Test # 4, memory Flashcards

(44 cards)

1
Q

Darwin

A

evolution of both physical and mental traits

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2
Q

what is intelligence

A

the ability to make adjustments, or modify old ones, in accordance with the results of its own individual experience. it is hard to learn without some intelligence-like construct

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3
Q

cognition

A

latin for knowledge or thinking. cognition may lead to actions not clearly explained by external stimuli

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4
Q

cognitive ethology

A

the study of the ability of animals to express conscious thought and intention

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5
Q

animal cognition

A

models and constructs used to explain behaviors not characterized by simple s-are associations. think mark test

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6
Q

memory

A

our ability to respond to or recount information that was experienced earlier.

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7
Q

three components to memory

A

acquisition, retention, retrieval

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8
Q

acquisition:

A

exposure to stimuli or information

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9
Q

retention

A

the time period which information is retained in between acquisition and retrieval

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10
Q

retrieval

A

tests of memory for the original exprerience

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11
Q

how do we distinguish between learning and memory

A

in learning we manipulate the acquisition parameters. studies of memory will more likely focus on retention and retrieval.

in studies of learning often acquisition is varied between groups, retention is constant and long, and retrieval is constant. in memory acquisition is constant but retention is varied often between short and long and retreival is varied

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12
Q

studying memory in animals hunter 1913

A

subjects: raccoons, dogs, rodents

phase 1: training; subjects placed in a start box, light on above baited box immediate choice.

phase 2: memory; subject placed in start box, light on above baited box, delayed choice. can they remember which box hd the light on after the delay

results: dogs could remember for five I think, rats for ten, raccoons for twenty five

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13
Q

working memory

A

operates when info needs to be maintained long enough to complete a task (each individual test trial)

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14
Q

reference memory

A

long term retrieval of rules necessary for the use of incoming and acquired information (training)

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15
Q

Bloody Mary

A

remembering the items you just put in a Bloody Mary is your working memory. remembering the items needed o make a Bloody Mary is your reference memory

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16
Q

memory task delayed matching to sample test

A

first, the sample identifies the correct response on a trial, then the sample is removed before the subject is allowed to respond. the period of time in between seeing the correct sample and seeing the next samples is the delay. after the delay you are presented with a choice of samples and they are asked to identify the sample that matches the initial correct sample. this is done in free operant. chance alone is 50%. we need to see at least 80% to unsure learning above chance.

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17
Q

test sample duration

A

the amount of time they have initially to see the correct sample choice

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18
Q

test sample trial delay

A

the interval between the correct sample turning off and the choices appearing

19
Q

grant 1976

A

has a 1, 4, 8, or 14 second test sample duration. then had a 0, 20, 40, or 60 second test sample trial delay. then had choice.

results: as test sample duration increased, accuracy increased. as test sample trial delay increased accuracy decreased

20
Q

do the test stimuli in the dms determine accuracy? trace decay hypothesis:

A

presentation of a stimulus produces changes in the cns that decay after the stimulus is removed

weak stimuli will decay quickly: what did I eat for lunch last Wednesday

strong stimuli will be maintained in memory: what did you eat when you got food poisoning

21
Q

does training impact the dms: sarigson and white

A

trained on dms, test sample trial delay 0, 2, 4, 6 seconds, trained to 80% correct

test with 0-10 second sample trial delay

results: decay prominent in 0 second there was an immediate drop off after 0 for this group because they never learned that they need to remember so they would never remember for more than 0 seconds.

each group performed best when tested with their training delay

22
Q

what is learned on the dms task

A

general rule: ie choose the stimulus that is the same as the sample

specific rule: select green after green

23
Q

test of transfer

A

a test used to determine if a specific or general rule was learned.

once the dms is learned with two samples a new pair is umped with the same rule. does the subject generalize the rule?

for example, if it learns that when green shows in the test sample duration, then green shows over a box it will get food, then you try the same with yellow and see if it generalizes that any light means there is food, or if it remains specific and only learns green means food

24
Q

oden et al 1988 trials unique procedure

A

on each trial different stimuli serve as matching and non matching samples. do they learn the dms, this forces the organism to learn the general rule. if the organism can get to 80% you have guaranteed the organism has learned the general rule

25
Clark's nutcracker
store seeds and remember them
26
AD early
Hippocampus and Basal Forebrain (acetylcholine) limits rehearsal/consolidation and causes anterograde amnesia
27
AD late
still hippocampus and basal forebrain but retrieval becomes limited too, also amygdala and cortical areas/cortex I think, this causes emotional instability. this then is retrograde
28
which part of the brain does Korsakoff limit
thalamus, limits consolidation and causes anterograde amnesia
29
retrograde
the inability to recall information before the onset of the amnesic event. often caused from a brain injury like a seizure or blackout, spans minutes to years, the is a problem with recall, retrieval cues can help sometimes, harms the association cortex and retrieval
30
anterograde
the inability to form new declarative memories. damage to the hippocampus and amygdala, which causes consolidation deficit
31
beacon
following associaition of beacon with goal, right in front or on top
32
landmark
goal at fixed location away from stimulus
33
proactive interference
previously learned information disrupts new memories
34
retroactive interference
new memories disrupt previously learned information. shoot who did I meet when I first got to this party
35
long term potentiation
how does us pulling in external information effect downstream targets in the brain? bliss and loco 1973: stimulated hippocampus input, moderate strong and weak, when moderate no change in the downstream areas, when high there was long term potentiation meaning that there was enhanced synaptic strength and learning, with weak there is long term depression. this teaches us that external information can produce synaptic strength, lip increases the efficiency of memory paths, through experience we can take a small plastic spine and make it receptive to input, increase the number of dendritic spines - more places to receive messages, increase the size of the dendritic spine head, increase the excitatory receptor density on dendritic spine head
36
wilder and penfield
did thousands of brain surgeries on epilepsy found that medial temporal lobe stimulation induced specific memories when stimulated mri confirms that consolidated memory is stored and retrieved from the cortex cortical areas in the brain is where we store this information for retrieval
37
rehearsal circuit
hippocampus, basal forebrain, thalamus, amygdala
38
infantile amnesia and 2-5 years
in inintile amnesia 0-2 very little information is recounted explicitly, but as we found earlier very little information can occur 2-5 years some memories can be recounted, emotional memories are amplified via the amygdala
39
what are the symptoms of Alzheimer disease
impaired memory, disorientation, emotional flux, worsening of symptoms
40
nueropathologic symptoms of ad
atrophy: the brain gets smaller, tissue seems to degenerate cerebral arterial sclerosis: brain tissue hardens histological abnormalities: these intra and extra cellular features are the only way we diagnose the disease. the only real way to know is post mortem work to find amyloid plaques and neurofibrillary tangles
41
possible causes of ad
aging increases mutations, genes but only 4-8% so not a huge factor - although there are familial forms, brain damage, inflammation - an underlying facet that increases all of these factors although just targeting information is not enough
42
current treatments for AD
cholinesterase inhibitors: increase ach - we see a difficiancy in ach causes weak consolidation bmda antagonists: reduce glutamate cognitive and physical exercise immunotherapies against amyloid plaques: compounds that are built as antibodies to target the plaques and tangles, this usually slows but does not stop - we are still unsure if the plaques and tangles are even the cause or the worst part of the disease
43
korsakoffs syndrome
severe anterograde amnesia causes wild tales we see a degeneration of the mammillary bodies and lesions of the thalamic nuclei which are super important in the rehearsal circuit they usually have no retrograde amnesia so they have a rich tapestry of memories to pull on
44
causes of Korsakoff's
thiamine (vitamine b1) deficiency people with bad diets alcoholics get a lot of their calories from alcohol and they don't get enough thiamine, high proof alcohol effects the absorption of thiamine there is no treatment once you lost your maxillary bodies if someone is severely malnourished if you give them a push of glucose with no thiamine you will give them acute Korsakoff's