Traumatic Cataracts Flashcards Preview

Ocular Disease - Lens > Traumatic Cataracts > Flashcards

Flashcards in Traumatic Cataracts Deck (14):
1

What are signs of traumatic cataract?

(unilateral, bilateral?, lens permeability, ruptured capsule?, dislocation)

- Unilateral
- Change in lens permeability from trauma
- ruptured capsule and dislocation

2

Describe Vossius ring cataract

(Does lens rupture?, where is ring located on lens?)

- lens may not rupture
- ring of iris pigment on anterior surface

3

Describe Rosette cataract

(Where on lens is it seen, what happen to lens fibers after trauma)

- seen on posterior lens capsule
- due to trauma, lens fibers dissolve along liner of growth pattern

4

Describe Exfoliation

(What happens to lens after trauma, where does lens protein escape to and how does the immune system react?, What type of uveitis results?)

- lens capture rupture due to trauma
- cortical material in aqueous, opacity develops
- lens protein seen as foreign substance by immune system (phakoanaphylactic uveitis)

5

Describe Siderosis Lentis

(what causes it, what results as a result)
Hint: think rust

- caused by foreign body penetrating lens
- Rust forms underneath capsule leading to complete cortical cataract

6

Describe Chalcosis Lentis (Sunflower cataract)

(what causes it, what colors does opacities appear, what disease can cause this, what is the management)
Hint: think copper

- caused by excessive amounts of copper in eye
- yellow brown opacities in subcapsular cortex and "petal-like" spokes that extend to equator
- causes green-reddish brown discoloration
- Caused by Wilson's disease, drop from Copper sulfate or copper foreign body
- Tx: remove foreign body

7

Describe Subluxation and Luxation

(How its caused, what's the difference between the two)

- Subluxation: zonules rupture due to trauma (incomplete detachment). Lens pulled to one side
- Luxation: complete detachment of lens, may disclocate anterior/posterior

8

You have a patient that comes in with metal debris that's penetrated the eye resulting in rust forming underneath the lens capsule. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

D. Siderosis Lentis

9

You have a patient that has a lens pulled to one side due to trauma. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

F. Subluxation

10

You have a patient that has a ring on the anterior surface due to trauma but no rupturing. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

A. Vossius Ring

11

You have a patient that comes in with a painful uveitis after trauma. Upon SLE you see a ruptured lens capsule and material in the aqueous. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

C. Exfoliation

12

You have a patient that comes in with metal debris that's penetrated the eye resulting in yellow brown opacities in subcapsular cortex and sunflower (petal-like spokes). A SLE exam reveals green/reddish brown discoloration. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

E. Chalcosis Lentis

13

You have cataract that has lens fibers dissolve on the posterior lens capsule due to trauma. What type of traumatic cataract is this?

A. Vossius Ring
B. Rosette Cataract
C. Exfoliation
D. Siderosis Lentis
E. Chalcosis Lentis
F. Subluxation

B. Rosette Cataract

14

Describe Glauomflecken

(Where is the opacity located, what's it caused by (related to angle closure GLC), What can build up on material in aqueous do to the cell?)

- Supcapsular opacities
- Dramatic increase in IOP from acute angle closure causes stagnation in anterior chamber
- Build up of noxious material in aqueous leads to cell necrosis resulting in permenant subcapsular opacities