Valvular HD Flashcards

(76 cards)

1
Q

VHD

Main two types

A
  • Congenital
  • Acquired
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2
Q

VHD

S1

A

Mitral and tricuspid closure

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3
Q

VHD

S2

A

aortic and pulmonary V closure

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4
Q

VHD

Apex beat

A

5th ICS just medial to the midclavicular line

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5
Q

VHD

Peds murmurs heard above the nipple line

A

ejection systolic

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6
Q

VHD

peds murmurs heard below the nipple line

A

Pansystolic

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7
Q

VHD

Mitral valve normal size

A

4-6 cm2

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8
Q

VHD

Mitral stenosis PC

A
  • SOB
  • Orthopnea
  • PND
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9
Q

VHD

Pulse in MS

A

irregularly irregular due to AF

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10
Q

VHD

JVP in MS

A

prominent “a” wave if complicated by Pulm HTN

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11
Q

VHD

Palpation in MS

A

tapping apex, parasternal heave

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12
Q

VHD

Auscultation findings in MS

A
  • Loud S1
  • Opening snap
  • Rumbling low- pitched mid diastolic murmur with presystolic accentuation
  • Best heard in mitral area w expiration and left lateral position
  • Pulm HTN - Loud P2
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13
Q

VHD

S1 in MS

A

Loud

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14
Q

VHD

Murmur in MS

A

rumbling low pitched mid diastolic murmur w presystolic accentuation

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15
Q

VHD

murmur is MS is heard on

A

mitral area with expiration on left lateral position

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16
Q

VHD

Signs of Pulm HTN

A
  • Parasternal heave
  • Loud palpable P2
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17
Q

VHD

DDs of MS

A
  • Carey coombs Murmur in acute rheumatic carditis
  • Austin flint murmur in severe AR
  • Atrial myxoma
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18
Q

VHD

whats a carey coombs murmur

A

mitral V is swollen

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19
Q

VHD

Atrial myxoma

A

a tumor growing through the mitral valve

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20
Q

VHD

main cause of MS

A

Rheumatic fever

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21
Q

VHD

MS is mostly affected among

A

Females> males

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22
Q

VHD

severe stenosed MS

A

<1 cm2

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23
Q

VHD

Complications of MS

A
  • A fib
  • Pulm edema
  • IE
  • Stroke ( AF can form thrombus)
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24
Q

VHD

Ix of MS

A
  • ECG
  • 2D echo
  • CXR
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25
# VHD ECG on MS
P mitrale ( bifid P wave)
26
# VHD Ix of choice in Dx MS
2D echo
27
# VHD CXR on MS
small heart w an enlarged L/ atrium
28
# VHD Mx of MS
* Mx A fib * interventional Mx
29
# VHD Interventional Mx of MS
* PTMC ( Percutaneous transluminal mitral commisurectomy) * Valvotomy * Valve replacement ( rarely)
30
# VHD When is PTMC considered in MS
* isolated MS * Minimal MR * L/atrium free of thrombus * MV mobile and pliable
31
# VHD Most Pts w MS present during
PG
32
# VHD Two types of valve replacement
* metallic- Pt should be on long- standing warfarin * Bioprosthetic - durability is ~ 10 years.
33
# VHD why is bioprosthetic V not replaced on young females
it can get damaged during PG. so they are usually put in elderly
34
# VHD MR apex
thrusting displaced apex
35
# VHD Murmur in MR
pansystolic murmur at the apex radiating to the axilla
36
# VHD Etiology of MR
* RF * IE * Degenerative * IHD * dilated cardiomyopathy
37
# VHD Why does IHD cause MR
ruputre of papillary muscles
38
# VHD Complicated MR can lead to
heart failure
39
# VHD Ix for MR
* ECG - P mitrale, AF * CXR- LV and LA dilated * 2D echo
40
# VHD Mx of MR
* MV repair, MV replacement
41
# VHD Indications for surgery in MR
* Severe acute MR * Severe chronic symptomatic MR * ASx Chronic MR w evidence of progression of LV dilatation
42
# VHD How can an austin flint murmur in severe AR cause MS
When blood backflows through the aortic V, it can hit the MV causing a MS
43
# VHD PTMC
send a guiding wire and cute the stenosed V
44
# VHD Isovolumic contraction
The Ventricles will be contracting as a closed chamber until it's pressure increases to snap open the aortic and pulmonic valves
45
# VHD Ejection systole
When the pressure increases in the ventricles to open, blood will enter into the aorta and pulmonary artery
46
# VHD Pressure changes in the cardiac cycle is reflected in the
internal jugular V
47
# VHD waves in JVP
* a wave * c wave * v wave
48
# VHD Two descents in JVP
* X descent * Y descent
49
# VHD a wave
**due to atrial systole**. SVC and IVC are valveless. So when the atria is contracting during systeole, some of the blood will regurgitate and see a pulsatile wave
50
# VHD c wave
**during isovolumetric contraction.** when the ventricles contract as a closed chamber, due to the force, the TV and MV will be pushed out while remaining shut. this causes a reflex wave
51
# VHD V wave
during atrial filling
52
# VHD X descent
atrial relaxation
53
# VHD Y descent
ventricular filling
54
# VHD Abnormalities of JVP
* Non- pulsatile raised JVP * Loss of a wave * Prominent a wave * canon a wave * cv wave * sharp x descent
55
# VHD non- pulsatile raised JVP
SVC obstruction. due to the block, nothing from the atria will be transmitted to the int. jugular V. Also because of the block, blood gets backedup increasing JVP
56
# VHD loss of a wave
no proper atrial contraction like in A fib
57
# VHD Prominent a wave
tricuspid stenosis Pulm HTN
58
# VHD How can Pulm HTN cause a prominent a wave
Pulm artery pressure increase overtime. RV will undergo hypertrophy. High pressure in atria to push blood into RV coz the pressure in RV increase due to the hypertrophy. More blood regurgitated to the SVC
59
# VHD Canon a wave
in complete heart block No connection netween the atria and ventricles. Atria is unaware of what happened in the ventricles. So atria will contract while the TV and MV closed. ( It's the SA node that tells TV and MV to open for atrial systole) since there is a heart block, cardiac process not in sync. Pressure in atria increase more blood regurgitate to SVC.
60
# VHD cv wave
tricuspid regurgitation. c & v become one large wave together
61
# VHD sharp x descent
constrictive pericarditis
62
# VHD AR Sx
* Exertional dyspnea * palpitations
63
# VHD Pulse AR
* collapsing pulse * Prominent peripheral pulses
64
# VHD AR apex
* displaced and hyperkinetic ( thrusting ) * Palpitations
65
# VHD Murmur in AR
early diastolic M best heard at lower left sternal edge and patient sitting up and holding the breath on expiration
66
# VHD Peripheral signs of AR
* head nodding ( de Musset's sign) * Visible carotic pulsations ( Corrigan's sign) * Capillary pulsations (Quincke's sign) * Pistol shot femorals * examination of the pupils - argyll robertson ( syphillis) * Examine the joints and back for RA, ankylosing spondylitis
67
# VHD etiology of AR
* Acquired * Congenital
68
# VHD Congenital causes for AR
* Marfans - aortic root dilatation * Osteogenesis imperfecta
69
# VHD Acquired causes of AR
* Rheumatic heart disease * IE * Trauma * Aortic dissection * Severe HTN * Syphillid * Seronegative arthritis * Rheumatoid Arthritis
70
# VHD a longer and a louder murmur
severe lesion
71
# VHD How would you say is an AR is severe
* the longer the murmur the louder the murmur * Presence of the austin - flint murmur * Wide pulse pressure * Features of LV failure
72
# VHD Ix for AR
* ECG - LVH in advanced cases * CXR- LV dilatation and aortic rootdilatation in some cases * Echo
73
# VHD Mx of AR
* Medical - Mx HF ( ACEI, diuretics) * Surgical
74
# VHD When to consider surgery in AR
* acute AR * Sx patients * ASx pts w progressively increasing ventricular dilatation and declining LV function
75
# VHD PC of AS
* syncope * SOB * Chest pain
76
# VHD