Upper Respiratory Problems Flashcards

(48 cards)

1
Q

Deviated Septum

A

CROOKED NASAL SEPTUM, APPEARS BENT

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2
Q

Causes of a DEVIATED SEPTUM

A

CAUSED BY TRAUMA; ALSO CONGENITAL DISPROPORTION IN THE SIZE OF
THE SEPTUM VS. THE NOSE

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3
Q

SYMPTOMS OF A DEVIATED SEPTUM

A

OBSTRUCTION, NASAL EDEMA, DRYNESS OF THE
NASAL MUCOSA WITH CRUSTING/EPISTAXIS; IF SEVERE CAN CAUSE BLOCK DRAINAGE OF SINUS CAVITIES RESULTING IN INFECTIONS (SINUSITIS)

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4
Q

MANAGEMENT OF A DEVIATED SEPTUM

A

CONTROL OF NASAL ALLERGIES (ALLERGIC RHINITIS);

SEVERE SYMPTOMS REQUIRE NASAL SEPTOPLASTY (REALIGNMENT)

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5
Q

CAUSES OF A NASAL FRACTURE

A

TYPICALLY CAUSED BY TRAUMA

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6
Q

NASAL FRACTURE COMPLICATIONS

A

EPISTAXIS, AIRWAY OBSTRUCTION, MENINGEAL TEARS, COSMETIC DEFORMITY

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7
Q

CLASSIFICATIONS OF NASAL FRACTURES

A

UNILATERAL, BILATERAL OR COMPLEX

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8
Q

ASSESSMENTS OF NASAL FRACTURES

A

ASSESS ABILITY TO BREATH THROUGH EACH NOSTRIL, NOT EDEMA, BLEEDING,
HEMATOMA; MAY BE ECCHYMOSIS UNDER ONE/BOTH EYES ‘RACCOON EYES;
INSPECT FOR SEPTAL DEVIATION, HEMORRHAGE, OR CLEAR DRAINAGE (CHECK CLEAR DRAINAGE FOR CSF)

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9
Q

NASAL FRACTURES NURSING MANAGEMENT

A

REDUCE EDEMA, PREVENT COMPLICATIONS,

EMOTIONAL SUPPORT; APPLY ICE; MAY REQUIRE SURGERY FOR REALIGNMENT (SEPTOPLASTY, RHINOPLASTY)

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10
Q

NURSING MANAGEMENT: NASAL SURGERY (RHINOPLASTY, SEPTOPLASTY)

A

• OUTPATIENT PROCEDURE, REGIONAL ANESTHESIA
• INSTRUCT PATIENT TO NOT TAKE ASPIRIN-CONTAINING DRUGS OR NONSTEROIDAL ANTI-INFLAMMATORY DRUGS (NSAIDS) FOR 2 WEEKS BEFORE SURGERY.
• TISSUE ADDED OR REMOVED; USE OF PLASTIC IMPLANTS, NOSE LENGTHEN OR SHORTENED
• POST OP: MAY HAVE NASAL SPLINTS AND OR PACKING
• IMMEDIATE POSTOPERATIVE PERIOD INCLUDES ASSESSMENT OF RESPIRATORY STATUS, PAIN MANAGEMENT, AND OBSERVATION OF THE
SURGICAL SITE FOR HEMORRHAGE AND EDEMA.
• TEACHING IS IMPORTANT BECAUSE THE PATIENT MUST BE ABLE TO DETECT COMPLICATIONS AT HOME.

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11
Q

EPISTAXIS

A

Nosebleed

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12
Q

CAUSES OF EPISTAXIS

A

TRAUMA, FOREIGN BODIES, NASAL SPRAY ABUSE, STREET DRUG USE, ANATOMICAL MALFORMATION, ALLERGIC RHINITIS, TUMOURS

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13
Q

How does HTN affect epistaxis

A

HTN IS NOT A CAUSE BUT DOES MAKE CONDITION MORE DIFFICULT TO CONTROL

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14
Q

DRUG CONTRAINDICATIONS

A

ASPIRIN, NSAIDS, WARFARIN AND CONDITIONS PROLONGING BLEEDING TIME OR ALTERING PLATELET COUNTS PREDISPOSE PATIENTS TO EPISTAXIS

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15
Q

NURSING AND COLLABORATIVE MANAGEMENT: EPISTAXIS

A
  • KEEP THE PATIENT QUIET.
  • PLACE THE PATIENT IN A SITTING POSITION, LEANING FORWARD, OR IF NOT POSSIBLE, IN A RECLINING POSITION WITH HEAD AND SHOULDERS ELEVATED.
  • APPLY DIRECT PRESSURE BY PINCHING THE ENTIRE SOFT LOWER PORTION OF THE NOSE FOR 10 TO 15 MINUTES.
  • APPLY ICE COMPRESSES TO THE FOREHEAD AND HAVE THE PATIENT SUCK ON ICE.
  • APPLY DIGITAL PRESSURE IF BLEEDING CONTINUES.
  • OBTAIN MEDICAL ASSISTANCE IF BLEEDING DOES NOT STOP.
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16
Q

ALLERGIC RHINITIS

A

REACTION OF THE NASAL MUCOSA TO ALLERGENS

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17
Q

TYPES ALLERGENS THAT CAUSE ALLERGIC RHINITIS

A
  • SEASONAL: POLLEN – TREES, GRASSES, FLOWERS

* PERENNIAL: PET DANDER, DUST MITES, MOLDS, COCKROACHES

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18
Q

CLINICAL MANIFESTATIONS OF ALLERGIC RHINITIS

A
  • NASAL CONGESTION; SNEEZING; WATERY, ITCHY EYES AND NOSE; ALTERED SENSE OF SMELL; THIN, WATERY NASAL DISCHARGE
  • NASAL TURBINATES APPEAR PALE, BOGGY, AND SWOLLEN
  • CHRONIC EXPOSURE TO ALLERGENS: HEADACHE, CONGESTION, PRESSURE, POSTNASAL DRIP, NASAL POLYPS
  • PATIENT MAY COMPLAIN OF COUGH, HOARSENESS, SNORING, OR RECURRENT NEED TO CLEAR THE THROAT
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19
Q

NURSING AND COLLABORATIVE MANAGEMENT: ALLERGIC RHINITIS

A

• IDENTIFY AND AVOID TRIGGERS OF ALLERGIC REACTIONS

DRUG THERAPY (TABLE 29-2)
• NASAL SPRAYS, LEUKOTRIENE RECEPTOR ANTAGONISTS (LTRAS), ANTIHISTAMINES, AND DECONGESTANTS TO MANAGE SYMPTOMS
• INTRANASAL CORTICOSTEROID AND CROMOLYN SPRAYS (DECREASE INFLAMMATION LOCALLY)
• PROVIDE INSTRUCTIONS ON PROPER USE OF NASAL INHALERS (THEY CAN CAUSE REBOUND EFFECT FROM PROLONGED USE).

  • IMMUNOTHERAPY (“ALLERGY INJECTIONS”) MAY BE USED IF DRUGS ARE NOT TOLERATED OR ARE INEFFECTIVE.
  • INVOLVES CONTROLLED EXPOSURE TO SMALL AMOUNTS OF A KNOWN ALLERGEN THROUGH FREQUENT (AT LEAST WEEKLY) INJECTIONS WITH THE GOAL TO DECREASE SENSITIVITY
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20
Q

ACUTE VIRAL RHINITIS

21
Q

ACUTE VIRAL RHINITIS IS CAUSED BY

A

VIRUSES THAT INVADE THE UPPER RESPIRATORY TRACT; SPREAD BY AIRBORNE DROPLET SPRAYS EMITTED WHILE BREATHING, TALKING, SNEEZING, OR COUGHING OR BY DIRECT HAND CONTACT

22
Q

RISK FACTORS ACUTE VIRAL RHINITIS

A
WINTER MONTHS (MORE TIME INDOORS), CHILLING,
FATIGUE, EMOTIONAL STRESS, IMMUNOCOMPROMISE
23
Q

CLINICAL MANIFESTATION OF ACUTE VIRAL RHINITIS

A

TICKING IRRITATION, SNEEZING OR DRYNESS OF NOSE FOLLOWED BY RHINORRHEA, NASAL CONGESTION, WATERY EYES, ELEVATED TEMPERATURE, HEADACHE, MALAISE

24
Q

THREE VIRUSES OF INFLUENZA

25
CLINICAL MANIFESTATIONS OF INFLUENZA
* ONSET ABRUPT; SYSTEMIC SYMPTOMS OF COUGH, FEVER, MYALGIA, HEADACHE, SORE THROAT * IN UNCOMPLICATED CASES, SYMPTOMS SUBSIDE WITHIN 7 DAYS; OLDER ADULTS MAY EXPERIENCE PERSISTENT WEAKNESS OR LASSITUDE.
26
MOST COMMON COMPLICATION OF INFLUENZA
PNEMONIA
27
TREATMENT OF INFLUENZA
* SUPPORTIVE CARE, SYMPTOMS RELIEF – REST, FLUIDS, NUTRITION, ANTIPYRETICS, ANALGESICS * ANTIVIRALS I.E. TAMIFLU (TO PREVENT OR DECREASE SYMPTOMS)
28
NURSING AND COLLABORATIVE MANAGEMENT OF INFLUENZA
* HANDWASHING | * INFLUENZA VACCINATION
29
SINUSITIS IS DUE TO WHAT?
BLOCKAGE OF THE NASAL PASSAGE (INFLAMMATION), SECRETIONS ACCUMULATE – IDEAL ENVIRONMENT FOR THE GROWTH OF VIRUSES, BACTERIA, FUNGI
30
EXAMPLES OF ACUTE SINUSITIS
URTI, ALLERGIC RHINITIS, SWIMMING, DENTAL MANIPULATION
31
EXAMPLES OF CHRONIC SINUSITIS
PERSISTENT INFECTION R/T ALLERGIES, NASAL POLYPS
32
CLINICAL MANIFESTATIONS OF ACUTE SINUSITIS
SIGNIFICANT PAIN, PURULENT NASAL DRAINAGE, NASAL OBSTRUCTION, CONGESTION, FEVER, MALAISE, SINUSES TENDERNESS, HEADACHES, PAIN WITH CHANGE OF POSITION
33
CLINICAL MANIFESTATIONS OF CHRONIC SINUSITIS
FACIAL PAIN, NASAL CONGESTION, INCREASED DRAINAGE; SEVERE PAIN AND PURULENT DRAINAGE ARE OFTEN ABSENT • SYMPTOMS MAY MIMIC THOSE SEEN WITH ALLERGIES • DIFFICULT TO DIAGNOSE BECAUSE SYMPTOMS MAY BE NONSPECIFIC; PATIENT IS RARELY FEBRILE
34
NURSING AND COLLABORATIVE MANAGEMENT OF SINUSITIS
- ENVIRONMENTAL CONTROL - APPROPRIATE DRUG THERAPY – ANTIBIOTICS, DECONGESTANTS, NASAL STEROIDS - PATIENT INTERVENTIONS • INCREASE FLUID INTAKE • NASAL CLEANING TECHNIQUES AND IRRIGATION - PERSISTENT COMPLAINTS MAY REQUIRE ENDOSCOPIC SURGERY
35
TYPES OF SINUSES
- FRONTAL - ETHMOIDAL - SPHENOIDAL - MAXILLARY
36
TYPES OF OBSTRUCTION OF THE NOSE AND | PARANASAL SINUSES
- POLYPS | - FOREIGN BODIES
37
POLYPS
BENIGN MUCOUS MEMBRANE MASSES
38
POLYPS CLINICAL MANIFESTATIONS
NASAL OBSTRUCTION, NASAL DISCHARGE | (USUALLY CLEAR MUCUS), SPEECH DISTORTION
39
POLYPS TREATMENTS
TOPICAL STEROIDS REDUCE INFLAMMATION; SURGICAL REMOVAL
40
FOREIGN BODIES OBSTRUCTING THE NOSE OR PARANASAL SINUSES CLINICAL MANIFESTATIONS
* INORGANIC MAY GO UNDETECTED | * ORGANIC PRODUCE LOCAL INFLAMMATION, NASAL DISCHARGE
41
FOREIGN BODIES OBSTRUCTING THE NOSE OR PARANASAL SINUSES TREATMENTS
SHOULD BE REMOVED THROUGH ROUTE OF ENTRY (SNEEZE WITH OPPOSITE NOSTRIL CLOSED, BLOWING NOSE)
42
WHAT SHOULD NOT BE DONE WITH FOREIGN BODIES OBSTRUCTING THE NOSE OR PARANASAL SINUSES?
DO NOT IRRIGATE OR PUSH OBJECT UP NOSE (RISK FOR ASPIRATION OR AIRWAY OBSTRUCTION)
43
ACUTE PHARYNGITIS
* ACUTE INFLAMMATION OF THE PHARYNGEAL WALLS * MAY INCLUDE TONSILS, PALATE, AND UVULA * CAN BE CAUSED BY A VIRAL (MOST COMMON), BACTERIAL (“STREP THROAT”), OR FUNGAL INFECTION (CANDIDIASIS)
44
CLINICAL MANIFESTATIONS OF ACUTE PHARYNGITIS
* RANGE FROM “SCRATCHY THROAT” TO SEVERE PAIN * APPEARANCE NOT ALWAYS DIAGNOSTIC; CULTURES NEEDED * RED THROAT +/- EXUDATES * DIFFERENTIATE FROM CANDIDA (WHITE PATCHES)
45
NURSING AND COLLABORATIVE MANAGEMENT OF ACUTE PHARYNGITIS
* INFECTION CONTROL * SYMPTOMATIC RELIEF – INCREASED FLUIDS, ANALGESICS, COOL SOFT FOODS, AVOID ACIDIC JUICES * PREVENTION OF SECONDARY COMPLICATIONS (TREAT BACTERIAL STREP THROAT)
46
PERITONSILLAR ABSCESS
A COMPLICATION OF ACUTE PHARYNGITIS OR ACUTE TONSILLITIS WHEN BACTERIAL INFECTION INVADES ONE OR BOTH TONSILS
47
DANGERS OF PERITONSILLAR ABSCESS
TONSILS MAY ENLARGE SUFFICIENTLY TO THREATEN AIRWAY PATENCY
48
PERITONSILLAR ABSCESS CLINICAL MANIFESTATIONS
PATIENT EXPERIENCES A HIGH FEVER, LEUKOCYTOSIS, AND CHILLS