UNIT 1-ECG AND CODE MANAGEMENT Flashcards

1
Q

Identify this rhythm

A

Artifact

  1. You will see artifact on the monitor when leads and electrodes are not sexure, or if there is muscle activity (shievering) or electrical interferce
  2. Artifact is a distortion of the baseline and waveforms seen on the ECG
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2
Q

What are your nursing interventions when you have Artifact on your ECG?

A

Because accurate interpretation of cardiac rhythm is difficult when artifiact is present. If artifact occurs, check the connections in the equipment. You may need to replace the electrodes if the conductive gel has dried out.

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3
Q

What are the four properties of cardiac cells that enable the conduction system to start an electrical impulse, send it through the cardiac tissue, and stimulate muscle contraction?

A
  1. Automaticity
  2. Excitability
  3. Conductivity
  4. Contractility
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4
Q

What is automaticity?

A

The ability to initate an impulse spontaneously and continuously

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5
Q

What is excitability?

A

Is the ability to be electrically stimulated

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6
Q

What is conductivity?

A

The aility to transmit an impusle along a membrane in an orderly manner

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7
Q

What is contractility?

A

The ability to respond mechanically to an impulse

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8
Q

Describe the function of the conduction system?

A
  1. Normal cardiac impulse begins in the sinoatrial (SA) node in the upper right atrium.
  2. It spreads over the atrial myocardium via interatrial pathways and internodal pathways, causing atrial contraction.
  3. The impulse then travels to the atrioventricular (AV) node, through the bundle branches. It ends in the purkinje fibers, which transmits the impulse to the ventricles.
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9
Q

Dysrhythias result from disorders of….

A

impusle formation, conduction of impulses or both

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10
Q

The SA node is the normally the ___1___ of the heart? firing at how many times per min?

A
  1. Pacemaker
  2. 60-100
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11
Q

What should we know about secondary pacemakers from another site?

NOT CLEAR ON THIS

A

Can fire in two ways
1. If the SA node fires more slowly than a secondary pacemaker, the electrical signals from the secondary pacemaker may “escape” The secondary pacemaker will then fire automatically at its intrinisc rate.
2. Another way that secondary pacemakers can start is when they fire more rapidly than the normal pacemaker of the SA node. “triggered beats” (early or late) may come from an ectopic focus or accessory pathway which is outside the normal conduction pathway in the atria, AV node or ventricles. The results in a dysrthmia, which replaces the normal sinus rhythm.

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12
Q

What is the intrinsic rate of the AV node or the His- Purkinje system?

A

AV node fires at a rate of 40 to 60 times per minute or the His-purkinje system at a rate of 20-40 times per min

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13
Q

The autonomic nervious system play an important role in the rate of….. in the cardiac system.

A
  1. Impulse formation
  2. Speed of conduction
  3. Strength of cardiac contraction
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14
Q

What are the components of the autonomic nervous system that affect the heart?

A
  1. Vagus nerve fibers of the parasympathetic nervous system and nerve fibers of the sympathetic nervous system
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15
Q

How does stimulation of the vagus nerve which a part of the parasympathetic nervous system affect the heart?

A
  1. Decreases rate of firing of the SA node
  2. Slowed impusle conduction of the AV node
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16
Q

How does stimulation of sympathetic nerves in the sympathetic nervous system affect the heart?

A
  1. Increases rate of SA node
  2. Increases impulse conduction of AV node
  3. Increase cardiac contractility
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17
Q

What are some sympathetic nervous system responses?

A

Stressed state
1. Pupils expand
2. Fast & shallow breathing
3. Heart pumps faster
4. Gut INACTIVE

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18
Q

What are some parasympathetic nervous system symptoms?

A

Calm
1. Pupils shrink
2. Slow deep breaths
3. Heart slows
4. Gut ACTIVE

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19
Q

What is Telemetry monitoring?

A

It is the observation of the patients HR and rhythm at a site distant from the patient

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20
Q

What are the 2 types of systems used for telemetry monitoring?

A
  1. Centralized monitoring
  2. Bedside advanced alarm systems
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21
Q

Centeralized monitoring system- ECG requires….

A

The nurse or a telemetry tech to contionusly observe a group of patients ECGs at a central location

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22
Q

Advanced alarm system bedside monitoring does not require….

A

Constant survallience. These systems have the cability of detecting and storing data. Advanced alarm systems provide different levels of detection of dysrhythimas, ischemia or infarction.

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23
Q

What do we need to remember about computerized monitoring systems?

A

They are not fail proof. We should frequently assess all monitored patients for any signs of hemodynamin instability

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24
Q

What should we know about the P wave?

A
  1. It is the 1st waveform that is normally seen.
  2. This wave should be upright (postive deflection) with a rounded top in lead
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25
Q

The P wave represents what part of the cardiac conduction system?

A

The P wave represents the SA node sending out an electrical impulse and represents atrial depolarization (contraction)

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26
Q

What should we know about the QRS wave (or complex)

A
  1. Normally follows the P wave.
  2. Can take on many different forms
  3. Normal QRS complexes are usually pointy and skinny in width.
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27
Q

The QRS complex represents what in the cardiac conduction system?

A

This complex represents ventricular depolarization (contraction) The ventricles depolarize from the endocardial layer (inside of the heart) to the epicardial layer (outermost layer of the heart)

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28
Q

How do we calculate the HR on an ECG

A
  1. Count the number of QRS complexes in 1 min
  2. QRS complexes in 6 seconds and multiply by 10
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29
Q

How does regularity determined when looking at the ECG?

A

Regularity can be determined by counting the boxes between the waveforms being measured, such as p wave to pwave or qrs to qrs complex (r to r). A regular rhythm will have the same number of boxes or equal space between waveforms or complexes

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30
Q

What are important concepts and questions to consider with our assessment of cardiac rhythm?

A
  1. Interpret the rhythm AND evaluate the clinical status of the patient
  2. Is the patient hemodynamically stable?
  3. Determine cause of dysrthymia
    • This is a priority. For example, tachycardias may be the result of a fever and may cause a decrease in cardiac output and hypotension. Electrolyte distubances can cause dysrthmias and if not treated can be life-threatening
  4. Treat the patient, not the monitor
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31
Q

How should we assess the cardiac rhythm?

A

When assessing a cardiac rhythm, use a consistent and systematic approach.
1. Pwave
- look for its presence, is it upright or more inverted? Is there one for every QRS complex or more than one? Are there atrial fibrillatory or flutter waves?
2. P-R interval
- Is it normal duration or prolonged
3. Ventricular rate and rhythm?
- Is it regular or irregular
4. QRS complex
- Measure the duration of the QRS complex. Is it normal or prolonged
5. ST segment
- Is it isometric (flat), elevated, or depressed
6. Q-T interval
- Measure the duration of the Q-T interval is it normal or prolonged
7. T wave
- Is it upright or inverted

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32
Q

Identify this rhythm

A

Normal Sinus Rhythm which refers to a rhythm that starts in the SA node at a rate of 60-100 times per min and follows the normal conduction path way

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33
Q

Identify this dysrhythmias

A

Sinus Bradycardia

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34
Q

What do we need to know about sinus bradycardia

A
  1. Sinus bradycardia may be a normal sinus rhythm in aerobically trained athletes and in some people during sleep
  2. May be associated with some disease states
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35
Q

What is sinus bradycardia?

A

It occurs in response to a parasympathetic nerve stimulation and certain drugs, carotid sinus massage, valsalva maneuver, hypothermia, increased introcular pressure, vagal stimulation and administration of certain drugs like adrenergic blockers, calcium channel blockers

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36
Q

What are some common disease states associated with sinus bradycardia?

A
  1. Hypothyroidism
  2. Increased ICP
  3. Hypoglycemia
  4. Inferior myocardinal infarction (MI)
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37
Q

What are the manifestations of sinus bradycardia

A
  1. Hypotension
  2. Pale, cool skin
  3. Weakness
  4. Angina
  5. Dizziness or syncope
  6. Confusion or disorienatation
  7. SOB

Clinical significance of sinus bradycardia depends on how the patient tolerates it.

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38
Q

Symptomatic bradycardia refers to a HR that is….

A

Less than 60beats/min and is inadequate for the patients condition, causing the patient to experience symptoms

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39
Q

Treatment of sinus bradycardia includes?

A
  1. Atropine (anticholinergic drug) usually given to symptomatic patients
  2. Pacemaker
  3. Stop offending drugs
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40
Q

Identify this rhythm?

A

Sinus Tachycardia

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41
Q

Identify this rythem

A

Sinus Tachycardia

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42
Q

What do we need to know about sinus tachycardia

A
  1. The conduction pathway is the same in sinus tachycardia as that in normal sinus rhythm.
  2. The p wave is normal, procedes each QRS complex and has a normal shape and duration
  3. The P-R interval is normal and the QRS complex has a normal shape and duration
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43
Q

What is the rate of sinus tachycardia?

A

101-180beats/min

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44
Q

What causes sinus tachycardia?

A
  1. The discharge rate from the sinus node increases because of vagal inhibition or sympathetic stimulation
  2. Sinus tachycardia is associated with physiologic and psycholotic stressors such as exercise, fever, pain, hypovolemia, anemia, hypoxia, hypoglycemia, MI, heart failure, hyperthyridism, axiety and fear
  3. It can also be an effect of drugs such as epi, norepinephrine, atropine, caffeine, theophylline, or hydralazine, sudafed
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45
Q

What are manifestations of sinus tachycardia?

A
  1. Dizziness
  2. Dyspnea
  3. Hypotension
  4. Angina in patients with CAD

Clinical significance of sinus tachycardia depends on the patients tolerance of increased HR.

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46
Q

What is the treatment of sinus tachycardia

A
  1. Guided by cause for example for the patient who is experiencing tachycardia from pain… effective pain managment will treat tachycardia
  2. Vagal maneuver
  3. B-adrenergic blockers
    • Metoprolol can be given to reduce HR and decrease myocardial oxygen consumption
47
Q

Identify this rythem

A

PSVT- Paroxysmal Suptraventricular tachycardia

48
Q

What is PSVT?

A

Is a dysrhythmia startin gin an etopic focus anywhere above the bifurcation of the bundle of HIS. Identification of the ectopic focus is often difficult even with a 12 lead.

49
Q

What is the HR of someone experiencing PSVT

A

151-220 beats/min and the rhythm is regular or slightly irregular

50
Q

What does the rhythm present with PSVT?

A

The P wave is often hidden in the preceding T wave. If seen, it may have an abnormal shape. The P-R interval may be shortned or normal, and the QRS complex is usually normal.

51
Q

Identify this rhythm?

A

PSVT

52
Q

Why do PSVT occur?

A
  1. Because of a reentrant phenomenon (reexcitation of the atria when there is a one-way block)
  2. Usually a PAC triggers a run of repeated premature beats
53
Q

What is a PSVT?

A

Paraoxysmal refers to an abrupt onset and ending. Termination is sometimes folled by a brief period of asystole

54
Q

In a normal heart PSVT is associated with…

A
  1. Overexertion
  2. EMotional stress
  3. deep inspiration
  4. Stimulants such as caffeine and tobacco

PSVT is also associate dwith rheumatic heart disease, dig tox, CAD, or cor pulmonale

55
Q

What are the manifestations of PSVT?

A
  1. HR is 150-220 beats/min (add for clarification)
  2. HR >180 leads to decreased cardiac output and stroke volume
  3. Hypotension
  4. Dyspnea
  5. Angina

Clinical significance of PSVT dpends on the associated symptoms

56
Q

What is the treatment of PSVT?

A
  1. Vagal stimulation
  2. IV Adenosine #1 Drug
  3. IV b-adrenergic blockers (Sotalol)
  4. Calcium channel blockers (Diltizem)
  5. Amiodarone
  6. DC cardioversion (direct current)
    • used if vagal stimulation and drug therapy are ineffective and the patient becomes hemodynamically unstable.
57
Q

Identify this rhythm

A

Effect of Adenosine
1. Explain that the patient may feel chest pressure afte med is given
2. Injection site should be as close to heart as possible
3. Monitor patients ECG continously brief periods of asystole is common
4. Assess for flushing,dizziness, chest pain or palpitation

58
Q

Identify this rhythm

A

Atrial flutter which is a atrial tachydysrthythmia identifed by recurring, regular sawtooth-shaped flutter waves that originate from a signle extopic focus in the right atrum or less commonly, the left atrium

59
Q

What is the atrial rate of atrial flutter

A

200-350 beats per min

60
Q

What is the ventricular rate of atrial flutter?

A

150 beats/min

61
Q

How is atrial flutter discribed?

A
  1. Atrial rhythm is regular and ventricular rhythm is usually regular
  2. The P-R interval is variable and not measureable
  3. The ORS complex is usually normal. Becuase the AV node can delay signal from the atria, there is usually some AV block in a fixed ratio of flutter waves to QRS complexes
62
Q

How is adenosine given?

A

IV closest to the patient givin in 1-3 seconds followed by a rapid flush of 20ml of saline.

1st dose 6mg, 2nd dose 12 mg,

63
Q

Atrial flutter is typically associated with?

A
  1. Chronic lung disease
  2. Cardiomyopathy
  3. Hyperthyroidism
  4. Hypertention
  5. PE
  6. drugs
    • digoxin, quindine, and epi

Can occur in a healthy heart but its rare.

64
Q

What is the treatment of atrial flutter

A
  1. Pharmacologic agent
    • Calcium channel blocker and b adrenergic blockers, antidysrthymic
  2. Electrical cardioverson may be performed to convert the atrial flutter to sinus rhythm in an emergency ( The patient is hemodynamically unstable) and electively.
  3. Radiofrequency catheter ablation is a treatment of choice for atrial flutter. Usuing a low-voltage, high-frequency form of electrical engery. The tissue is ablated (or destroyed) the dysrhythmia is ended and normal sinus rhythm is restored.
65
Q

Patients with atrial flutter at at an increased risk of?

A

Stroke because of the risk of thrombus formation in the atria from the stasis of blood. Warfarin is given to prevent stroke in patients with atrial flutter

66
Q

What is the primary treatment goal of atrial flutter?

A
  1. Slow ventricular response by increase AV block.
67
Q

Identify this ryrthm

A

Atrial Fibrillation

  1. Most common clnically significant dysrhymia w/ respect to morbidity and mortality rates and economic impact
  2. The dysrhythmia may be paroxymal and end spontanously or be persistent lasting longer than 7days
68
Q

What causes Afib and what can it cause if left untreated?

A
  1. As with atrial flutter– causes a decrease in CO and increased risk of stroke
  2. Often develops acutely with thyrotoxicosis, alcohol intoxication, caffeine, electrolyte disturbances, stress, cardiac surgery
  3. A fib results in a decrease in CO because of ineffective atrial contractions (loss of atrial kick) and/or a rapid ventricular response. Thrombi (clots) form in the atria because of blood stasis. An embolized clot may develop and pass to the brain, causing a stroke.
69
Q

Afib is usually occurs in the patient with….

A
  1. Heart disease
  2. CAD
  3. Rheumatic heart disease
  4. Cardiomyopathy
  5. HTN heart disease
  6. HF
  7. Pericadiitis
70
Q

How is afib characterized?

A

1.By a total disorganization of atrial electrical activity due to multiple ectopic foci resulting in loss of effective atrial contraction
2.During atrial fibrillation, the atrial rate may be as high as 350 to 600 beats/min
3.P waves are replaced by chaotic, fibrillatory waves.
4.Ventricular rate varies and the rhythm is ususally IRREGULAR

71
Q

When the ventricular rate is between 60-100 beats/min in a fib this is considered….

A

Atrial fibrillation with controlled ventricular response
example b

72
Q

Atrial fibrillation with a ventricular rate greater than 100 beats/min is atrial fibrillation with a …… response

A

rapid or uncontrolled ventricular response aka AFIB with RVR

example c

73
Q

What is the treatment of Atrial Fibrillation?

A
  1. Slow the ventrial response by increasing the AV block
  2. Drugs to control ventricular rate and/or convert to sinus rhythm (amiodarone and ibutilide most common)
  3. Electrical cardioversion
  4. Anticoagulation
  5. Radiofrequency ablation
  6. Maze procedure with cryoblation
74
Q

What is the goal of treatment for afib?

A

Goal of treatment includes a decrease in a ventricular response (to less than a 100 beats/mins) prevention of stroke and conversion to sinus rhythm, if possible.

75
Q

If a patient is in atrial fibrillation for longer than 48 hours what must we initate before the cardioversion?

A

anticoagulation therapy with warfarin for 3 to 4 weeks before

76
Q

What might be ordered in order to rule out the presence of clots in the atria before a cardioversion?

A

A transesophageal echocardiogram

if there are no clots present we can proceed

77
Q

What happens if drugs or cardioversion cannot convert afib to a normal sinus rhythm?

A

Long-term anticoagulation therapy is required and warfarin is drug of choice

78
Q

Identify this rhythm?

A

PVC: Premature ventricular contraction which is a contraction coming from an ectopic focus in the ventricles. It is the premature (earlY)occurence of a QRS complex

A PVC is wide and distorted in shape compared to a qrs complex coming down the normal conduction pathway

79
Q

What are PVCs that arise from different foci and appear in a different shape from each other called?

A

Mulifocal PVC

80
Q

PVCs that have the same shape are called?

A

Unifocal PVCs

81
Q

When every other beat is a PVC, the rhythm is called…

A

Ventricular bigeminy

82
Q

When every third beat is a PVCs it is called…

A

Ventricular trigeminy

83
Q

What occurs when there are three or more consecutive PVCs?

A

Ventricular tachycardia

84
Q

PVCs are associated with….

A
  1. Stimulants such as caffeine, alcohol, nicotine, aminophylline, epi, isoproterenolm and digoxin, electrolye imbalances, hypoxia, heart disease, emtional stress, fever, exercise
  2. Disease states like MI, mitral valve prolapse, HF and CAD
85
Q

Treatment for PVCs include?

A
  1. Correct cause
  2. Antidysrthmics

Usually not harmful in the patient with a normal heart

86
Q

Identify this rhythm?

A

Ventricular Tachycardia

87
Q

Ventricular rate in vtach is how many beats/min

A

150 to 250 beats/min
Rhythm may be regular or irregular

88
Q

What causes torsades de pointe

A

Mg deficiency. Can only give mg if you know what the mag level is

89
Q

Ventricular tachycardia is associated with….

A
  1. Heart disease
  2. Electrolyte imbalances
  3. Drugs
  4. CNS disorders
90
Q

Patients in Vtach can be considered __1__ or __2__

A
  1. Stable (has a pulse)
  2. Unstable (no pulse)
91
Q

Identify this rhythm

A

Ventricular fibrillation

Vfib which is a severe derangment of the heart rhythm characterized on ECG by irregular waveforms of varying shapes and amplitude. This represents the firing of multiple ectopic foci in the ventricle. Mechanically the ventricle is simply quievering with no effective contraction, and consequently no CO occurs.

Lethal dysrhythmia- HR not measurable. Irreglar and chaotic.

92
Q

VFIB is associated with

A
  1. MI
  2. Ischemia
  3. Disease states
  4. Procedures
93
Q

How does a patient present in vfib?

A

Unresponsive, pulseless and apneic

If not treated rapidly, dealth will result

94
Q

How do you treat VFIB?

A
  1. Treat with immediate CPR and ACLS
  2. Defibrillation
  3. Drug therapy (epinephrine, vasopression)
95
Q

Identify this rhythm

A

Asystole- represent the total absence of ventricular electrical activity

Occasionally P waves are seen
No ventricular contraction occurs because depolarization does not occur

96
Q

How do patients with asystole present?

A

Unresponsive, pulseless, and apneic

Lethal and requires immediate tx
You should always assess rhythm in more than one lead..

97
Q

What is the prognosis of a patient in asystole?

A

Poor

98
Q

Asystole is usually the result of…

A

Advanced cardiac disease, severe conduction disturbance, or end stage HF

99
Q

How is asystole treated?

A

Treat with immediate CPR and ACLS measures

Epinepherine and or vasopressin
Intubation

100
Q

Identify this rhythm
HINT NO PULSE

A

Pulseless electrical activity- Is a situation in which organized, electrical activity is seen on the ECG, but there is no mechanical activity of the ventricles and the patient has NO PULSE

Prognosis is POOR unless the underlying cause is quickly identified and treated

101
Q

What are your Hs and Ts?

MUST KNOW
6H
5p

A

H’s
1. Hypovolemia
2. Hypoxia
3. Hydrogen ION (acidosis)
4. Hyper-/hypokalemia
5. Hypoglycemia
6. Hypothermia

T’s
1. Toxins(drug overdose)
2. Tamponade (cardiac)
3. Thrombosis (MI and pulmonary)
4. Tension Pneumothorax
5. Trauma

102
Q

What is the treatment of PEA?

A
  1. CPR followed by intubaiton and IV epinephrine
  2. Treatment is directed toward correction of the underylying cause
103
Q

Identify this rhythm

A

3rd degree AV heart block (complete heart block)- Constitutes one form of AV dissociation in which no impulses from the atria are conducted to the ventricles. THe atria are stimulated and contract independently of the ventricles. the ventricular rhythm is an escape rhythm, and the ectopic pacemaker may be above or below the vifurcation of the bundle of his

Atrial rate is usually a sinus rate of 60-100
Ventricle rate depends on the site on the site of the block. if its the AV node the rate is 40-60 beats/min and if its in the his-purkinje system it is 20-40 beats/min

103
Q
A
104
Q

Describe the characteristic of thrid degree AV heart block on the ECG

A
  1. Atrial and ventricular rhythms are regular but unreleated to each other
  2. The P wave has a normal shape
  3. The P-R interval is variable and there is no relationship between the Pwave and the QRS complex.
  4. THe QRS complex is normal if an escape rhythm is initated at the bundle of his or above it is widened if an escape rhythm is initatied below the bundle of his.
105
Q

3rd degree AV heart block (complete heart block) is associated with…. and results in… which can lead to what….

A
  1. Associated with severe heart disease (CAD, MI), some systemic disease (amylodosis), certain drugs (digoxin, b-adrenergic drugs, calcium channel blockers)
  2. Usually results in decreased cardiac output with subsequent ischemia, HF, and shock
  3. Can Lead to syncope, HR and shock
106
Q

How is 3rd degree AV heart block treated?

A
  1. Pacemaker
  2. Drugs to increase heart rate if needed while awaiting pacing. (atropine, dopamine,epi) usually temporary to increase HR and support BP until temporary pacing is started. Will need a permenant pacemaker asap.
107
Q

What is a pacemakers purpose?

A
  1. Control heart rate when conduction is compromised
  2. Single chamber pacemaker- paces either the atria or ventricle
  3. Dual chamber pacemaker- paces both atrum and ventricle
108
Q
A
109
Q

What are CPR ratio for an adult?

A

30:2

110
Q

What is our CPR rate for an adult?

A

100-120 compressions per min

111
Q

How many breaths per 6 seconds

A

2

112
Q

How many J do we charge our biphasic defibrillator to?

A

120-200J

113
Q

How many J do we charge our monophasic defibrillator to?

A

360