The Psychology of Stress Flashcards

1
Q

How would you define stress?

A

A psychological and physiological reaction that occurs in response to a threat, i.e., when an individual perceives that environmental demands tax or exceed his or her adaptive capacity.

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2
Q

What is a stressor?

A

A stimulus or event that causes a stress reaction. This can be a thought, it doesn’t have to be external.

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3
Q

What is the difference between acute and chronic stress?

A

Acute: sudden, typically short-lived, threatening event
Chronic: ongoing environmental demand

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4
Q

Do you think that acute stressors are worse for our health than chronic stressors?

A

No, the fact that they’re short-lived means that our body can recover relatively well and is not as concerning to our health. Sapolsky writes about how our stress-response is more adapted to handle these sorts of stressors.

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5
Q

Why is chronic stress concerning for our health?

A

These are ongoing demands which repeatedly activate your stress-response. These chronic stressors are much more challenging for our mental and physical health.

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6
Q

Differentiate minor and major life stressors. Historically, what has health psychology focussed on?

A

Major life events are big events in your life (good or bad).
Minor life events are more the day-to-day hassles.
Health psychology has focused on major life stressors in the past. Now minor life stressors are being recognised.

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7
Q

What’s the main measure of major life stressors?

A

The Social Readjustment Rating Scale

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8
Q

Describe one of the earliest studies linking major life stressors to disease (Rahe et al., 1964).

A

Using archival, retrospective data to study major life stressors leading up to the development of TB.
The TB patients had a lot more major life events in the years prior to developing TB, compared to the control group.

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9
Q

Can you determine causality from the study by Rahe et al? (Linking major life stressors to disease)

A

No, it was using retrospective archival data. We can’t tease out what happened first, their health could have been driving life events. (although this pattern has been demonstrated numerous times e.g. people who had heart attacks also had more life stressors before illness onset)

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10
Q

The accumulation of ____ severe but ____ frequent hassles can be ____ harmful to health as _____ life events.

A

less…more…more…major

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11
Q

How can we measure major life events in childhood? What do results from this questionnaire correlate to?

A

One of the most commonly used questionnaires is the Adverse Childhood Experience (ACE) Questionnaire. Greater than 4 typically puts you at higher risk of mental and physical illness.
Those with higher ACE scores were more likely to experience depression, use antidepressant prescriptions, smoke and suffer from alcoholism.

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12
Q

Odgers & Jaffee (2013) believe that exclusive focus on major traumatic events ignores the role of…

A

…routine or ongoing stressors, which may have equally, or perhaps more, profound effects on children’s development owing to both the frequency and the duration of their exposure.

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13
Q

List 3 examples where routine adversity impacts health.

A
  • Mothers who report more minor hasses are more aggressive and short tempered with their children.
  • Environmental hassles are linked to children’s poorer outcomes independently from more major forms of abuse.
  • Daily stressors linked to outcomes such as coronary heart disease.
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14
Q

In the lecture, what was the measure of minor stressors?

A

The Perceived Stress Scale 4. One of the most commonly used stress measures.

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15
Q

There is a clear linear relationship between perceived stress scores and…

A

…perceived health status.

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16
Q

Stress scores are going __ over time. (comparing to Cohen’s original sample in 1983)

A

Up

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17
Q

How valid are these stress measures? Are they just measuring out self perceptions of things or are they tied to biological markers of stress?

A

They do seem to be valid, for example change in perceived stress is correlated to change in amygdala grey matter density, an important structure for threat detection. Amygdalae less active in people with greater reduction in perceived stress after mindfulness intervention.

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18
Q

What’s the word for positive stress?

A

Eustress

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19
Q

Eustress is more akin to _________.

A

Challenge.

20
Q

What is the word for negative stress?

A

Distress

21
Q

Distress is more akin to ______.

A

Threat.

22
Q

What is the concept of anti-fragility?

A

Where organisms benefit from stress.
Stress can inoculate a person to future stress. If you try to remove all the stress from a child’s environment how will they learn to cope with stressors.

23
Q

Are the experiences of eustress and distress similar?

A

They are quite different and even have different vascular profiles.

24
Q

What are 6 factors that increase or decrease the stress response?

A
  • Controllability
  • Locus of control
  • Uncertainty
  • Ambiguity
  • Complexity
  • Volatility
25
Q

Why is a species with a well-developed frontal lobe disadvantaged when it comes to stress?

A
  • We have more variable and chronic stressors
  • These stress systems work great in evolutionarily less sophisticated animals, but less great in evolutionarily more sophisticated animals because of our more complex and stressful societies, our ability to imagine and think into the future, and the rigors of a modern stressful world
  • Humans also live longer, have more exposure to stress and more accumulated effects
26
Q

Stress affects health through ____ primary pathways.

A

Four

27
Q

The four primary pathways which stress affects health are:

A

Behavioural
Neural
Hormonal
Immune changes

28
Q

How might stress affect health through behavioural changes?

A

Changes in the big four; eat worse, smoke more, drink more and exercise less. These changes would then affect our health.
It can also lead to inattention which can lead to accidents.

29
Q

How might stress affect health through physiological (neural and hormonal) changes?

A

Stress influences two main physiological systems:
1. The neural-based SAM system
2. The hormone-based HPA axis
Stress influences health through multiple converging and interacting systems, like the two above.

30
Q

Fill the gaps:

___________ Nervous System –> Adrenal _______ –> ___________/______________

Hormone System –> _______ Cortex –> _____________

A

Sympathetic…Medulla…Epinephrine/Norepinephrine

Adrenal…Glucocorticoids

31
Q

What does SAM stand for?

A

Sympathetic-Adrenal-Medullary

32
Q

What are the effects of theSAM system on the body?

A
Increased feelings of activation/arousal
Increased heart rate
Increased blood pressure & vascular resistance
Increased sweating
Increased attention and pupil dilation
Decreased digestion
33
Q

What does vascular resistance mean?

A

Vascular resistance refers to when the veins and arteries become stiff, so that blood is able to travel more quickly.

34
Q

How might chronic stress affect your blood pressure?

A

We want our cardiovascular system to be reactive meaning that when needed our heart rate, blood pressure and peripheral resistance increase.
However, if activated too much and not given adequate time to recover it can lead to health issues.
If your stress-response keeps activating repeatedly then your blood pressure may not come down; this elevated blood pressure can lead to a whole host of problems included atherosclerosis (narrowing of arteries) by buildup of cholesterol.

35
Q

Is there any evidence linking stress to atherosclerosis? Which study was this? What was measured?

A

A study from 1983 (Kaplan, Manuck et al.) on macaque monkeys provides supportive experimental evidence for this.
Healthy monkeys were randomly assigned to either the unstressed control group or the experimental group.
Experimenters took regular measures of blood pressure and cholesterol, and at the end of the 21 month experiment they measured the thickness of the coronary artery (intimal thickness).

36
Q

A study from 1983 (Kaplan, Manuck et al.) on macaque monkeys provides supportive experimental evidence linking stress to atherosclerosis. What were the results of this study?

A

There were significant differences in thickness of the coronary artery. There were no differences in circulating cholesterol levels or bimonthly blood pressure readings.

37
Q

A study from 1983 (Kaplan, Manuck et al.) on macaque monkeys provides supportive experimental evidence linking stress to atherosclerosis. What are the implications for humans?

A

The study forms the basis of a large body of literature suggesting that more frequent, pronounced, and prolonged increases in blood pressure and heart rate - due to SAM activation - initiate atherosclerosis and can lead to Coronary Heart Disease.

38
Q

How does stress trigger hormonal changes?

A

With stress, the hypothalamus launches a hormonal cascade, resulting in the release of glucocorticoids from the adrenal glands into the bloodstream.
Cortisol increases access to energy reserved - via increases in blood sugar - among other functions.

39
Q

Outline the HPA axis.

A

H: Appraisal of stress sends signal to hypothalamus
P: Hypothalamus sends releasing factor (corticotropin releasing hormone CRH) to pituitary gland
A: Pituitary sends ACTH (adrenocorticotropic hormone) through the bloodstream to the adrenal gland.
The adrenal gland releases cortisol into the bloodstream.

40
Q

What are the effects of activating the hypothalamic-pituitary-adrenal (HPA) axis?

A
  • Enhances effects of epinephrine and norepinephrine on the heart
  • Increased utilisation of glucose and fatty acids into bloodstream
  • Decreased growth, reproduction hormones
  • Decreased immune response/inhibited inflammatory response
41
Q

How do SAM system and HPA axis compare in regards to timing?

A

Sympathetic nervous system is instigated in the brain and is mediated by actual physical linkages of the different axons of neurotransmitters throughout the body, so this is a fast response (milliseconds).
Hormone system which is also triggered in the brain but it releases slower acting molecules through the blood system (roughly 20 minutes). It initially supports the SAM system.

42
Q

Fill the gaps:
The ___ system is mostly linked with short-term bursts of activity; focused consequences for _______________ system.
The ___ ____has more systematic and _______ relations with the body; it’s involved in trying to restore __________ and has widespread consequences for ______ __________.

A

SAM; cardiovascular

HPA axis; complex; allostasis; energy utilisation

43
Q

Prolonged activation of the HPA axis…

A

… raises cortisol.

44
Q

What are the effects of prolonged cortisol activation?

A
Cardiovascular reactivity
Insulin resistance
Weight loss or gain
Immune system impairment
Growth problems
Reproductive problems
Cognitive problems via destruction of neurons in the hippocampus
Depression
45
Q

Cushing’s Disease is due to…

A

… too much cortisol due to HPA-axis dysregulation. It’s a pituitary problem, it keeps signalling to the adrenals. Lot’s of sugar in the blood it gets stored as adipose.

46
Q

How would you recognise Cushing’s Disease?

A

Extreme weight gain and sometimes a dowager hump.

47
Q

During stress, your ________ also secretes _________ which is involved in suppressing _____________.

A

Pituitary; prolactin; reproduction