Trauma & Orthopaedics Flashcards

(195 cards)

1
Q

Risk factors for primary OA?

A

obesity, advancing age, female gender, and manual labour occupations

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2
Q

Differentials for OA in the hands?

A

De Quervain’s tenosynovitis, rheumatoid arthritis, and gout

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3
Q

Differentials for OA in the hip?

A

trochanteric bursitis, radiculopathy, spinal stenosis, or iliotibial band syndrome

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4
Q

Differentials for OA in the knee?

A

meniscal or ligament tears, or chondromalacia patellae

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5
Q

General differentials for OA?

A

inflammatory arthropathies (e.g. rheumatoid arthritis), crystal arthropathies (e.g. gout or CPPD), septic arthritis, fractures, bursitis, or malignancy (primary or metastatic)

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6
Q

Classic radiological features of OA?

A

Loss of joint space
Osteophytes
Subchondral cysts
Subchondral sclerosis

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7
Q

Outline management of OA

A

Conservative:
weight loss, strengthening exercises, local heat packs, joint support, physio

Medical:
simple analgesics and NSAIDs
intra-articular steroid injections (can cause steroid flare)

Surgical:
mainstay of management is with arthroplasty, however other options include osteotomy and arthrodesis (joint fusion)

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8
Q

What is the most important adage to remember for the surgical management in traumatic orthopaedic complaints?

A

‘Reduce – Hold – Rehabilitate’

In the context of high-energy injuries, this is precluded by resuscitation following ATLS principles

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9
Q

Reduction involves restoring the anatomical alignment of a fracture or dislocation of the deformed limb.

Reduction allows for what 4 things to occur?

A
  1. Tamponade of bleeding at the fracture site
  2. Reduction in the traction on the surrounding soft tissues, in turn reducing swelling
  3. Reduction in the traction on the traversing nerves, therefore reducing the risk of neuropraxia
  4. Reduction of pressures on traversing blood vessels, restoring any affected blood supply

Fracture reduction is typically performed closed in ED. However, some fractures need to be reduced open intraoperatively

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10
Q

What are the clinical requirements for fracture reduction?

A

Analgesia
- where regional or local blockade is sufficient and easily provided (e.g. phalangeal/metacarpal/distal radius fractures), this is the method of choice

short period of conscious sedation often in anaesthetic room

3 staff members - one to perform the reduction manoeuvre and one to provide counter-traction, with a third person needed to apply the plaster.

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11
Q

What is meant by ‘hold’ in fracture management?

A

generic term used to describe immobilising a fracture

consider whether traction needed - e.g. where the muscular pull across the fracture site is strong and the fracture is inherently unstable

most common ways to immobilise a fracture are via simple splints or plaster casts

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12
Q

When applying a plaster cast, the most important principles to remember are what?

A

For the first 2-weeks, plasters are not circumferential: (not always the case in children)
- They must have an area which is only covered by the overlying dressing, to allow the fracture to swell; if not the cast will become tight (and painful) overnight, and if left the patient is at risk of compartment syndrome

If there is axial instability ( the fracture is able to rotate along its long axis), e.g. combined tibia-fibula metaphyseal fractures or combined radius-ulna metaphyseal fractures, the plaster should cross both the joint above and below:
- usually termed ‘above knee’ or ‘above elbow’ plasters, respectively, preventing the limb to rotate on its long axis

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13
Q

What is important to consider when initiating fracture immobilisation?

A

Can the patient weight bear?

Do they need thromboprophylaxis?
If the patient is immobilised in a cast and is non-weight bearing, it is common to provide thromboprophylaxis

Have you provided advice about the symptoms of compartment syndrome?

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14
Q

What is the most important investigation when investigating an acute monoarthritis?

A

joint aspiration

The aspirate can be sent for white cell count and MCS, as well as light microscopy (for crystals)

aspiration of prosthetic joints should be done in theatre due to infection risk

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15
Q

What will synovial fluid appear like in a non- inflammatory arthritis, inflammatory arthritis and septic arthritis?

A

non- inflammatory arthritis - clear/straw coloured
inflammatory arthritis - clear/cloudy yellow
septic arthritis - turbid

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16
Q

What will the WCC look like in a non- inflammatory arthritis, inflammatory arthritis and septic arthritis?

A

non- inflammatory arthritis - moderate <2000
inflammatory arthritis - high >2000
septic arthritis - very high >50,000

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17
Q

What is septic arthritis?

A

infection of a joint most commonly caused by S. aureus

It is important that it is identified and treated quickly as it can cause irreversible articular cartilage damage or overwhelming sepsis and mortality

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18
Q

What are Spondyloarthropathies?

A

group of conditions comprising of Psoriatic Arthritis, Ankylosing Spondylitis, Reactive Arthritis, and Enteropathic arthropathy

seronegative conditions (RF negative)
associated with HLA-B27

all can present with “axial arthritis” (those affecting the spinal and SI joints)

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19
Q

What is haemarthrosis?

A

Bleeding into a joint cavity

most commonly due to trauma although can also be caused by bleeding disorders and anti-coagulation

may also be a concurrent ligamentous or meniscal injury that has specifically caused the bleeding (e.g. ACL containing a genicular artery)

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20
Q

When is a fracture considered to be ‘open’?

A

when there is a direct communication between the fracture site and the external environment

most often through the skin – however, pelvic fractures may be internally open, having penetrated in to the vagina or rectum

may become open by either an “in-to-out” injury, ( sharp bone ends penetrate the skin from beneath) or an “out-to-in” injury, where a high energy injury (e.g. ballistic injury or a direct blow) penetrates the skin

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21
Q

What are the most common open fractures?

A

tibial, phalangeal, forearm, ankle, and metacarpal

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22
Q

Why is the rate of infection so high following open fractures?

A

direct contamination, reduced vascularity, systemic compromise (such as following major trauma) and need for insertion of metalwork for fracture stabilisation

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23
Q

What should you check for on examination of an open fracture?

A

neurovascular status
overlying skin / tissue loss
evidence of contamination - marine, agricultural, and sewage contamination is of the highest importance
identify need for plastics early

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24
Q

The Gustilo-Anderson classification can be used to classify open fractures. Outline Types 1 through to 3C

A

Type 1: <1cm wound and clean

Type 2: 1-10cm wound and clean

Type 3A: >10cm wound and high-energy, but with adequate soft tissue coverage

Type 3B: >10cm wound and high-energy, but with inadequate soft tissue coverage

Type 3C: All injuries with vascular injury

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25
How can open fractures be managed?
1. rescucitation and stabilisation 2. realignment and splinting 3. reassess neurovascular status 4. broad spectrum abx and tetanus vaxx if not up to date 5. photograph wound and remove gross debris 6. dress wound with saline-soaked gauze
26
Definitive surgical management of open fractures requires debridement of the wound and the fracture site, removing all devitalised tissue present. When should this happen?
either immediately if contaminated with marine, agricultural, or sewage material, or <12-24 hours in all other cases early surgical exploration by vascular if evidence of vascular compromise
27
When should soft tissue coverage of open fractures happen?
within 72 hours, or as guided by plastic surgeon advice
28
What is compartment syndrome?
critical pressure increase within a confined compartmental space any fascial compartment can be affected, however the most common sites affected are in the leg, thigh, forearm, foot, hand and buttock
29
What is the pathophysiology of compartment syndrome?
typically occurs following high-energy trauma, crush injuries, or fractures that cause vascular injury - can also be due to tight casts, DVT, and post-reperfusion swelling Fascial compartments are closed and cannot be distended so extra fluid = increase in the intra-compartmental pressure veins compressed first, then nerves, then arteries as pressure matches the diastolic pressure
30
The most reliable symptom of compartment syndrome is severe pain. Describe this pain. Aggravating factors?
severe pain, disproportionate to the injury not readily improved with initial measures (such as analgesia, elevation to the level of the heart, and splitting a tight cast) pain is made worse by passively stretching the muscle bellies traversing the affected fascial compartment
31
Compartment syndrome is a clinical diagnosis. What diagnostic test can be used when there is uncertainty?
intra-compartmental pressure monitor used in atypical presentations or if the patient is unconscious / intubated CK may also aid diagnosis
32
What are normal compartmental pressures?
0-8 mmHg
33
How should compartment syndrome be managed?
early recognition and immediate surgical treatment via urgent fasciotomies other steps: Keep the limb at a neutral level with the patient High flow O2 Augment BP with bolus of IV crystalloid fluids ( transiently improves perfusion of the affected limb) Remove all dressings / splints / casts, down to the skin Opioid analgesia
34
What should be done post-fasciotomy?
skin incisions are left open and a re-look is planned for 24-48 hours - assess for any dead tissue that needs to be debrided Monitor renal function closely, due to the potential effects of rhabdomyolysis or reperfusion injury
35
What is osteomyelitis?
infection of the bone - mostly acute bacterial origin in adults, the vertebrae are the most commonly affected bones (in children, long bones) caused by haematogenous spread, direct inoculation (such as following an open fracture or penetrating injury), or direct spread from nearby infection (such as a contiguous joint)
36
Most common causative organism of osteomyelitis?
staph aureus most common P. aeruginosa - intravenous drug users Salmonella spp - patients with sickle cell disease
37
Risk factors for osteomyelitis?
diabetes mellitus immunosuppression (such as long term steroid treatment or AIDS) alcohol excess IVDU
38
What is Potts disease?
infection of the vertebral body and intervertebral disc by Mycobacterium tuberculosi Patients will present with back pain +/- neurological features MRI gold standard investigation
39
X-rays are often performed for osteomyelitis although they have poor accuracy - any signs tend to only be visible from ~7-10 days post-initial infection. What might they show? What other investigations can be performed?
osteopaenia, periosteal thickening, endosteal scalloping, and focal cortical bone loss Definitive diagnosis can be achieved through MRI imaging Gold standard diagnosis is from culture from bone biopsy at debridement (or curettage where there are associated ulcers) - important to check for TB and fungus in immunosuppressed
40
How can osteomyelitis be managed?
If the patient is clinically well, patients will require long-term IV abx (>4 weeks) tailored to any cultures available If the patient clinically deteriorates, the limb shows evidence of deterioration, or imaging shows progressive bone destruction, then surgical management may be required
41
Complications of osteomyelitis?
septic arthritis or soft tissue infections overwhelming sepsis recurrence of infection - esp with early discontinuation of abx children may develop growth disturbances as a result of premature physeal closure
42
How will patients with chronic osteomyelitis present? How can it be managed surgically?
localised ongoing bone pain and non-specific infection symptoms (e.g. malaise or lethargy) may be a draining sinus tract and they may have difficulties in mobility Mx: local bone and soft tissue debridement for definitive source control, alongside extensive long-term abx
43
What is a radiculopathy?
a conduction block in the axons of a spinal nerve or its roots motor axons = weakness sensory axons = parasthesia
44
What is the distinction between radiculopathy and radicular pain
Radiculopathy is a state of neurological loss and may or may not be associated with radicular pain. Radicular pain is pain deriving from damage or irritation of the spinal nerve tissue, particularly the dorsal root ganglion.
45
What can cause radiculopathy?
most commonly due to nerve compression Intervertebral disc prolapse - repeated minor stresses on lumbar spine that predispose to rupture of the annulus fibrosus and sequestration of nucleus pulposus Degenerative diseases of the spine – spinal canal stenosis - 80% of the population over 55 years old have degenerative changes between C5/6 and C6/7 Fracture – either trauma or pathological Malignancy – most commonly metastatic Infection – extradural abscesses, osteomyelitis (most commonly TB (‘Pott’s disease’), or Herpes Zoster
46
What can you assess for on examination of cauda equina syndrome?
pinprick sensation in the perianal dermatomes (reduced) anocutaneous reflex (diminished or absent) anal tone (reduced) rectal pressure sensation (reduced)
47
Red flags for CES?
Faecal incontinence Urinary retention (painless, with secondary overflow incontinence) Saddle anaesthesia
48
Red flags for infection as a cause of radiculopathy?
Immunosuppression Intravenous drug abuse Unexplained fever Chronic steroid use
49
Red flags for fracture as a cause of radiculopathy?
Chronic steroid use Significant trauma Osteoporosis or metabolic bone disease
50
Red flags for malignancy/mets as a cause of radiculopathy?
New onset after 50 years old Systemic symptoms Hx of malignancy
51
The differential diagnosis for radicular pain should include pseudoradicular pain syndromes: these are conditions that do not arise directly from nerve root dysfunction, but cause radiating limb pain in an approximate radicular pattern. Give some examples *REVIEW CARD
Referred pain Myofascial pain Thoracic outlet syndrome Greater trochanteric bursitis Iliotibial band syndrome Meralgia paraesthetica Piriformis syndrome
52
Most IV disc prolapses can be managed non-operatively. What are the indications for surgery?
unremitting pain despite comprehensive non-surgical management progressive weakness new or progressive myelopathy (compression of the spinal cord)
53
What can be used for symptomatic mx of radiculopathy?
Amitriptyline is usually first line, or pregabalin and gabapentin as alternatives benzodiazepines (often diazepam) or baclofen for muscle spasms physio
54
What is degenerative disc disease?
the natural deterioration of the intervertebral disc structure Often related to ageing : Progressive dehydration of the nucleus pulposus Daily activities causing tears in the annulus fibrosis Injuries or pathology resulting in instability
55
What are the cascade of changes seen degenerative disc disease?
1. Dysfunction – outer annular tears and separation of the endplate, cartilage destruction, and facet synovial reaction 2. Instability – disc resorption and loss of disc space height, along with facet capsular laxity, can lead to subluxation and spondylolisthesis 3. Restabilisation – degenerative changes lead to osteophyte formation and canal stenosis
56
What are the potential signs of degenerative disc disease?
local spinal tenderness or contracted paraspinal muscles, hypomobility, or painful extension of the back or neck Further disease progression may demonstrate signs of worsening muscle tenderness, stiffness, reduced movement (particularly lumbar region), and scoliosis
57
What is Lasègue test?
also known as the straight leg raise , used to assess for disc herniation in patients presenting with lumbago with the patient lying down on their back, the examiner lifts the patient’s leg while the knee is straight A positive sign is when pain is elicited during the leg raising +/- ankle dorsiflexion or cervical spine flexion
58
When is imaging warranted for suspected degenerative disc disease?
Red flags present Radiculopathy with pain for more than 6 weeks Evidence of a spinal cord compression Imaging would significantly alter management MRI spine is the gold standard investigation however the majority of cases do not require imaging
59
When are spine radiographs recommended?
history of recent significant trauma, known osteoporosis, or aged over 70 years
60
Analgesia and physiotherapy is the mainstay of management. When would referral to pain clinic be indicated?
continued pain after 3 months despite analgesia
61
What is the most commonly used classification system for fractures of the cervical spine?
AO classification
62
Give some differentials for patients presenting with cervical neck pain
fracture, cervical spondylosis, cervical dislocation, or whiplash injury
63
What is a Jefferson fracture?
burst fracture of the atlas, usually unstable It is caused by axial loading of the cervical spine resulting in the occipital condyles being driven into the lateral masses of C1. They are often associated with head injuries - think 'silly Jeff diving headfirst into the shallow end'
64
What is a Hangman's fracture?
also called traumatic spondylolisthesis of the axis fracture through the pars interarticularis of C2 bilaterally, usually with subluxation of C2 on C3 caused by cervical hyperextension
65
What are odontoid peg fractures?
common cervical fractures, most common in older patients
66
What is the imaging for suspected cervical spine fractures?
Perform a CT scan in adults, if suggested by Canadian C-spine rules Perform MRI for children, if suggested by Canadian C-spine rules
67
How should C-spine fractures be managed?
3-point C-spine immobilisation initially to prevent damage to spinal cord Non-operative management can be appropriate for stable injuries: Rigid collars or halo vests Unstable fractures are usually treated operatively by fusing across the injured segment of the spine to the uninjured segments above and below
68
What is the most common area for a spinal fracture?
thoracolumbar junction (T11–L2)
69
What 3 columns can the spine be split into when assessing the stability of a fracture?
Anterior column – anterior longitudinal ligament and the anterior half of the vertebral body and disc Middle column – posterior half of the vertebral body and disc, and posterior longitudinal ligament Posterior column – comprised of the posterior elements (the posterior ligamentous complex, including the facet joint capsule, ligamentum flavum, and interspinous and supraspinous ligaments) and the intervening vertebral arches
70
What are the 3 types of thoracolumbar fracture according the AO classification?
Type A – compression injuries Type B – distraction injuries Type C – translation injuries
71
Which patient group do clavicle fractures occur in?
very common fractures mainly adolescents and young people second peak in 60+ age group due to osteoporosis
72
How can clavicle fractures be classified?
Allman classification Type I (75%)– fracture of the middle 1/3 of clavicle (weakest segment) - generally stable Type II (20%)– fracture involving the lateral 1/3 of the clavicle -when displaced, often unstable Type III (5%) - medial 1/3 of the clavicle - commonly associated with multi-system polytrauma - as the mediastinum sits directly behind this fracture site, they can be associated with neurovascular compromise, pneumothorax, or haemothorax
73
How do clavicle fractures usually displace?
The medial fragment will often displace superiorly, due to the pull of the sternocleidomastoid muscle, whilst the lateral fragment will displace inferiorly from the weight of the arm
74
Due to the subcutaneous location of the clavicle, it is important to specifically look for open injuries or threatened skin. How does 'threatened' skin present?
tented, tethered, white, and non-blanching skin
75
How are clavicle fractures assessed? How are they managed? Healing time?
X-rays - both anteroposterior and modified-axial views Tx: usually conservative - sling to support elbow and improve deformity - early mobilisation of shoulder to prevent frozen shoulder - surgery for open fractures or bilateral fractures to enable weight bearing Usually heal in 4-6 weeks
76
Major complication of clavicle fractures?
non-union - most associated with distal 1/3 fractures also neurovascular injury and pneumothorax
77
Most common site of shoulder fracture? Risk factors?
proximal humerus majority of proximal humeral fractures are low energy injuries in elderly patients (FOOSH) due to osteoporosis Same as for other osteoporotic fractures: older female, early menopause, long term steroids, recurrent falls
78
How might proximal humerus fractures present? How would you investigate?
elderly patient following FOOSH pain around the upper arm and shoulder, with restriction of arm movement and an inability to abduct their arm damage to the axillary nerve can result in loss of sensation in the lateral shoulder (“Regimental Badge”) and loss of power of the deltoid Investigations: trauma: urgent bloods incl G&S pathology suspected: calcium and myeloma screen X-ray: AP, lateral scapular, and axillary views
79
How should proximal humeral fractures be managed?
Most managed conservatively initial immobilisation with early remobilisation including pendular exercises around 2-4 weeks requires correctly applied polysling that allows their arm to hang- gravity will aid the reduction of the fragments Surgical fixation is indicated in patients with displaced, open, or neurovascularly compromised fractures
80
What surgical repair would be indicated for proximal humeral fracture patients with multiple segment injuries?
open reduction internal fixation (ORIF) - preferred in a head splitting fracture intermedullary nailing -preferred if the fracture involves the surgical neck, or if the fracture is combined with a humeral shaft fracture
81
Complications of proximal humeral fracture?
reduced range of motion - extensive physiotherapy required, often a year of rehab avascular necrosis of the humeral head following an injury disrupting the blood supply (from the anterior and posterior humeral circumflex arteries) -hemiarthroplasty or reverse shoulder arthroplasty
82
Shoulder dislocations account for over 50% of major joint dislocations which present to ED - if not managed correctly they can lead to chronic joint instability and chronic pain. What is the most common type of dislocation?
anteroinferior (95%) - classically caused by force being applied to an extended, abducted, and externally rotated humerus posterior much less common - seizures or electrocution
83
How do shoulder dislocations present?
painful shoulder, acutely reduced mobility, and a feeling of instability OE: asymmetry loss of shoulder contours (from a ‘flattened deltoid’) and an anterior bulge from the head of the humerus
84
What associated injuries can shoulder dislocations cause?
Bony: 1. Bony Bankart lesions - fractures of the anterior inferior glenoid bone, present in those with recurrent dislocations 2. Hill-Sachs defects- impaction injuries to the chondral surface of the humeral head, occur in anterior glenohumeral dislocations, traumatic dislocations Labral, ligamentous, and rotator cuff: 1. Soft Bankart lesions- avulsions of the anterior labrum and inferior glenohumeral ligament 2. Glenohumeral ligament avulsion 3. Rotator cuff injuries occur frequently in anterior dislocations
85
How are shoulder dislocations investigated? Management?
Investigations: X-ray: a trauma shoulder series is required - at least 2 views performed - AP, Y-scapular, or axial views (The Y view is very useful for differentiating between anterior and posterior dislocations) If labral or rotator cuff injuries are suspected - MRI Tx: closed reduction, immobilisation and rehabilitation broad arm sling for 2 weeks
86
What sign on x-ray suggests posterior shoulder dislocation?
light bulb sign - humerus fixed in internal rotation
87
Complications of shoulder dislocation?
chronic pain, poor mobility and recurrence adhesive capsulitis nerve damage rotator cuff injury
88
The rotator cuff is a group of 4 muscles that support and rotate the glenohumeral joint. Name these muscles
Supraspinatus – abduction Infraspinatus – external rotation Teres minor – external rotation Subscapularis – internal rotation
89
Rotator cuff tears are common. How can they be classified?
acute (lasting <3 months) or chronic (lasting >3 months) tears either partial thickness or full thickness tears full thickness tears can be further classified into small (<1cm), medium (1-3cm), large (3-5cm), or massive (>5cm or involves multiple tendons) tears
90
How do patients with rotator cuff tears present? Differentials? Risk factors?
pain over the lateral aspect of shoulder and an inability to abduct the arm above 90 degrees more common in the dominant arm OE: tenderness over the greater tuberosity and subacromial bursa DDx: shoulder fracture, persistent glenohumeral subluxation, brachial plexus injury, or radiculopathy Risk factors: age, trauma, overuse, and repetitive overhead shoulder motions
91
What specific tests can be used to assess for rotator cuff tears?
Jobe’s test (the “empty can test”, tests supraspinatus) Gerber’s lift-off test (tests subscapularis) Posterior cuff test (tests infraspinatus and teres minor)
92
How should rotator cuff tears be investigated? Mx?
Investigations: X ray to exclude fracture USS for presence and size of tear MRI Mx: < 2 weeks = conservative (physio and analgesia) > 2 weeks = surgery - arthroscopically (allowing for earlier recovery) or via open approach (preferred in large/complex tears)
93
Main complication of rotator cuff tears?
Adhesive capsulitis
94
Adhesive capsulitis (frozen shoulder) is a condition in which the glenohumeral joint capsule becomes contracted and adherent to the humeral head. Who does it commonly present in? How can it be categorised?
more common in women peak onset is between 40-70yrs old Primary adhesive capsulitis (idiopathic) Secondary adhesive capsulitis – rotator cuff / biceps tendinopathy, subacromial impingement syndrome, previous surgery or trauma, or joint arthropathy
95
How does adhesive capsulitis present?
progresses in three stages (an initial painful stage, a freezing stage, and finally a thawing stage) generalised deep and constant pain of the shoulder that often disturbs sleep OE: loss of arm swing and atrophy of deltoid limited range of motion, mainly affecting external rotation and flexion of the shoulder Adhesive capsulitis is a self-limiting condition, management is typically conservative and rarely requires surgical intervention
96
DDx for adhesive capsulitis?
Acromioclavicular pathology – a more generalised pain may be present with weakness and stiffness related to pain Subacromial impingement syndrome (rotator cuff tendinopathy, subacromial bursitis) – preserved passive movement and hx of repetitive overuse/external compression of subacromial space risk factors Muscular tear – the weakness often persists when the shoulder pain is relieved Autoimmune disease –polyarthropathy and systemic symptoms
97
Neck of femur fractures are associated with a high one year mortality and the patient cohort are often elderly with multiple co-morbidities. How do they present? What key differential should be considered?
trauma (usually low energy) acutely painful hip that is shortened and externally rotated with an inability to weight bear pain may be in groin, thigh or referred to knee DDx: alternative fracture -pubic ramus fractures, acetabulum, femoral head and femoral diaphysis
98
Blood supply to the NOF is retrograde. Which blood vessel is responsible for the majority of the supply?
medial circumflex femoral artery lies directly on the intra-capsular femoral neck displaced intra-capsular fractures disrupt the blood supply to the femoral head and can cause avascular necrosis
99
How can intracapsular fractures be further classified?
Garden classification I - Non-displaced , Incomplete II - Non-displaced, complete III - Complete fracture, partial displacement IV- Complete fracture fully displaced
100
Investigations for NOF fractures?
Bloods + CK if long lie suspected Urine dip, CXR and ECG in elderly patients - cause of fall + pre-op workup Imaging: X rays - AP and lateral view of affected hip, AP pelvis
101
Non-operative conservative management is rarely recommended for NOF fractures, as the benefits of surgical intervention nearly always outweigh the potential conservative management. What is the surgical tx of displaced subcapital NOF fractures?
Hip Hemiarthroplasty - Replacement of the femoral head and neck via a femoral component fixed in the proximal femur
102
What is the surgical tx of Inter-trochanteric and Basocervical NOF fractures?
Dynamic Hip Screw (or short IM nail) - lag screw into the neck, a sideplate, and bicortical screws. The lag screw is able to slide through the sideplate, allowing for compression and primary healing of the bone
103
What is the surgical tx for Non-displaced intra-capsular NOF fractures?
Cannulated hip screws - Three parallel screws in an inverted triangle formation
104
What is the surgical tx for Sub-trochanteric NOF fractures?
Anterograde Intramedullary Femoral Nail - titanium rod is placed through the medullary cavity of the femur for stabilisation
105
How should NOF patients be managed post-op? Complications?
jointly with ortho-geriatricians early rehab with physios and OTs Complications: Immediate - pain, bleeding, leg-length discrepancies, and potential neurovascular damage Long term - joint dislocation, aseptic loosening, peri-prosthetic fracture, and deep infection/prosthetic joint infection
106
OA is the most common cause of disability in older adults in the Western World. What are the risk factors for OA of the hip?
Systemic – Increasing age (>45 yrs), obesity, female gender, genetic factors, vitamin D deficiency Local – History of trauma to the hip, anatomic abnormalities, muscle weakness or joint laxity, participation in high impact sports
107
How does OA of the hip present?
Pain - most commonly groin, but can be lateral hip or deep buttocks aggravated by weight bearing and worse at end of day, relieves with rest stiffness, grinding, crunching OE: antalgic gait, pain on passive movement, reduced ROM, may have fixed flexion deformity in end stage disease
108
DDx for hip OA?
Trochanteric bursitis – lateral hip pain radiating down the lateral leg, with associated point tenderness over the greater trochanter Gluteus medius tendinopathy – lateral hip pain with point tenderness over the muscle insertion at the greater trochanter Sciatica – low back pain and buttock pain, often radiates down the posterior leg to below the knee. Diagnosis is made with the straight leg raise to produce Lasègue’s sign Femoral neck fracture – history of trauma or known severe osteoporosis, the patient will be unable to weight bear due to pain and the limb will appear shortened and externally rotated
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Signs of hip OA on x-ray?
Narrowing of the joint space Osteophyte formation Sclerosis of the subchondral bone Subchondral bone cysts
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Management of hip OA? Surgical complications?
WL, exercise, pain control (WHO stepladder) physio to slow disease progression and improve joint mechanics Surgery: hip replacement (arthroplasty or hemiarthroplasty) Posterior Approach (to glut medius) – The most common approach as rehabilitation is often fast due to preservation of the abductor mechanism, but risk of sciatic nerve damage Complications: thromboembolic disease / bleeding, infection, dislocation, loosening of the prosthesis, and leg length discrepancy
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How long do modern hip replacements last?
15-20 years
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The true pelvis contains the rectum, bladder and uterus in females, as well as the iliac vessels and the lumbosacral nerve roots. Knowing this, what are the potential complications of pelvic fractures?
life-threatening haemorrhage, neurological deficit, urogenital trauma, and bowel injury
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Pelvic ring injuries are most often caused by high energy blunt trauma, such as road traffic accidents or falls from height. What should you do during your examination?
full neurovascular assessment of the lower limbs including checking anal tone -sacral nerve roots and iliac vessels can frequently be injured abdo injuries, urethral injuries, and open fractures (incl “internal open fractures” into the rectum or vaginal vault) look for any surrounding ecchymosis or developing haematoma present (e.g. perineal, scrotal or labial)
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What can cause low energy pelvic fractures?
avulsion fractures - reported as a sudden severe pain, poorly localised to the hip/pelvis, felt whilst performing a rapid, powerful movement, such as starting to run
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How should suspected pelvic fractures be investigated?
3 X-rays needed to assess pelvic ring - AP, inlet and outlet view However CT usually performed in trauma setting so negates need for x-ray
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Indications for operative management of pelvic fracture? What may a haemodynamically unstable pelvic fracture patient require?
life threatening haemorrhage, unstable fractures, open fractures, and associated fractures with an associated urological injury interventional radiology or trauma laparotomy +/- retroperitoneal packing
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What is the most commonly used classification for pelvic fractures?
The Young and Burgess classification
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Complications following pelvic fractures?
urological injury, venous thromboembolism, and long-standing pelvic pain
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The knee joint is the most commonly affected joint by osteoarthritis. How should it be managed?
Initial management is with analgesia and physiotherapy, however total knee replacement (TKR) is the standard treatment for advanced osteoarthritis TKR lasts for at least 10 years Partial (unicondylar) replacement may be indicated for those with disease localised to either the medial or lateral compartment
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How does patellofemoral OA present?
Anterior knee pain, worse with activities that put pressure on the patella, such as climbing a flight of stairs
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How do ACL tears present? Specific clinical tests?
athlete with a history of twisting the knee whilst weight-bearing rapid joint swelling (highly vascular ligament rupture = haemarthrosis) significant pain Lachman’s Test (more sensitive) and Anterior Drawer Test
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How should ACL tears be investigated and managed?
Investigations: - plain film radiograph (AP and lateral) - exclude bony injuries, any joint effusion, or a lipohaemarthrosis - Segond fracture (bony avulsion of the lateral proximal tibia) is pathognomic - MRI scan GOLD STANDARD Mx: - RICE - strength training of quads and cricket pad knee splint for comfort - surgical reconstruction following 'prehabilitation'
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Complications of ACL tear / surgery?
post-traumatic OA
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Which is the most commonly injured ligament of the knee? What is the usual mechanism of injury?
MCL - valgus stabiliser external rotational forces are applied to the lateral knee, such as a impact to the outside of the knee MCL injuries can be graded from one to three: Grade I – mild injury, minimally torn fibres and no loss of MCL integrity Grade II – moderate injury, incomplete tear and increased laxity of the MCL Grade III – severe injury, complete tear and gross laxity of the MCL
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How does an MCL tear present? On examination?
hearing a ‘pop’ with immediate medial joint line pain swelling after a few hours OE: increased laxity when testing the MCL via valgus stress test extremely tender along joint line but may be able to weight bear
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Management of MCL tear? Complications?
Grade1 : RICE Grade 2: analgesia, knee brace, return to full exercise in 10 weeks Grade 3: analgesia with a knee brace and crutches, any associated distal avulsion = surgery considered, return to full exercise in 12 weeks Complications: instability in the joint and damage to the saphenous nerve.
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What is a Colles' fracture?
extra-articular fracture of the distal radius with dorsal angulation and dorsal displacement, within 2cm of the articular surface also includes an avulsion fracture of the ulnar styloid typically fragility fracture caused by FOOSH
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What is a Smith's fracture?
extra-articular fracture of the distal radius with volar angulation of the distal fragment (the reverse of a Colles fracture), with or without volar displacement caused by falling backwards and planting the outstretched hand behind the body, causing a forced pronation type injury
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What is a Barton's fracture?
intra-articular fracture of the distal radius with associated dislocation of the radio-carpal joint described as volar (more common) or dorsal (less common), depending on whether the volar or dorsal rim of the radius is involved
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The neurological examination for a suspected distal radius fracture should include the following nerves being assessed:
Median nerve: motor – abduction of the thumb sensory – radial surface of distal 2nd digit Anterior interosseous nerve: opposition of the thumb and index finger (OK sign) Ulnar nerve: motor – adduction of the thumb (‘Froment’s Sign’) sensory – ulnar surface of the distal 5th digit Radial nerve: motor – extension of IPJ of thumb sensory – dorsal surface of 1st webspace
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DDx for distal radius fractures?
Forearm fracture (such as Galeazzi or Monteggia fractures) Carpal bone fractures Tendonitis or tenosynovitis Wrist dislocation
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Which 3 measurements on X-ray help to diagnose distal radius fractures?
Radial height <11mm Radial inclination <22 degrees Radial (volar) tilt >11 degrees CT or MRI can be used for more complex fractures
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Management of distal radius fractures?
traction and manipulation under anaesthetic - under conscious sedation with a haematoma block or Bier’s block Stable and successfully reduced fractures = below-elbow backslab cast, then radiographs repeated after 1 week to check for displacement Significantly displaced or unstable fractures can require surgical intervention (or intra-articular step of the radiocarpal joint >2mm) = open reduction and internal fixation
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The main complications following distal radius fractures are:
Malunion- poor realignment leads to a shortened radius compared to the ulnar, leading to reduced wrist motion, wrist pain, and reduced forearm rotation - can be treated with corrective osteotomy Median nerve compression, more common in patients who heal in a significant degree of malunion Osteoarthritis, especially with intra-articular involvement from the original fracture
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What is carpal tunnel syndrome? Risk factors?
compression of the median nerve within the carpal tunnel of the wrist, due to a raised pressure within this compartment pain, numbness, and paresthesia in the lateral 3½ digits palm is often spared, due to the palmar cutaneous branch of the median nerve branching proximal to the flexor retinaculum and passing over the carpal tunnel RF: female gender, increasing age, pregnancy, obesity, and previous injury to the wrist
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Aggravating and relieving factors for carpal tunnel?
worse during night symptoms can often be temporarily relieved by hanging the affected arm over the side of the bed or by shaking it back and forth
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What can be seen on examination of carpal tunnel?
sensory symptoms can be reproduced by either percussing over the median nerve (Tinel’s Test) or holding the wrist in full flexion for one minute (Phalen’s Test) late stages- weakness of thumb abduction (due to denervation atrophy of the thenar muscles) and / or wasting of the thenar eminence
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DDX for carpal tunnel?
Cervical Radiculopathy - C6 nerve root involvement may produce similar sxs however will likely have an element of neck pain / involve the entire arm length Pronator teres syndrome (median nerve compression by pronator teres) - extend to the proximal forearm and sensation of the palm will also be reduced Flexor carpi radialis tenosynovitis - distinguished by tenderness at the base of the thumb
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Investigation and management of carpal tunnel? Complications of surgery?
Clinical diagnosis uncertain cases - nerve conduction studies Mx: wrist splint at night hand therapy steroid injections carpal tunnel release surgery in persistently symptomatic pts Complications of carpal tunnel surgery include recurrence, persistent symptoms (from incomplete release of ligament), infection, scar formation, nerve damage, or trigger thumb.
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Ankle fractures are a common injury, more common in younger males or older females. How can they be classified?
isolated lateral malleolar fractures, isolated medial malleolar fractures, bimalleolar fractures and trimalleolar fractures (medial + lateral + posterior) Weber classification for lateral fractures: Type A = below the syndesmosis Type B = at the level of the syndesmosis Type C = above the level of the syndesmosis more proximal = more unstable so Type C almost always need surgery
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Investigations and management of ankle fractures?
X-ray (ankle must be fully dorsiflexed for this)- AP and lateral view, check for evidence of talar shift If complex fracture - CT Mx: immediate fracture reduction, below knee back slab Conservative management will often be opted for in: Non-displaced medial malleolus fractures Weber A fractures or Weber B fractures without talar shift Those unfit for surgical intervention Surgery: ORIF - for displaced or open or talar shift
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What are ankle sprains?
ligamentous injuries classified into high ankle sprains (which are injuries to the syndesmosis) or low ankle sprains, which are injuries to the anterior talofibular ligament (ATFL) and the calcaneofibular ligament (CFL) present following an inversion injury on a plantarflexed ankle with fingertip tenderness distal to the malleoli
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What is Achilles tendonitis? RF?
inflammation of the Achilles (calcaneal) tendon The classical case of tendonitis or rupture occurs in an unfit individual who has a sudden increase in exercise frequency - 'weekend warriors' Other risk factors include poor footwear choice, male gender, obesity, or recent fluoroquinolone use (for tendon rupture)
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What are the most commonly used indicators of a clinical Achilles tendon rupture? How can it be diagnosed?
Simmonds test (loss of plantarflexion) and a palpable ‘step’ in the Achilles tendon Clinical diagnosis or USS
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What is the most common cause of infracalcaneal pain? How can it be diagnosed? Mx?
Plantar fasciitis Clinical diagnosis X ray to look for plantar heel spur - abnormal loading MRI for fascial thickening Initial management is conservative, however corticosteroid injections or plantar fasciotomy can be considered if no improvement
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Features of L3 nerve root compression?
Sensory loss over anterior thigh Weak hip flexion, knee extension and hip adduction Reduced knee reflex Positive femoral stretch test
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Features of L4 nerve root compression?
Sensory loss anterior aspect of knee and medial malleolus Weak knee extension and hip adduction Reduced knee reflex Positive femoral stretch test
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Features of L5 nerve root compression?
Sensory loss dorsum of foot Weakness in foot and big toe dorsiflexion Reflexes intact Positive sciatic nerve stretch test
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Features of S1 nerve root compression?
Sensory loss posterolateral aspect of leg and lateral aspect of foot Weakness in plantar flexion of foot Reduced ankle reflex Positive sciatic nerve stretch test
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When should sciatica be referred for an MRI?
4-6 weeks of conservative management and no improvement
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how may supraspinatus tendonitis (subacromial impingement) present on x-ray?
calcification of the supraspinatus tendon consistent with prolonged inflammation patient will likely exhibit the 'painful arc'
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Twisting sporting injuries followed by delayed onset of knee swelling and locking are strongly suggestive of what?
menisceal tear McMurrays test will be positive Arthroscopic menisectomy is the usual treatment
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rupture of which ligament will cause the tibia to lie back on the femur?
PCL rupture mechanism = hyperextension injuries (e.g. knee hitting dash) paradoxical anterior drawer test
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How does Chondromalacia patellae present?
Teenage girls, following an injury to knee e.g. Dislocation patella Typical history of pain on going downstairs or at rest Tenderness, quadriceps wasting
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How do tibial plateau fractures occur?
Occur in the elderly (or following significant trauma in young) Mechanism: knee forced into valgus or varus, but the knee fractures before the ligaments rupture Varus injury affects medial plateau and if valgus injury, lateral plateau depressed fracture occurs
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In a child with an asymptomatic, fluctuant swelling behind the knee the most likely diagnosis is what?
Baker's cyst
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How does facet joint pain present?
May be acute or chronic Pain worse in the morning and on standing On examination there may be pain over the facets. The pain is typically worse on extension of the back
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How does spinal stenosis present?
Usually gradual onset Unilateral or bilateral leg pain (with or without back pain), numbness, and weakness which is worse on walking. Resolves when sits down. Pain may be described as 'aching', 'crawling'. Relieved by sitting down, leaning forwards and crouching down Clinical examination is often normal Requires MRI to confirm diagnosis
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Any patient presenting with symptoms of intermittent claudication not worsened by increasing exertion =
neurogenic not ischaemic
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Severe sharp back pain worse on movement with positive straight leg raise test - what is the diagnosis and mx?
prolapsed disc arrange physio, no need to scan unless red flags
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How should discitis be investigated?
MRI imaging Assess for endocarditis e.g. with transthoracic echo or transesophageal echo
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A 23-year-old rugby player falls directly onto his shoulder. There is pain and swelling of the shoulder joint. The clavicle is prominent and there appears to be a step deformity. Dx?
Acromioclavicular joint (ACJ) dislocation
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Dupytren's contracture presents with thickening of the palm and an inability to full extend the metacarpophalangeal joints, usually the little and ring fingers. What can cause it?
manual labour phenytoin treatment alcoholic liver disease diabetes mellitus trauma to the hand
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commonly used method of analgesia for patients with a neck of femur fracture?
iliofascial nerve block aim of this is to reduce the use of opioids analgesics e.g. morphine, which is particularly helpful in elderly patients
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Increasing hip pain at rest, together with increased serum calcium and alkaline phosphatase are most likely to represent what?
metastatic tumour to bone Chondrosarcomas do occur in the pelvis but are not associated with increased serum calcium and typically have a longer history
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A 73-year-old man presents with pain in the right leg. It is most uncomfortable on walking. On examination he has a deformity of his right femur, which on x-ray is thickened and sclerotic. His serum alkaline phosphatase is elevated, but calcium is within normal limits. This is a typical hx of which condition?
Paget's disease
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Likely cause of bone pain with: 1. normal ALP and calcium 2. raised ALP but other parameters normal 3. raised ALP and calcium
1. osteoporosis 2. paget's disease of the bone 3. mets to bone
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What is an essential part of the management for all ankle fractures?
they should be promptly reduced to remove pressure on the overlying skin and subsequent necrosis
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Extracapsular hip fracture (subtrochanteric fracture) should be managed using what?
intramedullary device
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In paediatric practice, fractures may also involve the growth plate and these injuries are classified according to the Salter-Harris system. Outline Salter Harris classes 1-5
I - Fracture through the physis only (x-ray often normal) II - Fracture through the physis and metaphysis III - Fracture through the physis and epiphysis to include the joint IV - Fracture involving the physis, metaphysis and epiphysis V - Crush injury involving the physis (x-ray may resemble type I, and appear normal) Straight through physis Above Lower Through all 3 Everything crushed
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injury pattern of a greenstick fracture?
Unilateral cortical breach only
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How may fat emboli present?
post-trauma esp long bone fractures Respiratory: Tachycardia Tachypnoea, dyspnoea, hypoxia usually 72 hours following injury Pyrexia Dermatological: Red/ brown impalpable petechial rash Subconjunctival and oral haemorrhage/ petechiae CNS : Confusion and agitation Retinal haemorrhages and intra-arterial fat globules on fundoscopy
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Simmonds' triad is used to examine for an Achilles tendon rupture. What does it include?
It includes palpation of the Achilles tendon (examining for a gap), observing for an abnormal angle of declination (i.e. the foot is more dorsiflexed than the other), and performing the calf squeeze test.
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Investigation for Cauda Equina?
MRI spine within 6 hours
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Phalen's test is used to asses carpal tunnel syndrome. What does this involve?
The patient's wrist is held in maximum flexion (reverse prayer sign) for 30-60 seconds. The test is positive if there is numbness in the median nerve distribution
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Children and young people with unexplained bone swelling or pain:
consider very urgent direct access X-ray to assess for bone sarcoma (within 48 hours)
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Colle's key points?
Dorsally Displaced Distal radius → Dinner fork Deformity
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Colle's complications?
early: median nerve injury: acute carpal tunnel syndrome presenting with weakness or loss of thumb or index finger flexion compartment syndrome malunion rupture of the extensor pollicis longus tendon late: osteoarthritis complex regional pain syndrome
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common cause of lateral knee pain in runners?
iliotibial band syndrome
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If scaphoid fracture is suspected, but imaging is inconclusive, how should you proceed?
Referral to orthopaedics and repeat imaging in 7-10 days
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Leriche syndrome is atherosclerotic occlusive disease involving the abdominal aorta and/or both of the iliac arteries. How does it present?
Classically, it is described in male patients as a triad of symptoms: 1. Claudication of the buttocks and thighs 2. Atrophy of the musculature of the legs 3. Impotence (due to paralysis of the L1 nerve)
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A scaphoid fracture is a type of wrist fracture, typically arises as a result of a fall onto an outstretched hand (FOOSH). Why are they so high risk?
80% of the blood supply is retrograde - derived from the dorsal carpal branch (branch of the radial artery) risk of avascular necrosis
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How do patients with scaphoid fractures present?
pain over the radial aspect of wrist (base of thumb) and loss of grip strength OE: maximal tenderness over anatomical snuffbox wrist joint effusion pain on telescoping of thumb pain on ulnar deviation of wrist
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How should scaphoid fractures be investigated and managed?
Investigations: X-ray: 'Scaphoid views': posterioranterior (PA), lateral, oblique (with wrist pronated at 45º) and Ziter view MRI definitive to confirm dx Mx: immobilisation with a Futuro splint or standard below-elbow backslab review by orthopods undisplaced = cast for 6-8 weeks displaced = surgery
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De Quervain's tenosynovitis is a common condition in which the sheath containing the extensor pollicis brevis and abductor pollicis longus tendons is inflamed. It typically affects females aged 30 - 50 years old. How does it present? Mx?
pain over radial styloid process and painful abduction of thumb against resistance Mx: analgesia steroid injection immobilisation with a thumb splint (spica) may be effective
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Which drug tx can cause Dupytren's contracture as a side effect?
phenytoin
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Outline the different types of hip dislocation
Posterior dislocation (90%): affected leg is shortened, adducted, and internally rotated Anterior dislocation: affected leg is usually abducted and externally rotated. No leg shortening Central dislocation
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Mx of hip dislocation?
ABCDE Analgesia Reduction under GA within 4 hours to reduce the risk of avascular necrosis Long-term management: Physiotherapy to strengthen the surrounding muscles
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Complications of hip dislocation?
Sciatic or femoral nerve injury Avascular necrosis Osteoarthritis: more common in older patients Recurrent dislocation: due to damage of supporting ligaments
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What is meralgia parasthetica?
syndrome of paraesthesia or anaesthesia in the distribution of the lateral femoral cutaneous nerve Hx: Sxs over upper lateral aspect of thigh Burning, coldness, tingling, or shooting pain Numbness Deep muscle ache Usually aggravated by standing, and relieved by sitting OE: Symptoms reproduced by deep palpation just below the ASIS (pelvic compression) and by hip extension
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What is the most common upper limb injury in children under the age of 6? Mx?
Subluxation of the radial head (pulled elbow) distal attachment of the annular ligament covering the radial head is weaker in children at this age Mx: analgesia and passive supination of the elbow joint whilst the elbow is flexed to 90 degrees
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Which finger joint most commonly dislocates?
PIP
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In what ways can a PIP joint dislocate?
Laterally - damages collateral ligaments Dorsally - damages flexor tendon , can cause swan neck deformity Volarly - damages extensor tendon
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How can you investigate and manage phalanx dislocations?
examine : Elson's test, lateral stress test, sensation testing X-ray Splinting
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What causes knee dislocations? How should you investigate and manage?
usually high energy injury e.g. knee hitting dash - high risk of neurovascular damage e.g. to popliteal artery neurovascular assessment and x-ray Mx: reduce and stabilise ASAP assess limb perfusion - cap refill and peripheral pulses, can do doppler USS can do delayed ligamentous reconstruction