Test 3 - Respiratory Dysfunction Flashcards

(65 cards)

1
Q

General Aspects of Respiratory Infections

Described according to the anatomical area involved:

A
Upper respiratory tract
Nose, pharynx, larynx, 
upper trachea
Lower respiratory tract
Lower trachea, bronchi and
 bronchioles, alveoli
Croup syndromes
Infections of the epiglottis or larynx
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2
Q

In infants younger than __ months, maternal antibodies offer some protection.

A

In infants younger than 3 months, maternal antibodies offer some protection.
Espeically if the mom is breast feeding

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3
Q

In infants age __ to ___ months, the infection rate increases.

A

In infants age 3 to 6 months, the infection rate increases.

They don’t have immune system, exposure to more things, and losing maternal antibodies, mother may go back to work - baby go to daycare
In toddlers and preschoolers, there is a high rate of viral infections
Preschool, touching things,eating things, picking their nose, poor hygene and spread germs

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4
Q

In children older than __ years, there is an increase in GABHS and Mycoplasma pneumoniae infections.

A

In children older than 5 years, there is an increase in GABHS and Mycoplasma pneumoniae infections.
In school, it gets passed around
Increased immunity develops with age.

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5
Q

Size to children’s respiratory tract

A

Diameter of airways is smaller
Distance between structures is shorter, allowing organisms to rapidly move between
Short and open eustachian tubes

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6
Q

How do children get respiratory diseases

A
Immune system deficiencies
Allergies, asthma
Cardiac anomalies
Cystic fibrosis
Exposure to infections in daycare
Exposure to second-hand smoke
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7
Q

Seasonal Variation - respirtaory infection are most common during which seasons?

A

Seasonal Variation

Most common during winter and spring

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8
Q

Mycoplasma infections more common in which seasons?

A

Mycoplasma infections more common in fall and winter

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9
Q

Asthmatic bronchitis more frequent in which in of weather?

A

Asthmatic bronchitis more frequent in cold weather

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10
Q

RSV season typically which season?

A

RSV season typically winter and early spring

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11
Q

Generalized signs and symptoms and local manifestations differ in young children are…?

A

Apnea
Fever - you see it in younger children due to initial immune response (in neonate you see drop in temperature)
Anorexia, vomiting, diarrhea, abdominal pain
Cough, sore throat, nasal blockage or discharge
Respiratory sounds

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12
Q

Respiratory assessment should include:

A

RR, depth, rhythm,and effort
HR, O2 sat, hydration status,
Body temperature
Activity level and comfort

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13
Q

Nursing Interventions for Respiratory Infections

A

Ease the respiratory effort
Positioning (lay them up), suction, oxygenation (decrease effort), having them cough, trying to calm the toddler down, turn the lights down and put on a movie (distraction), managing fever

Manage fever
Tylenol, uncover them, put fan in the room, wet/cool towel on forehead and axella
Promote rest and comfort
Decrease metabolic demand - turn off the light, give transitional object, help them feel secure, distractions (movies, music)

Control infection
Make sure the visitor wash hands, make sure the visitors aren’t sick, keep kids out of the room, clamp down on visitors during flu seasons

Promote hydration and nutrition
Offer snacks (pt often don’t have appetite), if hard to breath usually don’t offer water but rather ice chips so they don’t aspirate.
Hydration over nutrition - push them to drink not to eat

Provide family support and teaching

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14
Q

Home management for Upper Respiratory Tract Infections (URIs)

A

Clear secretions -parents make sure knows how to use ball syringes (suction the kid’s nose)
Humidification - Cool mist only (warm causes bacteria and mold growth, and dangerous bc mist can burn them)
Small, frequent feedings
Fever management
Avoid OTC “cold” medicines

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15
Q

Pharyngitis - what is causes and risks

what is the clinical manifestations?

A

Causes and risks
Often viral
Gr A β hemolytic strep(strep throat)
Risk for Rheumatic fever, Acute Glomerulonephritis

Clinical manifestations
Abrupt onset, fever, HA, sore throat
Tonsils, pharynx inflamed, covered with exudate

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16
Q

What is diagnosis of pharyngitis

A

Diagnostics

Although 80-90% are viral, rapid strep test should be done

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17
Q

Therapeutic management for pharyngitis is…?

A

Oral PCN if strong suspicion of bacterial infection

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18
Q

Nursing considerations for pharyngitis is…?

A

Nursing considerations
Warm saline gargles, cool compresses, tylenol/motrin, encourage PO fluids, rest
Teach about administration of meds
Strep is contagious for 24 hours after antibiotics are started

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19
Q

Etiology and clinical manifestation for Tonsillitis?

A

Tonsillitis
Etiology: often occurs with pharyngitis, common in young children, viral or bacterial

Clinical Manifestations:
Enlarged tonsils (“kissing”)
Difficulty swallowing/breathing
Mouth breathing
Sleep apnea
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20
Q

Therapeutic management for Tonsillitis?

A

Therapeutic management - antibiotics
Tonsillectomy/Adenoidectomy
Nursing care: pain relief, minimize bleeding, close observation of breathing, cool clear fluids, observe for bleeding, avoid emesis and clearing of throat

avoid chips or things that can scratch the throat, or vomiting, causing the scab to come off.

Observe for frequent swallowing - that may be due to bleeding

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21
Q

Influenza Clinical manifestations

A

Clinical manifestations
Mild mod or severe, dry throat, cough, general myalgia and malaise, fever, and chills (every fiber of your body hurts)
Lasts 4-5 days minimum

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22
Q

Therapeutic management for influenza

A

Symptomatic treatment - fluids, tylenol

Antiviral medications lessen severity

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23
Q

Prevention for influenza

A

Yearly flu vaccines in children over 6 months of age
Family are the “vaccines for the young infant” - infant can’t get the flu vaccine, so people around the baby need to be vaccinated
Health care worker vaccines

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24
Q

Croup Syndromes

A

Croup Syndromes - fall and winter

Croup
croup won’t kill you, more towards infants
Croup gets worst for 3 nights and then gets better
Make sure to get a good history for pt w stridor - inspiratory sound (hear it on inspiration, sounds like a gasp)

Causes: foreign body - potato chips, lego, etc

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25
Epiglottitis
Epiglottitis Sicker, drooling, and can kill you, you want to leave them alone - never stick anything in mouth or cause pain, dont stick a tongue blade, it would reflexively swell Get steroids Characterized by hoarseness, barking cough (sounds like a seal), inspiratory stridor, and varying degrees of respiratory distress Croup syndromes affect the larynx, trachea, and bronchi Epiglottitis, laryngitis, laryngotracheobronchitis (LTB), tracheitis
26
Acute Epiglottitis-
Acute Epiglottitis- A Medical Emergency Clinical manifestations Rapid progression Sore throat, pain, tripod positioning, retractions Inspiratory stridor, mild hypoxia, distress Watch for: absence of spontaneous cough, drooling, agitation/anxiety Therapeutic management Potential for respiratory obstruction Nursing considerations – do not attempt to inspect the throat! Keep child as calm as possible. Allow position of comfort Prevention requires Haemophilus influenzae type b (Hib) vaccine, incidence greatly decreased since vaccine
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Acute Laryngotracheobronchitis (LTB)
Acute Laryngotracheobronchitis (LTB) Most common croup syndrome Generally affects children younger than 5 years of age Organisms responsible RSV, parainfluenza virus, Mycoplasma pneumoniae, influenza A and B viruses Manifestations of LTB Inspiratory stridor Suprasternal retractions Barking or seal-like cough Increasing respiratory distress and hypoxia Can progress to respiratory acidosis, respiratory failure, and death Therapeutic Management of LTB Airway management Maintain hydration (oral or IV) High humidity with cool mist Nebulizer treatments Epinephrine (racemic) – rapid onset, peak effect at 2 hours Steroids
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Infections of the Lower Airways
Considered the“reactive”portion of the lower respiratory tract Includes bronchi and bronchioles Cartilaginous support is not fully developed until adolescence Constriction of airways
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Bronchitis
``` Also known as tracheobronchitis Definitions – inflammation of the large airways Causative agents – primarily viral Clinical manifestations Dry hacking cough Worsens at night Mild, self-limiting disease ```
30
Bronchiolitis and RSV
Definitions: common acute viral illness, maximum effect and the bronchiolar level Respiratory syncytial virus (RSV) : occurs cyclically in winter and spring Most common cause of hospitalization in <1 year of age Severe RSV at <1 y is a significant risk of developing asthma
31
Pathophysiology of Bronchiolitis and RSV
Pathophysiology Affects the epithelial cells which swell and protrude into the lumen Lumina fill with mucus Leads to hyperinflation (they can breath out but can’t get the air out), hypoxia (bronchial with mucous not exchanging), atelectasis
32
Diagnostics of Bronchiolitis and RSV
Diagnostics Nasopharyngeal swab - you don’t want the mucous, you want the nasopharyngeal cell CXR: hyperinflation
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Therapeutic management of Bronchiolitis and RSV
Therapeutic management Most managed at home, Humidified O2, Periodic suctionin- especially before feedings, Fluids PO or IV, Bronchodilators? Prevention of RSV: prophylaxis (synagis), handwashing Nursing considerations: isolation, close monitoring, teach mom to provide care at home, suctioning
34
Pneumonia
Inflammation of the pulmonary parenchyma Causative organism varies greatly by age category The most useful classification is etiologic agent: Viral, Bacterial, Mycoplasma, Aspiration of foreign substances Clinical Manifestations Vary depending on Age, Etiology, Systemic reaction to infection, Extent of the lesions, Degree of obstruction
35
Pertussis (Whooping Cough)
Pertussis (Whooping Cough) Caused by Bordetella pertussis Highly contagious In the United States, it occurs most often in children who have not been immunized Highest incidence is in spring and summer Hallmark: severe, paroxysmal coughing, followed by characteristic “whoop”, Young infants may present with apnea - Heart rate will drop, stats will drop Post-tussive emesis common, often vomit afterwards Three phases Catarrhal - Increase secretion, running nose and eyes, look like any type of cold Paroxysmal - last a long time Convalescent -
36
Asthma
Asthma Chronic inflammatory disorder of the airways Recurring episodic symptoms Wheezing, Breathlessness, Chest tightness, Cough (especially at night) Limited air flow or obstruction that reverses spontaneously or with treatment Bronchial hyper responsiveness Narrowed airways causes forced expiration. Air trapping Reduced alveolar air exchange Inflammation now recognized as playing a key role in asthma
37
Asthma Severity Classification in Children 5 Years and Older
Step I: Intermittent asthma Sx <2 days/week, no limitations on ADL Step II: Mild, persistent asthma Sx > 2x/wk but not daily, night time 1-2x/mo, some limitations to ADLs Step III: Moderate, persistent asthma Daily symptoms, freq night sx, minor limitations to ADLs Step IV: Severe, persistent asthma Continued daytime sx, freq night sx, use of relievers several times per day, extreme limitations on ADL
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Asthma Diagnostic evaluation
``` Diagnostic evaluation Dyspnea, wheeze, cough PFTs, PEFR, allergy testing Therapeutic management Pharmacologic therapy Nonpharmacologic therapy Allergen/trigger control ```
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Asthma Nursing considerations
Nursing considerations Importance of patient and family teaching Need to control triggers in hospital Smoking cessation
40
Asthma Drug Therapy for Asthma
Drug Therapy for Asthma Long-term control medications Preventive (Controller) DAILY use, even when feeling well Quick relief medications Rescue (Reliever) - albuterol, xopenex Metered-dose inhaler (MDI) Always use with spacer/mask Corticosteroids - First line treatment Significant improvement of all asthma parameters Albuterol, terbutaline Β Adrenergic agonist, rapid onset treatment for acute bronchospasm ``` Dilates bronchioles prevent EIB (exercise-induced bronchospasm) - exercise induced ``` Long-term bronchodilators (salmeterol [Serevent]) Not used in children <12 years Theophylline (monitor serum levels) rarely used, causings significant agitation, and tachycardia, if serum too high pt vomits only in ED when not responding to maximal therapy ``` Leukotriene modifiers (singulair) Mediates inflammation, given in combination with β agonist, steroids for long term control ```
41
Asthma Intervention
Asthma Intervention Exercise/play Evaluate for EIB (exercise induced bronchospasm) Chest physical therapy (CPT) Breathing exercises - encourage cough and deep breathing
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Status Asthmaticus
Status Asthmaticus Respiratory distress continues despite vigorous therapeutic measures Sweating, remains sitting upright, refusal to lie down, agitation, absence of breath sounds, only speaking a few words at a time, level of conciousness is deminishing (meaning not enough air is getting in)= immediate intervention Emergency treatment is epinephrine 0.01 mL/kg subcutaneously (maximum dose, 0.3 mL) Magnesium sulfate - infuse over 20 minutes usually Potent muscle relaxant, decreases inflammation Monitor for side effects, can cause severe hypotension Vital signs q10minutes for an hour Concurrent infection in some cases - pt may also have pneumonia, thus start pt on antivirals
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Acute Asthma Care
Acute Asthma Care Calm nursing presence - pt and family might get anxious seeing you etc. Auscultation of breath sounds, air movement Monitor with pulse oximetry Titrate O2 - Choice of O2 delivery devices Allow older children to sit up if they are more comfortable in that position Allow parents to remain with children Oral vs IV fluids
44
Goals of Asthma Management
Goals of Asthma Management Avoid exacerbation, Avoid allergens Relieve asthmatic episodes promptly, Relieve bronchospasm Monitor function with a peak flow meter Self-management of inhalers, devices, and activity regulation Support child, adolescent, and family Identifying Asthma Triggers Viral infection, Exercise, Perfumes, Smoke One of the most important components of decreasing episodes of exacerbations Differs in each person - was there a weather change, different setting, food, etc Common triggers (not an exhaustive list) Viral infections (most common trigger for infants) “he never gets these symptoms unless he has cold, Perfumes, strong odors Smoke: tobacco, marijuana, wood, BBQ, fires, Exercise, Changes in temperature, Animal dander - cats.dog, Dust and mold, Pollen, Food allergies
45
Cystic Fibrosis (CF)
Cystic Fibrosis (CF) Exocrine gland dysfunction that produces multisystem involvement Most common lethal genetic illness among Caucasian children Autosomal recessive trait - need a defective gene from both parents Child inherits a defective gene from both parents, with an overall incidence of 1:4 Approximately 3% of the U.S. Caucasian population are symptom-free carriers
46
Pathophysiology of CF
Pathophysiology of CF Predominantly affects the respiratory tract and the pancreas - produce cement like mucous Increased viscosity of mucous gland secretions Responsible for many of the clinical manifestations Elevation of sweat electrolytes Increase in several organic and enzymatic constituents of saliva Increase in sodium and chloride in saliva and sweat Exocrine Gland Dysfunction in CF
47
Clinical Manifestations of CF
Clinical Manifestations of CF Pancreatic enzyme deficiency - doesnt let them digest fat Progressive chronic obstructive pulmonary disease (COPD) associated with infection Sweat gland dysfunction Failure to thrive - due to fighting all infection, they’re not absorbing food Increased weight loss despite increased appetite Gradual respiratory deterioration
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Presentation of CF
Presentation of CF Wheezing respiration; dry, nonproductive cough, Generalized obstructive emphysema Patchy atelectasis, Cyanosis Clubbing of fingers and toes Repeated bouts of bronchitis and pneumonia Seem like “sickly baby” - aren’t gaining weight, are sick often Meconium ileus - Earliest recognizable manifestation of CF Prolapse of the rectum - due to the stool Distal intestinal obstruction syndrome Excretion of undigested (fat) food in stool - Stool is bulky, frothy, and foul smelling Wasting of tissues (not absorbing fat, thus not absorning vitamin A,D,E,K (fat soluble)) Delayed puberty in females Sterility in males Parents report that children taste “salty” - when kissing their child Dehydration, Hyponatremic or hypochloremic alkalosis, Hypoalbuminemia
49
Diagnostic Evaluation of CF
``` Diagnostic Evaluation of CF Quantitative sweat chloride test - make them sweat and test the sweat Chest x-ray Pulmonary function tests (PFTs) Stool fat and/or enzyme analysis Barium enema ```
50
Treatment Goals for CF
Treatment Goals for CF Prevent or minimize pulmonary complications, Adequate nutrition for growth, Assist the child in adapting to a chronic illness
51
Respiratory Manifestations of CF
Respiratory Manifestations of CF Present in almost all CF patients, but the onset and extent vary Viscous secretions are difficult to expectorate and obstruct bronchi and bronchioles Cause atelectasis and hyperinflation Stagnant mucus leads to destruction of lung tissue Stagnant mucus provides a favorable environment for bacteria growth
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What is respiratory progression
Respiratory Progression Gradual progression follows chronic infection Bronchial epithelium is destroyed Infection spreads to peribronchial tissues, weakening the bronchial walls Peribronchial fibrosis Decreased exchange of O2 and CO2 - end up on oxygen all the time, kids become slim
53
What is further respiratory progression
``` Further Respiratory Progression Chronic hypoxemia causes contraction and hypertrophy of muscle fibers in pulmonary arteries and arterioles Pulmonary hypertension Cor pulmonale Pneumothorax Hemoptysis ```
54
Respiratory Management of CF
Respiratory Management of CF CPT (chest physiotherapy) and postural drainage - Therapy vests - vibrate to help move the mucous Bronchodilator medication, Forced expiration Aggressive treatment of pulmonary infections Home IV antibiotic therapy, Aerosolized antibiotics Steroids or non-steroidal anti-inflammatory drugs (NSAIDs) Transplantation
55
Gastrointestinal (GI) Tract from CF
Gastrointestinal (GI) Tract Thick secretions block ducts, leading to cystic dilation, degeneration, and diffuse fibrosis Prevents pancreatic enzymes from reaching the duodenum - have to take handful and pancreatic enzyme Impaired digestion and absorption of fat, or steatorrhea, occurs Impaired digestion and absorption of protein, or azotorrhea, develops Earliest manifestation may be meconium ileus Endocrine function of the pancreas is initially unchanged Eventually, pancreatic fibrosis - could kill off the pancreas occurs; may result in diabetes mellitus Focal biliary obstruction results in multilobular biliary cirrhosis - may cause heart, liver transplant Impaired salivation
56
GI Management in CF
GI Management in CF Replacement of pancreatic enzymes High-protein, high-calorie diet, as much as 150% of the recommended daily allowance (RDA) Treat constipation Reduction of rectal prolapse Salt supplementation Treat gastroesophageal reflux- Place the patient in the upright position after meals
57
Endocrine Management of CF
Endocrine Management of CF | Monitor blood glucose levels, Diet, Exercise, Regular eye examinations
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Prognosis of CF
Prognosis of CF Estimated life expectancy for a child born with CF in 2008 was 37.4 years CF continues to be a progressive, incurable disease Organ transplantation has increased the survival rate Heart–lung and bilateral lung transplantation Liver and pancreas transplantation For lung transplants, the survival rate is 75% at 1 year and 55% at 3 years following transplantation. Maximize health potential Nutrition, Prevention and early aggressive treatment of infection, Pulmonary hygiene
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Family Support for the Child with CF
Family Support for the Child with CF Coping with the emotional needs of the child and family Developmental care Child requires treatments multiple times each day Frequent hospitalizations Implications of genetic transmission of disease
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What is the difference between long term control medication, quick relief medications and metered dose inhaler?
Long-term control medications Preventive (Controller) DAILY use, even when feeling well Quick relief medications Rescue (Reliever) - albuterol, xopenex Metered-dose inhaler (MDI) Always use with spacer/mask
61
What is corticosteriods?
Corticosteroids - First line treatment | Significant improvement of all asthma parameters
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What is albuterol/terbutaline?
Albuterol, terbutaline Β Adrenergic agonist, rapid onset treatment for acute bronchospasm ``` Dilates bronchioles prevent EIB (exercise-induced bronchospasm) - exercise induced ```
63
What is sameterol/servent?
Long-term bronchodilators (salmeterol [Serevent]) Not used in children <12 years
64
What is theophyline?
Theophylline ***(monitor serum levels) rarely used, causings significant agitation, and tachycardia, if serum too high pt vomits only in ED when not responding to maximal therapy
65
What is Leukotriene modifiers (singulair)?
``` Leukotriene modifiers (singulair) Mediates inflammation, given in combination with β agonist, steroids for long term control ```