Trauma N Burns Flashcards

(65 cards)

1
Q
  1. Q1. What is the first priority after securing the airway in a burn patient?
A
  1. A1. Managing life-threatening injuries and aggressively resuscitating with large volumes of intravenous fluids.
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2
Q
  1. Q2. Why is fluid administration critical in burn patients?
A
  1. A2. It is crucial for patient survival and preventing renal failure.
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3
Q
  1. Q3. What type of shock is caused by burns and what changes are observed?
A
  1. A3. Hypovolemic shock, characterized by loss of circulating plasma volume, hemoconcentration, massive edema formation, decreased urine output, and depressed cardiovascular function.
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4
Q

When are fluid losses greatest after a burn injury?

A

In the first 12 hours after burn injury, stabilizing after 24 hours.

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5
Q
  1. What are the components of the primary survey in trauma care?
A
  1. The primary survey in trauma care involves assessing Airway, Breathing, Circulation, Disability (neurologic status), and Environment/exposure.
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6
Q
  1. Describe the goal of the primary survey in trauma care.
A
  1. The goal of the primary survey is to rapidly identify and manage life-threatening conditions or injuries.
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7
Q
  1. What does the secondary survey involve?
A
  1. The secondary survey is a comprehensive head-to-toe assessment that includes a neurologic examination, performed after stabilization efforts from the primary survey.
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8
Q
  1. Explain the difference between blunt and penetrating trauma.
A
  1. Blunt trauma results from high-energy impacts such as MVCs and falls, causing injuries due to deceleration, shearing, and other forces. Penetrating trauma involves direct entry of objects into tissues.
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9
Q
  1. What are the types of motor vehicle collision (MVC) traumas?
A
  1. MVC traumas include head-on, rear impact, side impact, rotational impact, and rollover, causing injuries above and below the waist.
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10
Q

What injuries are associated with blunt thoracic trauma?

A

Blunt thoracic trauma commonly affects the chest wall, lungs, heart, pericardium, and airways, often leading to significant morbidity and mortality.

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11
Q

What is tension pneumothorax?

A

Tension pneumothorax occurs when air enters the pleural space and cannot escape, leading to progressive collapse of the affected lung and displacement of mediastinal structures.

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12
Q

What are the symptoms of tension pneumothorax?

A

Symptoms of tension pneumothorax include hypotension, decreased breath sounds on one side, hyperresonance on percussion, and tracheal deviation.

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13
Q
  1. How is tension pneumothorax managed emergently?
A
  1. Emergent management of tension pneumothorax includes needle decompression followed by chest tube thoracostomy.
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14
Q
  1. What is pericardial tamponade and how is it managed?
A
  1. Pericardial tamponade restricts cardiac filling, leading to low cardiac output and hemodynamic instability. Management involves pericardiocentesis to relieve pressure on the heart.
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15
Q
  1. What are the anesthetic considerations for massive hemothorax?
A
  1. Massive hemothorax requires adequate fluid and blood resuscitation before chest tube placement to prevent hypotension and further bleeding complications.
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16
Q

How should anesthesia be managed in patients with cardiac rupture?

A

Patients with cardiac rupture require rapid prehospital transport and immediate surgical intervention due to high mortality rates in the prehospital setting.

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17
Q
  1. What are the hemodynamic goals in the management of traumatic aortic rupture?
A
  1. Hemodynamic management in traumatic aortic rupture aims for a systolic blood pressure (SBP) less than 100-120 mm Hg and a heart rate less than 100 bpm to minimize the risk of extending the aortic injury.
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18
Q
  1. How are tracheal injuries typically managed in the acute setting?
A
  1. Tracheal injuries may be managed with intubation to bridge the transected tracheal parts or surgical correction. Total transection requires rapid surgical retrieval of the distal segment for lifesaving mechanical ventilation.
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19
Q
  1. What are the preferred pharmacological agents used to achieve hemodynamic control in traumatic aortic rupture?
A
  1. Short-acting β-blockers, calcium channel blockers, and/or vasodilators like nitroprusside are used to achieve hemodynamic control in traumatic aortic rupture, aiming to stabilize blood pressure and heart rate to prevent further aortic damage.
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20
Q

Q1:** What is the primary goal during the primary survey of trauma care?

A

A1:** Identifying and rapidly managing life-threatening conditions or injuries.

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21
Q

Q2: What imaging techniques are utilized during the primary survey to assess injuries?

A

A2: Ultrasound and radiography.

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22
Q

Q3: Why is coordination between surgical and anesthesia teams essential during the primary survey?

A

A3: To ensure efficient management and preparation for potential surgical intervention.

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23
Q

Q4: When does the secondary survey begin in trauma care?

A

A4: After the primary survey and the initiation of resuscitative and stabilization efforts.

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24
Q

Q5: What does the secondary survey focus on?

A

A5: A more complete head-to-toe assessment, including a neurologic examination.

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25
**Q6:** What causes blunt trauma?
**A6:** Direct impact, deceleration, continuous pressure, shearing, and rotary forces.
26
**Q7:** How should all blunt trauma victims be treated regarding their cervical spine?
**A7:** They should be suspected of having an unstable cervical spine.
27
**Q8:** What is the third most common type of blunt injury?
**A8:** Blunt chest trauma.
28
**Q9:** What percentage of trauma-related deaths is chest trauma a major contributor to?
**A9:** 50% of trauma-related deaths.
29
**Q10:** What is a common complication of blunt thoracic trauma?
**A10:** Pneumothoraces, present in as many as 40% of all blunt thoracic injuries.
30
What significant sign indicates a developing pneumothorax during surgery?
Reduced tidal volumes and decreased oxygen saturation.
31
**Q12:** Why should nitrous oxide be avoided in patients with suspected thoracic trauma?
**A12:** It can increase the size of a pneumothorax during positive pressure ventilation.
32
**Q13:** What determines the extent of damage in penetrating trauma?
**A13:** The type of wounding instrument, the velocity of the projectile, and the tissue characteristics it passes through.
33
What is negative imbibition pressure and how does it affect fluid loss in burn injuries?
Negative imbibition pressure is a strong negative interstitial pressure within injured tissue causing fluid absorption. It results in large fluid losses and demands during resuscitation. The magnitude of burn injury increases this negative pressure, leading to early hypovolemia despite fluid therapy.
34
Q: What do current guidelines for fluid resuscitation not fully account for, and what happens to fluid volume after a burn injury?
Current fluid resuscitation guidelines, especially those based on crystalloids, do not fully account for early fluid loss, leading to a controlled hypovolemic state in the first 12-16 hours post-burn. Tissue edema peaks between 24-48 hours, and fluid is gradually returned to circulation and excreted over 7-14 days, potentially causing lung dysfunction due to hypervolemia.
35
Q: What are the characteristics and hemodynamic alterations of severe burn shock?
Severe burn shock is both distributive and hypovolemic in nature, with increased systemic vascular resistance (SVR) due to the release of catecholamines, antidiuretic hormone, and hemoconcentration, which compounds the adverse effects of the shock phase.
36
What state develops 48 to 72 hours after a burn injury and what are its characteristics?
A hyperkinetic and hypermetabolic state develops 48 to 72 hours after burn injury, characterized by decreased vascular permeability, increased heart rate, and decreased systemic vascular resistance (SVR), resulting in increased cardiac output.
37
What heart rate is considered normal in a burn patient?
100-120
38
What fluid resuscitation formula is most commonly used in the burn patient?
Parkland formula= 4ml/kg crystalloids for every% TBS burned. Given over 24hours half of which is given in the first 8 hours.
39
What percentage of TBSA can burns be managed with oral intake or 1.5 times hourly fluid requirement?
<15%
40
Fluid resuscitation formulas in the burn patient call for a target urine output of what?
0.5ml/kg/hr in adults (or 30-50ml/hr) and 1ml/kg/hr in children
41
What are the signs of resuscitation failure in the burn patient?
low urine output, repeated episodes of hypotension or need for vasopressors, worsening of base deficit, or fluid infu- sion in excess of predicted resuscitation needs in the first 24 hours
42
In regards to fluid resuscitation, when does the risk for abdominal compartment syndrome increase?
When administered fluid volumes exceed 250ml/kg/day
43
After what administered volume should you check cardiac function in the burn patient?
6ml/kg/%TBA
44
What are the normal abdominal pressures and which necessitate intervention?
Normal: 5-10mmhg Intervention: >25mmhg
45
What defines ACS?
Abdominal pressure exceeding 25mmhg with new onset organ disfunction (oliguria, decreased lung compliance)
46
Patients with high voltage injuries should be assessed for what?
Rhabdo, compartment syndrome, vertebral fractures
47
Before initiating frostbite warming, what temperature should the body be heated to?
35 degrees celcius
48
Significant difference between children and adult patients in burns?
Children have higher surface area to volume ratio, thinner skin, greater fluid requirements and fluid loss to evaporation, higher incidence of hypothermia
49
Besides TBSA burned, what is the most important morbidity factor?
Age
50
General metabolic factors in the burn patient?
increased resting energy expenditures, increased myocardial oxygen consumption, marked tachy- cardia, increased body temperature, glycolysis, proteolysis, lipolysis, and futile substrate cycling.
51
Nutritional needs of the burn patient are increased by what percent?
200% increase in basal energy requirement
52
In the preop assessment of the burn patient, special attention should be paid to what?
time and extent of burn injury, airway evaluation, presence of inhala- tion injury, quantity of fluid received, current resuscitation regimen and the patient’s response, vascular access/sites, and tolerance of enteral feeding and NPO status
53
In regards to the burn patients airway, what can be said regarding the musculature?
Patients masseter and neck muscles can be stiff/contracted, making displacement to the submandibular area difficult
54
During the acute phase of burn injury (first 24-48hours) what can be said about CO? How does this effect pharmicokinetics?
Decreased CO and intravascular volume decreasing metabolism and clearance of drugs.
55
After resuscitation of the burn patient, they go into the hyperdynamic phase. What does this entail pharmacologically?
Drugs dependent on organ blood flow (propofol, fentanyl) will have faster clearance and may require increased dosing.
56
What happens to plasma proteins in the burn patient?
Albumin decreases, while AAG increases twofold.
57
What happens to phase I and II reactions in the burn patient?
Decreased phase I reactions (oxidation, reduction, hydroxylation, demethylation), phase II is unaffected (conjugation, glucuronidation, and sulfation).
58
What are the effects of burns on NMBA?
Succs generally avoided after 24-48 hours of burn due to severe Hyperkalemia. NDNMBA require increased dosages due to up regulation of ACH and fetal (neuronal) ach receptors, increased AAG levels, and increased clearance by liver and kidneys
59
What is the dose of rocuronium in the burn patient?
1.2mg/kg-1.5mg/kg
60
What is the onset time of ROC in the burned patient compared to the unburned?
90 seconds in the burned patient compared to 60 in the unburned.
61
Why do patients with extensive burns have difficulty thermorefulating (neurologically)
The inflammatory response to large burns causes an increase in the hypothalamic core temperature set point and the metabolic rate is increased to maintain this increased temperature.
62
What can be given to persistent post operative pain that is resistant to opiates?
0.25mg/kg of ketamine IV.
63
Mild shock base deficit
2-5 mmol/L
64
Moderate shock base deficit?
6-14 mmol/L
65
Severe shock base deficit
>14mmol/L