Unit 3 Flashcards

(149 cards)

1
Q

Stroke volume is impacted on which specific 3 things

A

1 myocardial contractility
2 preload
3 SVR-afterload—resistance the ventricles must overcome in order to circulate

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2
Q

What 3 things are comprised in preload, after load, and contractility

A

Preload—venous return, fluid volume, atrial contraction
After load—resistance to ejected blood—systemic vascular resistance, health of vessels, vasoconstriction
Contractility—sympathetic stimulation—NE, parasympathetic stimulation- AcH, drugs—beta blockers calcium channel blocker

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3
Q

Hat is central perfusion

A

Ability of heart and large vessels to deliver oxygenated blood to body organ and tissues
—affected by cardiac output
—both mechanical and electrical components of the pump

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4
Q

What is tissue perfusion

A

Blood that flows through arteries and capillaries into target tissues
—mist have adequate arterial pressure

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5
Q

What are some things that impair central perfusion

A

Occurs due to altered conduction, reduced myocardial contraction, ineffective heart valves, decreased intramuscular volume. Or systemic vascular resistance
Ex: ventricular fibrillation, endocarditis, severe dehydration, hemorrhage

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6
Q

What happens to occur tissue perfusion

A

Arterial blockage
Stroke, myocardial infarction

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7
Q

A nurse is caring for a client with a surgical wound healing by secondary intention. Which finding would indicate proper healing during the granulation phase?
A. Approximation of wound edges.
B. Fibroblast activity with capillary formation.
C. Thick scar tissue forming with irregular borders.
D. Platelet aggregation forming a clot.

A

B

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8
Q

A patient is admitted with erythema, swelling, and warmth around a surgical site. What is the priority nursing intervention?
A. Administer prescribed NSAIDs.
B. Apply a warm compress to reduce swelling.
C. Assess for systemic manifestations of infection.
D. Perform wound care with aseptic technique.

A

C

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9
Q

A nurse is assessing a patient with a pressure ulcer on the sacrum. The ulcer is full-thickness, has slough present, and exposes subcutaneous tissue but no muscle or bone. What stage should the nurse assign?
A. Stage 2
B. Stage 3
C. Stage 4
D. Unstageable

A

B

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10
Q

A nurse is educating a client about the manifestations of localized inflammation. Which of the following signs should the nurse include?
A. Pain
B. Erythema
C. Pallor
D. Loss of function
E. Fever

A

A b d

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11
Q

A nurse is implementing interventions to prevent the development of pressure injuries in an immobile patient. Which actions should the nurse take?
A. Reposition the patient every 2 hours.
B. Place a pillow under the patient’s calves to elevate the heels off the bed.
C. Massage reddened areas to improve circulation.
D. Use a lift sheet when repositioning the patient.
E. Keep the patient’s skin clean and dry.

A

A

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12
Q

Which factors can delay wound healing? (Select all that apply)
A. Smoking
B. Malnutrition
C. Adequate perfusion
D. Diabetes
E. Advanced age

A

A b d e

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13
Q

A nurse is caring for a patient with atopic dermatitis experiencing intense pruritus. What is the priority nursing intervention?
A. Administer prescribed corticosteroids.
B. Teach the patient to avoid environmental triggers.
C. Apply a topical antihistamine.
D. Encourage the patient to wear loose clothing.

A

A

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14
Q

A patient presents with a newly formed wound exhibiting erythema, swelling, and exudate formation. What is the nurse’s priority action?
A. Perform a culture of the wound.
B. Apply a moist wound dressing.
C. Encourage the patient to increase protein intake.
D. Administer prescribed antibiotics.

A

A

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15
Q

A nurse is caring for a bedridden client at high risk for pressure injuries. Which intervention should the nurse prioritize?
A. Monitor the client’s serum albumin levels.
B. Apply a foam dressing to the sacrum.
C. Turn the client every 2 hours.
D. Educate the client about hydration and nutrition.

A

C

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16
Q

Which finding is most indicative of systemic inflammation in a patient with cellulitis?
A. Localized warmth and redness at the site of infection.
B. Elevated WBC count and fever.
C. Clear exudate draining from the affected area.
D. Thick scar tissue forming over the wound.

A

B

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17
Q

A nurse is educating a patient about healing by tertiary intention. Which scenario best demonstrates this process?
A. A clean surgical wound closed immediately with sutures.
B. A wound left open due to infection, then later sutured.
C. A pressure ulcer that closes gradually with granulation tissue.
D. A superficial wound that heals with minimal scarring.

A

B

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18
Q

Which laboratory test result is most relevant for monitoring systemic inflammation in a patient with rheumatoid arthritis?
A. Hemoglobin level.
B. C-reactive protein (CRP).
C. Sodium level.
D. Platelet count.

A

B

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19
Q

Which of the following factors may delay wound healing? (Select all that apply.)
A. Advanced age
B. Diabetes
C. High protein diet
D. Smoking
E. Low hemoglobin levels

A

A b d e

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20
Q

A nurse is caring for a patient with a pressure ulcer exhibiting tunneling and slough. What is the priority nursing action?
A. Measure the depth of the wound and document findings.
B. Debride the wound to remove dead tissue.
C. Administer prescribed antibiotics.
D. Apply a hydrocolloid dressing to the wound.

A

A

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21
Q

A nurse is caring for a patient with localized inflammation after a minor injury. Which is the priority intervention?
A. Administer prescribed NSAIDs.
B. Encourage fluid intake to promote healing.
C. Apply a cold compress to reduce swelling.
D. Perform range of motion exercises to prevent stiffness.

A

C

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22
Q

T/F inflammation does not mean infection

A

T

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23
Q

What are three types of inflammation and give an example of each

A
  1. Acute—paper cut (no scar)
    2 subacute—scab—same as acute but healing weeks to months
    3 chronic — months to years (autoimmune disease)
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24
Q

What are the 4 inflammation responses in order

A

1 histamine and prostaglandins released
2 capillaries dilate clotting begins
3 chemotactic factors attract phagocytic cells
4 phagocytes consume pathogens and cell debris

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25
What stage is capillaries dilate and clotting begins in the inflammation response
2
26
What stage in the inflammation response is when histamine and prostaglandin release
1
27
What stage in the inflammation response is phagocytes consume pathogens and cell debris
4
28
What inflammation response stage is chemotactic factors attract phagocytic cells
3
29
What are the inflammation manifestations of local inflammation Elevated WBC Warmth Swelling Fever Malaise Swelling Erythema (redness) Loss of function
Erythema Warmth Pain Swelling Loss of function
30
T F fever is d/t angiotensin response
F Fever is d/t cytokine response—helpful; tells us the body is attempting to respond
31
What are the two inflammation. Manifestations
Local and systemic
32
What lab values are correlated to systemic inflammation
CBC WBC—elevated CRP—elevated and detects systemic inflammation
33
What does CRP lab value do with inflammation tracking
Marker for inflammation in body we want it to trend But it is not specific
34
What is RICE with inflammation
Rest Ice Compress Elevate
35
How do you manage inflammation and treat
Protein Fluids NSAIDS Antipyretics
36
What is Allergic Contact Dermatitis
Lesasion appears 2-7 days after allergen contract Red papules and plaques Purities
37
How do you treat Allergic Contact Dermatitis
Corticosteroids and antihistamines PO and topical
38
What is Atopic dermatitis
Generic chronic relapsing Environment triggers Intense pruitis
39
How do you treat atopic dermatitis
Reduce environmental stressors, corticosteroids
40
What are the wound classifications
Surgical vs nonsurgical Acute vs chronic Depth of tissue loss Skin tear
41
What are the three depths of tissue loss
Superficial—epidermis Partial thickness—extends to dermis Full thickness—deep layers of tissue destruction
42
Healing process What are the phases of primary intention
Initial phase—clot formation, platelet matrix forms, WBCs flood the area Granulation phase—fibroblast migration, collagen secretion, formation of capillaries Maturation—collagen remodeling, scar strengthens
43
What are the qualities of secondary intention of healing process (Trauma.infection after healed the opened and infected)
Edges cannot be approximated Greater inflammation response leads to more debris, cells and exudate Greater defect and gaping wound edges lead to more granulation tissue and a larger scar
44
What is tertiary intention healing process mean (delayed primary)
Occurs when contaminated wound is left open and sutured closed after infection is controlled Also occurs when a primary wound becomes infected or opens Largest and deepest scar WOUND VAC
45
T/F dryness is the enemy of wound healing
T Most wound healing is good for skin formation
46
What does it mean to be an unstageable wound
Slough and eschar must be removed to expose the base of the wound for true depth to be determined Will be stage 3 or 4 If on heals, stable eschar should not be removed
47
A nurse is assessing a bedridden client and notes an open area on the sacrum with visible subcutaneous fat and slough present around the wound edges. Which stage should the nurse assign to this pressure ulcer
C
48
A nurse is caring for a client with limited mobility. Which of the following actions should the nurse take to help prevent pressure ulcer formation (SATA) A reposition the client every 4 hours B place a pillow under the client’s calves to elevate the heels off the bed C massage any reddened areas to promote circulation D keep the head of the bed elevated at 30 degrees or lower, if possible E ensure the client’s skin remains dry and clean
B D E
49
A nurse is providing care to a client with a surgical wound. which of the following findings would indicate that the wound is healing by primary intention A the wound is open with granulation tissue forming at the base of the B the wound edges are approximated, with minimal scar formation C the wound is left open to drain and gradually closes from the edges D the wound shows thick scar tissue forming with irregular borders
B
50
What are the kidney functions
Regulate the volume and composition of the ECF Excrete waste products Control blood pressure Make erythropoietin Activate vitamin d Regulate acid-base balance
51
What are the two types of renal impairment
Partial or complete impairment of kidney function that results in the inability to excrete metabolic waste products and water
52
What are the lab alterations of acute/chronic renal failure
BUN and creatinine and K+ increase
53
what are the staging of CKD
Stages 1-5 Gradual, irreversible decline in renal function Staging determined by GFR End-stage Renal disease (ESRD) kidneys can no longer function on their own.
54
What are the major causes and risk factors of CKD
Diabetes, hypertension, glomerulonephritis, other urinary disease. CKD affects all body systems
55
what are the manifestations of CKD of lab tests
Increased BUN and creatine Lethargy, fatigue, impaired thought process, and headaches Elevated triglycerides Increased K and Mg
56
What are the clinical manifestation of CKD
Anemia, risk for bleeding, infections, Dyspnea, GO impacts, neuro impacts, urea crystals on skin —pruritis from calcium -phosphate deposits
57
Hat are the comorbidities of CKD
Cardiovascular disease Osteoporosis Hypertension
58
What does calcium and phosphate in CKD cause
Causes CKD mineral and bone disorder (CKD-MBD)
59
What are the nutrition management of CKD
Protein—avoid high protein Fluids—yesterdays output + 600 ml Sodium: 2-4 grams/day Potassium 2-3 grams/day (unless hemodialysis) Phosphorous restrictions 1G/day in ESRD
60
What medications treat anemia in CKD
Erythropoietin, iron supplements, folic acid
61
Hyperkalemia medication in CKD
Kayexalate
62
What medication should treat mineral and bone disease
- vit d, phosphate binders
63
What medication for CKD to treat dyslipidemia
Statins
64
What are some implications for treating symptoms of CKD
CKD causes the potential from drug toxicity Drug doses and frequency adjusted based on CKD severity Toxicity may result as drug levels increase
65
What are some nephrotoxic medications
NSAIDS, vancomycin, chemo, calcium carbonate, IV contrast
66
Why are some interventions in nursing care in CKD
Measure vital sings—daily BPs Daily weights Strict intake and output
67
What is the goal of dialysis
To correct fluid and electrolyte imbalances and remove waste products Two methods: hemodialysis or peritoneal dialysis
68
what is the difference between AV fistulas and AV grafts
AV fistulas—preferred access, need 3 months to heal before use, assess for thrill and bruit, no BP or lab draws in that arms AV graft—synthetic graft forms a bridge between vein and artery. Tends to get infected or become throbogenic, infections may require removal
69
What are teh nursing assessments for hemodialysis before treatment, during treatment, after treatment
Before treatment—fluid status, condition of vascular access, vital signs, labs During treatment- two large bore needles in fistula, heparin is added to prevent clotting, during dialysis take vital signs are least every 30-60 minutes After treatment— fluid status, viral signs, labs (potassium)
70
What are the assessments of clients on peritoneal dialysis
Assess fluid status, vital signs, catheter site, labs (weekly) Monitor inflow and outflow, document intake and output, maintain aseptic dressing changes
71
What are some complications of peritoneal dialysis
Exit site infection—redness, tenderness, drainage Peritonitis—abdominal pain, rebound tenderness, etc. hernias—due to increased abdominal pressure Lower back problems—due to increased abdominal pressure Bleeding—common with initial catheter placement, ongoing is a problem Pulmonary complications—decreased lung expansion, Atelectasis Protein loss
72
What are some advantages of hemodialysis
Rapid fluid removal Rapid removal of urea and creatinine Effect potassium removal Less protein loss Lowering triglycerides Home dialysis possible
73
What are some disadvantages of hemodialysis
Vascular access problems Diet and fluid restrictions Heparinizations may be necessary Hypotension during dialysis Added blood loss that contributes to anemia Surgery for access
74
Where is a peritoneal dialysis placed
Placed surgically through the anterior abdominal wall
75
What are the phases of peritoneal dialysis
Inflow0 2 to 3 L over 10minutes Dwell (equilibrium) 20-30 minute-8 hours Drain 15-30 minutes
76
What are the two systems of peritoneal dialysis
Automated peritoneal dialysis—during sleep Continuous ambulatory peritoneal dialysis—manual exchange four times a day
77
What do you assess and care for clients on peritoneal dialysis
Assess- fluid status, vitals, catheter site, labs (weekly is stable, daily if unstable Monitor inflow and outflow Document I/Os
78
What are some advantages for peritoneal dialysis
Immediate initiation Less complicated than hemodialysis Portable system Fewer diet restrictions Short training Less cardiovascular stress
79
What are some disadvantages for peritoneal dialysis
Bacterial or chemical peritonitis Protein loss into dialysate Exit site and tunnel infections Self image problems w catheter placement Hyperglycemia Catheter can migrate
80
What are some contraindications for patients with peritoneal dialysis
Contraindicated in patient w multiple abdominal surgeries, trauma, unrepaired hernia
81
What are some complications of dialysis overall
Hypotension Muscle cramps Blood loss Hepatitis C infection control Emotional distress
82
What are the three components of perfusion
1 pump 2 volume 3 vascular tone
83
What is the cardiac output equation
Cardiac output=HRxSV
84
What are the three things that stroke volume is impacted by
Myocardial contractility—how hard the myocardium contracts for a given preload Preload—amount of blood entering the ventricles during diastole After load—resistance the ventricles must overcome in order to circulate blood
85
What affects preload
Venous return Fluid volume Atrial contraction
86
What affects after load
Resistance to ejected blood =systemic vascular resistance Health of vessels (atherosclerosis) Vasoconstriction
87
What affects contractility
Sympathetic stimulation —norepinephrine Parasympathetic stimulation —AcH Drugs -beta blockers, calcium channel blockers
88
What is the difference between central perfusion and tissue perfusion
Central—ability of heart and large vessels to deliver oxygenated blood to organs and tissues (Affected by cardiac output, both mechanical and electrical components) Tissue—blood that flows through arteries and capillaries into target tissues (must have adequate arterial pressure—product of cardiac output and systemic vascular resistance
89
What types of medication might help perfusion
Antiplatelets Anticoagulant Vasodilators Statins Digoxin Aspirin Antidysrhythmics Ace inhibitors Dieurtetics Calcium channel blockers
90
What is virchow triad with venous thrombosis
Venous stasis: valve problem or muscle problem Hypercoaguablility: blood clots more easily Endothelial damage: direct or indirect; stimulates platelet cascade
91
What are the Clincal manifestations of superficial thrombosis What are Clincal manifestations for deep VTE
Superficial—usually is varicosity, palpable firm chord like veins, area around the vein is itchy, painful, red and warm, edema is rare Deep VTE—present in deep veins, tenderness, venous distention/congestion, deep in reddish color, edema and “heaviness” in extremities, may have no physical signs
92
What is the difference between thrombu and embolus
Thrombus is called an embolus when it becomes mobile.
93
What are serious complications of superficial thrombus
VTE=pulmonary embolism (PE), stroke, sudden cardiac death
94
What are preventative interventions of thrombus
1. Early and progressive mobilization 2. Compression stockings 3. Sequential compression devices 4. Prophylactic antiplatelet/blood thinning medications
95
What do you do if there is a known thrombus
Extremity assessment, neuro checks, pulmonary assessment
96
What are the interventions of thrombus
Administer medications as prescribed Evaluate lab values Educate on risks of nicotine, hormone therapy, and inactivity Educate on s/s of DVTs and further complications
97
What are some considerations of enoxaparin (lovenox)
Thrombin inhibitor Give subq Use caution in patient with hex heparin-induced thrombocytopenia
98
What is the class and considerations of warfarin
Vitamin K antagonist orally Give at the same time each day
99
What is the class and considerations of apixaban (eliquis) and rocaroxaban (xarelto)
Factor Xa inhibitors oral Good for prevention or treatment of VTEs
100
What is an ischmic stroke
Inadequate blood flow Injury to blood vessel wall and formation of clot or embolic (mobile clot lodges in cerebral artery)
101
What is a hemorrhage stroke
Commonly caused by hypertension, vascular malformation, or disrupted coagulation (unable to clot) Bleeding into brain
102
What are some risk factors for ischemic strokes
Perfusion and DVT
103
What does BEFAST mean
Balance Eyes Face Arms Speech Time (time to call for ambulance)
104
What are some manifestations of right brain damage
Paralyzed left side Left side neglect Rapid performance, short attention span Impaired judgement
105
What are some manifestation of left brain damage
Paralyzed right side Impaired speech Aware of deficits Impaired comprehension
106
What are some managements of stroke
Can they swallow safely Are they impulsive Can they reposition themselves How do they communicate
107
What is the class and considerations of heparin
Thrombin inhibitor (indirect) Give IV for existing blood clots Give subq for prophylaxis
108
What is the difference between primary hypertension and secondary hypertension
Primary—idiopathic, accounts for 90-95% of cases Risk factors—altered endothelial function, inc SNS activity, inc sodium, obesity, tobacco Secondary—specific cause (pregnancy, drug, hepatic disease, renal disease) sudden onset treatment is aimed at removing underlying cause
109
What are the categories of hypertension, levels
Normal <120 and/or <80 Elevated 120-129 and or <80 Hypertension stage 1 130-139 and/or 80-89 Hypertension stage 2 140+ and/or 90+
110
What are some clincial manifestions of hypertension
Fatigue, dizziness, palpitations, angina, Dyspnea
111
What are organs are at highest sensitivity to HTN and what do for diagnosis
Brain, kidneys, eyes, heart Extensive HandP Urinalysis Blood sugar, cbc, bmp, lipid, ecg
112
What are compactions to the kidneys in hypertension
Microalbuminuria Proteinuria inc creatine (>=1.5)
113
What are complications of HTN in the brain
Stroke and transient ischemic attack
114
What are complications of HTN in the heart
CAD, heart failure, left ventricular hypertrophy
115
What are some complications of HTN in the abdomen
Aneurysm, aortic dissection
116
T/F is penile erectile dysfunction a complication of hypertension
T
117
What is the life’s simple 7 in multidisciplinary care of HTN
Stop smoking, get active, control cholesterol, manage blood pressure, eat healthy, lose weight, reduce blood sugar
118
What is the dash diet
Low fat, whole grain, fish, poultry, beans, low sodium
119
What do diuretics, ace inhibitors, CCBs and beta blockers do to affect blood pressure
Duiretics—affect stroke volume (reduce preload) —monitor electrolytes Ace inhibitors—affect cardiac output (reduce preload) —may not be well tolerated Beta blockers—decrease HR CCB—decrease SVE (reduce after load)
120
What is the first line therapy
Use non pharmacological and 1 medication —introduce second medication depending on stage and response
121
What is emergency crisis hypertension
SBP >180 and or DBP >120 Emergent when target organ disease exists in Encephalopathy, brain hemorrhage, MI, HF, acute renal failure, aortic aneurysm, retinopathy
122
What are the management of emergency HTN
Hospitalization —IV antihypertensives Invasive BP monitoring with arterial line Assess hourly urine output Slowly lower BP to avoid stroke, renal failure, and HF
123
What are the characteristics of left-sided HF
Caused by impaired contractility, valve abnormalities, and hypertension Systolic failure—it is a failure of left ventricle to empty adequately during systole EF <45% Fluid back up into the lungs
124
What are the characteristics of right sided HF
Failure of right ventricle Rest of the body, fluid back up into the venous system Most common cause left sided HF
125
What are some manifestations in left sided HF
Increased HR Extra heart sounds Confusion Cough Crackles Pleural effusion Shallow respirations Anxiety, depression Dyspnea Fatigue Nocturnal Orthopnea Nocturnal Dyspnea
126
What are the manifestations of right sided HF
Increase HR Anasarca Ascites Edema,weight gain Hepatomegaly JVD, murmurs Anorexia, G bloating Anxiety, depression Fatigue Nausea, RUQ pain
127
Diuretics remove fluid, what is something we need to do before administering diuretics
Know K+ prior to administration
128
What fluid describes fluid inside the cells, has potassium and phosphate , contains higher concentration of proteins
Intra cellular fluid Two third of the body’s water if found in the ICF
129
What is the daily fluid requirement of men and women
3.7 men 2.7 women
130
What is the daily fluid output
0.5-1.5ml/kg/hr Normal range 800-2000ml per day Pediactrics 1-2 ml/kg/hr Both urinate every 6hrs
131
What conditions where we would see fluid volume deficit
Ocurrs with: 1 abnormal body fluid loss 2 inadequate intake 3 shift from plasma to interstitial fluid Diabetes insipid is, GI losses, hemorrhage, inadequate intake, insensible water loss or perspiration, osmotic diuretics, third space fluid shifts
132
What manifestation would we see with fluid volume deficit
Cap refill, confusion, restlessness, lethargy Cold clammy skin, postural hypotension, increase pulse, seizures, coma, thirst, dry mucus membranes, urine concentration, weight loss
133
What are some implementations of fluid volume deficit
Record I and Os Monitor vital signs Oral rehydration in mild cases Moderate-severe rehydration Fall precautions Provide prescribed diet Frequent oral cares
134
What are some causes of fluid volume excess
Long-term corticosteroid use Cushing syndrome HF Primary Polydipsia Renal failure
135
What will we see with fluid volume excess
Bounding pulse, inc HR, confusion, headache, lethargy, Dyspnea, crackles, pulmonary edema Edema, JVD, muscle spasms, polyuria, extra heart sounds, weight gain
136
What are some implementations of fluid volume excess
Daily weights, record I and Os, monitor vital sings, administer diuretics, sodium fluid restrictions, implement fall precautions, skin care-frequent position changes, elevate edematous extremities
137
What takes priority in FVE
Cardiovascular checks take priority
138
What are plasma electrolytes
Sodium H, K l, Ca l, Mg l Cl h,
139
What are intracellular electrolytes
K h, Na l, Mg l
140
Normal range of Na and what are the functions of Na
136-145 1 maintain water volume in ECF 2 electrical transmission of nerve impulses 3 maintaining acid-base balance of blood 4 regulate relationship of Na and K placement
141
Normal potassium range and what are the functions
3.5-5 1 nerve impulse conduction 2 cardiac electrical activity regulation 3 skeletal and smooth muscle contraction 4 regulate acid-base balance
142
What are manifestations and interventions of hyperkalemia
Bilateral muscle weakness (begins in quads) Abdominal distention Decreased bowel sounds Constipation Dysrhythmias ECG abnormalities (U waves flattened, inverted T waves, ST depression) Treat with PO or IV KCl Increase K foods, treat underlying cause
143
What are the manifestations of hyperkalemia
Bilateral muscle weakness Abdominal cramps Diarrhea Dysrhythmias Cardiac arrest if severe ECG abnormalities (peaked T waves, widened QRS Stop Po and IV k intake Increase potassium excretion (loop/thaizide diuretic) Administer insulin, dextrose, albuterol Treat underlying cause
144
What is the normal value of calcium and what are the functions
8.2-10.2 Need vit d, magnesium, phosphorus, and vitamin K are needed for absorption 1. Build strong bones and teeth 2 facilitates blood clotting 3 essential for nerve impulse transmission 4 activation of certain enzymes
145
What are the manifestations and interventions of hypocalcemia
Numbness, finger/toe tingling Positive Chvostek sign Hyperactive reflexes Muscle twitch/cramping Carpal and pedal spasms Positive trousseaus sign Seizures Laryngospasm Dysrhythmias Treat w PO and IV mediations Gluconate+vit d to aid in oral absorption Adjust diuretics from loop to thiazide
146
What are the manifestations and interventions of hypercalcemia
Anorexia, nausea, vomitting, Constipation, fatigue, diminished reflexes Lethargy, LOC, confusion, personality change Treat with PO or IV calcitonin, biphosphonates, prednisone) Restrict calcium intake Increase weight bearing activity, maintain hydration
147
What is the normal value of magnesium and the functions
1.3 to 2.1 1 needed for normal function in muscles, nerves, and cardiac conduction 2 supports immunity 3 assist in blood clotting 4 required for calcium and vitamin d absorption
148
What does the numonic mean create bind at the ren fair
BUN and creating values most affect renal failure
149
What does the numonic bro please, what the cost for the VR
Blood pressure=cardiac output x systemic vascular resistance