Triage, fluid and oxygen therapy Flashcards

(57 cards)

1
Q

Define triage

A

CLASSIFICATION OF PATIENTS TO DETERMINE PRIORITY OF NEED
AND THE OPTIMAL ORDER IN WHICH THEY SHOULD BE TREATED

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2
Q

Before arrival, the following information about the patient should be gotten via phone: (7)

A

Short history
Breed
Sex
Age
Approx. weight
Instructions for a safe arrival
Phone nr

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3
Q

Triage categories

A

Categories can vary dependent upon literature.

Green = non-emergent
Yellow = urgent but not life-threatening
Orange = very urgent, potentially life-threatening
Red = immediate care needed, life-threatening
Black = dead on arrival

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4
Q

What does primary triage involve broadly?

A

Airways
Breathing
Circulation
Disability

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5
Q

MBSA =
(5)

A

MBSA (major body system assessment)

 Respiratory system
 Cardio-vascular system
 Nervous system
 Urinary/reproductive system
 Other parameters/changes

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6
Q

Describe respiratory system triage

A

Observation from a distance as well as auscultation of the trachea and lungs in order to detect hypoxemia and
hypoventilation.

 Open airways
 Stertor (above larynx)
stridor (larynx or below)
 Respiratory rate and pattern
 + mucous membranes
 Auscultation
 Palpation of the thorax

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7
Q

Stertor vs stridor

A

Stertor (above larynx)
stridor (larynx or below)

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8
Q

parameters for assessment of hypoperfusion: (5)

A

 Colour of mucous membranes
 Capillary refill time (CRT)

 Pulse-presence/quality
- Femoral pulse
- Dorsal metatarsal pulse

 Heart rate
 Heart auscultation

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9
Q

Parameters of shock (7)

A

mm can vary
crt typically prolonged
heart rate elevated
resp. rate elevated
peripheral pulse weak or absent
BP decreased
lactate elevated

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10
Q

Color of mucous membranes.
What do the varying colors indicate?

A

brown mm = methemoglobinemia

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11
Q

Triage of the nervous system involves? (4)

A

Assessment of:
 Mentality (stupor, coma etc.)
 Cranial nerves (anisocoria etc.)
 Movement
 Pain/deep pain (in paralysis, paresis cases espesh)

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12
Q

stupor vs coma

A

stupor = unconscious, reactions to external manipulations can be present

coma = unconscious,
no reactions to external manipulations except for potentially some autonomic reflexes

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13
Q

Triage of the Urinary/reproductive system, 4 main issues to check for.

A

Big painful bladder

Uterine prolapse
Dystocia

Persistent erection/paraphymosis (dogs, chinchillas especially)

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14
Q

hyperthermia vs fever

A

hyperthermia = elevated temperature due to external factors

fever = under control of the body’s own thermoregulation center

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15
Q

Modified ATT (Animal Trauma ‘Triage) score

A

The animal trauma triage (ATT) score is a veterinary illness severity score that numerically classifies the degree of trauma in an attempt to quantify mortality risk probability.

Parameters Assessed in mATT:
Perfusion
Cardiovascular
Respiratory
Neurological
Gastrointestinal (GI)/Urogenital
Musculoskeletal

Ranges from 0 (normal) to 18 (severely compromised).

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16
Q

MGCS (Modified Glasgow Coma Scale)

A

is an adaptation of the human Glasgow Coma Scale, specifically designed for veterinary use to assess the level of consciousness and neurological function in animals.

The MGCS evaluates three main neurological parameters:

Motor Activity
Brainstem Reflexes (Pupillary Light Reflex and Ocular Position)
Level of Consciousness

total score ranges from 3 to 18.
18 indicates a normal neurologic function, while 3 indicates severe brain dysfunction or deep coma.

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17
Q

Primary stabilization includes (4)

A

IV catheter
O2 therapy
Fluid therapy
Analgesia

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18
Q

Blood sample diagnostics that may be used for triage: (7)

A

 PCV/Total Solids
 Glucose
 Lactate
 Blood gases
 UREA
 Blood smear
 Electrolytes

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19
Q

aFAST/tFAST

A

abdominal and thoracic focused assessment with sonography for trauma/triage

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20
Q

Describe secondary triage.

A

After first stabilization, do a secondary examination. Did you miss something? Recheck and cover any unassessed bases.

The goal of secondary triage is to provide a more detailed evaluation of a patient’s condition, prioritize further treatment, and ensure that medical resources are allocated efficiently.

2nd triage gives you further info for communication with owners
 Prognosis
 Cost Etc…..

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21
Q

Why exactly is oxygen therapy important during primary stabilization?

A

PROLONGED HYPOXEMIA AND POOR TISSUE OXYGEN DELIVERY MAY RESULT IN
MULTIPLEORGAN FAILURE AND THEREFORE SHOULD BE TREATED IMMEDIATELY.

Oxygen is an IMPORTANT THERAPEUTIC TOOL IN THE MANAGEMENT OF EMERGENCY AND CRITICAL CARE PATIENTS.

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22
Q

Goal of Oxygen therapy. (5)

A

 Its aim is to increase the fraction of inspired oxygen (FiO2).

 To improve PaO2(partial pressure of arterial O2).

 To improve hemoglobin saturation.

 To increase oxygen delivery to tissues.

 To avoid hypoxemia, tissue hypoxia and lactic acidosis.

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23
Q

Indications for oxygen therapy. (4)

A

 PaO2 less than 80 mm Hg (Blood gas
analyzer)

 Pulse oximetry reading less than 95%

 Respiratory distress symptoms

 All shock patients

24
Q

Respiratory distress symptoms

A

 Increased respiratory rate and effort
 Extended head and neck posture

 Cyanosis(indicates severe hypoxemia)
 Flaring of the nares
 Open mouth breathing

 Changes in respiratory pattern
 Abnormal findings in auscultation

25
Non-invasive and invasive methods of oxygen therapy depends on (5)
 Choice depends on FiO2 desired (fraction of inspired O2)  Length of treatment  Equipment available  Type of patient  In most cases FiO2 30-40% provides sufficient oxygenation
26
What does Long –term O2 therapy require?
requires humidification  Avoids drying of nasal mucosa and degeneration of respiratory epithelium.  Avoids impaired mucociliary clearence
27
Non-invasive O2 therapy methods: Flow-by oxygen
 Oxygen hose near the mouth or nostrils  Flow rate of 2-3 L /min provides FiO2 of 25%  Well tolerated by most patients especially cats  Short term administration
28
Non-invasive methods: Oxygen mask
 Can be used in any patient who lying still and/ or tolerates the mask  More suitable in short term oxygen administration  2-6 L/min flow recommended (patient size)  Possible to reach 40-50%FiO2  minimal equipment required  Needs supervision
29
Non-invasive methods: Oxygen hood or self-made cage
 Easily made in hospital  Leave 2-5 cm from the top open to allow humidity and CO2 elimination  Not tolerated by all patients  Temperature inside collar area may increase rapidly  Maintenance flow 2-5L/min  FiO2 obtained is 30-40%
30
Non-invasive methods: Oxygen cage
Suitable for patients suffering for severe stress and not tolerating other methods (cats)  FiO2 40-50%  Disadvantages - cost  Hyperthermia can develop easily  Possible lack of patient access
31
Invasive methods: Nasal prongs
 Human nasal prongs can be used  Well tolerated by most dogs  Easily dislodged  Unknown FiO2, Possibly higher than flow by oxygen
32
Invasive methods: Nasal catheter
 If O2 is needed more than 24h  Simple to place and requires minimal equipment  Catheter should be changed every 24-48h  Flow rates 50-150ml/kg can provide FiO2 30-70%  Higher flow can be irritating and cause sneezing  Humidification is needed
33
Invasive methods: Trans-tracheal oxygen
 Cathether is inserted through the cricothyroid ligament or caudal to it (between 3rd-5th ring)  Needs experience  May need sedation  Aseptic technique  O2 flow rates of 50 ml/kg/min provide FiO2 40-60%  Risks associated with sedation and infection  Lesions or damage of trachea
34
Invasive methods: Tracheostomy tube
* Needs experience * Need sedation * Aseptic technique * Risks associated with pneumomediastinum, infection and dislodging.
35
If you can't get a patient's pulse oximetry reading over ? with non-invasive O2 therapy then consider what?
If you can't get a patient's pulse oximetry reading over 95% with non-invasive O2 therapy then consider sedation and intubation.
36
Describe Oxygen toxicity.
 Excessive oxygen can be toxic to pulmonary epithelium. - Destruction of cells causes inflammatory reaction.  Inflammatory mediators result in increased tissue permeability and due to this: pulmonary edema, atelectasis, fibrosis and pulmonary function disorders, can result.  FiO2 (fraction of inspired oxygen) more than 50% should not be administered for longer than 24-48 h.  Neonates are more sensitive to O2 toxic effects.
37
Aims of fluid therapy. (4)
MAINTAIN ADEQUATE PERFUSION IN THE BODY RESTORING FLUID BALANCE/TREATMENT OF DEHYDRATION RESTORING ELECTROLYTE IMBALANCES RESTORING NORMAL BLOOD CIRCULATION AND CARDIAC FUNCTION IN SHOCK AND HYPOVOLEMIA
38
total body water percentage intracellular fluid percentage extra cellular fluid percentage interstitial fluid percentage plasma percentage of total body water
total body water percentage 60% intracellular fluid percentage 40% of the above extra cellular fluid percentage 20% interstitial fluid percentage 15% of the above plasma percentage of total body water 5%
39
Indications for isotonic fluids. (4)
 Quick correction of hypovolemia  Treatment of dehydration and electrolyte imbalances  General anaesthesia  Correcting metabolic acidosis
40
Dangers of isotonic fluids. (3)
Lung edema: fluid overload (cats, small dogs), cardiogenic diseases Dilution of blood in anemia and thrombocytopenia cases further exacerbating tissue hypoxia Trauma patient with lung contusion/edema (may worsen it) or brain contusion/edema (may increase intracranial pressue)
41
Describe hypertonic fluids such as NaCl
 5-7,5% solutions  Increases osmotic pressure  Water moves from tissues to blood stream, causes cellular dehydration so you must administer isotonic fluids concurrently via separate IV.  Quickly increases the volume in the blood vessels.  Quick way to fix hypovolemia Used in Head trauma patients to get "water off the brain". Careful in bleeding patients.  Dog: 4-5ml/kg  Cat: 2-4ml/kg  Time of action 30-60min after IV administration
42
Describe Colloids.
Synthetic involve big polysaccharide molecules in isotonic fluid. The bigger the molecule, the longer it stays in the blood vessel. E.g: Dextran-40, Dextran-70, Starch, Hetastarch etc. Non-synthetic colloid options include:  Natural  Fresh frozen plasma  Human albumin
43
Indications for colloids
 Rapid increase in blood volume needed  Hypovolemia  Hypotension  Hypoproteinemia
44
Dangers of colloids include: (6)
Kidney Damage Coagulation Issues Allergic Reactions Volume Overload: leading to pulmonary edema or other forms of fluid retention Electrolyte Imbalance: may lead to hypokalemia or hyperchloremia. Increased Mortality Risk: In some studies, the use of certain colloids has been associated with an increased risk of mortality.
45
Describe blood products
Fresh whole blood- 10-20ml/kg - RBC; WBC; platelets, coagulation factors, plasma protein (albumin) Packed red blood cells (pRBC)-5-10ml/kg - Erythrocytes (PCV 70-80%) Fresh-frozen plasma- 10-20ml/kg (frozen quite soon after collection) Frozen plasma-10-30ml/kg (frozen 8h+ after collection)
46
Indications for transfusion of whole blood versus packed red blood cells?
whole blood - acute hemorrhage, anemias with coagulopathy, hypoalbuminemia or thrombocytopenia packed red blood cells - anemia without coagulopathy
47
Indications for transfusion of plasma?
coagulopathy and DIC
48
Alternative routes for fluid administration (5)
dorsal pedal intraosseous jugular/ central venous line NG or NE tube subcut
49
Define dehydration. Symptoms? Causes?
loss of fluids into the interstitial space from cells (can involve cellular crenation) Symptoms: decreased skin turgor, dry mucous membranes, enophthalmos, weak pulse, hypotension, tachycardia, weakness Causes: diarrhea, vomiting, kidney dysfunction, use of diuretics, diabetes, insufficient fluid intake etc.
50
Define hypovolemia. Symptoms? Causes?
Hypovolemia: insufficient amount of blood in blood vessels (intravascular space) Symptoms: pale mucous membranes, prolonged CRT, weak pulse, tachycardia (Cats!), weakness, cold extremities Causes: severe dehydration, blood loss, vasodilatation.
51
Dehydration versus hypovolemia
Dehydration primarily involves a loss of fluid from the intracellular space and the extracellular space. Hypovolemia specifically refers to a reduction in the intravascular fluid volume.
52
Describe the treatment of Hypovolemic shock and Fluid therapy.
Low volume/hypotensive resuscitation uses isotonic crystalloids.  Aiming for a mean blood pressure of 60-90 mmHg  Fluid rate: boluses 15-20ml/kg within 10-15min  Repeated up to 4 times with reassessments in between If isotonic crystalloids do not work, Add colloids:  Dogs: 5-10ml/kg  Cats: 1-5ml/kg Hypertonic fluids: Do not use in hypernatremia and severe dehydration!  Dogs: 4-5ml/kg  Cats: 2-4ml/kg If blood loss due to active bleeding, try to stim it and Consider blood products.
53
Fluid therapy and Cardiogenic shock
Stop or decrease diuretics first. Consider: does the patient even need fluid therapy. Remember in cardiogenic shock, its not that there isnt enough fluid, its that the heart can pump it properly. Offer water to drink or in wet food. If needed: crystalloids  20-35ml/kg/ 24h Avoid colloids because pulmonary permeability is increased so increased risk for lung edema.
54
Fluid therapy and Cardiopulmonary arrest
Administration using crystalloids is reasonable when suspected hypovolemia.  Conservative doses: 20ml/kg over 15- 20min But You won`t save patient with only fluids!  Cardiopulmonary resuscitation and drugs!  Prognosis is bad anyway so don't focus on fluids in this situation.
55
Fluid therapy and head trauma
Head trauma patients Usually present in hypovolemic shock cause these are usually bleeding patients. Aim of fluid therapy:  Maintain cerebral perfusion  Reduce cerebral ischemia  Reducing and maintaining normal mean arterial BP Isotonic fluids: low volume resuscitation Hypertonic fluids (decrease intracranial pressure)  Dogs: 4-5ml/kg  Cats: 2-4ml/kg  Remains in vasculature for 1h (alternative is Mannitol) Colloids only if really needed; combination of hypertonic solution and colloid.
56
Fluid overload is when
A Normovolemic patient is receiving too much fluids.  Increase in body mass >10% Clinically: edemas and effusions, changes in mental status. Predisposed patients:  Heart failure  Kidney failure in anuria or oliguria  Hypoalbuminemia  Vasculitis Fluid leaks out of vessels into tissues resulting in fluid overload in the two prev.
57
In shock patients: Decreased renal perfusion due to shock, can cause...? Kidney contusions can cause...? Damage to the lower urinary tract can cause...?
pre-renal azotemia renal azotemia post-renal azotomia