Unit 1 Review Flashcards

1
Q

Types of joints and examples.

A
  • Fibrous joints, immovable and joined by tough collagen tissue Eg: suture in skull.
  • Cartilaginous joints, 2 bones joined together by cartilage, limited movement Eg: symphysis pubis and between ribs and costal cartilage.
  • Synovial joints, true joints, greatest range of motion Eg: elbow, knee, hip.
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2
Q

Parts of a synovial joint and their function

A

Articular cartilage: protects bone tissue and reduces friction when bones move

Joint capsule: made of fibrous tissue that surrounds the joint, prevents unwanted material from entering joint and adds stability

Synovial fluid: lubricant for moving surfaces

Ligaments: fibrous bands that connect bone to bone and adds stability

Bursae: fluid filled sacs that reduce friction Eg; tendons rub over bones

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3
Q

Osteoarthritis article - main points and where OA is most commonly found

A

Point 1: address the question of what osteoarthritis is
Point 2: provide an overview of the epidemiology of OA particularly risk factors for development
Point 3: consider role of exercise on join tissue and disease management

OA is most commonly found in knees, hips and hands

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4
Q

Types of muscles

A

Smooth: surrounds body’s internal organs, involuntary, and doesn’t get tired
skeletal: muscle attached to bone, voluntary, and appears striated under microscope
cardiac: only found in heart, involuntary

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5
Q

Types of muscle contractions

A

Concentric contraction: muscles shorten (flexion)

Eccentric contraction: muscles lengthen (extension)

Isometric contraction: muscles length does not change, remains static. Holding a contraction

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6
Q

Agonist/Antagonist examples, importance, and how they work together to produce movement at joints

A

Ex: in knee flexing agonist are hamstrings, antagonist are quadricepts
Ex: in elbow flexion agonist is biceps, antagonist is triceps
Ex: in dorsiflexion agonist is tibiales anterior, antagonist is gastrocnemius and soleus.

Agonist is the muscle moving and antagonist are the muscles that are lengthening when agonist muscles contract.

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7
Q

Origin/Insertion - where are they in relation to skeleton and examples

A

Origin: the point at which muscle attaches to more stationary bone

Insertion: the point at which muscle attaches to the bone that’s moved the most

Ex: biceps: origin is near shoulder, insertion is on the radius

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8
Q

Parts of Sarcomeres

A

Actin, myosin, troponin, tropomyosin

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9
Q

Motor unit

A
  • Refers to motor neuron, it’s axon and muscles fibres it stimulates (components to stimulate a muscle)
  • can either turn on all muscle fibres the motor unit is attached to or none (all or none law)
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10
Q

What is sliding filament theory

A
  • Describes how muscular contraction occurs
  • Muscle fibres do not shorten but instead overlap to appear shorter
  • Myosin filaments (thick) create cross bridges with actin filaments (thin)
  • Myosin attaches, rotates, detaches and repeats.
  • this process makes them overlap
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11
Q

Different Nutrients and purpose

A

Carbohydrates: provide energy for use by cells in form of glucose (used first as an energy source)

Fat: used as an energy source after carbohydrates (if not used will be stored in fat cells)

Protein: used for catalysant chemical reactions, transporting of materials, build new tissue (protein will be used for energy if no fat or carbs)

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12
Q

The three metabolic pathways and which types of activities prioritize each

A

ATP-PC (anaerobic alactic) - short bursts of energy (0-10 seconds) Eg: sprints, jumping, weights

Glycolysis (anaerobic lactic) - longer bursts of energy (0-90 seconds) Eg: hockey, middle distance track 400m, 800m

Cellular Respiration - long sustained activity Eg: marathons

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13
Q

Where do the metabolic pathways occur in the body

A

ATP-PC occurs in the cytoplasm of the muscle cells

Glycolysis occurs in the cytoplasm

Cellular respiration occur in the cytoplasm and mitochondria of the cell (depends which step)

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14
Q

How much net ATP does each metabolic pathway produce

A

ATP-PC - 1 ATP molecule per molecule of glucose

Glycolysis - 2 ATP per molecule of glucose

Cellular respiration - net gain of 38 molecules of ATP per molecule of glucose

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15
Q

Chemical equation for each metabolic pathway

A

ATP-PC: PC + ADP -> ATP + creatine

Glycolysis: C6H12O6 + 2ADP + 2P -> 2C3H6O3 + 2ATP + 2H2O (product is lactic acid)

Cellular respiration: C6H1206 + 602 -> 6C02 + 6H20 + 38 ATP

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16
Q

Advantages and disadvantages of each metabolic pathway

A

ATP-PC advantages: allows for quick intense muscle contraction and produces little amount of ATP quickly, requires no oxygen
Disadvantage: lasts very short amount of time, only good for short bursts of energy

Glycolysis advantages: longer bursts of energy and doesn’t require oxygen
Disadvantages: build up of lactic acid which causes fatigue, still not long term energy production

Cellular respiration advantages: net gain of ATP is the most, can sustain very long activities,
Disadvantages: longest pathway and requires oxygen

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17
Q

Each step of cellular respiration and products

A

Step 1: Glycolysis 2 ATP produced, except pyruvic acid converted to Acetyl CoA

Step 2: Krebs cycle - Acetyl CoA broken down to yield 2 ATP

Step 3: Electron Transport Chain (ETC) - 34 ATP produced

18
Q

Lebel the structures of the heart

A
19
Q

Name the blood vessels of the heart and summary of what they do

A

Arteries: carry blood away from heart, in systemic circulation it’s oxygenated blood and in pulmonary circulation it’s deoxygenated blood

Veins: carry blood towards heart, in systemic circulation is deoxygenated blood, and in pulmonary circulation it’s oxygenated blood

Capillaries: smallest blood vessel (only one cell thick). Allows exchange of gas, nutrients, and waste between blood and tissue

20
Q

What is Skeletal muscle pump

A

It helps in the return of blood back to the heart through the veins.

With each contraction of skeletal muscle pump, blood is pushed back to heart

21
Q

What is the pathway of blood

A

Deoxygenated blood comes into the superior and inferior vena cava then to the right arium, moves into the right ventricle and is brought into the pulmonary arteries to be sent out to the lungs.
After picking up oxygen, the blood travels back to the heart through the pulmonary veins into the left atrium, to the left ventricle and into the aorta and descending aorta to be sent out to the body’s tissues.

22
Q

How does the heart beat initiate

A

Sinoatrial node (SA node): specialized region of tissue in right atrium. Initiates electrical signal that lead to contraction (pacemaker of the heart)

Atrioventrical node (AV node): tissue that transmits signal from atria to ventricle.

23
Q

What is ECG

A

Process of producing electrocardiogram. Which is a recording of the hearts electrical activity

24
Q

What is heart disease and risk factors

A

Heart disease can be:

Artherosclerosis - gradual narrowing of arteries resulting from accumulation of plaque on the lining of blood vessels

Myocardial infarction - blood flow in coronary circulation becomes blocked
Results in tissue dying due to lack of oxygen

Risk factors: Smoking, Elevated blood lipids, Hypertension, Family history, Physical inactivity

25
Q

Effects of training on cardiovascular system

A
  • Heart rate (HR): rises during activity
  • Cardiac output (Q): The amount of blood pumped out by each ventricle in
    one minute. Rises during training
  • Stroke Volume (SV): amount of blood pumped by each ventricle with each
    heart beat. Rises during activity
  • Blood pressure (BP): Rises during
    increased activity
  • Distribution of blood flow: blood is redirected to skeletal muscles
26
Q

Factors that influence cardiac output

A

Heart rate, stroke volume

27
Q

How do you calculate cardiac output

A

Cardiac output (Q) = Heart Rate (HR) x Stroke Volume (SV)
(Unit mL/min or L/min)

Stroke Volume (SV) = Cardiac output (Q) ÷ Heart Rate (HR)
(Unit mL)

Heart rate (HR) = Cardiac output (Q) ÷ Stroke Volume (SV)
(Unit bpm)

28
Q

How do you calculate stroke volume

A

Stroke Volume (SV) = End Diastolic Volume (EDV) - End Systolic Volume (ESV)

(Unit is mL/beat)

29
Q

Expected differences between trained vs sedentary individuals. Both at rest and during exercise

A

On average an athletes heart rate will be lower than a non-athlete at rest and at max.

On average an athletes SV will be higher than a non-athlete at rest and at max

On average an athletes Q will be more or less the same as a non-athlete at rest, but at max will be higher than a non-athlete

30
Q

In total does the distribution of cardiac output to various regions during progressive exercise to maximum power increase?

A

Yes

31
Q

What is the role of the respiratory system

A

It supplies O2 to blood
Remove CO2 from blood
Regulate blood pH

32
Q

Pathway of air

A

Down throat, into trachea, tranches is then divided so sir goes into both lungs.

33
Q

Differences in partial pressures of gasses

A

Alveoli are surrounded by capillaries. Gases will diffuse from area of high concentration to low concentration between alveoli and capillaries.
The concentration is referred to the partial pressure of each gas

34
Q

Types of respiration and brief explanation

A

External respiration: exchange of gases between external environment and respiratory organs

Internal respiration: exchange of gases at tissue level, between cells and blood

Cellular respiration: process in which cells use O2 to generate energy

35
Q

What is the mechanism of breathing - inspiration

A

(Inhaling)

  • Air is brought into lungs externally
  • Contraction of diaphragm and intercostal muscles increase volume of thoracic cavity.
  • increased volume = decreased pressure
  • air will flow from area of hight pressure to low pressure
36
Q

What is the mechanism of breathing - Expiration

A

(Exhaling)

  • Air is exploded from lungs into external environment
  • Relaxation of the diaphragm and intercostal muscles move thoracic cavity back up and in
  • decreased volume = increased pressure
  • air will be forced out of the lungs
37
Q

What is VO2max

A

Maximal volume of oxygen that the body can use in one minute, per kg of body weight

A trained individual can increase their VO2max which allows them to perform more intense exercise for a longer duration

38
Q

What is Oxygen deficit and debt

A

Oxygen deficit is the oxygen needed to be an ideal amount during intense exercise. Oxygen debt is the oxygen needed to restore all energetic system.

39
Q

What is Onset of Blood Lactate Accumulation (OBLA)

A

OBLA is the point at which lactate levels begin to accumulate rapidly in blood.

40
Q

Describe how muscles contract

A
  • electrical signal is sent to muscle fibre
  • this releases calcium ions
  • The calcium ions attached to troponin on the actin
  • This displaces tropomyosin to reveal binding sites
  • by using a ADP and phosphate molecules, the phosphate is dropped, which causes the myosin head to attach to the binding site
  • The two filaments then glide until ATP releases the bond
  • ATP is then converted to a TP plus phosphate which gives energy to the myosin head for the next muscle contraction.