VD and Problems w/Peripheral Circulation Flashcards

(53 cards)

1
Q

Peripheral artery disease is a result of?

A
  • atherosclerotic plaque
  • thromboemboli
  • trauma
  • infections
  • inflammation
  • vasospastic disorders
  • congenital
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2
Q

Peripheral artery disease is a manifestation of?

A

atherosclerosis

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3
Q

What is atherosclerosis?

A

a thickening/hardening of arteries

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4
Q

Atherosclerosis is a slow process that begins?

A

in early adulthood, most people do not know they have it

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5
Q

Assessment considerations for PAD?

A
  • brain
  • heart
  • kidneys
  • mesentery
  • limbs
  • frequently seen in DM and elderly patients
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6
Q

PAD causes:

A
  • partial or totally obstruction
  • lumen narrows
  • infraction
  • accusation of: lipid, calcium deposits, fibrous tissue, stenosis, thrombosis
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7
Q

Who is at risk for PAD?

A
  • tabacco * big factor
  • family
  • age 70
  • obesity
  • HTN
  • HLD
  • DM
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8
Q

PAD asymptomatic/ symptomatic manifestations:

A
  • pain in distal muscle groups: worse when walking
  • ulcers
  • numbness/tingling
  • hairloss
  • pallor
  • cool to touch
  • no pulse
  • paralysis
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9
Q

PAD structural changes:

A
  • hair loss distal to the occlusion
  • thick opaque nails
  • shiny dry skin
  • skeletal muscle atrophy
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10
Q

PAD skin color changes:

A
  • pallor with elevation

- dependent rubor

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11
Q

PAD pulse changes:

A
  • diminished or absent blew area of stenosis/obstruction

- cool extremity distal to occlusion

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12
Q

PAD sensation changes:

A
  • paresthesia: numbness/tingling
  • ulcerations
  • gangrene
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13
Q

With rubor the blood vessel cannot?

A

constrict they stay dilated.

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14
Q

Intermittent claudication is a hallmark symptom for PAD and is?

A

limping pain

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15
Q

What is it when the arterial occlusions develop gradually, and there is less risk of sudden death b/c this ______ ______ may develop.

A

collateral circulation

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16
Q

Over time with collateral circulation continued decreased perfusion results in?

A

ischemia and tissue death

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17
Q

DX of PAD:

A
  • doppler
  • ultrasonography
  • magnetic resonance angiography (MRA)
  • CT
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18
Q

An MRA is able to?

A

isolate blood vessels and vein in 3D

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19
Q

Treatment of PAD:

A
  • thrombolysis
  • percutaneous
  • cilostazeol (aspirin)
  • bypass graft
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20
Q

What is thrombolysis?

A

a thrombolytic agent injected directly into thrombosis to lyse the clot

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21
Q

What is percutaneous?

A
  • angioplasty: balloon expanded stenosis
  • stent placement: assess perfusion/pain
  • bypass graft: rerouting blood flow
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22
Q

Nursing management of PAD:

A
  • pain relief
  • maintain tissue integrity
  • tabacco cessation
  • stress reduction
  • avoid hot temperature
  • avoid constrictive clothing
  • avoid crossing legs
23
Q

Six P’s for acute arterial occlusion:

A
  • pain
  • pallor
  • pulselessness
  • poiikilothermia (cool temp)
  • paresthesia
  • paralysis
24
Q

_______ is vasospasm causing vasoconstriction, and occurs with cold or stress?

A

raynauds syndrome

25
_____ _____ is when blood vessel become inflamed, swell, and become blocked (thrombi).
buergers disease
26
Burgers disease puts you at risk for?
- infection | - gangrene
27
Abdominal aortic aneurysm (AAA) is a?
localized out pouching sac dilation formed on the arterial wall.
28
AAA is a result of?
- atherosclerotic plaque - trauma - disease - congenital
29
Risk factors for AAA:
- genetic basis | - HTN
30
AAA grows an average of?
0.4 cm/year
31
Clinical manifestations of AAA:
- some people may not have any - feeling there heart beat in their abdomen when lying down - report feeling an abdominal mass or abdominal throbbing - mottling of toes - IMPENDING RUPTURE: severe pain pain or abdominal pain, persistent of intermittent, often midline - Rupturing anerysm: constant, intense back pain, falling BP, decreasing hematocrit - MOST IMPORTANT INDICATION: pulsating mass in the abdomen, also systolic bruit may be heard of mass
32
What is the MOST IMPORTANT indication for AAA:
pulsating mass in the abdomen.
33
DX of AAA:
- ultrasonography | - CT
34
Treatment of AAA:
- observation - monitor BP - education - anti-hypertensives - surgery
35
AAA can be confused with?
MI
36
For AAA ultrasound every 6 months and monitor BP is small but if larger than?
5.5 cm or significantly enlarging surgical intervention is required.
37
Nursing management for AAA:
- education r/t diet, smoking, meds
38
Postop considerations for AAA:
bleeding, circulation, neuro changes, pain management
39
Venous thrombosis is?
formation of blood clot in the venous system
40
People at risk for DVT:
- stasis of blood - vessel wall injury - hormone therapy - smoking - dehydration - varicose veins
41
Manifestations of DVT:
- pain - swelling/edema - increase in tempature - larger leg - gray brown skin
42
DX of DVT:
- D dimer - ultrasonography - MRA
43
a d dimer that is positive:
clots when lyse
44
Treatment of DVT:
- heparin - warfarin - thrombotic therapy - surgery
45
Goal for patients with DVT of warfarin:
- INR 2 to 3 - with heart valve INR 2.5 to 3.5 - check INR every month
46
heparin does not dissolve a clot that is?
already formed.
47
aPTT for heparin:
21 - 35 sec
48
Venous Ulceration complication:
cellulitis/dermatitis may complicate healing
49
Venus ulcers develop as a result of?
increased venous pressure or external trauma
50
Venous ulcerations cause the skin to be?
dry, cracked and pruritic
51
Treatment of venous ulceration?
- dressing for debridement - hyperbaric oxygenation - compression
52
With PAD venous ulceration develops as a result of
ischemia and may lead to gangrene
53
Treatment of ulceration
keep dry and usually not debriefed until revascularization procedures are performed.