Treatment Guidelines Flashcards

(38 cards)

1
Q

What features indicate pt is at high risk of being peri-arrest? (5)

A

Hypotension/shock, syncope, ischemic CP or ischemia on 12 lead, heart failure: increased JVP/pulmonary edema, arrhythmias

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2
Q

In what patient population is Atropine potentially harmful?

A

Heart transplant patients. (Denervation of parasympathetic pathways)

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3
Q

What is BRASH syndrome?

A

Bradycardia, renal failure, AV blockade, Shock, Hyper K

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4
Q

What are some reversible causes of bradycardia? (5)

A

Hypoxia, increased parasympathetic tone, drugs/overdose, Hyper K, Myocardial ischemia

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5
Q

What is circum-rescue collapse

A

collapse of a pt in VF or cardiac arrest either before, during or shortly after rescue from cold environment

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6
Q

At what body temperature will a pt likely develop cardiac arrhythmias progressing to VF

A

30 degrees

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7
Q

Why should Epi be limited to 3 doses in hypothermic patients?

A

Drugs are metabolized more slowly and there is a potential for toxic build-up

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8
Q

At what ETCO2 level should an improvement in CPR quality be considered

A

10mmHg

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9
Q

What are the components of the Post Arrest checklist? (4)

A

Airway: Check tube position, air entry, tube tie
Breathing: 1/4-1/3 of BVM, 10-12/min, SPO2 of 94%, PEEP 5cmH2o
Circulation: Rhythm-5 mins for SVT, Map 65, 12 lead
Disability:HOB 30 degrees, Glucose, Temp neutral

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10
Q

How much fluid should an adult be resuscitated with?

A

20mL/kg

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11
Q

Cerebral and cardiac dysfunction accompanied by prolonged systemic ischemia (hypoperfusion/cardiac arrest) is known as?

A

Post cardiac arrest syndrome

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12
Q

What is the most common cause of traumatic cardiac arrest?

A

Haemorrhage

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13
Q

What are the reversible causes of traumatic arrest?

A

Hypovolemia, Hypoxia, Tension Pneumothorax

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14
Q

What is the transport window for a pt in cardiac arrest secondary to a traumatic cause?

A

15 minutes (20 in Vancouver Coastal)

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15
Q

When should a pelvic binder be applied in traumatic arrest?

A

After other reversible causes are treated, unless pelvic fx is suspected as being a leading cause of hemorrhage.

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16
Q

What is coagulopathy?

A

A derangement in hemostasis resulting in either excessive bleeding or clotting

17
Q

What is the calculation for initial and subsequent defibrillation in pediatric patients

A

2J/kg, repeat at 4J/kg

18
Q

Where are the 2 sites for needle thoracentesis placement

A

2nd intercostal mid-clavicular

5th intercostal mid-axilla

19
Q

What are the 6 criteria for recognition of life extinct (ROLE)

A
No palpable pulse for 90 s
No heart sounds for 90s
No breath sounds/resp effort for 90 s
Fixed non-reactive pupils
No response to central stimuli
Observe Asystole or PEA < 30 for 60s
20
Q

What is the Pediatric assessment triangle (PAT)

A

General appearance (Tone, Inconsolability, Gaze, Color)
Work of breathing
Skin (Circulation)

21
Q

How do you calculate mean and lower BP limits in pediatrics?

A

Mean: 80+ (2x Age (years))
Lower: 70+ (2x Age (years))

22
Q

How do you calculate the ET tube size in pediatrics?

A

Uncuffed:(Age/4)+4
Cuffed: (Age/4)+3

23
Q

What is the SIRS criteria?

A

Systemic Inflammatory Response Syndrome

Tachypnea, Tachycardia, Fever, Increased WBC

24
Q

What is qSOFA

A

Quick Sequential Organ Failure Assessment

Altered LOC, Tachypnea, Hypotension

25
Exposure to CO with levels above (?) require conveyance to hospital?
Above 10%
26
How long does CO remain bound to hemoglobin?
4-5 hours at R/A 1-2 hours at 100% O2 20 mins in hyperbaric chamber
27
How does a hyperbaric chamber treat CO poisoning?
By producing a 10x increase in the amount of O2 dissolved in plasma, thereby increasing oxygen delivery to the tissue, and increasing CO elimination.
28
What are normal CO levels for a smoker?
2%, may be as high as 9% in heavy smokers
29
What causes a right shift (reduced affinity) in the oxyhemoglobin dissociation curve? (5)
``` Low pH High temp High CO2 High 2,3-BPG Low affinity hb variants ```
30
Organophosphates and Carbamates inhibit what? | Leading to what?
Acetylcholinesterase, causing stimulation of muscinaric (parasympathetic) and nicotinic (sympathetic) receptors
31
What is the mneumonic for organophosphate toxicity?
``` Sludge and the killer B's Salivation Lacrimation Urination Defecation GI upset Emesis Bronchospasm Bronchorrhea Bradycardia ```
32
What is the goal of Atropine in treating organophosphate poisoning? What receptors does it affect?
Control secretions, and correct bradycardia and hypotension. Reverses muscinaric, but not nicotonic
33
What are some symptoms of beta-blocker OD? (7)
``` Bradycardia Hypotension Mental status changes Ventricular dysrhythmias Cardiogenic shock Bronchospasm Hypoglycemia ```
34
What are common ECG findings in beta-blocker OD?
PR prolongation, QRS prolongation, bradydysrhythmia
35
What are the steps in treating beta-blocker OD? | What treatments are unlikely to be successful?
Glucagon-increasing cardiac inotropy by activating adenyl cyclase by a secondary mechanism bypassing beta-blockade Calcium-improve BP and contractility Sodium bicarb- Wide QRS, increases sodium and initiation of action potential Mag sulfate-Torsades, Epi infusion- Increases chrono/ino/dromo-tropy Atropine and pacing are unlikely to be successful
36
What are the common S&S of TCA OD?
Sedation/Unconciousness Seizures Wide complex tachycardia Hypotension
37
What is the cardiac consequence of TCA OD?
Sodium channel blockade
38
What are the common ECG finding of TCA OD?
QRS> 100ms Deep S in I and aVL Tall R in aVR Tachycardia