Test 2 week 6-9 Flashcards
What do you assess when looking at A-Airway
- is airway patent
- secretions
- mucous & vomit
- swelling
- tongue (LOC)
What are interventions to maintain Airway
- open airway (head tilt chin lift & jaw thrust)
- suction
- oropharyngeal airway
- nasopharygeal airway
- HOB flat, no pillows
What do you assess when looking at breathing
- respiratory rate, depth & rhythm
- accessory muscle use
- quick listen
- SpO2
What are interventions for breathing
- position (high fowlers/semifowlers, tripod)
- Coached breathing - PEEP
- Oxygen
- Bronchodialtors
- Bipap
- intubation
- ventilation - ambu bag
What do assess when looking at circulation
- skin colour, temp, turgor
- capilary refill
- pulse
- blood pressure
What are interventions for circulation
- fluids (maybe)
- Drugs - Epi (increase HR), norepi (constrict BV), Inotropic/anti inotropic, antiarrythmias
- 12-lead ECG
- IV access
- cardioversion
*cardiac monitor
What do you assess when looking at disability
LOC
AVPU
GCS
Antiarrythmic drugs
REMINDER
Amniodarone - tachy ventricular rhythm
Lidocaine - tachy ventricular
Adenosine - tachy atrial
Atropine - increase HR (increase symp)
BB, CA channel - decrease rate
What is COPD
And the two diseases seen
Chronic obstructive pulmonary disease
* is a respiraotry disorder largley caused by smoking and is characterized by progressive, paritally reversible airway obsturction and lung hyperventilation, systemic manifestations and increasing frequency and severeity of exacerbations
* Chronic bronchitis - chronic inflmmation of lower respriatry tract - excessive mucus secretion, cough & dyspnea
* Pulmonary emphysema - destruction of alveoli, enlargment of distal airways & breakdown of alveolar walls
What is the etiology of emphysema
Smoking & inherited alpha1-antitrypsin (anti-protease) deficiency
What is chronic bronchitis
- large and small airway obstruction associtaed with chronic irritation from smoking & recurrent infection
- hx of productive cough for at least 3 consecutive months in at least 2 consecutive years
- acute exacerbations with pruluent sputum, increase in SOB, fatigue, chest congestion, fever/chills
What are clinical manifestations of COPD
& early, middle, late stages
Primary :
* cough
* sputum production
* dyspnea on exertion
Additional
* weight loss - d/t increase WOB, and interference with eating
* Chest pain - hyperinflation, loss of lung elasticity & therefore recoil
* prolonged expiratory wheeze, crackles
* Tripod positon - accessory msucle use
* Pursed lip breathing - helps prevent airway collaps
* hypoxemia, hypercapnia, cyanosis, polycythemia
* right sided heart failure form pulmonary vasocontriciton & increased PAP
Early - fatigue, exercise intolerance, cough, sputum, SOB
Middle - progressively more dyspnea with frequent infections
Late - chronic respiroatry failure, death usually 2nd to exacerbation by infection
How can you improve ventilation in COPD
**Diaphragmatic Breathing **- abdominal breathing, focuses on using diaphram instead of accessory muscle to achieve max inhalation and slow respiratory rate (tense abdomen on exhalation)
Pursed-lip breathing - prolong expiration, prevent brochiolar collapse, assist with dyspnea, - allows for effective coughing, reduce fatigue
**Postural Drainage **- promotion of airway clearance- percussion and vibration are used after the client assumes a postural drainage positon to assit in loosening the mobilized secretions
What can occur when giving someone with COPD too much oxygen
Normally, the accumulation of CO2 is a stimulate of the resp. system. however in ppl with COPD who have diminished ability to exhale properly, can have chornically higher levels of CO2 and they develop a tolerance. For these people the drive to breathe is hypoxemia - thus admisntering oxygen to patients with COPD can weaken their drive to breathe - BUT need to maintain O2 saturation
What are S/S of pneumonia in older adult
Dyspnea, chills, altered mental status (lethargy, confusion, stupor), tachypnea, hypotension, hypo/hyperthermia
What is Pneumonia
Pneumonia is an acute inflmmation of the lung parenchyma by a microbial agent - can have community-acquired or hospital-aquired pneumonia, aspiraiton pnuemonia, or opportunisitc & fungal
Symptoms of pneumonia & treatment
Sudden onset of fever, chills, cough producing purulent sputum, pleuritic chest pain, crackles
Treatment
* antibiotic treatment
* oxygen
* antipyretics (ASA, asprin, Tylenol)
* maybe analgesics
* proper nutrition
What are symptoms and characteristics of pleural pain
- Abrupt onset
- unilateral, localized to lower and lateral part of chest - possibly referred to shoulder
- usually worsened with chest movement
- tidal volume is small
- breathing is rapid
- reflex splinting of the chest may occur
What is a pleural effusion
- abnormal collection of fluid in the pleural cavity - formation exceeds rate of removal
- accumulation of fluid comes from lung, paritetal pleura, peritoneal cavity or decreased removal by lymphatics
- fluid can be exudate, purulent, chyle, sanguinous
What are clinical manifestations of pleural effusion
- DEPENDENT ON CAUSE
- fever, increase WBC (if infectious)
- fluid decreases lung expansion on affected side - decreased movement of chest wall
- pleuritc pain
- hypoxemia, dyspnea
- dullness on percussion
- absent or decreased breath sounds
- Empyema (pocket of pus) - fever, night sweats, wt loss, cough
What is the Dx, Tx, & collab care for pleural effusion
DX & TX
* CXR, US, CT
* thoracentesis
* chest tube drainage
* rapid removal of fluid can cause hypotension, hypoxemia, pulmonary edema
Collab Care
* treat undelrying cause
* pleurodesis - prevent reaccumulation of fluid 2nd to sclerosing pleural space
* chest tubes
* antibiotics
* suppoortive - analgesia, O2, IV, antipyretic (if needed)
What are the 4 types of pneumothorax
Primary Sponatenous- usually in taller young men, 10-30 year old, ruptured bleb
Secondary spontaneous- underlying lung disease, more serious
Traumatic- penetrating or non-penetrating, may be accompanied by hemothorax
Tension - air can enter but can’t leave, area on visceral or parietal pleura as a one - way valve, life threatening, medisatinal shift, compression of great vessels (vena cava & aorta), heart and both lungs
Clinical manifestations of pneumothorax
Tachycarida, dyspnea, resp distress, chest pain, decreased air entry
Tension - severe distress, tachycardia , hypotension, tracheal shift, mediastinal shift
What is a hemothorax
Pleural effusion of blood in pleural cavity 2nd to chest injury, surgery, malignnacy, rupture of a big vessel
can have minimal (300-500cc usually reabsorbs),
moderate (500-1000cc - signs of lung compression & loss of intravascular volume, drainage & fluid replacement, maybe surgery,
large ( 1000cc +, bleeding from intercostal or mammary artery, immediate drainage, fluid replacment (possible autotransfusion) & surgery )