Trauma Flashcards

1
Q

What are the ABCDE’s of trauma care?

A
A - airway
B - breathing
C - circulation
D - Disability (neuro)
E - environment
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2
Q

What should you do after primary survey completed with trauma pt?

A

complete head-to-toe and neuro exam

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3
Q

Direct impact,
abrupt deceleration, continuous pressure,
shearing and rotational forces

A

blunt trauma

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4
Q

important thing to consider about trauma victims?

A

assume unstable c-spine until confirmed!!

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5
Q

thoracic blunt trauma consideration?

A

40% have pneumothorax that can’t be seen so avoid N2O!!

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6
Q

Hypotension, sub-cu emphysema, unilateral ↓BS, ↓ chest wall motion, distended neck veins, tracheal shift

A

tension pneumo

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7
Q

Rx for tension pneumo?

A

emergent needle aspiration 2nd ICS (above 3rd rib), MCL and chest tube

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8
Q

precaution you should take regarding meds used with pericardial tamponade pts?

A

careful during induction - dont want to knock out compensation - use ketamine, etomidate.
no prop/versed/etc

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9
Q

where is subxiphoid approach pericardiocentesis?

A

between xiphoid process and L costal margin 30-45 d angle

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10
Q

lethal triad?

A

acidosis
hypothermia
coagulopathy

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11
Q

damaged control surgery components

A

immediate control of bleeding
prevent lethal triad
limit crystalloid, increase products

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12
Q

what is FAST and why is it used?

A

focused assessment with sonography in trauma

-rapid assessment of blood to look for internal bleeding, cost effective and sensitive

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13
Q

A (ABCDE’s) major assumptions

A

no turning back, full stomach, c - spine concerns

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14
Q

best way to intubate trauma pt?

A

RSI with paralytic, stabilize neck!

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15
Q

B - breathing goals?

A

↓ TV, ↓ PIP (< 32 cm H20), avoid 02 toxicity

prevent barotrauma and ards

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16
Q

most deaths r/t to circulation are due to

A

coagulopathies, not on admit

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17
Q

stage I shock

A

blood volume normalized by shifting fluids

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18
Q

CV depression d/t ischemia,

thrombosis, toxins, cellular damage

A

Stage II (progressive) hem shock

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19
Q

Stage III (irreversible) hem shock

A

ATP depleted,
cellular death
[pt will die immediately or later down the road]

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20
Q

Minimize bleeding by maintaining
SBP

A

85-90 mmHg

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21
Q

When bleeding controlled, maintain bp

A

SBP >100 mmHg and HR <100

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22
Q

Replace EBL with

A

1:1 PRBCs, 3:1 crystalloid

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23
Q

using these fluids result in rapid restoration but ↑ risk of pulmonary
edema and bleeding.

A

Colloids

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24
Q

fluids best for perfusion

A

Isotonic crystalloids

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25
Maintain BG __ for suspected TBI or | cerebral edema.
<150 mg/d
26
Elevated PT on admission tells us pt more likely to have
massive hemorrhage, injury and poor perfusion | state
27
causes of Trauma-Induced Coagulopathy (TIC)
Dilution Hypothermia & acidosis TBI Shock
28
how to AVOID DILUTIONAL | COAGULOPATHY
Damage control resuscitation (DCR) | - DCS, rewarming, restricted crystalloids, permissive hypotension, balanced transfusion, massive transfusion protocol
29
Until targeted transfusion is available, empirically transfuse:
PRBCs/plasma/platelets at 1:1:1 (units) infusion
30
ASA recommends INR and plt count?
INR of ≤1.5*, PLT ct >50,000
31
Hypothermia probably alters
plt function and decreases fibrin formation
32
1°C drop causes
5% ↓ in clotting reactions
33
2 things in trauma pt that cause significant coagulopathy
Acidosis (pH <7.l) + hypothermia
34
pH of 7.2 does what to clotting
clotting function ↓ to 50% of noraml
35
pH of 6.8 does what to clotting
↓ to 20% of normal clotting
36
what does the T-T complex do?
activated protein C (APC) pathway
37
activated protein C (APC) pathway
Inhibits V & VIII Promotes fibrinolysis results in systemic anticoagulation
38
4 components of Assessment of blood consumption (ABC) score
Penetrating injury SBP <90 HR >120 Positive FAST
39
ABC score ≥ 2 =
↑ risk of needing massive | transfusion
40
TXA dose >12 years
1 gm bolus then 1 gm over 8 hrs
41
txa dose <12 years
5 mg/kg bolus then 2 mg/kg/hr for 8 hrs
42
timeframe TXA must be administered
<3 hours post-injury
43
according to BROHI what should you do?
check and make sure TXA bolus has been given! if not, then give bolus over 10 mins
44
Code red pack A MTP
6 U PRBCs | 4U FFP
45
code red pack B MTP
6 U prbc 4 u FFP 10 plt 2 cryo pools
46
what GCS should you intubate?
<8
47
C HTN = ICP
>10; Treat ICP >25 mmHg
48
clinical targets of neuro trauma pt
-Maintain MAP at 70-75 mmHg to maintain CPP at >50 mmHg -Moderate hyperventilation (PaCO2 of 30-35 mmHg) -Ventriculostomy (EVD) for monitoring and venting
49
drugs to avoid neuro trauma?
N2O ketamine etomidate
50
how to treat increased ICP?
ncremental propofol, moderate hyperventilation, mannitol (0.25–1 gm/kg), furosemide, head elevation.
51
TREAT ALL TRAUMA PTS AS
C-SPINE INJURED | UNTIL PROVEN OTHERWISE.
52
consideration when using succ with spinal chord injuries?
Fasciculations | can worsen SCIs
53
spinal shock triad?
Hypotension, bradycardia, hypothermia
54
anesthesia considerations for spinal chord injury pts?
prepare difficult intubation document pre op deficits heavy blood loss
55
anesthesia med considerations for spinal chord injury?
avoid succ and N2O | consider ket and dexmed
56
``` Massive SNS response d/t stimulus below level of spinal injury (frequently r/t bladder), most common above T5 ```
AUTONOMIC HYPERREFLEXIA
57
AUTONOMIC HYPERREFLEXIA triad
Hypertension, bradycardia, dysrhythmias
58
AUTONOMIC HYPERREFLEXIA can occur
``` during N2O/opioid GA or regional anesthesia (not seen with volatile agents) ```
59
rx HTN crisis with
direct acting vasodilators
60
Major risks: ortho
Hemorrhage, shock, fat emboli, PE emboli (especially with pelvic and long bones)
61
Injury to areas “junctional” to the trunk (pelvis, groin, perineum, axilla, neck)
JT is non-compressible
62
what does JUNCTIONAL | TOURNIQUET do
Compresses the aorta at the umbilical level
63
Most intra-op trauma deaths r/t
hyperkalemia, hypocalcemia, | acidosis.
64
later deaths from trauma r/t
PIICS (persistent inflammatory, immunosuppressed catabolic syndrom