Week 1 Flashcards

(44 cards)

1
Q

FOGS

A

-Assessing pt. cognitive status –> should be assessed at beginning to drive the rest of the eval
F = Family report of memory loss
O = Orientation to person, place, time
G = General info recall
S = Spelling (WORLD forward and backward, or counting task)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Components of the Neuromuscular Assessment

A
  • cognitive assessment
  • communication assessment
  • cranial nerve assessment
  • sensory assessment
  • motor control assessment
  • functional assessment
  • coordination assessment
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Aphasia

A

Impairment in Broca’s area (expressive language) or Wernicke’s area (receptive language)
-difficulty with spoken language, reading, writing, hand gestures/sign language

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Dysarthria

A

Problems with the motor component of speech

-can’t articulate well, makes their speech slurred

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Why assess cranial nerves?

A
  • Allows examiner to localize brainstem dysfunctions
  • May be affected by a wide range of conditions including trauma, infection, CVA, tumor, intracranial inflammation
  • Dysfunction in certain CN may help to rule in or rule out certain diagnoses
  • CN assessment is not necessary for all patients
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

3 Primary roles of sensation in movement

A
  • Guide selection of motor responses for effective interaction with the environment via feedforward info
  • Adapt movements and shape motor programs through feedback for corrective action
  • Protect the organism from injury
  • Assessment of sensation aids us in diff-dx as well as looking at sensory integrity
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Deficits in sensory integrity result it..

A

poor motor planning, organization, and performance

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Indications for sensory assessment

A

-Impaired locomotion
-Impaired joint mobility or integrity
-Impaired motor control/motor function
-Impaired muscle performance
-Impaired neuromotor development
-Impaired reflex integrity
-Impaired posture
-Impaired ventilation, respiration, circulation
Pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What do we assess during sensory assessment?

A
  • Afferent inputs
  • Peripheral sensory processing –> superficial sensation, deep sensation, combined cortical
  • Cortical sensory processing –> DCML and ALS
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Combined cortical

A
  • stereognosis: object recognition
  • tactile localization
  • two-point discrmination
  • barognosis (recognition of weight)
  • graphesthesia (identification of traced figure)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

ALS

A
  • crude touch: pain, temperature, tickle, itch, sexual sensation
  • activated primarily by mechanoreceptors, thermoreceptors, nocioreceptors
  • small, slow afferent fibers
  • crosses over almost immediately in spinal cord
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

DCML

A
  • discriminative, finely graded sensation, precise location
  • large, rapidly conducting fibers
  • ascends ipsilaterally to medulla, synapses, then crosses over
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Deep Tendon Reflexes

A
  • involuntary, predictable, specific
  • ‘2’ is ‘normal’
  • hyperreflexia may indicate UMN lesion
  • hyporeflexia may indicate LMN lesion
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

DTR Grading Scale

A
0 = absent; no reflex response
1 = minimal response
2 = moderate response
3 = brisk, strong response
4 = clonus
*grades of 1-3 are considered WNL for some patient, combined with other findings may indicate something more specific
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Muscle tone

A

Resistance of a resting muscle to passive elongation or stretch
-3 main categories: hypotonicity, hypertonicity, and dystonia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Factors that contribute to normal muscle tone:

A
  • physical inertia
  • intrinsic mechanical-elastic stiffness of mm. and connective tissue
  • active muscle contraction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Hypotonicity

A
  • decrease in tone below resting level
  • indicative of LMN lesion (could be in anterior horn, spinal nerves, peripheral nerves, or NMJ)
  • flaccidity
  • diminished DTRs
  • fibrillations on EMG
  • hyperextensibility or “floppy” limbs and joints
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Hypertonicity

A

Increase in tone above resting levels

  • 2 types: spasticity (clonus, clasp-knife); rigidity (cogwheel, lead pipe)
  • indicative of UMN lesion (pyramidal pathways, cerebellum, or basal ganglia)
  • abnormal timing
  • paresis or plegia
  • brisk DTRs, +Babinski
  • dysynergic patterns of movement
19
Q

Dystonia

A

Hyperkinesis due to CNS injury; Impaired or disordered tone that fluctuates in an unpredictable pattern

  • repetitive involuntary movements: usually twisting or writhing
  • periods of dystonic posturing (sustained abnormal postures caused by contractures of muscles that may last minutes, hours, or permanently
20
Q

Coordination of movement

A

Ability to execute smooth, accurate, controlled motor responses
-requires coordinated effort of all the components of the motor system

21
Q

Intralimb coordination

A

Using one UE to to brush the hair requires coordination at the shoulder, elbow, wrist, and fingers

22
Q

Interlimb coordination

A

Integrated performance of two or more limbs working together- LEs/UEs during walking

23
Q

Visual motor coordination

A

Integration of visual and motor activities with the environment to accomplish a goal (writing a letter, driving a vehicle)
-eye-hand coordination

24
Q

Nonequilibrium coordination tests

A

address both static & mobile and gross & fine motor components of movement with the subject not attempting to maintain balance

25
Equilibrium coordination tests
address static and dynamic components of balance and posture
26
Three main areas that impact higher level processing and execution of coordinated motor responses:
-cerebellum -basal ganglia -DCML All three work together with and provide input to the cortex for coordinated movement
27
Ataxia
most common term used for cerebellar impairments affecting gait, posture, or patterns of movement (can be caused by other things than cerebellum)
28
Dysarthria
disorder of the motor component of speech, can be slow, slurred, hesitant with inappropriate pauses -manifestation of cerebellar pathology
29
Dysdiadochokinesia
unable to perform rapid alternating movements (quickly switching between muscle groups) -manifestation of cerebellar pathology
30
Dysmetria
inability to judge distance or range, can be hyper or hypometric -manifestation of cerebellar pathology
31
Dyssynergia
movement broken into parts rather than smooth simple activity -manifestation of cerebellar pathology
32
Gait ataxia
wide BOS, arms may be in high guard, steps are irregular, unsteady, veering -manifestation of cerebellar pathology
33
Nystagmus
rhythmic, involuntary quick eye movements | -manifestation of cerebellar pathology
34
Rebound phenomenon
check reflex | -manifestation of cerebellar pathology
35
Tremors
involuntary, oscillating movements - intention: during volitional movements - postural: back and forth oscillations of body in standing
36
Akinesia
inability to initiate movement (freezing) | -manifestations of basal ganglia pathology
37
Athetosis
slow, involuntary writhing, twisting, wormlike movements | -manifestations of basal ganglia pathology
38
Bradykinesia
decreased amplitude of velocity of movement | -manifestations of basal ganglia pathology
39
Chorea
involuntary, rapid, irregular, jerky movements | -manifestations of basal ganglia pathology
40
Dystonia
involuntary contractions of agonist/antagonist causing abnormal posturing -manifestations of basal ganglia pathology
41
Hemiballismus
large amplitude sudden violent flailing motions of arm/leg on 1 side -manifestations of basal ganglia pathology
42
Rigidity
increased muscle tone | -manifestations of basal ganglia pathology
43
Resting tremors
``` pill rolling (you see their tremors when they are still, volitional movement makes them go away) -manifestations of basal ganglia pathology ```
44
DCML manifestations
- gait disturbance - +Romberg due to proprioceptive loss - dysmetria - visual feedback can compensate so they mask DCML pathology well a lot of the time