Uterine Pathology Flashcards

(26 cards)

1
Q

what two abnormalities may be indistinguishable from hyperplasia?

A

endo cancer and polyp

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2
Q

most common cause of abnormal uterine bleeding?

A

hyperplasia

in peri- and postmenopausal women

may be caused by unopposed estrogen hormone replacement therapy

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3
Q

during saline infusion sonohysterography what approach is used?

A

transvaginal

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4
Q

failure of fusion consists of?

A

uterus didelphys: complete dublication of uterus, cervix, and vagina

bicornuate uterus: most common Mullerian anomaly, single vagina, one or two cervices, and variable lake of fusion of upper uterine cavity

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5
Q

sonographic findings of endometritis

A

endo appears prominent, irregular, with small amount of fluid

pus in cul-de-sac

enlarge cystic ovaries

dilation of tube (5mm or more)

tubo-ovarian complex

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6
Q

Example of a persistant structure that arises from the caudal remnants of the mesonephric (Wollfian) duct?

A

gartners duct cyst

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7
Q

Rare malignancy arising from the myometrium?

A

leiomyosarcoma

may be indistinguishable from fibroid

clue is relatively rapid growth of mass in post-menopause

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8
Q

Clinical signs of leiomyomas

A

menometrorrhagia

frequent urination

enlarged uterus

increasing pain with degenerative changes

infertiliy or spontaneous abortions

alteration in normal menstrual flow

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9
Q

sonographic findings of cervical cancer

A

normal in early disease

enlarged uterus

similar to cervical myoma

hydronephrosis

involvement of other pelvic organs

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10
Q

What is failure of dissolution?

A

median septum fails to dissolve after fusion of the two separate Mullerian ducts, septate uterus

least severe Mullerian anomaly is a slightly protruding into the uterine cavity, arcuate uterus

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11
Q

The uterus, fallopian tubes and upper vagina develop from what?

A

Mullerian ducts

(paramesonephric ducts)

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12
Q

what is endometrial hyperplasia?

A

proliferation of endometrial glandular tissue

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13
Q

typical patient of adenomyosis

A

30-50 yrs old

dysmenorrhea and irregular bleeding

parous women

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14
Q

Exposure associated with poor pregnancy outcome, cervical cancinoma and breast carcinoma, and a T-shaped uterus?

A

DES (diethylstilbestrol) syndrome

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15
Q

Are the ovaries affected in the presence of mullerian anomalies?

A

No, do not develop from Mullerian ducts

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16
Q

most common gyn malignancy, usually occurs in postmenopausal women?

A

endometrial carcinoma

present with early postmenopausal bleeding

increased risk between estrogen and malignancy

17
Q

third most common GYN malignancy?

A

cervical cancer

20-30 yrs old

most common symptom is post-coital bleeding

18
Q

Failure of formation demonstrates?

A

complete agenesis:

absence of vagina, cervix, uterus, and fallopian tubes

partial agenesis:

range of anomalies, most common is unicornate uterus and single fallopian tube

19
Q

hyperplasia clinical signs are similar to endo cancer, sonographic findings differ?

A

smooth boarders

more homogeneous

premenopause > 14mm

tamoxifen > 10mm

postmeno estrogen only > 5mm

postmeno w/ estrogen and progesterone > 8mm

20
Q

Commonly acquired after surgical procedure or uterine trauma and associated with heavy vaginal bleeding?

A

uterine arteriovenous malformation

21
Q

causes of endometritis

A

PID

retained products

postprocedural complication

vaginitis

22
Q

In the case of uterine anomalies, what other system should be evaluated for anomalies?

A

urinary system

23
Q

abnormality found in posttraumatic or postsurgical historiesincluding uterine curettage?

A

synechiae or ashermans syndrome

24
Q

sonographic findings associated with synechiae

A

bright echoes within endo

better identified during secretory phase or under SIS

bridging bands of tissue that distort cavity

25
What are the four categories of anomalous internal genitalia development?
failure of formation failure of fusion failure of dissolution failure of structures to disappear
26
risks associated with endometrial cancer
obesity and anovulatory cycles estrogen replacement history of hyperplasia tamoxifen therapy family history