Trauma Flashcards

(94 cards)

1
Q

What are the causes of dilated neck veins in a trauma patient?

A

Tension pneumothorax
Tamponade
Myocardial contusion (cardiogenic)
MI (cardiogenic)
Air embolism

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2
Q

What is the cardiac box

A

Nipples
Clavicles
Costal cartilages.

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3
Q

What are the indications to activate the MTP?

A
  • Replacement of 50% of blood volume within 1 hour.
    • Replacement of 100% of blood volume within 24 hours.
    • Haemorrhagic shock with active bleeding
    • Use of 4 units of RBC within 4 hours.
    • Physician concern
      • Majorly injured patient with high likelihood of transfusion requirement.
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4
Q

ATMIST - what does it stand for

A

Age
Time of incident
Mechanism of injury
Injuries sustained.
Signs and symptoms.
Treatment.

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5
Q

What does AMPLE stand for

A

Allergies
Meds
PMHx or pregnant
Last meal
Environment - patients family, responders

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6
Q

What is damage control and what are the principles?

A

A deliberate and pre-emptive set of non-traditional maneuvers used to reverse the pre-terminal effect of exsanguination, massive injury and shock

Goal is to temporize management of major injuries with directed resuscitation and staged surgery, to allow for the resuscitation and restoration of normal physiology

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7
Q

Why is temporary abdominal closure used?

A

Prevent heat and moisture loss and protect the abdominal viscera

Reduce the risk of abdominal compartment syndrome in a patient that is likely to have received a large amount of IVF resuscitation and undergo tissue interstitial oedema

Difficulty or inability to close the abdomen due to point B

Damage control surgery has taken place and a relook procedure is required

Stapled off bowel ends

Packs in place for haemorrhage control

Vascular shunts created for named vessels requiring definitive repair

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8
Q

Explain the pathophysiology of trauma

A

Ebb and Flow

Ebb
Short duration of severe shock and reduction in enzyme activity and O2 consumption
Hypovolaemia leads to decreased tissue perfusion causing release of catecholamines
Ebb is treated by good resuscitation

Flow (2 parts)

Hormonal mediators such as sympathetic system, cortisol, ADH and aldosterone leads to increased fluid conservation via sodium retention
Hyperdynamic state stimulated by inflammation

Catabolic phase - Fat/protein mobilization and weight loss
- Increased urine nitrogen excretion

Anabolic phase - Fat/protein store restoration and weight gain
- Normal/high - BGL, glucose production, FFA, insulin, catecholamines
- High - glucagon, O2 consumption, CO, temperature
Normal lactate

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9
Q

What is the definition of shock

A

Inadequate delivery of oxygenated blood to the tissues causing cellular hypoxia

Initially leading to reversible ischaemic injury then irreversible damage

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10
Q

What factors are used to grade hypovolaemic shock

A

Heart rate
Blood pressure
Pulse pressure
Respiratory rate

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11
Q

At what temperature and pH does the coagulation cascade stop working?

A

pH < 7.2
Temperatures < 34

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12
Q

What does FFP contain?

A

Volume 250mls
All clotting factors
Natural pro and anticoagulants (protein C and S, anti-thrombin and tissue factor pathway inhibitor)
Fibrinogen 800mg
Albumin

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13
Q

What does cryoprecipitate contain?

A

Volume 20mls
Fibrinogen
vWF/Factor VIII complex
Factor VIII

Benefit - rich source of fibrinogen in 1/8 of the volume.

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14
Q

How does tranexamic acid work?

A

TXA binds to plasminogen inactivating it
- prevents it being converted to plasmin
- plasmin breaks down fibrin
- thus assists with clot stabilisation

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15
Q

What are the risks of transfusion?

A

Hyperkalaemia
Infection
- HIV, CMV, EBV
Haemolytic transfusion reactions
- very rare with ABO compatability testing.
Immunomodulation in transplant patients
Graft versus host disease

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16
Q

How does TEG and ROTEM work?

A

○ Blood sample in cup with pin which oscillates creating fibrin strands.
○ For TEG the cup oscillates whereas for ROTEM the pin oscillates.
○ TEG uses a mechanical sensor whereas ROTEM uses and optical sensor.
○ In both tests a “clot activator” is given to stimulate the clot.

They are thus very similar tests - evaluating the formation of a clot around a pin.

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17
Q

Explain the TEG graph

A

R time
- Measures time for the blood to begin clotting after the addition of a clotting activator.
- Measures the beginning of fibrin formation.
- Prolonged in clotting factor deficiencies - thus treated with FFP

K time
- Kinetical phase
- Measures time it taken for the clot to reach a certain level of firmness (amplitude of 20mm)
- Measures fibrinogen level - treated with cryoprecipitate.

Alpha angle
- Slope between R and K
- Reflects rate at which fibrin cross linking occurs. Normally 50-70 degrees
- Represents fibrinogen
- Angle is decreased in low fibrinogen levels - treated with cryoprecipitate.

TMA - time to maximum amplitude
- Measures strength and firmness of the blood clot.
- Represents platelets (80%) and fibrin (20%)
- If MA is decreased - thrombocytopenia or reduced platelet function
- Treat with platelets or desmopressin.

AY30
- Measures clot lysis at 30 minutes
- Measure of fibrinolysis/plasminogen activation
Treated with tranexamic acid.

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18
Q

What is the definition of the massive transfusion protocol?

A

Definition
- administration of 100% of blood volume within 4 hours.
- administration of 50% of blood volume within 1 hour.

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19
Q

What are the end points for the MTP?

A

Active surgical bleeding has been controlled
Temp < 35
pH > 7.3
Fibrinogen > 1.5
INR < 1.5
Hb > 80

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20
Q

When assessing a limb trauma - what are hard signs of vascular compromise?

A

Pulselessness
active bleeding
Expanding haematoma.
Thrill or bruit.
Unilateral cool and pale periphery.

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21
Q

What dose the mangled limb severity score look at?

A

Score > 7 predicts high liklihood of needing an amputation

Skeletal/soft tissue injury
- low energy
- medium energy
- high energy
- very high energy

Limb ischaemia
- pulse reduced but perfusion normal
- no pules, perfusion cold
- insensate

Shock
- SBP > 90
- SBP transiently < 90
- SBP < 90

Age
- <30
- 30-50
- 50+

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22
Q

What are risk factors for a wound infection in an extremity?

A

Severity of injury
Delay from injury to surgical care (> 6hrs)
Failure to use prophylactic abx
Inappropriate wound toilet
Lack of coverage of bony structures
Inappropriate wound closure (e.g. primary closure in contaminated/contused wounds)
Open fracture wounds that are more likely to get infected

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23
Q

What is the pathophysiology of compartment syndrome?

A
  • Is an increase in the hydrostatic pressure within an enclosed oesteofascial space causing decreased perfusion of muscles and nerves within a compartment.
    • The pressure within the compartment increases until the intramuscular arteriolar pressures are exceeded
    • Means blood is not entering capillaries.
    • Cascade of events
      ○ Initially local trauma leading to bleeding and oedema - results in increased interstitial pressure
      ○ Vascular occlusion and subsequent myoneural ischaemia develops.
    • Leads to muscle ischaemia.
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24
Q

What are the landmarks for a lower limb fasciotomy - and what nerves do you need to avoid on both sides

A

Lateral
- 2cm below fibula head
- lateral malleolus.
- avoid common peroneal nerve and superficial peroneal nerve at distal aspect of the wound

Medial
- 2cm below tibial tuberosity
- 2cm above medial malleolus
- avoid the GSV and saphenous nerve

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25
What objectives can you achieve with a resuscitative thoracotomy?
- Cardiac tamponade decompression - Control intra-thoracic bleeding - Control air embolism or bronchopleural fistula - Permit open cardiac massage - Allow temporary descending aorta occlusion to limit abdominal haemorrhage
26
What its the WEST guidelines for an indication for a resuscitative thoractomy?
- Indication ○ Penetrating trauma <15 CPR ○ Blunt trauma <10 minutes of CPR ○ Penetrating neck or extremities with <5 minutes CPR ○ CPR with signs of life - Against Inverse of above
27
What are the endpoints for a resuscitative thoractomy?
○ Unrepairable cardiac or great vessel damage occurred ○ Massive head injuries identified (probably shouldn't start if identified prior) - No ROSC after 15 minutes of surgical procedure
28
Explain the Tiles classification of pelvic fractures
- Based on pelvic ring stability. - Type A - Complete stable ring with intact posterior arch. ○ A1 - iliac wing ○ A2 - pubic ramus ○ A3 - transverse fracture of the sacrum - Type B ○ Rotational instability (vertically stable) ○ B1 § Externally rotated unilateral. i.e. open book. ○ B2 § Internally rotated unilateral § Associated with bladder injury and shock. ○ B3 § Bilateral rotation. - Type C ○ Vertically unstable ○ C1 - unilateral disruption of posterior arch. ○ C2 - unilateral disruption of posterior arch with contralateral posterior arch incomplete disruption. - C3 - bilateral complete posterior arch disruption with spinopelvic dissociation.
29
How do you manage a liver trauma?
- Push, pack, pringle, then total vascular isolation. - Push ○ Restore the anatomy of the liver - Packing ○ Dry, folded packs with radio-opaque marker ○ Remove within 1-3 days (can cause infection) ○ Complications § Reduced CO if too tight and compressing IVC. § Respiratory deterioration if diaphragmatic movement restricted too much - Pringle ○ Diagnostic - hepatic artery/portal vein versus hepatic vein/caval bleeding. ○ Timing § 1 hour max § Less ischaemia with 10 minutes on 5 minutes off. ○ Sometimes need to finger fracture to get access to deeper bleeders within the parenchyma. ○ Can staple off or ligasure bleeding segments along anatomical planes. ○ Anatomical lobectomy has high mortality § Situations where anatomical lobectomy is needed □ Left lateral segment extensive injury - can't pack. □ Non-viable liver segment post-packing. □ Almost free segment. □ Devitalised liver at pack removal. - If ongoing bleeding despite Pringle's being applied ○ Means there is an injury to either a hepatic vein or retrohepatic IVC - very difficult injury to control with high mortality even in experienced hands ○ I would placed additional packs anteriorly to see if this can achieve haemostasis and allow transfer. ○ If this does not § Try and ascertain what side of the liver blood is coming from - consider mobilising right of left lobe to tie of corresponding hepatic vein. ○ If concern that injury is to retrohepatic IVC § Consider total hepatic exclusion Clamp inferior IVC above right kidney and suprahepatic IVC - repair IVC.
30
How do you perform a right medial visceral rotation and what does it allow access to?
○ Procedure § Mobilise the hepatic flexure and ascending colon via dividing the line of Toldt § Then mobilise the SB mesentery from caecum/RLQ to ligament of Treitz and reflect right colon to LUQ § Then Kocherise the Duodenum ○ Provides exposure to § Entire duodenum - particularly D3 and D4 □ Accessing D3 with SM vessels there □ Need to be retracted upwards § Pancreas - Head, neck and proximal body § Access to IVC and renal vessels Right kidney
31
How do you clamp the supra-coeliac aorta in trauma?
§ Open Pars flaccida § The oesophagus is retracted to the left § The peritoneum overlying the left crus is incised and the muscle fibers are split to expose the aorta. § The adventitia around the aorta is exposed and then the aorta is clamped with a Satinsky clamp. ****Often in reality it will be difficult to get a clamp on so you can use an "aortic compressor" and compress while you wait for help*****
32
How do you access the renal arteries surgically?
Right or left medial visceral rotation
33
How do you access the infrahepatic IVC?
Right medial visceral rotation with a Kochers maneuvre
34
How would you control bleeding from the retroehepatic IVC?
Right medial visceral rotation with a Kochers maneuvre and then clamp the infrahepatic IVC Mobilise the coronary ligaments superiorly and then clamp the suprahepatic IVC Mobilise the right lobe of the liver to identify injury - suture with 4/0 Prolene
35
How do you place a Shunt?
Select an appropriate shunt - the shunt length should be 3x the length of the defect. - the diameter of the tube should be 2/3 the diameter of the injured vessel. - should be an ETT, NG, chest drain - Place silk sutures around each third (i.e. 1/3 along and 2/3 along)
36
How do you manage a colonic injury found at trauma?
- Can be divided to non-destructive or destructive injuries ○ Non-destructive - <50% of the circumference of the bowel without any devascularisation - primary repair ○ Destructive - >50% of the circumference or with devascularisation - segmental resection - Colostomy as definitive procedure rather that anastomosis dependent on patient factors at time of re-look/definitive surgery. - Stomas should be placed laterally and at the level of the umbilicus and away from wounds.
37
What is the AAST liver grading system?
1. Subcapsular haematoma < 10% SA Laceration < 1cm depth 2. Subcapsular haematoma 10-50% SA Laceration 1-3cm depth. Intraparenchymal haematoma <10cm. 3. Subcapsular haematoma > 50% SA Laceration > 3cm depth Intraparenchymal haematoma > 10cm Active bleeding contained in parenchyma or vascular injury (AV fistula/pseudoaneurysm) 4. Parenchymal disruption 25-75% of a lobe. Active bleeding into the peritoneal cavity. 5. Parenchymal disruption > 75% Juxtahepatic venous injury - retrohepatic IVC and central hepatic veins.
38
What complications can occur with liver packing in trauma?
Reduced CO if too tight and compressing IVC. Respiratory deterioration if diaphragmatic movement restricted too much.
39
How do you grade a splenic trauma?
1. Subcapsular haematoma <10% SA Laceration < 1cm in depth. 2. Subcapsular haematoma 10-50% SA Laceration 1-3cm. Intraparenchymal haematoma < 5cm. 3. Subcapsular haematoma > 50% SA Laceration > 3cm. Intraparenchymal haematoma > 5cm. 4. Any injury + splenic vascular injury with contained blush in capsule. Parenchymal laceration with segmental/hilar involvement and >25% of spleen devascularised. 5. Shattered spleen Any injury + splenic vascular injury with blush into peritoneal cavity.
40
What is the criteria for non-operative management of a splenic trauma?
- Haemodynamically stable. - CT performed to classify the injury severity. - No other injuries which require surgery - Transfusion requirement is < 2 packed red cells. - Has to be in a facility which trauma laparotomy can be performed
41
Indications for angioembolisation?
Therapeutic - ongoing bleeding - dropping Hb - tachycardia - Blush on CTA - Pseudoaneurysm Prophylactic embolisation - Grade 4 or 5 injury - Reduces liklihood of these patients failing non-operative management.
42
What is the pathogenesis of OPSI?
- Insufficient opsonization filter function - Delayed and impaired production of immunoglobulins. - Lack of splenic macrophages. - Reduced number of memory B cells - usually produce IgM to promote clearance of encapsulated bacteria.
43
What are the operative options for pancreatic trauma?
NOM - For grade 1 and 2 found on imaging at time of surgery - Consideration to ERCP and stenting Operative - Grade 3/distal injuries with duct transection - distal pancreatectomy +/- splenectomy (as reduces operative time) - Grade 4/5 - pyloric exclusion with gastrojejunostomy.
44
What fracture is duodenal injuries associated with
Chance fracture of L1/2
45
What are some signs found at surgery of a duodenal injury
Bile staining of the tissues. Retroperitoneal haematoma Periduodenal haematoma. Periduodenal air bubbles
46
How are duodenal injuries graded?
1. Haematoma involving single portion of duodenum Partial thickness laceration, no perforation. 2. Haematoma involving more than one portion Disruption <50% of circumference. 3. Disruption of 50-75% of circumference of D2 Disruption of 50-100% of circumference of D1, D3, D4 4. Disruption of >75% of D2 Disruption involving ampulla or distal bile duct. 5. Massive disruption of duodenopancreatic complex. Devascularisation of the duodenum.
47
What are the key factors when considering how to repair a duodenal injury
Proximity to ampulla Injury charactersitics i.e. simple versus complex. Circumference Associated injuries - pancreas, biliary tree.
48
What are your options to duodenal trauma?
- Simple suture closure of simple wounds - Closure of longitudinal duodenotomy transversely if length <50% of circumference. - Primary anastomosis of complete transection - Diversion (gastrojejunostomy + pyloric exclusion, Bilroth II) - Consider cholangiogram if close to the ampulla/bile duct.
49
AAST grading for renal injury?
1. Subcapsular haematoma Parenchymal contusion No laceration 2. Laceration <1cm with no urine extravasation Peri-renal haematoma confined to Gerotas fascia. 3. Laceration >1cm with no urine extravasation Any injury with renal vascular injury or active bleeding confined to Gerota's fascia. 4. Parenchymal laceration into collecting system with urine extravasation. Renal pelvis laceration and/or complete uretero-pelvis disruption. Segmental renal vessel injury. Active bleeding beyond Gerotas fascia. Segmental infarction secondary to vessel thrombosis. 5. Shattered kidney. Devitalised kidney. Hilar vessel injury.
50
Why is non-operative management favourable/often successful in renal trauma?
Haematoma displaces renal tissue Closed retroperitoneal space to tamponade it. Kidney is rich in tissue factor - thus stimulates the extrinsic coagulation cascade aiding in haemostasis
51
What electrolyte disturbances do you get with an intraperitoneal bladder injury?
High creatinine High urea Low sodium - peritoneum re-absorbs urine (which is usually low in sodium) resulting in a dilution hyponatraemia
52
What are CI to non-operative management of extra-peritoneal bladder injuries?
Bladder neck injury Presense of bony fragments in bladder wall. Infected urine Associated female genital tract injury.
53
How do you grade oesophageal injury?
Grade 1 - contusion/haematoma. Partial thickness laceration Grade 2 - full thickness laceration < 50% circumference. Grade 3 - full thickness laceration >50% circumference. Grade 4 - segmental loss/devascularisation < 2cm. Grade 5 - segmental loss/devascularisation > 2cm.
54
What is the broad management of tracheobronchial fistula?
Proximal injury - Stent - Repair primarily Distal bronchial injury - Will often resolve with a chest drain alone - If persistent - or impaired ventilation - Posterolateral thoracotomy and repair with monofilament suture OR segmental lobar resection. If they don't settle with multiple drains - need to perform bronchoscopy to confirm proximal injury then operate.
55
How are diaphragmatic injuries graded?
1. Contusion 2. Laceration < 2cm. 3. Laceration 2-10cm. 4. Laceration >10cm with < 25cm2 tissue loss 5. Laceration > 10cm with > 25cm2 tissue loss
56
What is a flail chest and why is it a problem?
3 or more contiguous ribs all fractured in 2 spots creating a floating segment which loses mechanical continuity and thus move paradoxically.
57
What are the indications for rib fixation in trauma?
Early - Respiratory failure - either impending or diagnosed. - Severe deformity of chest wall. - Failure to wean off ventilator. Late - Mal or non-union of the ribs (i.e. painful clicking)
58
What are the benefits of acute rib fixation?
- Reduced need for intubation. - Easier to wean ventilator. - More likely to wean ventilator. - Reduced mortality, chest morbidity (pneumonia), LOS.
59
What is the pathophysiology of a flail chest?
- A flail chest is 3 or more contiguous ribs broken in 2 or more places creating a segment of chest which moves independently and movers paradoxically with breathing. - When the patient breaths in the flail segment gets sucked in, damaging the lung parenchyma worsening the chest injury. This also results in ineffective ventilation.
60
What can occur with a broncho-pulmonary fistula?
Depends on the pressure differential If from bronchus to vein - will get air embolism. If from vein to bronchus - will get massive haemoptysis.
61
What symptoms/signs does an air embolism cause?
- Focal/lateralizing neurology - Sudden CV collapse - air embolism to coronaries. - Frothy blood sample - Neurology in the absence of a head injury and the presence of a chest injury. - Fundoscopy - air in retinal vessels.
62
What is the aetiology of spinal shock and neurogenic shock
Spinal shock - occur due to inflammation of the cord causing vasoconstriction - results in flaccid paralysis, loss of sensation and reflexes, as well as autonomic instability. - is temporary - can occur at any level Neurogenic shock. - occurs in high spinal cord injuries due to disruption of the sympathetic outflow - results in hypotension and bradycardia. - generally don't get flaccid paralysis and loss of sensation.
63
What factors determine traumatic brain injury severity?
Loss of consciousness Amnesia. Imaging. GCS
64
What is severe TBI?
LOC and post traumatic amnesia > 24 hours with GCS 8 or less
65
How do you calculate the cerebral perfusion pressure? What is normal?
MAP - ICP Normal is 60-70mmHg
66
What is the pathophysiology of TBI?
Primary insult. Mechanical injury leads to excessive glutamine release which results in cellular influx of calcium ATPase pump failure occurs which drives cellular oedema. - The blood-brain barrier is disrupted which drives cerebral oedema - CPP will drop if the MAP is not elevated. - This can result in cerebral ischaemia. Lactic acid levels increase from anaerobic metabolism.
67
Why do you have to increase the MAP in patients with TBI?
To counter the increased ICP as CPP = MAP - ICP
68
What are the indications for a rural centre Burr hole?
Neurologically deteriorating patient Sizebale intracranial haematoma. Nearest surgical unit > 2 hours away.
69
What are the indications for a Burr hole?
GCS 8 or less with epidural haematoma causing midline shift.
70
What are the landmarks for Burr hole drainage?
Frontal - 10cm above the pupli and in the mid-pupillary line Parietal - 5cm superior to the ear Temporal - 3cm superior to the posterior zygomatic arch.
71
What is the classification of Blast injury?
Primary - Due to direct pressure or Barotrauma from the shockwave of the blast - At risk organs are gas filled - Lung, bowel, tympanic membrane, ocular injuries, concussion. Secondary - Caused by projectiles and debris propelled by the explosion - fragmentation injury - Results in penetrating trauma, lacerations, fractures. Tertiary - Displacement injury - caused by displacement of the body from the blast wind - Results in blunt trauma and fractures - Can also cause acceleration/deceleration injuries, soft tissue destruction. Quaternary - Due to injuries from event during trauma - Burns, inhalation of toxic gas or smoke, exacerbation of pre-existing injuries. Quinary - Injury caused by exposure to toxins from blast - Ionizing radiation, radiation sickness etc.
72
What is blast lung?
Blast lung injury results in tissue disruption at the capillary-alveolar interface. Causes - pulmonary oedema, parenchymal damage and air embolism. Clinical diagnosis based on hypoxia, respiratory distress, and bilateral lung infiltrates on Chest Xray. Supportive management - ICU, mechanical ventialtion, low pressure ventilation to avoid barotrauma, chest physio, nebulisers.
73
What is the pathophysiology of Rhabdomyolysis?
Myoglobin is released into the plasma from crushed muscle which overhelms the haptoglobin pick up rate. Results in - Nephrotoxity and AKI
74
What is the treatment of Rhabdomyolysis?
Aggressive fluid resus - 2ml/kg/hour May need dialysis May need fasciotomies
75
Hard signs of injury in the neck?
Vascular - Rapidly expanding, pulsatile, or largesu haematoma - Refractory shock. - Palpable thrill or bruit. - Shock unresponsive to IV fluid resuscitation. - Absent or diminished radial pulse. - Neurological deficit consistent with cerebral ischaemia. Hard signs of aero-digestive injury - air bubbling from the wound. - massive haemoptysis. - respiratory distress
76
What are the zones of the neck?
1. Sternal notch to lower border of cricoid cartilage 2. Cricoid cartilage to angle of the mandible. 3. Angle of the mandible to base of the skull.
77
What incision would you perform for a zone 1 penetrating neck injury in an unstable patient?
Median sternotomy If you are having difficultly accessing the left proximal subclavian you can perform an infra-clavicular extension.
78
How do you expose the carotid artery?
Incision along medial border of SCM with lateral mobilisation of the SCM. The common facial vein is the landmark used to identify the carotid bifurcation - this should be ligated - omohyoid should be divided.
79
How do you access the central compartment of the neck?
Incision along medial border of SCM which is lateralised. Three structures need to be divided - middle thyroid vein. - inferior thyroidal artery - omohyoid.
80
Denver grading system for blunt cerebrovascular injury?
1. Intimal transection with <25% of luminal narrowing. 2. Dissection or intramural haematoma with >25% of luminal narrowing or visible intimal flap 3. Pseudoaneurysm or a haemodynamically insignificant AV fistula. 4. Complete vessel occlusion. 5. Vessel transection or a haemodynamically significant fistula.
81
Signs and symptoms which suggest blunt cerebrovascular injury?
Arterial haemorrhage from neck and nose or mouth. Expanding neck haematoma. Cervical bruit. Focal neurological deficit. Horners syndrome. Stroke seen on imaging.
82
What is the NEXUS criteria to clear the C-spine
- 2 exam findings ○ No posterior midline tenderness ○ No abnormal neurological findings. - 3 reasons which would make findings invalid. ○ No painful distracting injury. ○ Normal level of alertness. - No evidence of intoxication.
83
What are the ABCs of cervical spine imaging?
A - Visually trace 4 lines (anterior, posterior, spinolaminar, and posterior spinous space) - Look for widening between vertebra B - Overt fractures or lucency - Disruption of C1 C - Widened of narrowed disc spaces. S - Widened pre-vertebral space - can be a sign of blood or oedema associated with a fracture. - C2 space should measure <7mm - C6 space should measure < 22mm.
84
What are the three zones for REBOA
1. Left subclavian to coeliac 2. Coeliac to renal vessels - NO GO ZONE 3. Renal vessels to bifurcation
85
What are the extubation criteria?
- S - secretions are minimal - O - oxygenation is good - A - alert - A - airway is not injured or compromised. - P - pressure or parameters are satisfactory - Tidal volume etc.
86
Describe Jackson Burn classification
Jackson's Burn Wound Mode 3 zones Coagulation - Protein coagulation and cell death - Irreversible necrosis Stasis - Viable but at risk due to impaired perfusion and inflammation - Can be saved with good aggressive and early resuscitation Hyperaemia - Outermost zone with minimal injury - Viable tissue with expected full recovery
87
What are the injuries you can get from an electrical burn?
- Entry and exit burn - Clots to blood vessels and damage to nerves - Damage to muscles - causing rhabdomyolysis and compartment syndrome - Muscle spasm causing dislocations and compression fractures. - Cardiac rhythm problems
88
What is the pathophysiology of an alkali burn?
o Mechanism * Alkalis cause liquefactive necrosis * Hydroxide ions saponify cell membrane lipids and denature proteins. * Resulting in deep tissue penetration and ongoing damage. o Tissue effects * Liquefaction allows the agents to spread rapidly through tissue planes * Can continue causing damage long after contact unless neutralised or removed. o Clinical course * Damage is often underestimated initially due to the limited surface signs * Tissues may appear pale and slippery before progressive necrosis occurs. * Examples - oven cleaners, drain cleaners, industrial agents
89
What is the pathophysiology of an acid burn?
o Mechanism * Cause coagulation necrosis * Hydrogen ions denature proteins, leading to eschar formation. * The eschar limits deep penetration of the acid. o Tissue effects. * Injury tends to be more superficial compared to alkalis * Coagulation of proteins forms a thick eschar that impedes further spread. o Clinical course * Burn is usually well demarcated and painful. * May evolve over hours depending on concentration and contact duration.
90
What are the problems you can get associated with inhalation injury?
* Inhalation injury can be very morbid - associated with burns in an enclosed space o Facial burns o Singed hair o Airway oedema/stridor. o Carbonaceous sputum/oral deposits. o Carboxyhaemoglobin > 10% o Hoarse voice. Burns in an enclosed space can result in carbon monoxide poisoning * Can have a normal sats as CO has displaced oxygen * Should measure carboxyhemoglobin.
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What is the modified parkland formula?
4 x TBSA burn x kg - give first half over 8 hours and next half over next 16 hours. - titrate to a urine output of 1ml/kg/hour (or 2ml/kg/hour for children)
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When should you refer a patient to a burns center?
- >10% TBSA adult - >5% TBSA child - Special areas - face, hands, feet, perineum, major joints. - Circumferential limbs/chest. - Inhalation injury - Electrical/chemical burns. - Extremes of age. - Burns with complex co-morbidities
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What is the anatomy and aetiology of a Zenker's diverticulum
Killian’s triangle is between oblique fibers of the thyropharyngeus, and horizontal fibers of the cricopharyngeus muscle (both of which make up the inferior constrictor) Discordinated relaxation between the pharyngeus muscles and cricopharyngeus results in a pulsion diverticulum.
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