topic 6 Flashcards

(69 cards)

1
Q

What is a stimulus?

A

something that causes a change in an organism’s internal or external environment.

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2
Q

Why is it important that organisms can respond to stimuli?

A

Organisms increase their chance of survival by responding to stimuli.

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3
Q

What is a tropism?

A

A growth response of a plant towards (positive) or away from (negative) a directional stimulus.

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4
Q

Summarise the role of growth factors in flowering plants.

A

Hormone-like substances (e.g., auxins like IAA) produced in growing regions, transported via phloem or diffusion, regulating growth in response to directional stimuli.

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5
Q

Describe how indoleacetic acid (IAA) affects cells in roots and shoots.

A

In shoots: high IAA stimulates cell elongation; in roots: high IAA inhibits cell elongation.

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6
Q

Explain gravitropism in flowering plants.

A

IAA moves to the lower side of the root/shoot, stimulating cell elongation in shoots but inhibiting in roots, causing shoots to bend away from gravity and roots towards gravity.

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7
Q

Explain phototropism in flowering plants.

A

IAA moves to the shaded side, stimulating cell elongation in shoots and inhibiting in roots, causing shoots to bend towards light and roots away.

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8
Q

Describe taxes.

A

A directional response towards or away from a stimulus.

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9
Q

Describe kinesis.

A

A non-directional response involving changes in speed or rate of turning in response to stimulus intensity.

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10
Q

Explain the protective effect of a simple reflex.

A

Rapid, automatic, involves few neurones and synapses, protecting from harm.

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11
Q

Describe the structure of a Pacinian corpuscle.

A

Concentric layers of connective tissue around a sensory neurone ending.

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12
Q

How does a generator potential form in a Pacinian corpuscle?

A

Pressure deforms lamellae, opens stretch-mediated Na+ channels, Na+ enters, depolarises, triggers action potential if threshold reached.

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13
Q

What does the Pacinian corpuscle illustrate about receptors?

A

They respond only to specific stimuli and produce a generator potential leading to an action potential.

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14
Q

Compare light sensitivity of rods and cones.

A

Rods more sensitive (summation of many rods to one neurone); cones less sensitive (one cone to one neurone).

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15
Q

Compare visual acuity of rods and cones.

A

Rods: low acuity (signals merged); cones: high acuity (separate signals).

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16
Q

Compare colour sensitivity of rods and cones.

A

Rods: monochromatic; cones: trichromatic (red, green, blue).

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17
Q

Define myogenic cardiac muscle.

A

It contracts without external nervous stimulation.

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18
Q

Describe the conduction system of the heart.

A

SAN generates impulse → atria contract → non-conducting tissue → AVN delays → Bundle of His → Purkyne tissue → ventricles contract from apex.

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19
Q

Where are chemoreceptors and baroreceptors located?

A

Aorta and carotid arteries.

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20
Q

Explain how chemoreceptors and pressure receptors affect heart rate.

A

Detect pH/CO2 or blood pressure changes → medulla → sympathetic/parasympathetic nerves to SAN → adjust heart rate.

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21
Q

Describe resting potential in a neurone.

A

Inside negative relative to outside due to Na+/K+ pump and differential membrane permeability.

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22
Q

Explain how resting potential is established.

A

3 Na+ out, 2 K+ in by active transport; K+ leaks out, few Na+ leak in.

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23
Q

Describe depolarisation.

A

Stimulus opens Na+ channels; Na+ influx; if threshold reached, action potential fired.

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24
Q

Explain all-or-nothing principle.

A

Action potential occurs only if threshold reached; same size each time; stronger stimulus increases frequency, not amplitude.

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25
Explain saltatory conduction.
Myelinated axons: depolarisation only at nodes of Ranvier, speeding up impulse.
26
What happens in multiple sclerosis to impulse speed?
Myelin damaged → slower or blocked conduction → slower response.
27
Define refractory period.
Time after an action potential when no new impulse can be generated.
28
Why is the refractory period important?
Ensures discrete impulses, limits frequency, and ensures one-way transmission.
29
Describe synaptic transmission at cholinergic synapse.
Depolarisation → Ca2+ influx → vesicles release ACh → binds receptors → Na+ channels open → depolarisation.
30
How is acetylcholine removed?
Hydrolysed by acetylcholinesterase → components reabsorbed.
31
Why is synaptic transmission unidirectional?
Neurotransmitter released from presynaptic side; receptors only on postsynaptic membrane.
32
What is spatial summation?
Multiple presynaptic neurones release neurotransmitter to reach threshold at one postsynaptic neurone.
33
What is temporal summation?
One presynaptic neurone releases neurotransmitter repeatedly in a short time to reach threshold.
34
Explain inhibition at a synapse.
Inhibitory neurotransmitter opens Cl- or K+ channels → hyperpolarisation → harder to reach threshold.
35
Describe muscle contraction in antagonistic pairs.
Agonist contracts, pulling bone; antagonist relaxes. Allows controlled movement and posture.
36
Name the two protein filaments in a myofibril.
Actin (thin) and myosin (thick).
37
Where are Z-lines, M-lines, and H-zones found?
Z-line: ends of sarcomere; M-line: middle; H-zone: only myosin.
38
Explain the banding pattern in myofibrils.
I-band: actin only (light); A-band: actin + myosin (dark); H-zone: myosin only.
39
Explain role of ATP in muscle contraction.
ATP hydrolysis releases energy for myosin head movement; new ATP breaks cross-bridge.
40
What is the role of phosphocreatine?
Rapidly regenerates ATP from ADP during short bursts of intense activity.
41
Compare slow and fast muscle fibres.
Slow: many mitochondria, aerobic, fatigue-resistant. Fast: anaerobic, less mitochondria, fatigue quickly.
42
What is homeostasis?
Maintenance of stable internal conditions within narrow limits.
43
Explain why temperature must be controlled.
Enzyme activity depends on temperature; too hot = denature; too cold = less kinetic energy.
44
Explain why blood pH must be controlled.
Enzyme shape depends on pH; denature outside optimal range.
45
Explain why glucose levels must be controlled.
Too low: not enough respiration; too high: dehydration.
46
Describe negative feedback.
Receptors detect deviation; effectors counteract; conditions return to normal.
47
Give an example of positive feedback.
Childbirth: oxytocin increases contractions; blood clotting.
48
Name the hormones controlling blood glucose.
Insulin, glucagon, adrenaline.
49
Explain insulin's role.
Binds to receptors, inserts glucose channels, stimulates glycogenesis.
50
Explain glucagon's role.
Binds to receptors, activates enzymes for glycogenolysis and gluconeogenesis.
51
Explain adrenaline's role.
Binds to receptors, activates adenylate cyclase → cAMP → activates enzymes for glycogenolysis.
52
What is the second messenger model?
Hormone binds to receptor → adenylate cyclase → cAMP → activates enzymes.
53
Compare type I and type II diabetes.
Type I: no insulin produced. Type II: receptors lose sensitivity.
54
Why can't insulin be taken orally?
Protein, so digested by enzymes in stomach.
55
How is type II diabetes controlled?
Diet, exercise, weight loss, drugs increasing insulin sensitivity.
56
Describe ultrafiltration.
High pressure forces water, glucose, ions out of blood at Bowman's capsule.
57
Describe selective reabsorption of glucose in PCT.
Na+ out by active transport; Na+ in with glucose by cotransport; glucose diffuses into blood.
58
Why does a diabetic's urine contain glucose?
Carrier proteins saturated; excess glucose remains in filtrate.
59
What is osmoregulation?
Control of blood water potential using ADH.
60
Describe ADH action.
Binds collecting duct receptors → inserts aquaporins → increases water reabsorption.
61
What is the loop of Henle's role?
Creates Na+ gradient in medulla; water reabsorbed from collecting duct.
62
Why do desert animals have long loops of Henle?
More Na+ gradient, more water reabsorption.
63
Where is ADH made and released?
Made in hypothalamus; released by posterior pituitary.
64
How does temperature affect conduction speed?
Higher temp → faster diffusion; too high → denatures proteins.
65
Explain what a refractory period ensures.
One-way transmission and limits frequency.
66
Why is spatial summation important?
Ensures weak stimuli can still trigger an action potential.
67
Explain why an axon with a larger diameter conducts faster.
Less resistance to ion flow.
68
What is saltatory conduction?
Impulse jumps between nodes of Ranvier, speeding up conduction.
69
Describe the advantage of second messengers.
Amplifies signal, rapid response.