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Test 1 Flashcards

1
Q

5 factors that impact probing depth

A
  1. inflammation
  2. probe diameter
  3. Tapered vs parallel
  4. Hand force
  5. Band width
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2
Q

Furcation probe measures what type of bone loss?

A

Horizontal

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3
Q

What is the average width of the PDL in an adult?

A

0.17mm

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4
Q

Amount of movement seen in class I mobility?

A

0.2 - 1 mm

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5
Q

Amount of movement seen in class II mobility?

A

greater than 1 mm

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6
Q

Amount of movement seen in class III mobility?

A

greater than 1 mm plus axial displacement

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7
Q

How do you calculate CAL?

A

PD + (distance between GM -> CEJ)

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8
Q 
GM = 2 mm coronal to CEJ
PD = 5
CAL = ?
A

CAL = 3mm

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9
Q 
GM = 4 mm apical to CEJ
PD = 2
CAL = ?
A

CAL = 6mm

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10
Q

What is the biological width made up of?

A

Junctional epithelium + CT attachment = 2mm

Bottom of sulcus to crest of alveolar bone

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11
Q

What 3 things might happen if violation of the biological width occurs?

A
  1. Inflammation
  2. Increased probing depth
  3. Inconsistent resorption of alveolar bone
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12
Q

Steroids act on what in the inflammation cascade?

A

Phospholipases -> Arachidonic acid

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13
Q

NSAIDS/Asprin inhibit what in the inflammation cascade?

A

Cycooxgenase

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14
Q

Singulair inhibits what in the inflammation cascade?

A

5-Lipoxygenase

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15
Q

Cytoplasmic granules of basophils/mast cells contain what 5 things?

A
  1. Histamine
  2. Platelet activating factor
  3. Heparin
  4. TNF alpha
  5. SRS-As (Leukotriene C4, D4, E4)
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16
Q

Monocytes/macrophages synthesize and release which cytokines/lymphokines?

A
  1. IL-1 (osteoclast activation, B-lymph/CD8 activation)
  2. IL-6 (Osteoclast activation)
  3. TNF-alpha (activator of endothelium/inflam mediator)
  4. INF (interferes with virus replication)
  5. Lipid mediators of inflammation: prostaglandins, leukotrienes, and plateley activating factors (PAF)
17
Q

What is the key to the immune response and activates macrophages?

A

CD4 lymphocytes

18
Q

What is the general name for soluble, locally active polypeptides that regulate cell growth, differentiation and or function?

19
Q

What does IL-1 do?

A

pro inflammatory, stimulates osteoclasts, fibroblasts, and macrophages

20
Q

What does IL-6 do?

A

Pro inflammatory, simulates B and t cells

21
Q

What does IL-8 do?

A

Pro inflammatory, attracts and activates PMNs

22
Q

what does TNF alpha do?

A

Pro inflammatory, activates osteoclasts

23
Q

What does PGE2 do?

A

Vasodilation, pyrogenic, release mediator from mast cells, cell mediated cytotoxicity

24
Q

What is the average rate of CAL in patients with untreated periodontitis?

A

0.1 - 0.3 mm F/L per year
0.3 mm interproximal per year
(Will lose 3.5 times more teeth than pts who receive treatment)

25
How long after plaque formation does it take for a T cell lesion to form and clinical signs of gingivitis?
End of the 1st week
26
What type of pocket is seen with horizontal bone loss?
Suprabony pocket
27
What type of pocket is seen with vertical bone loss?
intrabony pocket
28
Why type of intrabony pocket has the best regenerative capacity?
3 wall
29
What is the most common bony defect involving posterior teeth?
Interdental crater (usually mandibulars)
30
3 examples of drugs that cause gingival overgrowth
1. Phenytoin sodium - Dilantin 2. Cyclosporine - Sandimmune 3. Ca+ channel blockers - Nifedipine
31
Normal distance between CEJ and bone crest?
1.5-2 mm
32
What bacteria increases as levels of progesterone increase?
Prevotella intermedia (uses progesterone as a substitute for Menadione)
33
what 2 things are P. intermedia associated with?
1. gingival inflammation | 2. pyogenic granuloma (pregnancy tumor = hyperplastic gingival mass) formation
34
What is the most common etiologic agent of infectious endocarditis?
alpha hemolytic strept
35
3 types of patients that need a premed
1. artificial heart valves 2. history of infective endocarditis 3. congenital heart conditions (unrepaired defect, residual defect after surgery, within 6 months of valve surgery, cardiac transplant)
36
5 acute periodontal diseases
1. acute pericoronitis 2. acute herpetic gingivostomatitis 3. ANUG 4. acute periodontal abscess 5. acute gingival abscess
37
4 common symptoms of acute perio disease
1. pain 2. swelling/edema 3. lymphadenopathy 4. fever
38
What 2 bacteria produce proteinases that cause perio abscesses?
P. gingivalis and P. intermedia

Perio (3 decks)

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