Flashcards in Vascular Path Robbins Part 3 Deck (41)
dilation of venous plexus at anorectal jxn
-pain, bleeding, may ulcerate
thrombophlebitis- most where?
(also called phlebothrombosis)
-venous thrombosis and infl
-90% in deep v's in legs- can be completely asymptomatic!!
risk factor for DVT in LE?
-prolonged activity/immobilization- single most important!
-systemic hypercoagulability (may also increase risk)
-mechanical factors that slow venous return- CHF, pregnancy, obesity
most common serious clinical complication of DVT?
-pulmonary embolism- often 1st manifestation of thrombophlebitis
migratory thrombophlebitis - assoc with?
-assoc with cancer- hypercoagulability!
-venous thromboses appear at 1 site, disappear, and reappear at another site
-adenocarcinomas of lung, ovary, pancreas
superior vena cava syndrome- caused by? effects
-neoplasms or aortic aneurysms that compress or invade the SVC
-dilation of v's of head, neck, arms with cyanosis
-pulm vessels may be compressed- resp distress
inferior vena cava syndrome-caused by? effects?
-neoplasms that compress or invade IVC
-thrombosis of hepatic, renal, or LE v's that moves cephalad
-hepatocellular carcinoma and RCC- grow within v's!
-obstruction- LE edema, distention of superficial collateral v's of lower abdomen, proteinuria (with renal v involvement)
lymphangitis- what is it? caused by? manifested by?
acute infl elicited by the spread of bacterial infections into lymphatics
-group A B-hemolytic streptococci- most common
-dilated and filled with exudate of neutrophils/monocytes
-red, painful subcutaneous streaks and painful enlargement of draining LNs (lymphadenitis)
primary lymphedema- due to?
isolated congenital defect or familial Milroy disease (heredofamilial congenital lymphedema)
-lymphatic agenesis or hypoplasia
secondary or obstructive lymphedema- due to?
blockage of a previously normal lymphatic
-surgical procedures that remove regional groups of LNs
-postinfl thrombosis and scarring