements intially, initially reduce load on the knee in flexed position (squat)

22
Q

Patient advise- achilles

A

In insertional: avoid excessive DF to prevent compressive load
Avoid plyometric movements to reduce SSC/Tensile load, but continue running at a tolerable level
Put more weight through heel so not using PF
Orthotics- limited benefit however AFO can be useful in longitudinal tears in early phases of rehab

23
Q

pain management

A

Reduced motor responses, more severe in reactive phase, manage the load on the tendon- avoid compressive load i.e. stretching, cut out activities involving SSC
anti-inflammatory to regulate tenocyte activity
isometric exercise

24
Q

treatment: isometrics

A

5X45 sec hold with 1 min rest
significant reduction in acute pain 45 mins as compared with isotonic
well tolerated- can be tailored to patients preferred ROM
Useful for reactive tendons

25
Q

treatment:Alfredson protocol

A

3X15 reps, unilateral, 3 sec contraction
2 min rest between sets
5 min rest between exercises
2 exercises: heel drops of step with straight and bent knee
twice daily
12 weeks
limitations: lots of reps, very time consuming, often poor compliance

26
Q

treatment: Heavy slow resistence

A
  • 3 x a week
  • 3 second concentric and eccentric (6s total)
    contraction
  • 2-3 min rest between sets
  • RPE of 8 on last 2 reps
    Protocol:
  • Week 1: 3 x 15 RM
  • Week 2-3: 3 x 12 RM
  • Week 4-5: 4 x 10 RM
  • Week 6-8: 4 x 8 RM
  • Week 9-12: 4 x 6 RM
    3 exercises:
  • Resisted seated heel raise
  • Straight leg heel raise
  • Straight leg heel raise in standing with barbell and
    forefoot on weight
27
Q

Treatment: monitoring load and progression

A

symptoms led number of reps, >12 reps= add external load, education RE normal pain, pain monitoring model by Silbernagel

28
Q

Sedentary people

A

 Heavy external loads may be less appropriate
 Greater structural changes – correlated to
functional ability
 Tend to respond faster
 If progressing to HRT – rest days should be
incorporated