THA Flashcards
How does anterior approach compare to posterior approach?
What is the main con of each?
Anterior slightly better/faster at rehab -> NO DIFF after 3 months
Anterior - high rate of femoral failure
Posterior - higher rate of dislocation
What is the average failure rate %/year of THA?
1%/year
In what scenario should you consider cementing a cup?
Previously irradiated
What should the average size be for the following to have good bone ingrowth:
- ) Metal porosity %
- ) Depth of pores
- ) Gap
Rules of 50’s!
- ) 50%
- ) 50-150 microns
- ) < 50 microns
What is the consequence of wear rate of modern PE (UHMWPE) with use of larger femoral heads?
NO consequence of bigger femoral heads!
(typically with old poly the larger femoral heads led to higher volumetric wear and small heads led to high linear wear)
What is the main type of wear that occurs with ceramic on poly THA bearings - contributes MOST to osteolysis?
Adhesive wear
What is the osteolytic threshold (that is the wear rate under which osteolysis is not seen)?
< 0.1 mm/year
What are the main molecules/mediators involved in osteolysis?
TNF-alpha
IL1-beta
IL-6
What technical error is particularly bad for MoM/hip resurfacing?
Vertical cup placement -> edge loading!!
What is the main mediator cell for:
- ) Osteolysis due to PE
- ) ALVAL
- ) Macrophage
- ) Lymphocyte (delayed Type IV hypersensitivity)
* *Realize that ALTR (which is the condition and ALVAL is the histologic dx) can result in XR with signs of osteolysis! PE is not the only thing that causes osteolysis!
What is the best study to look at pseudotumor in MoM hips?
MARS MRI
What is the cause of trunionosis?
What is the treatment?
Micromotion at modular head/neck jxn of metal head w/ trunion -> fretting corrosion = corrosion due to small cyclic motion disrupting the protective oxide layer. See Co:Ch levels 4-5:1
Treatment is revision w/ placement of Ceramic head w/ titanium sleeve
What bad consequence can happen due to destruction from metallosis (MoM or trunionosis)? And what is the revision surgery typically needed?
Loss of abductors
Constrained liner
If a patient has footdrop postop and you think it is due to hematoma what is the first thing that you do?
TAKE THEM BACK TO OR AND DECOMPRESS IT!
Don’t order any imaging studies if you know from presentation!
What are the two possible HO ppx treatments? And when should they be given?
- ) Indomethacin (variable dosing) given for 1-6 weeks postop (look out for people w/ kidney issues and AVOID this!)
- ) Radiation - 7 Gy to be given 24 hrs preop or 48 hrs postop (very small risk of cancer!)
* *There is NO role for late use of these - if they form HO…just wait for it to mature and take out later
What is the cause of most LLD postop?
Perceived! -warn the patients that they may feel long b/c they have been living short!
Tx -> observe, stretch and let tissue accommodate
What is a possible cause of anterior hip pain after THA? And what are the possible tx’s?
Iliopsoas impingement/tendonitis.
Can verify w/ injection
1.) Get crosstable lateral XR to eval for cup overhang -> if overhand > 8 mm revise!
2.) Tenotomy
In the case of performing a revision for PE wear w/ osteolysis and lytic defects behind a well-fixed, well-positioned cup what should you do?
LEAVE THE CUP!!! EVEN IF BIG OSTEOLYTIC DEFECT BEHIND IT!! (High morbidity if you do this!)…..if the poly locking mechanism doesn’t exist b/c it’s an old cup -> CEMENT IN THE LINER
What is the #1 cause for THA revision longterm?
Aseptic loosening
What is the Paprosky definition of femoral bone loss? And what implant should be used for each?
Type 1: Intact metaphysis (any primary implant)
Type 2: Metaphyseal bone loss (cementless diaphyseal engaging stem)
Type 3A: > 4 cm diaphysis remaining
Type 3B: < 4 cm diaphysis remaining
(cementless diaphyseal engaging)
Type 4 -> Nonsupportive diaphysis (DFR or APC)
In a revision THA on the acetabular side, how much acetabulum do you need to use a hemispherical cup w/ screws?
> 2/3 rim intact
> 50% acetabular bone stock
What do you do if there is an intraop calcar fracture?
1.) Expose fracture!
If limited to calcar/metaphysis -> cable and keep same stem. WBAT!
If extends to diaphysis -> switch to a diaphyseal engaging stem!
What is the most common cause of acute PJI?
What is the most common cause of chronic PJI?
Acute -> Staph aureus
Chronic -> Staph epi (aka coagulase negative Staph)
What is MoA of TXA?
Who is it contraindicated in?
Competitively inhibits plasminogen (antifibrolytic) by binding to the lysine binding site
Contraindicated in renal failure and pt w/ active VTE
**NO evidence that it increases the risk of VTE…but decrease intraop blood loss and need for transfusion!